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180 Cards in this Set

  • Front
  • Back
Peripheral
happening to sensory organs
Central
brain/brainstem, starts at 8th nerve
Functional/Non-organic
They truely believe they have hearing loss- all psychological
organic
physiological, elevated thresholds, not normal. most like peripheral
Atresia
Absense or closure of ear canal. Usually unilateral but can be bilateral
Atresia location
outer ear
atresia caused by
congential absence of external ear canal due to failur to form during embryologic development... alterations during development stage, manifests in different ways. genetics often involved (or syndromic)
Atresia complaints
none as an infant; may have classroom difficulties as a child
Atresia physical signs
atretic ear canal on affected side. may have microtia (deformed pinna?)
Atresia Audio problems
moderate severe conductive loss in affected ear. may also be bilateral
Atresia treatments
Elective surgery; CROS or BAHA hearing aid. preferential seating in classroom.
CROS
type of hearing aid- microphone on bad ear and sends signal to other ear. or if can hear in that ear it will amplify and send signal?
BAHA
newer BA- bone anchored. Put steel bolt in head and attach equivalent to bone ossilator, connected to microphone stimulates cochlea without needing conductive system.
Atresia audiogram
looks like a conductive hearing loss around 60-70 dB
Exotosis
• Bone-like growths arising from the lining of the ear canal
• Often found after repeated exposure to cold water
• Rarely cause hearing loss
• Are generally not removed
may lead to secondary problems like impacted cerumen
Impacted Cerumen
A build up of wax deep in the ear canal. If it's severe enough it can occlude the ear canal and hairs are coated with wax-- debri will collect foreign objects.
an acquired disorder of the external aud canal that occurs when there is an overaccumulation of cerumen that becomes impacted somewhere along the canal.
new wax is
soft not dark
older wax
dark and still soft
old wax
hard and dark
Impacted cerumen location
outer ear canal
impacted cerumen causes
cleaning ears with cotton swabs
impacted cerumen complaints
difficulty hearing on affected side
impacted cerumen physical signs
cerumen plug; unable to visualize tympanic membrane
impacted cerumen audio
mild conductive loss on affected side.
treatments for impacted cerumen
removal of cerumen by health service provider; over-the-counter softening agents; instruct patient not to clean canals with cotton swabs
impacted cerumen audiogram
looks like a mild conductive loss around 30 dB
Otitis Externa (Swimmers Ear)
infection of the tissues lining the external ear canal. these infections are usually caused by bacteria or fungus or viral (like herpes zoster)
painful and may be accompanied by swelling and/or fluid discharge
Usually a dermatological issue but becomes an audiological issue when swells ear canal shut and change in pH balance in ear and throws everything off (sensitive to change)
otitis externa treatment
topical or oral antibiotics depending on the severity. earplugs while swimming may also help prevent otitis media
Treated with dry agents and time
Otitis Media
Inflammation of the middle ear, usually accompaned by fluid (effusion); acquired from poor eustachian tube function; common in children.
Serous otitis media
fluid in the middle ear is clear and noninfected with bacteria
acute otitis media
fluid in the middle ear is infected with bacteria
mucoid otitis (purulent)
fluid becomes thickened or pusslike (glue ear)
chronic otitis media
if fluid remains in the middle ear for an extended period of time (over 8 weeks)
Otitis media causes
poor eustachian tube function and fluid build up in middle ear; may become infected by way of the eustachain tube though coughing or sneezing. common in young children
otitis media complaints
difficulty hearing; lack of attention in school. Fever, tugging on ears, irritability
otitis media physical signs
tympanic membrane may be retracted, reddened, thickened, ruptured; may have visible discharge
otitis media audio
mild to moderate conductive loss in both ears if bilateral. may also be unilateral
otitis media treatments
medical observation; may need antibiotics for infection or PE tubes for chronic/reocurring cases
Perforation
puncture or rupture of the tympanic membrane. can vary in size. PE tube may act like a small perforation
Can be chronic or short term
location of perforation
tympanic membrane
perforation causes
sharp object, slap to the ear, otitis media, or PE tube
trauma to eardrum.. racquetball, water skiing, bomb and explosion-- enequal pressure--
perforation complaints
usually not noticed. may have secondary problem if water gets in ear.
