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43 Cards in this Set
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- Back
Motor Speech Disorders |
Speech disorders that occur due to neurologic impairment that affects: - motor planning or programming, OR –Neuromuscular execution -includes Dysarthria & Apraxia |
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**Dysarthria** |
Collective name for group of neurologic speech impairs -reflect abnormalities in strength, speed, range, steadiness, tone / accuracy of movements req'd for all 5 aspects of speech. -Characterized by slow, weak, imprecise, or uncoordinated movements of the speech musculature |
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Dysarthria affects: |
Motor execution (=Specification of precisely how the motor plan is to be achieved: –Which muscles contract –How much (movement range and direction) –When (timing)) =Direct activation of motoneurons, muscle contraction, and movement |
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Types of Dysarthria |
-Flaccid -Spastic -Ataxic -Mixed -Hypokinetic -Hyperkinetic -Unilateral UMN |
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**Factors that Impact Dysarthria** |
*Age of onset (congenital or acquired (at any age)) *Cause (vascular, traumatic, infectious, neoplastic) *Natural course (Developmental (CP), Recovering (stroke/TBI), Stable/Chronic (adult CP), Degenerative (ALS), Exacerbating-Remitting (some MS) *Site of Lesion *Underlying neurological diagnosis *Pathophysiology (spasticity, flaccidity, ataxia, tremor, dysmetria, involuntary movements) *Speech subsystems involved *Perceptual characteristics *SeverityFactors |
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**Apraxia of Speech** |
Neurologic motor speech disorder reflecting an impaired capacity to plan / program sensorimotor commands needed for directing movements that result in phonetically & prosodically normal, volitional speech. |
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Apraxia affects: |
Motor Planning (=Selection of appropriate movement strategies in light of intended goals and prevailing physical conditions) |
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**Intelligibility vs. Comprehensibility** |
INTELLIGIBILITY•Info only f/ speech signal COMPREHENSIBILITY•Consideration of info f/ speech signal + context. |
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Darley, Aronson, & Brown |
-Developed use of auditory-perceptual assessment to characterize types & physiologic bases of dysarthrias
•Their clusters of deviant perceptual dimensions formed impt framework for diagnosis & management of dysarthrias. |
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Aspects of Disability / Disorder |
Impairment (body), Activity (limitation), & Participation (restriction - society), |
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Role of SLP in MSDs |
*Differential diagnosis *Intervention -Rehab -Mgmt / compensatory strategies -Maint. -Advocacy |
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Gray Matter vs. White Matter |
Grey = Cells & Dendrites White = Axons |
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Action Potentials vs. Synaptic Transmision |
Action Potential: Electrical info down axon Synaptic Transmission: Chemical info f/1 neuron to another |
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Meninges |
*Dura *Arachnoid *Pia Mater Spaces: -Epidural -Subdural -Subarachnoid |
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Supratentorial Level of N.S. |
*Hemispheres, Lobes, & CN I & II *Basal Ganglia: Striatum (Caudate Nucleus & Putamen), Globus Pallidus *Thalamus: Rt. & Lft. |
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Posterior Fossa |
*Brainstem (Midbrain, Pons, Medulla) *& Cerebellum |
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Spine |
*Cervical: 8 S.N.s *Thoracic: 12 S.N.s *Lumbar: 5 S.N.s *Sacral: 5 S.N.s |
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Peripheral |
*CNs |
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**IMPT CNs for Speech & Swallowing** |
CN V - Trigemnial: Motor for jaw, Sensory to face, teeth, anterior tongue CN VII - Facial: Motor for face; taste; salivation CN IX - Glossopharyngeal: Motor of pharynx & larynx; Sensory to posterior tongue & upper pharynx; taste; salivation CN X - Vagus: Motor for palate; phonation; taste; & swallowing CN XII - Hypoglossal: Motor for tongue |
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**Name all Cranial Nerves** |
*CN I: Olfactory *CN II: Optic *CN III: Oculomotor *CN IV: Trochlear *CN V: Trigeminal *CN VI: Abducens *CN VII: Facial *CN VIII: Auditory/Vestubular *CN IX: Glossopharyngeal *CN X: Vagus *CN XI: Spinal Accessory *CN XII: Hypoglossal |
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**UMN vs. LMN** |
*UMN: -originate in motor cortex; -term. in CN nuclei of brainstem or ventral horn of spine *LMN: Final Common Pathway; every neuron that exits brainstem / spine -originate in CN nuclei of brainstem -terminate on head & neck muscles -originate in ventral horn of spine -terminate on all other muscles |
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**Speech Subsystems** |
*Respiratory *Phonatory *Resonatory *Articulatory *Prosody |
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**Direct Activation Pathway** |
*Pyramidal System = Conscious cntrl of voluntary motor •Direct cntrl f/ cortex (1 synapse bxn origin & FCP) •Tracts from cortex: Corticobulbar & Corticospinal *UMN |
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**Indirect Activation Pathway** |
*Extrapyramidal System = Automatic / Subconscious cntrl of muscle activities (posture, tone, & reflexes) •Indirect cntrl f/ cortex (multiple synapses bxn cortex & termination in FCP) •Tracts from cortex: Corticorubral to rubrospinal and Corticoreticular to reticulospinal *UMN |
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**General Functions of Cerebellum** |
Integrates & Coordinates execution of smooth, directed movements |
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**Motor Speech Examination** |
