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137 Cards in this Set

  • Front
  • Back
In an emergency for an epilepsy patient, what protocol should be done?
Remove any prosthetic appliances
Main objective is to remove objects around patient to prevent injury
No need to move the patient to the floor
Maintain airway- turn head to the side and use high evacuator for vomiting
Oxygen can be used
no restrains (passive)
prolonged intravenous lorazapam administered
What is appropriate dental management of seizure patient?
Schedule preferably within a few hours of taking anti-seizure meds
Try to be calm and prevent non irritating situations
Have patient report aura as soon as it is sensed
If questionable history or poorly controlled seizures, consult with physician
Be alert to effect of medications
Be prepared to handle a seizure
Questions
What type of seizures do you have?
When was you last seizure?
Do you know when a seizure is coming on?
What usually happens during your seizure?
Have you had a seizure during dental treatment?
Are there any special things I should avoid during dental treatment to prevent a seizure?
Nonsurgical treatment= scaling and root planing/ scrupulous home care
Change in drug prescription; takes about 1 year to decrease gingival enlargement
Surgical removal
gingivectomy- not a permanent solution
because often enlargement reoccurs
These are examples of ______.
Treatment of Phenytoin-induced gingival growth
Contributing factors for phenytoin-induced gingival growth are?
Bacterial plaque

Gingivitis

THE BETTER THE ORAL HYGIENE, THE LESS LIKELY LESIONS ARE TO OCCUR OR LESS SEVERE
name the two types of seizures?
1. Partial seizure
2. Generalized seizure
__________
simple partial seizures (consciousness*** is not lost)
Tingling** sensation in arm, finger, foot
Seeing flashing lights***
Speaking unintelligibly

complex partial seizures (consciousness is impaired**) – may last a couple of minutes
Patient sits motionless** or moves in inappropriate ways
Blank stare**
may evolve to Generalized Tonic-Clonic***
Partial
_____________
Absence seizure (petit mal)** non-convulsive* seizures
Blank stare
_______of eyes
Completely unaware they are having seizure
Brief unconsciousness***

Tonic-clonic (grand mal)**
Convulsive* seizures
Both types have loss of ______
Generalized Seizures
Rhythmic blinking
consciousness
Generalized ______-Clonic
_______= momentary sensory alteration involving ____ or smell preceding convulsion in 1/3 of patients.
loss of consciousness**
air forced out (________)
Eyes- roll back and to the side; pupils dilate
muscular rigidity
Urination may occur
Tonic
Aura
sight
epileptic cry
Generalized Tonic-_______
_______- beating movements
Time
lasts about ___ seconds
Respiration
breathing may stop
postconvulsive coma
becomes limp and comatose
postconvulsive phase
consciousness returns gradually with confusion
and headache
Clonic
Muscular spasms
90
A _______ is a convulsive disorder that results from a transient, uncontrolled alteration of the brain function. It is a sudden electrical discharge of neurons in the brain.
seizure
What is usual treatment for a seizure patient?
Medications
Usually taken for life
Children take meds for 1-2 years until seizure
free or until age of 16
Surgery
Vagus nerve stimulation-used when meds are not working. Similar to a cardiac pacemaker in the brain. DO NOT NEED ANTIBIOTIC PREMEDICATION
Does a patient with Vagus nerve stimulation need a premedication?
NOOOOOO
Name the three anticonvulsant medications that seizure patients take.
Phenytoin- Dilantin
Carbamazepine- Tegretol
Valproic Acid – Depakote
________________
⇨ Gingival overgrowth
Phenytoin- Dilantin
. ____________
⇨ Xerostomia, delayed healing
⇨ Drug interaction with
erythromycin ***
Carbamazepine- Tegretol
_________________-
⇨ Excessive bleeding (decreased platelet
aggregation), delayed healing.
⇨ Also used for migraine headaches, mood
stabilizers
Valproic Acid – Depakote
oral manifestations of epileptic patients...
Gingival overgrowth due to Dilantin

