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47 Cards in this Set

  • Front
  • Back
normal PR interval
3-5 small box (120-200 msec)
normal QRS
2-3 small box (80-120 msec)
normal QTc
9-11 small box (360-440 msec)
1 mm (small box) = ?
0.04 sec or 40 msec
to check p-wave, look at lead ___.
II (mainly), III, aVF
normal PR interval
3-5 small box (120-200 msec)
normal QRS
2-3 small box (80-120 msec)
normal QTc
9-11 small box (360-440 msec)
1 mm (small box) = ?
0.04 sec or 40 msec
to check p-wave, look at lead ___.
II (mainly), III, aVF
atrial fibrillation will show up as what ekg?
no p-wave with QRS complex irregular spacing (this is most common arrhythmia)
what's the HR jingle that Dubin taught you?
300, 150, 100...75, 60, 50 (from thick line to thick line)
bifid or notched p-wave in lead II shows ____ ____ _________.
left atrial enlargement.
what will you look for in right atrial enlargement?
tall p wave > 2.5 mm (small boxes) in lead II
what would you look for on ekg:
- left atrial enlargment
- right atrial enlargement
- AV block
- bifid P in lead II
- tall P wave > 2.5
- prolonged PR interval (> 5 boxes or 0.20 sec)
name some reversible causes of P-R prolongation (AV node block)
-drugs (beta blockers, ca channel blockers, digoxin)
-acute ischemia or infarction
what is an irreversible cause of PR prolongation?
degenerative or calcific dz of the conduction system (Lev's dz)
name the triad caused on the ekg by wolff-parkinson-white (WPW)
short PR (<120 msec)
wide QRS (>3 blocks)
delta wave (slurring at initial rise of R wave)
what does delta wave ALWAYS mean?
that the conduction is anterograde depolarization down the accessory pathway, bypassing the AV node (this is less common - 20% - and is more dangerous)
what happens to QRS complex in BBB?
also, differentiate b/w RBBB and LBBB
ventricle with BBB is depolarized late, so that you have an R(prime) for the later ventricle

you'll have:
- wider QRS (3+ boxes)
- for RBBB: look in V1 and V2 for "rabbit ears"
- for LBBB: look in V5 and V6 (not rabbit ears)
causes of BBB
- ischemia/infarct of BB
- eccentric hypertrophy of that ventricle (will take longer to depolarize causing wider QRS and RSR')
most common congenital dz presenting in adulthood
ASD (atrial septal defect) = shows up as R BBB/R eccentric hypertrophy (blood shunts from left to right atrium, filling RV with more blood)
what is the normal QTc? how do you calculate it?
360-440 msec

QT divided by square root of R-R interval
complication of long QT interval
Torsades de Pointes (poly morphic ventricular tachycardia)
causes of prolonged QT interval
- drugs (anti-arrythmics, anti-psychotics, TCAs)
- electrolyte abnormalites (low K, low Mg or low Ca)
what is the most common electrolyte abnormality leading to prolonged QT interval?
low K
name the drugs that prolong PR (block AV node)
beta blockers, Ca channel blockers, digoxin, amiodarone, clonidine
what do you look for on an ekg if it's volume overload (eccentric hypertrophy)?
LBBB or RBBB
what do you look for on an ekg if it's pressure overload (concentric hypertrophy)?
RVH, LVH
what is the voltage criteria for LVH and RVH?
LVH: R (V5 or V6) + S (V1 or V2) > 35 mm

RVH: Tall R > 5 mm or R>S in V1 or V2
what's the axis criteria for LVH and RVH?
LAD in LVH and RAD in RVH: “Overall QRS axis shifts to the hypertrophied ventricle”
name the 11 steps in ekg reading
-rhythm
-rate
-P wave morphology
-QRS axis
-PR interval
-QRS interval
-QT interval
-Chamber enlargement: LVH, RVH
-pathologic Q waves
-ST and T wave changes
-Precordial R wave progression
What are the 3 criteria for pathologic Q waves?
1. negative initial QRS
2. WIDER than 0.04 sec (40 msec) - one small box
3. deeper than 1/3 of subsequent R wave in same QRS
what are the 3 hallmarks of an evolving MI?
pathologic Q wave
ST elevation
inverted T waves
1)ST elevation =
2)ST elevation AND path Q waves =
3)path Q waves ONLY =
acute MI
evolving MI
old MI
ST depression signals _____ ____.

ST elevation signals _____.
1) subendocardial ischemia (this is the first part to have ischemia - since it's in the area that's farthest from coronary arteries) --> you'll see this before elevation

2) acute MI
what are the 3 things you see wrong in this ekg?
pathologic Q waves
ST elevation
inverted T waves

this is an evolving MI!!
The ST segment depression that is most characteristic of ischemia is:
a. downsloping
b. upsloping
c. horizontal
d. A and C
D. downsloping and horizontal

upsloping ST segment depression can occur in nl person during stress test
The most life-threatening cause of ST segment elevation is:
a. Acute Myocardial Infarction
b. Acute Pericarditis
c. Coronary vasospasm (Prinzmetal’s angina)
d. Early Repolarization
a. AMI
differentiate acute MI vs. pericarditis in terms of 1) ekg lead, 2) presence of pathologic Q waves, 3) PR morphology
acute MI = localized ST elevation (think only one artery/branch involved), path Q wave, NO PR depression

acute pericarditis = diffuse ST elevation, no path Q waves, depressed PR
what are the 3 things ST elevation could be?
-AMI
-pericarditis
-coronary vasospasm (Prinzmetal's angina)
if you see tall ST segment elevations in leads I, aVL and V2-V6, dx?
massive acute MI
three things to spot for evolving MI
pathologic Q wave
ST elevation
inverted T wave
what can a peaked/tall T wave (>10mm) mean?
high potassium (mainly!)
AMI
ischemia
what if you only see ST elevation?
acute MI!!
what could a tall R in V1/V2 indicate (precordial R wave progression)?
old posterior MI
RVH
RBBB
WPW
by itself, what are pathologic Q waves indicative of?
an old myocardial infarction