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16 Cards in this Set
- Front
- Back
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HyperK: Metoprolol, Atenolol
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Inhibit renin
Inhibit K uptake by cells |
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HyperK: KCl, Penicillin, Polycitra
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Increased K intake
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HyperK: Captopril, Lisinopril
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Inhibit conversion of Ang1-2
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HyperK: Heparin sodium
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Inhibits aldosterone synthase
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HyperK: Spironolactone, Eplerenone
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Aldosterone receptor antagonist
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HyperK: Amiloride, Triamterene
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Block ENaC (Less drive for K secretion)
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HyperK: Trimethoprim, Pentamidine
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Block ENaC
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HyperK: Ibuprofen, Celecoxib
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NSAIDS and COX2 inhibitors inhibit renin production
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HyperK: Digoxin
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Inhibits Na/K Atpase (less drive for secretion)
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HyperK: Cyclosporine, Tacrolimus
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Inhibit Na/K Atpase
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HyperK: Losartan, Valsartan
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Inhibit the Angiotensin's AT1 receptor
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Blocks Carbonic Anhydrase leading to Bicarbonate Wasting
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Acetazolamide
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Anesthesia, NSAIDS, Cyclosporine, Tacrolimus and ACE-I can result in what kidney problem due to changes in vascular resistance?
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Pre-renal AKI
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Aminoglycosides and Contrast can cause what?
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ATN which leads to intrinsic AKI
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How does contrast cause ATN and what are preventive measures?
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Causes vasoconstriction (ischemia) and is toxic to PCT
Prevent with: Hydration, N-acetylcysteine, low vol. and low osm. |
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What meds are responsible for 70% of all acute interstitial nephritis?
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Penicillins and Cephalosporins
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