Effects of accidents during seizures
scars of lips and tongue
fractured teeth
Hyperplasia usually occurs 42 % of the time. (Gingival overgrowth)
Begins as painless enlargement of gingival margin
Interproximal papilla become involved to cover the occlusal/incisal surface
Located in anterior more often than posterior and buccal rather than lingual
study
________= overdose of insulin
⇨ sugar levels less than 50 mg/dL
________=
⇨ sugar levels greater than 126 mg/dL
Diabetic shock/ hypoglycemia
Diabetic coma/ hyperglycemia
Diabetes mellitus may occur as a result of:
Genetic make-up- Type I or Type II (twin study)
Destruction of islet cells due to inflammation, cancer, or surgery
An endocrine disorder such as hyperthyroidism
Disease caused by the use of steroids
study
Islets of Langerhans=hormone producing cells of _______
Beta cells= endocrine cells which produce _____
pancreas
insulin
__________( represents 10% of diabetics) Absolute insulin deficiency
Type I
_____ (represents 90-95% of diabetics= 10 X more common than Type I) Result of insulin resistance with an insulin secretory defect
Type II
IGT-impaired glucose intolerance /malnutrition related
GDM-gestational diabetes mellitus=abnormal glucose tolerance during pregnancy…. Baby becomes fat!
study
Lack of insulin or insulin action allows:
Build-up of glucose in the tissues
________ leads to increase in glucose excretion and increase in urinary volume