perforation physical signs
small hole in tympanic membrane; may have discharge if related to otitis media
perforation audio
normal hearing to slight conductive loss on affected side.
perforation treatments
none. will usually heal itself (when smaller). swim plugs recommended when in water
tympanoplasty
type 1- patch hold in ear drum
type 5 - replace entire middle ear pretty much
Otosclerosis
Spongy growth of emporal bone around oval window. can encapsulate stapes or invate cochlea. begins in second to fourth decade. some hereditary link.
slow growing process, but if reduces movement of stapes it causes conductive hearing loss
Otosclerosis location
middle ear (stapes footplate)
otosclerosis causes:
bony growth of temporal bone around stapes footplate. appears in second to third decade. may have a genetic link; exacerbated during pregnacy (hormones)
otosclerosis complaints
difficulty hearing. may hear better in noisy situations
otosclerosis physical signs
may see pinkish glow otoscopically (blueish black) called Schwartz's sign
otoscerlosis audio
progressive mild to moderate conductive loss as fixation increases. may begin unilateral and progress to bilateral. characteristic bone conduction notch at 2000 Hz (Carharts notch)
otitis media augiogram
look like mild conductive hearing oss at 20-30 dB
perforation audiogram
looks like mild conductive loss
otosclerosis treatment
elective surgery to replace stapes with prosthesis (stapedecotmy). Hearing aids/BAHA alos options
peak age of onset for otosclerosis
20
sclerosis means
hardening
Cholesteatoma
Skin flap (squamous epithelium) growing in the middle ear, usually from retraction pocket. sloughed-off skin builds up and becomes infected. continues to grow in middle ear and may rupture tympanic membrane, producing smelly discharge. can spread into mastoid or temporal bone and considered a medical emergency
usually secondary to a perforation or otitis media
otorrhea
foul smelling discharge
otalgia
ear pain
cholesteatoma location
middle ear
cholesteatoma causes
skin flap grows into middle ear, eg. from retraction pocket in pars flaccida. sloughed-off skin becomes trapped casuing pseudotumor to grow; becomes keratinized and infected
cholesteatoma complaints
hearing loss on affected side. may have otalgia and smelly drainage from ear
cholesteatoma phsyical signs
whitish mass behind tympanic membrane. may have discharge in ear canal
cholesteatoma audio
normal to moderately severe conductive loss in affeted ear depenidng on how it affects ossicles
cholesteatoma treatments
surgery is required to remove growth. ossicular reconstruction may be option if needed. hearing aids also option if conductive hearing loss dur to removal of ossicles after surgery
#1 reason for sensorineural loss
presbycousis
#1 conductive disorder
otitis media
what percent of kids will have otits media before age 6
90%
if you have an ear infection before age one, what is the possibility of having chronic ear infections
50%
Eustachian tube dysfunction.. the cuase of otitis media has to do with
nursing and breathing at same time. need as a newborn and can grow out of it as get older. then their ET slants downwards as they grow
acute OM
0-21 days
subacute OM
22 days- 8 weeks
chronic OM
greater than 8 weeks
how many doctor visits per year for OM
30+ million
fluid effusion: serous
thin, watery, sterile
(drain out of middle ear lining, looks like bubbles with otoscopy)
fluid effusion: perulent
pus-like infected fluid
fluid effusion: mucoid
thick, mucus like
(advanced, sever, colorful. wait long enough it will perferate eardrum. called glue ear. can smell it across room)
acute otitis media is common with
male, caucasians, people who live in cold climates, and upper respiratory infections
cholesteatoma audiogram
looks like conductive hearing loss from normal to moderately severe depending on how it affects ossicles
For otitis media to occur
1. Eustachian tube has to become dysfunctional
2. must get it so that it is swelled shut and then the ET is easy to clog up when tissue is irriated.
