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115 Cards in this Set
- Front
- Back
what are the BP parameters for HTN
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>/= 140/90
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what are the BP parameters for PRE-hypertension
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systolic=120-139
diastolic=80-89 |
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what amt of adults in the US have HTN
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~ 25%
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the incidence of HTN increases with what
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AGE
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the incidence of HTN is higher in what population
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AFRICAN AMERICANS
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HTN is a risk factor for what other dz processes
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*CAD
*CHF *CVA *ESRD *arterial aneurysm |
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what population has the HIGHEST incidence of HTN
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black males
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what are the contributing factors to ESSENTIAL hypertension
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*increased SNS activity in response to stress
*overproduction of vasoconstictors or Na retaining hormones *high Na intake *LOW K and Ca intake *increased renin secretion *decreased endogenous vasodilators *concurrent dz (DM, obesity) |
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what are some endogenous vasodilators
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*NO
*prostaglandins |
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essential HTN accounts for what percent of all HTN
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95%
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if a pt is obese and NORMOtensive what is the result on the heart
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dilatation
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if a pt is lean and HYPERtensive what is the result on the heart
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hypertrophy
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if a pt is obese and HYPERtensive what is the result on the heart
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dilatation and hypertrophy leading to CHF
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what is the optimal range for BP
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<120 systolic
<80 diastolic |
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what is the normal range for BP
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<130 systolic
<85 diastolic |
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what is the high normal range for BP
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130-139 systolic
85-89 diastolic |
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what is the BP for stage I HTN
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140-159 systolic
or 90-99 diastolic |
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what is the BP for stage II HTN
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160-179 systolic OR
100-109 diastolic |
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what is the BP for stage III HTN
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>/= 180 systolic OR
>/= 110 diastolic |
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regarding tx goals for a normal pt what range would you want to get BP into
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<140/90
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regarding tx goals with co-existing dz what range would you want to get BP into
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<130/80
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what is the reasonable FIRST approach for tx of essential HTN in pts WITHOUT associated risk factors or evidence of end organ damage
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lifestyle modification
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what are lifestyle modifications that can be done in the treatment of essential HTN
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*wt loss
*increased physical activity *smoking cessation *decrease ETOH intake *adequate intake of Ca and K *dietary salt restriction |
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what is the BIGGEST factor for decreasing BP from lifestyle modifications in essential HTN
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weight loss
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with treatment of essential HTN and post-MI what classes of drugs are recommended
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*ACE inhibitor
*aldosterone antagonist *beta blocker |
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with tx of essential HTN and heart failure what are the recommended drug classes
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*ACE inhibitor
*aldosterone antagonist *ARB *beta blocker diuretic |
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with tx of essential HTN and high risk for CAD what are the recommended drug classes
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*ACE inhibitor
*beta blocker *Ca channel blocker *diuretic |
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with tx of essential HTN and DM what are the recommended drug classes
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*ACE inhibitors
*ARB *beta blockers *Ca channel blocker *diuretic |
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with tx of essential HTN and renal insufficiency what are the recommended drug classes
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*ACE inhibitor
*ARB |
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what tx of essential HTN and previous CVA what are the recommended drug classes
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*ACE inhibitor
*diuretic |
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what type of HTN has a KNOWN cause
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secondary HTN
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what percentage of HTN does secondary HTN account for
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<5%
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what type of HTN is often amenable to sx therapy
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secondary HTN
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what are the common etiologies for secondary HTN
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*renovascular dz
*hyperaldosteronism *aortic coarctation *pheochromocytoma *cushings syndrome *renalparenchymal dz *PIH |
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what is the MOST common cause of secondary HTN
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renovascular dz
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what is the BP in a hypertensive crisis
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>180/120
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with a hypertensive crisis higher BP are tolerated by what type of pt
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pts with chronic HTN
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what is HTN urgency
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elevated BP WITHOUT evidence of target organ damage
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what is HTN emergency
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evidence of target organ damage
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what is the tx goal in a hypertensive crisis
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*gradual reduction of BP
-20% reduction in the 1st hr -reduce to 160/110 range over next 2-6 hrs |
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regarding surgical and anesthetic risk HTN is associated with an increased risk of what
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silent ishcemia and infarction
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incidence of magnitude of what perioperative events is increased with pts with HTN
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hypertensive and hypotensive events
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there is little evidence that BP in what range increase complication rate in elective sx
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<180/110 but greater than normal
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what are concerns with the hypertensive pt with induction
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hypertension and hypotension
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what are the maintenance goals of anesthesia with a hypertensive pt
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minimize wide swings in BP that are common to pts with HTN