leads to increase to _____ especially in elderly

Also leads to metabolic acidosis which if severe, leads to coma and death=______
Hyperglycemia
dehydration
KETOACIDOSIS
Life expectancy of diabetic- live on average ___ years less than general population OR median age of death is 49 years old
22-24
________
Beta cells are destroyed and no _______ is secreted
Has a sudden onset of clinical symptoms
Often found in individuals under 40 years of age
In children it is preceded by sudden onset of growth spurt
Has a weak genetic component
_______ is leading cause of death
Body build is usually normal or thin due to lack of glucose to cells/ cellular starvation
type 1
NO INSULIN
Renal failure
Cardinal symptoms of Type ____:
_______ (excessive thirst)
_______(excessive urination)
_______ (excessive eating)
Weight loss=body shifts to metabolizing fat to “find” a source of glucose
Loss of strength
I
Polydipsia
Polyuria
Polyphagia
____________
Beta cells _____, but secrete low amounts of insulin and have insulin resistance
Generally occurs after the age of 40
Has either increased insulin resistance or decreased insulin secretion
Has a higher ________ component than Type I
Obesity plays a major role in the development-60-70% are obese
Most common cause of death in patients is ________/ by age 55, 1/3 of Type II diabetics die from coronary heart disease
remain
genetic
myocardial infarction
Cardinal symptoms of Type ____ diabetes is much less common and are more slowly developed
Usual symptoms:
Body build is obese
urination at night
blurred and/or decreased vision
Usual symptoms
Paresthesia (abnormal sensation such as burning, pricking, tingling)
Loss of sensation
Impotence*
Orthostatic hypotension*
2
Monitoring glucose
Glucometer- daily monitoring by patient who is diabetic
Finger prick/ insulin dose determined by
basis of the level of glucose
HbA1c lab test- long term assessment of glycosylation of hemoglobin A
Measures blood sugar control over the
preceding 2-3 months
6%- 8% HbAlc good range/ 20% is out
of control
study
Oral hypoglycemic agents- Type ___ diabetes= ______ (Glucophage)
Insulin therapy- injectable insulin- Type I diabetes
Exubera® inhaled insulin
Insulin pump- worn on a belt and sensors reflect the need for continuous insulin need
Implantable insulin pump
Pancreas transplantation
II
metformin
Medications for _______
Injectable= insulin
Injected because of it’s large molecular size
Novolog*
Humalog*
Humulin*
Lantus
Novolin
Oral preparations
4 groups according to their mechanism of action
Metaformin*
Glucagon*
Acarbose
Piogliatazone
Diabetes
_______________
__________ REACTION DUE TO EXCESS OF INSULIN/ BLOOD GLUCOSE LOW
Rapid onset
Patient has failed to eat , but has taken medication
Patient has symptoms such as confusion, cold sweats, headache and shaking
insulin shock
HYPOGLYCEMIC
Cardinal symptoms of Type ____ diabetes is much less common and are more slowly developed
Usual symptoms:
Body build is obese
urination at night
blurred and/or decreased vision
Usual symptoms
Paresthesia (abnormal sensation such as burning, pricking, tingling)
Loss of sensation
Impotence*
Orthostatic hypotension*
2
Monitoring glucose
Glucometer- daily monitoring by patient who is diabetic
Finger prick/ insulin dose determined by
basis of the level of glucose
HbA1c lab test- long term assessment of glycosylation of hemoglobin A
Measures blood sugar control over the
preceding 2-3 months
6%- 8% HbAlc good range/ 20% is out
of control
study
Oral hypoglycemic agents- Type ___ diabetes= ______ (Glucophage)
Insulin therapy- injectable insulin- Type I diabetes
Exubera® inhaled insulin
Insulin pump- worn on a belt and sensors reflect the need for continuous insulin need
Implantable insulin pump
Pancreas transplantation
II
metformin
Medications for _______
Injectable= insulin
Injected because of it’s large molecular size
Novolog*
Humalog*
Humulin*
Lantus
Novolin
Oral preparations
4 groups according to their mechanism of action
Metaformin*
Glucagon*
Acarbose
Piogliatazone
Diabetes
_______________
__________ REACTION DUE TO EXCESS OF INSULIN/ BLOOD GLUCOSE LOW
Rapid onset
Patient has failed to eat , but has taken medication
Patient has symptoms such as confusion, cold sweats, headache and shaking
insulin shock
HYPOGLYCEMIC
Stages of ________
Mild
* characterized by hunger, weakness, trembling, tachycardia, pallor and sweating
* Occurs before meals, during exercise, when food has been omitted or delayed
Moderate
* Patient becomes incoherent, uncooperative, belligerent
* Patient may injure him/herself or some one else
Severe
* Complete unconsciousness, with possible seizure
* Takes place during sleep, or after exercise, or after ingestion of alcohol.
* May need glucose IV
insullin shock
_____________
Hyperglycemia= build of glucose in blood
Slower onset than diabetic shock, but is more life threatening
Occurs from eating and neglecting to take medication
Symptoms are ketoacidosis, increased thirst and urination, fruity breath
Unable to manage infections and heal wounds
Body fluids become acidic
Decreased blood flow to area of injury
Severe acidosis leads to coma and death
diabetic coma
Diabetic emergency
If you are unsure which scenario is happening and patient has passed out or incoherent, go ahead and dispense _____ source ASAP
glucose
Cardinal symptoms of _____ - refer to physician
Findings which may suggest diabetes:
headache, dry mouth, blurred vision
progressive periodontal disease,
multiple periodontal abscesses
diabetic
What are some dental recommendations for brittle diabetics?
Patient with brittle diabetes:
may need special dietary instruction after periodontal or surgical procedures and may need antibiotic prophylaxis if they develop an infection
Appointment Management of diabetic
Major goal is to prevent insulin shock during dental appointment
Morning appointments-preferably right after breakfast
Confirm patient has eaten and taken insulin
Keep a source of sugar available
Patient with brittle diabetes:
may need special dietary instruction after periodontal or surgical procedures and may need antibiotic prophylaxis if they develop an infection
Limit use of local anesthetic to the well-controlled diabetic
study
Diabetic patient who is controlled and under care can receive any indicated dental treatment
Delay treatment ***when fasting blood glucose is < 70mg/dL or > 200 mg/dL
Recommend a liquid diet after extensive periodontal procedures and/or surgery (blend their normal diet in a blender)
Have patient report any symptoms while you are working
study
Delay treatment of diabetic when fasting blood glucose is < ___mg/dL or > ____ mg/dL
70
200
oral manifestations of _______
Accelerated periodontal disease in poorly controlled diabetics
Periodontal abscesses
Increased caries- increase sucrose level in parotid gland in times of unbalanced glucose levels
Xerostomia- due to increase of fluid depletion
Poor healing
Oral ulcerations
Candidiasis
Bacterial and fungal infections
Numbness of oral tissues- diabetic neuropathy (nerves are damaged)
Burning of oral tissues- diabetic neuropathy
Lichen planus- altered immune system
Children of diabetics have 28% more frequency of enamel hypoplasia
diabetics
Emergency management of diabetic patient
place pat. in supine position
administer oxygen
if conscious, give high sugar
not conscious rub high sugar on buccal mucosa
monitor and record vital signs
Call 911
insulin shock improves immediately
________ is autoimmune disease of unknown etiology that is characterized by:
symmetric inflammation of _____ joints
hands
feet
knees
rheumatoid arthritis
large
Pathophysiology of ______
Changes in _______ (inner lining of the joint capsule)
edema
thickening
folding
Marked infiltration of cells into capsule
lymphocytes
plasma cells (B and T cells)
Enzymatic activity of cells destroys cartilage
Healing leads to granulation tissue
_________“”
RA
synovium
squeaky wheels
Drug therapy for ____
1.aspirin= A LOT…. 5 tablets/ 4X day
2. NSAIDS
ibuprofen=Motrin, Advil,
3.COX 2 inhibitors =
Celebrex/
Vioxx (off the market now)
4. naproxen=Aleve
5. Injectable glucocorticoids- not long term
6. Gold compounds
7. Immunosuppressive= Methotrexate
RA
What are oral manifestations of RA?
TMJ involvement- because of erosion of the condylar head; found in up to 75% of patients.