3. seals off middle ear space and can't equalize pressure
4. creates air pocket (body hates)
5. will try to get rid of air pocked by absorbing air (by M.E. lining)
6. will create negative pressure (vacuum)
7. get retracted ear drum from neg. pressure (first seals off, can get pos. pressure, but then has vacuum process start, eardrum is pulled in)
8. since vacuum is created, no air pockets so now moisure lining is being sucked out.. pulls lining away from where it's suposed to be
9. create swimming pool for germs, dark warm, moist, dead skin cells to eat
10. whiteish business in fluid means bacteria is growing
OM otalgia comes a lot from
irritated and inflamed binding of skin around eardrum - that's where a lot of pain come from.
tell if babies have otalgia by
grabbing at ears, cry, irritability more than normal, not eating right
OM tympanic membrane can appear
red
yellow
retracted or bulging depending on stage
may see white stuff
what can you use to tell if PE tubes are working
tympanogram
how many OM cases will resolve by themselves
75-85%
otosclerosis audiogram
conductive loss from mild to moderate. may begin unilateral and progress to bilateral. characteristic bone conduction notch at 2000 Hz
Aerotitis Media (Barotrauma)
inability to get to equalize pressure (airplanes, scuba divers, won't open - not due to infection but due to external extreme pressure changes)
50% of people with PE tubes will get
tympanic sclerosis
tympanic sclerosis
scarring, build up on tympanic membrane may develop mild conductive loss.
recurrent otitis media
more than 3 episodes in 6 months
ossicular erosion
worst thing to do is let it sit too long and bones start to dissolve bc they are little, soft, flexible
Ossicular Disarticulation
separation of ossicular chain or break in one of the ossicles, typicall dislocation of the long process of the incus from the stapes
only disorder that the tympoanogram shows if your ear is too floppy
ossicular disarticulation
disarticulation location
middle ear (ossicular chain)
disarticulation cause
head injury, slap to ear, cholesteatoma
disarticulation complaints
hearing loss on affected side
disarticulation phsyical signs
none. may notice hyperflaccid tympanic membrane, otoscopically
disart. audio
moderately severe conductive loss in affected ear
disart. treatments
ossicular reconstruction may be option if needed. CROS hearing aid or BAHA options if surgery not done or if hearing loss continues after surgery
Cochlear otosclerosis
otospongiosis of temporal bone around oval window invades the inner ear and eithe produces toxins or alteration of the blood supply that destroy the hair cells. It's slow, progressive sensorineural HL.
no reversing, slowly grow, impede movement of basilar membrane
conductive loss inside ear and sensorineural loss in inner ear
NO CARHARTS NOTCH
PROGRESSIVE, SLOW GROWING
INVADES COCHLEA- TOXIC
cochlear otosclerosis location
middle ear around oval window and cochlea
cochlear otosclerosis caues
bony growth of temporla bone around oval window invades cochlea. toxins destroy hair cells in cochlea
cochlear otosclerosis complaints
progressive mild to moderate bilateral sensorineural loss, usually worse in mid frequencies. no carharts notch. if combined with stapes fixation, there may be a mixed hearing loss and carharts notch.
cochlear otosclerosis treatments
sodium flouride. hearing aids when hearing loss affect communication
Noise induced hearing loss (NIHL)
hearing loss that occurs from exposure to extremely loud impulsive type sounds, like an explosion or gunshot, or more commonly from longer term exposures to high level industrial military and or recreational noises, such as jackhammers, airplanes, machinery, or music. ONE OF THE MOST COMMON ACQUIRED SENSORINEURAL HEARING LOSSES
HL associated with NIHL results from
damage to stereocilia, tectorial membrane, metabolic changes to the hair cell, rupture of Reissner's membrane, or complete destruction of the organ of Corti
NIHL location
inner ear
NIHL causes
disruption of stereocilia, first on OHCs, then on IHCs, can produce biochemical changes in HCs or other structural damage to cochlea
NIHL compaints
difficulty hearing, especially in background noise. high pitched tinnitus
NIHL physical signs
none
NIHL audio
sensorineural typically bilateral. begins as a notched loss 3-6 kHz region; progressive, and eventuall affects a wider frequency range
NIHL treatments
none for exisitng damage. ear protection devices when exposed to noise/loud music to prevent further damage. should have hearing conservation program if in noisy work environment
NIHL is characterized by at notch at what Hz on auiodgram
4000 Hz
85 dBA for 8 hours
have to offer hearing protection
90 dBA 8 hours
don't have a choice to wear hearing protection. wear it or i'll fire you
95 dBA
only allowed 4 hours
100 dBA
2 hours
105 dBA
1 hour
110 dBA
half hour
rule for dBA
every increase in 5 dB, only be in it half as long
TTS- temporary threshold shifts
may occur when exposed to loud noise levels for a few hours as might occur from a music concert or loud stereo and you may notice that sounds seem muffled and you have tinnitus
may last 2 mins to 24 hrs.
