Microbiology Enteric Bacteria

Front 1

Helicobacter pylori symptoms

Back 1

most are asymptomatic, ulcers (addominal pain, tenderness, bleeding)

Front 2

Helicobacter pylori causitive agent

Back 2

Short,Spiral,Gram-negative,Microaerophile,Multiple polar flagella

Front 3

Helicobacter pylori pathogenesis

Back 3

survive in stomach, uses flagella to corkscrew through mucosal lining causing inflammation, persists for yrs

Front 4

Helicobacter pylori epidemiology

Back 4

fecal oral route (flies)

Front 5

Helicobacter pylori prevention

Back 5

no provention

Front 6

Helicobacter pylori treatment

Back 6

antibiotics

Front 7

Cholera symptoms

Back 7

severe diarrhea (which leads to muscle cramps)

Front 8

Cholera causitive agent

Back 8

Gram-negative,Curved bacillus,Salt tolerant,Tolerates high alkaline environment

Front 9

Cholera pathogenesis

Back 9

Large numbers must be ingested to effect disease due to sensitivity to stomach acid (adhere to epithelial lining in small intestine)

Front 10

Cholera epidemiology

Back 10

Fecal contamination of water

Front 11

Cholera prevention

Back 11

Adequate sanitation

Front 12

Cholera treatment

Back 12

replacement of fluids and electrolytes

Front 13

what destroys cholera toxin

Back 13

heat

Front 14

Shigellosis symptoms

Back 14

dysentery, headache, vomit, stiff neck, joint pain

Front 15

Shigellosis causitive agent

Back 15

S. flexnuri,S. boydii,S. sonnei,S. dysenterriae

Front 16

Shigellosis pathogenesis

Back 16

First step is phagocytosis of bacteria by cells in the large intestine,Cells transport bacteria beneath epithelium,Bacteria adhere to specific receptors near base of epithelial cells,Bacteria multiply at high rate,

Front 17

Shigellosis epidemiology

Back 17

fecal oral route (human to human)

Front 18

Shigellosis prevention

Back 18

Adequate sanitation

Front 19

Shigellosis treatment

Back 19

no vaccine

Front 20

what does shiga toxin inhibit

Back 20

protein synthesis

Front 21

does shigellosis have a high or low infecting dose

Back 21

low (can survive in the stomach)

Front 22

E Coli symptoms

Back 22

depends on virulence, vomitting, lose stool, severe cramps, bloody diarrhea

Front 23

E Coli causitive agent

Back 23

Most diarrhea causing E. coli fall into four groups

Front 24

E Coli pathogenesis

Back 24

Production of enterotoxin,Adherence to cells of small intestine

Front 25

E Coli epidemiology

Back 25

Person-to-person spread,Contaminated food and water,Unpasteurized milk and juices

Front 26

E Coli prevention

Back 26

Hand washing,Pasteurization of drinks,Proper food preparation

Front 27

E Coli treatment

Back 27

Replacement of fluids and electrolytes,Infants may require antibiotics

Front 28

Enterotoxigenic E. coli (ETEC)

Back 28

Most common cause of traveler’s diarrhea (Montezuma’s revenge).

Front 29

Enteroinvasive E. coli (EIEC)

Back 29

Disease closely resembles that of Shigella species

Front 30

Enteropathogenic E. coli (EPEC)

Back 30

Causes outbreaks in hospital nurseries and bottle fed infants

Front 31

Enterohemorrhagic E. coli (EHEC)

Back 31

Often produces severe illness due to production of potent group of toxins

Front 32

Salmonellosis symptoms

Back 32

Diarrhea,Abdominal pain,Nausea,Vomiting,Fever

Front 33

Salmonellosis causitive agent

Back 33

Motile,Gram-negative,Enterobacteria

Front 34

Salmonellosis pathogenesis

Back 34

Bacteria sensitive to stomach acid,Large number required for infection,found in small intestine

Front 35

Salmonellosis epidemiology

Back 35

Main source is FOOD (chicken)

Front 36

Salmonellosis prevention

Back 36

Adequate cooking kills bacterium

Front 37

Salmonellosis treatment

Back 37

vaccine

Front 38

Salmonellosis is associated with what type of shock

Back 38

septic shock

Front 39

Campylobacteriosis symptoms

Back 39

Fever,Vomiting,Diarrhea,Abdominal cramps,Dysentery

Front 40

Campylobacteriosis causitive agent

Back 40

Motile,Gram-negative,Curved bacilli,Cultivated from feces

Front 41

Campylobacteriosis pathogenesis

Back 41

Organisms penetrate intestinal epithelium,Bacteria multiply in and under cells and initiate inflammation,Guillain-Barré Syndrome

Front 42

Campylobacteriosis epidemiology

Back 42

Numerous foodborne and waterborne outbreaks have been reported,unpasteurized milk

Front 43

Campylobacteriosis prevention

Back 43

proper treatment of water and food

Front 44

Campylobacteriosis treatment

Back 44

subside without antimicrobial treatment in about 10 days

Front 45

does Campylobacteriosis have a high or low infecting dose

Back 45

low (500 organisms required to initiate disease)