Anterior open-bite possible
What is specific dental management for RA?
Short appointments
Allow patient to make frequent position changes for comfort
Sitting or semi supine
Physical supports such as rolled towels
Pre medication needed for joint replacements
Drug related side effects(RA)
Prolonged bleeding due to _____
Immunosuppressives may cause ________
Gold salts may cause _____
NSAIDS
oral ulcerations
stomatitis
________ is a Degenerative joint disease. It is the most common form of arthritis
etiology
Unknown
May be the end result of normal wear and tear on joints over a long period of time
Genetic predisposition
Obesity leading to overloading of joints
osteoarthritis
pathophysiology of _____
Articular cartilage becomes _____
Joint thins after a period
leading to softening of the cartilage
There is:
progressive splitting
and abrasion of cartilage down to the subchondral bone
The exposed bone becomes “_____” and sclerotic
New bone forms at:
the margin of reticular cartilage
in the non-weight-bearing part of the joint
This creates “spurs”, often covered by cartilage
This augments the degree of deformity
OA
thicker
polished
S&S of ___
Initial symptoms localized to one or two joints

Dull painful swelling of the joints lasting 15 minutes typically in the morning or after inactivity

Heberden’s nodes/Bouchard’s -painless bony growths

TMJ symptoms:
Crepitus
Stiffness
Pain during chewing
OA
Oral complications of ___
TMJ involvement= radiographic changes such as decreased joint space
Occasional pain
Crepitus
Pain upon wide opening
OA
Medical management of __
Acetaminophen (Tylenol) considered first line of defense
Aspirin or NSAIDS if Acetaminophen is not effective
Narcotics for acute flare ups
Joint replacement
OA
New guidelines
Given the potential adverse outcomes and cost of treating an infected joint replacement, the AAOS
recommends that clinicians consider antibiotic prophylaxis for all total joint replacement patients prior to any invasive procedure that may cause bacteremia.
However, patients with pins, plates and screws, or otherorthopaedic hardware that is not within a synovial joint are not at increased risk for hematogenous seeding by Microorganisms
study
Two thirds of PJI were the result of ____ infections
4.9% caused by STREP of possible oral origin most likely by physiological factors
Evidence is weak
STAPH
Prosthetic implants placed to restore esthetics or function ARE NOT considered at risk for bacterial seeding from oral invasive procedures and DO NOT require antibiotic coverage.
Example: breast and penile
study
S&S of ___
Joint inflammation and damage
Joint stiffness
Muscle and soft-tissue weakness
TMJ more frequently involved
High fever and/or rash
High level of white cells in the blood
Enlarged:
lymph nodes
liver
spleen
juvenille arthritis
S&S of ____
Arthritis:
the most common manifestation (76%)
Butterfly rash of:
nose
and cheeks
on areas of sun exposure
Recurrent pharyngitis
Oral ulcerations
Renal abnormalities
Neuropsychiatric symptoms
Pulmonary manifestations
SLE
Oral manifestations of ____
Oral lesions of lips and mucous membranes 5-25% of the time
Resemble lichen planus or leukoplakia
Xerostomia
Hyposalivation
Dysgeusia and glossodynia (burning or painful tongue)
SLE
Autoimmune disease classified with the rheumatic diseases which affects the salivary and lacrimal glands. (Dry mouth and dry eyes)
SJOGREN’S SYNDROME
oral manifestations of ______
Hyposalivation-less than 5% or normal quantity of saliva
Glossitis
Burning tongue-tongue become depapillated and fissured
Candidias of the tongue
Mucositis
Parotid gland hypertrophy
Angular cheilosis
Dysgeusia- taste dysfunction
SJOGREN’S SYNDROME
Glucocorticoids = (cortisol)
Cortisol regulates
Carbohydrate, fat, and protein ______
Inhibits _____
Maintains homeostasis during physical/emotional stress
metabolism
inflammation
_______= (aldosterone)
Aldosterone regulates:
sodium and potassium balance in the kidney
maintenance of extra cellular fluid
Mineralocorticoids
Primary adrenocortical insufficiency:
hypoadrenalism
* _____ disease
Secondary adrenocortical insufficiency: 5 times more common than primary
hypoadrenalism
* Pituitary disease or steroid therapy
Excess production of adrenal products: hyperadrenalism
*______ disease
Addison’s
Cushing’s
_______=deficiency of aldosterone and cortisol
Primary symptoms
weakness
fatigue
abnormal pigmentation***
Addison’s disease
Rare but life threatening in spite of steroid supplementation
⇨ If patient is challenged by stress, they may not have enough steriods to handle the stress
⇨ Considered a medical emergency that can result in death:
* profuse sweating
* sunken eyes
* severe hypotension