PTS- permanent threshold shift
may vary depending on type of noise as well as one's susceptibility, other health factors, or exposure to chemiclas or drugs that may damage the ear.
Acoustic trauma
permanent cochlear damage resulting immediately from single exposure to very high sound press. levels like gunshot/explosion
gradualling developing NIHL
slowly develops over time of repeated exposures to sound press. levels lower than those producing acoustic traumas.. common in factory or industrial work.
Presbycusis
hearing loss due to aging
percent of presbycusis over 65
25-40%
percent of presbycusis at 90
90%
Result of hearing impariment in elderly
psychological issues: depression, confusion, inattentivesness, tension, and negativism
Psyiological issues of poor health, reducd mobiliyt and reduced interpersonal communications have been associated
what percent of hearing impaired elderly actually received rehabilitation
20%
gender differences with presbycusis
womean lose LF and retain HF better. HF are worse in men
max output a heading aid can give
140 dB
Presbycusis locaiton
inner ear
Presbycusis causes
hearing loss due to agin, beginning around age 50 years. affects cochlear hair cells, stria vascularions, and or neural auditory pathways
Presbycusis complaints
difficulty hearing, esp. in background noise. high-pitched tinnitus
Presbycusis phsyical signs
none
Presbycusis audio
high frequency sensorineural loss. degree of loss and range of affected frequences increase slowly with age.
Presbycusis treatments
none for existing damage. hearing aids are primary treatment when loss is affecting communication
presbycusis diagnosed by
exclusion
4 types of presbycusis
1. sensory (cochlear)-degeneration of hair cells
2. neural- loss of chochlear neurons resutling in problems of transmission inforamtion coding
3. strial or metabolic - degeneration (atrophy) of stria vascularis in cochlea (stroke may cause this)
4. mechanical or cochlear conductive- results from alterations to cochlear mechanics..due to stiffening as aging, mechanical things wear out.
Meniere's Disease has to have these 4 things
a disorder that is characterized usually by four conditions:
a. episodes of vertigo often sever with vomiting. true rotary vertigo.. room spinning and won't stop - dizziness
b. LF sensorineural HL which can fluctuate in early stages
c. fluctuating aural fullness (pressure in ears)
d. fluctuating low pitched tinnitus (ocean roar)
Meniere's Disease
too high fluid pressure in middle ear
trying to pump a lot of fluid in small confined space.. causes imbalance hearing issues
ENDOLYMPATHIC HYDROPS
Meniere's Disease location
inner ear
Meniere's Disease causes
excessive buildup of endolymp in the scala media and vestibular labyrinths. may occur spontaneously, or from viral attack, or from late onset genetic link
Meniere's Disease complaints
vertigo, ocean roar tinnitus, aural fullness, and hearing loss in affected ear. vertigo may be debiliating
Meniere's Disease physical signs
none
Meniere's Disease audio
fluctuation, low frequency, sensorineural heairng loss, initially. may end up with a perment flat moderate sensorineural loss. often see best thresholds at 2000 Hz
Meniere's Disease Treatments
eliminate salt and caffeine from diet. may benefit from a prescribied diuretic. sever cases may require endolymphatic shunt operation or vestibular/cochlear nerve section
Hearing thresholds change dramatically and have whole life
Meniere's Disease
Ototoxicity
Hearing damage due to chemicals
Ototoxicity location
inner ear
Ototoxicity (from aminoglycosides) causes
systemic biochemical damage to cochlear and or vestibular hair cells from aminoglycoside antibiotic treatment
Ototoxicity complaints
slight hearing loss may be noticed. high frequency tinnitus often very sick from another medical disorder, which is why aminoglycosides were administered
Ototoxicity (from aminoglycosides) phsyical signs
none
Ototoxicity (from aminoglycosides) audio
permanent bilateral high frequency sensorineural loss. begins in ultra high frequency range. degree of loss and range of affected frequencies increase with dosage and time.