-Differential Diagnosis (rule out / consistent w/ medical findings?) -Chart Review -Px History (signs v. symptoms) -Oral Mech Exam (see other card) |
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**Oral Mechanism Exam** |
Examination of muscles innervated by CNs: (@ min.: CNs V, VII, IX, X, and XII) –Size, strength, symmetry, range, tone, steadiness, speed & accuracy –Observe at rest, During sustained postures, During movement (Jaw ROM, etc) •Intraoral exam •Non-speech tasks •Reflexes •Respiratory exam |
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Testing CN VII during Oral Mech |
Raise/lower eyebrows, Smile, Pucker, Lip closure against resistance, Puff cheeks full of air & @ rest (symmetry) (LMN Impair: Upper & lower facial weakness; Weakness is unilateral to lesion; UMN Impair: Lower facial weakness; Weakness is contralateral to lesion) Lagophthalmos: inability to shut eye completely |
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Testing CN III observations |
Ptosis, Double Vision (diplopia) |
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Testing CN V during Oral Mech |
Clench teeth and feel masseter & temporalis |
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Testing CN XII during Oral Mech |
Tongue deviation, atrophy, & fasciculations Tongue deviates to weak side LMN Impairment –Right side UMN Impairment –Right side |
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Testing CN IX during Oral Mech Exam |
Palatal Reflex (assymetrical, absent, etc.) |
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Testing CN X during Oral Mech Exam |
Sustained Phonation Wetness / Dryness of vocal quality |
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**Flaccid Dysarthria** |
*Resulting from LMN pathology (CN / SN, Cranial or Spinal Nuclei, Neuromuscular Junction -Can affect 1 or more speech subsystems: **Respiration: short phrases, monoloudness **Articulation imprecision **Laryngeal function: stridor, hoarseness, breathiness, diplophonia, monopitch **Velopharyngeal function: hypernasality / nasal air emission |
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Etiologies of Flaccid Dysarthria |
•Trauma, (surgical, head / neck injury) •Vascular disorders, (brainstem stroke) •Degenerative disease,(ALS) •Demyelinating disease, (Guillain-Barre’ syndrome) •Viral infections, (polio, herpes) •Neuromuscular junction disease, (myasthenia gravis) |
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Flaccid Dysarthria - CN V Lesions |
Typically not involved in isolation. Etiologies: stroke, infection, AVM, tumors, & skull fractures. •Unilateral Impair.: Jaw deviates toward weak side; Reduced contraction w/ bite •Bilateral Impair: Jaw may hang open @ rest; can't close mouth; weakness w/ jaw closure & chewing |
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Flaccid Dysarthria - CN VII Lesions |
•Etiologies: brainstem injuries, acoustic neuroma, herpes zoster, mono, vascular lesions, & trauma •LMN damage impacts both upper & lower face muscles •Unilateral impair: Ipsi hypotonia, unwrinkled forehead, droop eye/brow, eye [partially] open, asymmetry during movement *Impact on speech: –Unilateral impair: more visible than audible, mild artic imprec., Vowels OK -Bilateral impair: Artic imprecision, flutter of cheeks when talking |
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Flaccid Dysarthria - CN IX Lesions |
•Rarely damaged in isolation (b/c overlapping nuclei & proximity of other CNs: CNs IX & X impaired together) •Etiologies: similar to others for lower brainstem•Reduced / asymmetrical gag reflex •Pharyngeal elevation impact during swallowing •No taste f/ ipsilateral posterior 1/3 of tongue |
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Flaccid Dysarthria - CN X Lesions |
-Etiologies: tumors, trauma f/ surgery, infection, stroke, G-B syndrome, motor neuron disease, aneurysms *Nonspeech OME findings: Pharyngeal Branch: .UL: Soft palate hang low on side of lesion & uvula deviates to strong side; .BL: Palate hang low @ rest & moves minimally/not at all during phonation; Gag reflex difficult to elicit; Nasal regurgitation occur during swallowing -SLN branch: If recurrent laryngeal nerve branch isn’t affected, VFs may look normal; In unilateral lesions, affected VF vocal cord may appear shorter than normal; In bilateral lesions, both VFs may appear short & bowed –Unilateral RLN (w/ or w/o SLN): Affected VF weak/paralyzed; May have dysphagia; weak cough & glottal coup -Bilateral RLN (w/ or w/o SLN): Both VFs weak/paralyzed; Dysphagia; weak cough/coup, @ risk for airway compromise, Inhalatory stridor *Speech findings: -SLN & RLN affected: •Breathy / aphonia, Hoarseness, Reduced loudness, Diplophonia, Short phrases, Rapid vocal flutter•See these symptoms w/ UL damage & BL damage (worse) |
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Flaccid Dysarthria - CN XII Lesions |
Etiologies: neck lesions, surgery, trauma, stroke, infection, tumor -Unilateral lesions: Tongue weak (ipsilaterally) & deviate to weak side, –Bilateral lesions: Bilateral atrophy & fasciculations; Protrusion (very) ltd in range; lateralization & elevation impaired |
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Flaccid Dysarthria - Multiple CN Lesions |
•AKA “bulbar palsy” •In brainstem, but, multiple CNs can be affected in diseases such as MG & ALS |
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Flaccid Dysarthria - Spinal Nerve Lesions |
•Etiologies: SCI, MG, ALS, G-B Syndrome •Nonspeech OME findings: Rapid, shallow breathing; Nasal flaring; use of upper chest & shoulder neck muscles to breathe; Unable to sustain subglottal air px •Speech findings:Short phrases, red. loudness; Decr.pitch & loudness variability; speaking on residual air; Vx strained |
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Vagus (X) Nerve Branches |
•Pharyngeal: Motor to pharynx & soft palate •SLN (Superior laryngeal nerve) –Internal: Sensory to laryngopharynx & supraglottic structures –External: CT (cricothyroid muscle) •RLN (Recurrent laryngeal nerve): Motor & Sensory to all intrinsic laryngeal muscles (except CT) |