* nausea, vomiting
* circulatory collapse
⇨ Requires IV injection of glucocorticoid
adrenal crisis
Hypoadrenalism:
⇨ Patients undergoing stressful surgical procedures may need additional supplemental glucocorticoids
study
Prevention of Complications
Routine dental care can be provided for patients with adrenal insufficiency
Only those going under stressful situations such as surgery might need supplemental glucocorticoids
If additional glucoccorticoids are taken, they are taken within 2 hours before the procedure
study
____________
A patient who has been receiving high dose corticosteroid therapy
Also considered hyperandrenalism
* hypertension, heart failure, depression
* increased risk for osteoporosis
* weight gain
* round or moon shaped face
* “buffalo hump” on back
* acne
HYPERADREALISM CUSHING’S SYNDROME
Hyperadrenalism: (Cushing’s syndrome)
⇨Monitor blood pressure*** (increased likelihood of hypertension)
⇨ Address the risk of increased risk of periodontal involvement ***(osteoporosis)
study
Hyperthyroidism = _________
Hypothyroidism = ______ or cretinism)
Thyrotoxicosis
(Myxedema
______________
SYMPTOMS
Restlessness, fever, tachycardia, sweating, onto coma and or death.
* BEGIN EMERGENCY TREATMENT:
- cool patient with cold towels because of
elevation of body temperature
- injection of hydrocortisone
- call 911
thyrotoxic crisis
oral manifestations of ______
Osteoporosis
More aggressive perio
Extensive caries
Premature loss of deciduous teeth
Early eruption of permanent teeth
Early jaw development
Tumors found in midline of posterior dorsum of the tongue
thyrotoxicosis
S&S of ______
Signs and symptoms - eyes
retraction of upper lid
bright-eyed stare
lower lid lag
jerky motions of lid
Found in 50% of Grave’s patients and causes the most disability of the symptoms
graves disease
Patients with untreated or poorly managed thyrotoxicosis are highly sensitive to:
____________-
or other amines
Do not use local anesthetic with EPINEPHRINE or impregnated epinephrine cord if patient is not being managed with physician
epinephrine
____________________
Five to six more times common than hyperthyroidism
10% of women over the age of 40 have a thyroid hormone deficiency
Treated with:
thyroid extract of synthetic thyroid***
Hypothyroidism/Acquired or Congenital
Oral manifestations of ______
Increased tongue size
delayed eruption of teeth
malocclusion
gingival edema
cretinism
Examine gland- enlarged thyroid will feel ____ than the normal gland
Normal gland- will feel _____
Posterior dorsal region of tongue-examine for nodule that could be lingual thyroid disease.
Carcinoma- will feel ___ and are usually isolated swellings (estimated number of cases in the U.S. 18,400 with 75% being women)
softer
rubbery
firm
Recognition of ____ (coma):occurs in untreated severe hypothyroidism.
hypothermia
bradycardia
hypotension
epileptic seizure
seek immediate medical aid
hydrocortisone (100-300mg)
CPR
myxedema
_____________
Hypotension to normal, BP, bradycardia
Intolerance to cold
Intolerance to CNS depressant drugs
Unintentional weight gain
Edema of face, tongue, neck, goiter
Lethargic, fatigued, dry skin
Medical emergency=myxedema coma
HYPOTHYROIDISM
____________________
Hypertension, tachycardia
Elevated body temperature
Intolerance to epinephrine
Weight loss
Bulging eyes, goiter
Nervous, trembling, sweating
Medical emergency= thyroid storm
HYPERTHYOIDISM
____________ is a condition in which fluid accumulates in the brain
Hydrocephalus
CSF shunts do not appear to increase the risk of bacterial infection after dental procedures
American Heart Association issue a statement that pre medication is not needed before dental procedures (2003)
study
With the ___________ shunt in place, cerebrospinal fluid flows into the ventricular (collection) catheter and down the exit catheter, which shunts the fluid into the peritoneal cavity.
Ventriculoperitoneal
_______
Patients may complain of initial symptoms to dental hygienist of abnormal facial pain, visual disturbances
Dental care should be given during remission
According to impairment; i.e. wheelchair transfer
Short _______ appointments due to the fatigue factor in the afternoon
Management for multiple sclerosis
morning
oral manifestations of MS
Occur only 2-3% of the time
____ of the orofacial structures
Produces slow, irregular speech
Numbness
A progressive irreversible disease characterized
by degeneration of the brain cells and leading to
severe dementia
Alzheimer’s Disease
The Alzheimer’s patient should be placed in an aggressive preventive dentistry program ( in the early stages of the disease) which should include:
__ month recall
Oral examination
Prophylaxis
Fluoride gel application