Ototoxicity (from aminoglycosides) treatments
none for exisitng damage. may alter drug therapy if appropraite. hearing aids are the primary treatment when loss affects communication
other considerations for ototoxicity
age, kidney function (drugs build up in body), and exisitn ghearing loss (going to be bilateral)
drugs that can cause a sensorineural HL
antibiotics (aminoglycosides)
Diuretctics
aspirin causes TTS and tinnitus; reversible
NSAIDS
antimalarials
chemotherapy drugs
tinnitus current research
brain making up for lack of input
treated with diet in some caess
Maskers are available
tinnitus maskers
little white noise generators in ear all day long
Ototoxicity (from loop diuretics or asprin) causes
loop diuretics affect function of stria vascularis; asprin biochemistry of cochlear hair cells
Ototoxicity (from loop diuretics or asprin) complaints
noticeable hearing loss bc of the involvment of speech frequences. high frequency tinnitus is offten prominent sign of beginning ototoxicity from asprin
Ototoxicity (from loop diuretics or asprin) phsyical signs
none
Ototoxicity (from loop diuretics or asprin) audio
temporary mild to moderate relatively flat sensorineural HL
Ototoxicity (from loop diuretics or asprin) treatment
hearing should improve spontaneously when loop diuretic treatment is finished.
Sudden Sensorineural Hearing loss
rapid onset of hearing loss, considered a medical emergency. urge client to see doctor immediately (may be able to reverse in short window of time) often treatead with steroids, but must be soon after onset.
sudden sen. HL location
inner ear
sudden sensorineural HL causes
autoimmune disorder, viral attacks, or idipathic disorders that produce damage to cochlear hair cells. perilymp fistula can alter fluids and cause hearing loss and vertigo
sudden sensorineural HL physical signs
none
sudden sensorineural HL auido
sudden unilateral relataivcely flat mild to moderate sensorineural loss. may progress and or may spontaneous recover. may occur in other eat at a later date.
sudden sensorineural HL treatments
seek medical attention immediately to stabilize hearing and maximize potential for recovery. typicall treated with corticosteroids. hearing aid should be considered after hearing los has stabalized.
Acoustic Neuroma
a slow-growing benign tumor on the 8th nerve causes hearing loss and vestibular symptoms
son't wpread to other types of body, but can cause nerve damage that will affect hearing
Acoustic Neuroma location
8th cranial nerve usually arises from Schwann cells of veestibular branch of nerve
Acoustic Neuroma causes
benign tumor of unknonw origin. compresses on cochlear nerve; first affects outermost layer of high frequency fibers. damages cochlear blodd supply with continued growth causing cochlear loss
Acoustic Neuroma complaints
difficulty understanding speech, especially on the phone. unilateral high freq. tinnitus. may have sligh balance problem
Acoustic Neuroma phsyical signs
none
Acoustic Neuroma auido
unilateral or asymmetircal high frequency sensorineural loss. degree of loss and range of affect frequencies increase slowly as tumor grows.
Acoustic Neuroma treatments
surgical removal of tumor. with early detection and treatment. may be preserved
Acoustic Neuroma most likely found in two places
internal auditory meaturs (ear canal for 8th nerve) or CPA (cerebellopontine angle.. where the cerebellum and pons meet)
Acoustic Neuroma thresholds
may not chagne but word ID may be poor. disortions make intelligibility hard