Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
40 Cards in this Set
- Front
- Back
What are the four critical considerations regarding use of antiarrhythmics
|
1. Establish frequency and cause of arrhythmia incase it can be treated without drugs
2. Low therapeutic index 3. Assess treatment options including no treatment 4. Have rational expectations of therapy including electrophysiological status of the heart |
|
What are the four critical considerations regarding use of antiarrhythmics
|
1. Establish frequency and cause of arrhythmia incase it can be treated without drugs
2. Low therapeutic index 3. Assess treatment options including no treatment 4. Have rational expectations of therapy including electrophysiological status of the heart |
|
Estrogen increases are a factor for intoxication during a part of the female cycle
|
PMS
|
|
What are the physiologic causes of arrhythmias
|
1. Altered impulse formation (e.g. enhanced automaticity)
2. Altered impulse propogation (eg reentry due to ischemia or accessory pathway) 3. Afterdepolarizations (delayed [DAD] or early [EAD]) |
|
What are the physiologic causes of arrhythmias
|
1. Altered impulse formation (e.g. enhanced automaticity)
2. Altered impulse propogation (eg reentry due to ischemia or accessory pathway) 3. Afterdepolarizations (delayed [DAD] or early [EAD]) |
|
Pattern of drinking 5 or more drinks for men in two hours
|
Binge Drinking
|
|
What are the actions of antiarrhythmics
|
1. Decrease spontaneous (Phase 4) depolarization
2. Increase the threshold for depolarization 3. Increase the maximum diastolic potential 4. Change action potential duration |
|
Complete inability to recall
|
Total
|
|
What are the actions of antiarrhythmics
|
1. Decrease spontaneous (Phase 4) depolarization
2. Increase the threshold for depolarization 3. Increase the maximum diastolic potential 4. Change action potential duration |
|
How would one treat, in a physiological sense, arrhythmias resulting from altered impulse formation (increased automaticity)?
|
1. Reduce slope of phase 4 depolarization
2. Make diastolic potential more negative 3. Make threshold potential more positive |
|
How would one treat, in a physiological sense, arrhythmias resulting from altered impulse formation (increased automaticity)?
|
1. Reduce slope of phase 4 depolarization
2. Make diastolic potential more negative 3. Make threshold potential more positive |
|
Condition of a droopy heart due to over consumption of alcohol.
|
Cardiomyapathy
|
|
How would one treat, in a physiological sense, arrhythmias resulting from altered impulse propogation?
|
1. Decrease conduction velocity (drugs target damaged tissue more)
2. Increase refractory period |
|
How would one treat, in a physiological sense, arrhythmias resulting from altered impulse propogation?
|
1. Decrease conduction velocity (drugs target damaged tissue more)
2. Increase refractory period |
|
Irregular heart beat due to over consumption of alcohol
|
Arrhythmia
|
|
What is Wolf-Parkinson-Wright Syndrome?
|
Sort of like having a second AV node that is depolarized at a different time from the other node which then depolarizes the true AV node to result in an arrythmia
"Reentry due to accessory pathway" |
|
What is Wolf-Parkinson-Wright Syndrome?
|
Sort of like having a second AV node that is depolarized at a different time from the other node which then depolarizes the true AV node to result in an arrythmia
"Reentry due to accessory pathway" |
|
Scarred Liver
|
cirrhosis
|
|
How would one treat, physiologically speaking, early after depolarizations?
|
Shorten action potential duration (decrease AA dose)
|
|
How would one treat, physiologically speaking, early after depolarizations?
|
Shorten action potential duration (decrease AA dose)
|
|
Final metabolized break down of alcohol is water and
|
Carbon Dioxide
|
|
How would one treat, physiologically speaking, delayed afterdepolarizations?
|
Reduce Ca2+ overload, decrease dig. dose
|
|
How would one treat, physiologically speaking, delayed afterdepolarizations?
|
Reduce Ca2+ overload, decrease dig. dose
|
|
Stage when you have a low blood pressure and shallow breathing
|
Coma
|
|
Exaggerated psychological state of intoxication
|
Euphoria
|
|
Inflamed Liver
|
Hepa***is
|
|
Behavioral sign that a person needs help and should stop
|
Belligerent
|
|
Brake down of sugar into an alcohol
|
Fermentation
|
|
Alcohol is a ________ drug
|
Depressant
|
|
Stage with mental numbness, difficulty walking and vomit
|
Stupor
|
|
Blurred vison and dulled pain
|
Confusion
|
|
Males have more of this in their bodies
|
Water
|
|
The persons body ceases to breath with the end result as death
|
Respiratory arrest
|
|
Enzyme that helps to metabolize alcohol
|
Dehydrogenates
|
|
Substance where alcohol will not be absorbed in
|
Fat
|
|
Not waking up after this action is a sign of a dangerous condition
|
Vomiting
|
|
First degenerative stage that makes the liver fatty
|
Steatosis
|
|
Liver will brake down before this fat
|
Alcohol
|
|
stage when you have poor coordination and your confidence increases
|
Excitement
|
|
Black out when bits and pieces are recalled
|
Fragmentary
|