OH education- may be given to primary caregiver
Adjustment of prosthesis
Patients with advanced dementia may require sedation and short appointments
3
Management of ______ patients
Repeat instructions and explanations
Nonverbal communication can be helpful
Facial expression and body posture should demonstrate support and care
Positive nonverbal communication includes:
direct eye contact, smiling, touching the patient at the shoulders or arms
Alzheimer’s
Drug consideration for Stroke pat.- for oral surgery
aspirin-obtain pretreatment bleeding time(<__ minutes)
Warfarin (Coumadin)-obtain prothrombin time
<2.5 seconds PT or < 3.5 INR
20
Oral complications of ______
Might have difficulty in swallowing
Slurred speech
Right side damage leaves left side possibly neglected
Food and debri might accumulate in impaired area
Might see ______ of the carotid arteries on panoramic films. (Near C 3 or 4) This indicates a risk of stroke and patient may need referred
stroke patients
calcified plaques
emergency management of stroke patient
Patient can be given oxygen
EMS should be activated ASAP
Signs of a patient having a stroke:
Can not smile
Can not raise both arms 
Can not speak a simple sentence coherently
study
Medical management of stroke
First is prevention
Fruits, vegetables, exercise have significant protective effects
Reduce or eliminate high BP, smoking diabetes
Aspirin 81- 325 mg daily reduces stroke by 25% in the at risk population
After stroke
Anticoagulant therapy for thrombosis or embolism
Possibly corticosteroids (reduce cerebral edema)
Surgery
Stent
Rehabilitation
study
_____ is a neurologic deficit caused by sudden interruption of oxygenated blood to the brain
Focal necrosis of the brain tissue is the end result
This interruption is usually caused by
thrombosis of a cerebral vessel- 60-80% of strokes
embolism (blood clot)
or intracranial hemorrhage/aneurysm- 15% of strokes
Cerebrovascular Accident (CVA
Most commonly, a major stroke is preceded by one or two Transient Ischemic Attack (TIA) within a day to a week. There are some warning signs.
study
Described as a “mini-stroke”
Numbness of face, legs, arms, tingling or
speech disturbances lasting less than 10
minutes
Ischemia= temporary deficiency of blood flow to an organ or tissue.
TIA
modes of transmission for ___
Transmission by inhalation:
A typical infection:
inhalation of infected droplets
are then carried to the alveoli
bacteria settle
begin to multiply
Infection progresses locally
may involve regional lymph nodes
Infection is dependent on the number of organisms inhaled and the susceptibility of the individual
Most commonly by:
Cough or sneezing or talking
Smaller droplets evaporate readily
Bacteria float in the air
are easily inhaled
Transmission by ingestion:
rarely occurs since pasteurization of milk
TB
_______=The interval from infection to development varies:
from a few weeks to decades
most cases are due to reactivation of tubercle-could even be 20 years later
about 10% result from initial infection
transmission
oral manifestations of ____
Oral ulceration (uncommon)- deep irregular ulcer on the back side of the tongue
Tuberculosis involvement of:
cervical lymph nodes
submandibular lymph nodes involvement called scrofula- large and painful nodes that abscess and need drained
TB
What are dental modifications for TB?
No treatment planning modifications are required for these patients if disease is not active or patient has received proper medication for proper length of time
Dental patients are placed in 4 categories for management purposes.
Patient with recently diagnosed clinically active and sputum-positive TB
Patients with past history of TB
3. Patients with recent conversion to “+” TB skin test
4. Patients with signs and symptoms of TB
study
What are the three drugs used for TB?
RIFAMPIN (can cause leukopenia and thrombocytopenia=increase in gingival bleeding)
ISONIAZID
PYRAZIMAMIDE
These three drugs are given:
for 8 weeks if susceptible organisms are present
Followed by isoniazid and rifampin
(without pyrazimamide)
for the next 4 months
To complete a 6 month treatment
ADVERSE EFFECTS OF THESE DRUGS:
WITH THE LIVER , GI, AND XEROSTOMIA
These are oral manifestations of ________ substance abuser
Poor hygiene and neglect (caries)
Nutritional deficiencies resulting in glossitis/ loss of papilla/ angular cheilitis
Sweet musty odor*** associated with liver failure
Enlarged parotid glands** soft and non tender
Increased risks factor for development of squamous cell carcinoma in lateral boarder of tongue and floor of the mouth
alcohol
Modifications to Dental Treatment for ______________
Postpone treatment for intoxicated patients

Pain control a problem: aspirin may cause GI problems of bleeding; acetaminophen, up to ___day (limited dose)

_______can be used

A thorough head and neck exam for squamous cell carcinoma

Any non painful oral ulceration present for more than two weeks should be referred for biopsy

Recommend _________ mouth rinses
Alcoholics
4g/
Lidocaine
non alcohol
GUM® brand Chlorhexidine Gluconate Mouth Rinse -
Crest Pro-Health
Peroxyl
Biotene
Plax
Tom’s Natural mouthwash
Are all examples of _______ mouthrinses?
non alcoholic
Peridex™ Chlorhexidine Gluconate 0.12% Oral Rinse prescribing information

Recommended use is twice daily rinsing for 30 seconds, morning and evening after tooth brushing. Usual dosage is 15ml of undiluted Peridex Oral Rinse. Patients should be instructed to not rinse with water, or other mouthwashes, brush teeth, or eat immediately after using Peridex Oral Rinse. Peridex Oral Rinse is not intended for ingestion and should be expectorated after rinsing.
study
Dental Management of ______ User
Should not receive any treatment for at least ___hours after last use
Danger of myocardial ischemia and cardiac arrhythmia **
Don’t use local anesthetic with ___ if treatment occurs within 24 hours after use
Patient should not be prescribed addictive substances**
Cocaine
24
epi
Oral Manifestations of _________

_______ is characterized by broken, discolored and rotting teeth

The drug causes the salivary glands to ______, which allows the mouth's acids to eat away at the tooth enamel, causing cavities

Teeth are further damaged when users obsessively grind their teeth, binge on sugary food and drinks, and neglect to brush or floss for long periods of time

Increased incidence of perio
disease because of ______ to tissues causing permanent damage
Methamphetamine
Meth mouth"
dry out
vasoconstriction
Dental Treatment Modifications of _______
No treatment for at least ___ hours after drug administration ⇨ peak blood levels may last 24 hours

Caution with local anesthetics, nitrous, and prescription narcotics-wait 8 hours ⇨ could cause severe hypertension

Speak slowly , using low voice
Use slow movements and hands visible

No bright lights** ⇨ may cause them to react violently

Keep patient talking to avert paranoid thoughts

Often full mouth extraction indicated***
methamphetamine
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Dental Treatment Modifications for _______ users
Heavy users should refrain from use at least ______ before dental treatment with local anesthetic
Marijuana
one week
Acute effects of ______
Mental/ euphoria, relaxation, increased visual and auditory awareness
Ocular/visual- dry, bloodshot eyes, impairs tracking ability
Hormonal/ disrupts testosterone secretion, reduced sperm count
Increased appetite, ______ increased heart rate

Difficulty concentrating, disrupts short term memory
marijuana

dry mouth
Morphine
Heroin

Hydrocodone= Loratab®,Vicodene hydrocodone + acetaminophen

Codeine=Plain codeine (injection)
Tylenol#2,3,4®
codeine + acetaminophen

Oxycodone=
Percocet®
acetaminophen +oxycodone
Percodan®
aspirin +oxycodone
study
_____ is a Fat soluble form of morphine
heroin
Which two forms of Hep is contracted through feces, route of transmission is through feces-oral, and are not chronic infections?
Hep A and E
HAV=
infectious hepatitis
2-3 week period before onset of jaundice
communicability decreases after jaundice appears
Hep A
______:
before jaundice appears (1-2 weeks)
______:
while jaundice is present(GI symptoms for 2-8 weeks)
________:
without jaundice
Jaundice may appear in adults, rarely in children
Preicteric:
before jaundice appears (1-2 weeks)
Icteric:
while jaundice is present(GI symptoms for 2-8 weeks)
Anicteric:
without jaundice
Jaundice may appear in adults, rarely in children
(HBV); ________ virus:
DNA type virus
(HBsAg); Hepatitis B surface antigen
found on surface layer of virus
serum marker in ____ disease
carrier state***
Hepatitis B
acute
(Anti - HBs); ______- YOU
indicates immunity
or passive antibody
from HBIG (vaccination)

(HBcAg); ________
found in center/core of virus
marker for ____ disease
when found < 6 months
persistent infection
when found > 6 months
antibody to HBsAg
Hepatitis B core antigen
active
(HBeAg) ; ________
indicates high infectivity***
persists into carrier state
Hepatitis B “e” antigen:
Direct percutaneous inoculation-needle sharing, tattooing, body piercing
FIVE TYPES OF TRANSMISSION OF ____
HBV
OCCUPATIONAL TRANSMISSION
Usually through a sharps injury with contaminated blood
HBV and HCV are the only hep virus with occupational risk. HCV is at _____ risk
Risk of contracting HBV or HCV after an injury is 6-30%
less
Absorption of infective secretions; the most frequent route of transmission in the U.S. is by sexual activity is the most frequent mode of transmission for _____
HBV
____________
Patient undiagnosed for hepatitis but:
develops antibodies
permanent immunity
transient because:
the individual has a rapid immune response
strong immune response to the HBV
HBV is cleared before it can become established
May persist for decades causing liver damage and/or heptatocellular carcinoma
Transient Subclinical Infection
Postexposure Prophylaxis
Immediately “bleed “ the wound
cleanse with soap and water
Draw patient blood for:
serology (HBsAg) and
Draw blood from inoculated individual for:
serology (Anti-HBs)
Test results.....
HBsAg- patient:
no treatment

HBsAg+ patient:
no treatment if vaccinated
and have adequate level of Anti-HBs+ level
study
Post exposure Prophylaxis
Patient HBsAg+:
Practitioner unvaccinated
give HBIG within 48 hours: (Hepatitis B immune globulin. Passively delivered antibody that provides “instant” protection against HBV)
and begin HBV vaccination series
Patient HBsAg+:
practitioner vaccinated
with inadequate levels of Anti-HBs+
HBIG and vaccine booster
study
HCV - Hepatitis C Virus
Most common _______ blood borne infection in the United States
Illicit drug users (60% of HCV are in this group and HCV is four time more common than HIV)
chronic
HDV - ______:
cause of delta hepatitis
causes infection only in the presence of HBV (co-infection)
HD Ag:
___________
detectable in early acute delta infection
Delta virus
Delta antigen
HEV :
Cause of hepatitis E
Hepatitis E virus name for:
enterically transmitted
(through small intestine) nA-nB hepatitis
similar to HAV-fecal/oral
Anti – HEV:
Hepatitis E antibody
indicates past or present infection to HEV
study
No dental treatment other than urgent care for patients with active hepatitis unless patient is clinically recovered
***study
No treatment modifications are required for patients who have recovered from hepatitis/ STANDARD PRECAUTIONS
Patients with chronic active hepatitis or who are carriers:
⇨ liver becomes unable to
metabolize drugs
⇨ lidocaine becomes a factor
and ___ carps of 2% lidocaine
is the limited dose
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