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what needs to be done with central neuraxis anesthesia and a hypertensive pt
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special attention needs to be payed to fluid status d/t associated sympathetic block and decreased BP
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what can be used to control HTN intra-op with HTN pt
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*VA
*anti-hypertensives |
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what can be used to control hypotension intra-op with a HTN pt
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*adjust anesthetic depth
*fluid resuscitation *sympathomimetics -ephedrine -phenylephrine |
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what drugs that a HTN pt may be on prior to sx may not respond well to ephedrine or phenylephrine
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*ACE inhibitors
*ARB |
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what are some of the possible etiologies for post-op HTN
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*pain, agitation
*shivering *bladder or bowel distention *emergence excitement *pre-existing HTN *hypercapnia *increased ICP *autonomic dysreflexia |
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what should be anticipated in a pt who experienced an episode of intra-op HTN
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post-op HTN
(appropriate orders for antihypertensives should be written) |
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what is the normal SYSTOLIC pulmonary artery pressure
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18-25 mmHg
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what is the normal DIASTOLIC pulmonary artery pressure
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6-10 mmHg
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what is the normal MEAN pulmonary artery pressure
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12-16 mmHg
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what is the definition of pulmonary arterial HTN
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MEAN PA pressure > 25 ar rest OR
MEAN PA pressure > 30 with exercise |
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how common is primary or idiopathic pulmonary HTN
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RARE
(1-2 / 1,000,000) |
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what is the median survival following diagnosis of primary pulmonary HTN
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2.8 yrs
|
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who is primary pulmonary HTN predominantly seen in
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young women
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primary HTN occurs in the ABSENCE of what
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*L sided heart or valvular dz
*congenital heart dz *myocardial dz *resp dz *connective tissue disorder *thromboembolic dz |
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responsiveness of pulmonary HTN to vasodilators is determined how
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by admin of
*prostacyclin *NO *adenosine *prostaglandin E1 |
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what is defined as "responsiveness" to a vasodilator in pulmonary HTN
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a decrease in PVR and a mean PA pressure of >/= 20%
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what amt of pts with primary pulmonary HTN will have a positive response to vasodilators
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~25%
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what are the risks of hypoxemia in pulmonary HTN
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*R to L shunting thru a patent foramen ovale
*exertion resulting in increased o2 extraction in light of a relatively fixed CO *perfusion of poorly ventilated alveoli d/t VQ mismatch |
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what are the various tx for pulmonary HTN
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*o2 *diuretics
*anticoagulation *Ca channel blockers *phosphodiesterase inhibitors *inhaled NO *prostacyclins *endothelin receptor anatgonist *sx |
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with sedation of pt with pulmonary HTN what do you want to do
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*be judicious to avoid hypoxia and hypercapnia
*supply supplemental o2 |
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what agents are good for the induction of a pt with pulmonary HTN
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thiopental and propofol
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what agents for induction of a pt with pulmonary HTN may inhibit pulmonary vasodilation
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*ketamine
*etomidate |
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what are the pros with VA in the maintanence of a pulmonary HTN pt
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they are potent bronchodilators
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what are the cons with VA in the maintanence of a pt with pulmonary HTN
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they decrease systemic vascular resistance with R to L shunt and increase shunt
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what drugs do you want to avoid in the maintanence of anesthesia with a pulmonary HTN pt
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drugs which produce histamine release
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what does positive pressure ventilation do with pulmonary HTN pts
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improves oxygenation
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ventilation settings with a pulmonary HTN pt must be appropriate to avoid what things
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*hypoxia
*hypercapnia *acidosis *decrease in venous return |
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what monitors are appropriate in sx's with a pulmonary HTN pt
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invasive monitors are appropriate in all but minor sx
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what is the post-op care for a pulmonary HTN pt
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*adequate pain control
*continued intensive monitoring |
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with labor and delivery and pulmonary HTN what type of anesthetic management would you choose
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epidural analgesia with dilute local anesthetic and narcotic
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with a c-section and pulmonary HTN what anesthetic tech would you choose
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epidural is the most likely choice in the non-emergency setting
(general if emergency) |
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what type of anesthesia should you AVOID with a c-section and pulmonary HTN
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spinal anesthesia d/t sudden decrease in SVR
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what type of monitoring would you do for a c-section with pulmonary HTN
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*a-line
*pulmonary arterial catheter |
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what is the maternal mortality with pulmonary HTN
why? |
~50%
-primarily d/t CHF in labor and the early postpartum period |
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what is acute benign pericarditis
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*viral etiology
*does NOT involve a sig effusion or tamponade *rarely progressive |
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what type of presentation does pericarditis have following an MI
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acute presentation OR
delayed presentation (dresselers syndrome) |
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acute presentation of pericarditis following an MI is seen when
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1-3 days following transmural MI
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delayed presentation of pericarditis following an MI is seen when
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weeks to months after an MI
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what is the dx criteria for acute pericarditis
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*chest pain
*friction rub *EKG changes |
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what is the tx for acute pericarditis
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*salicylates
*NSAIDs *corticosteroids |
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when does a pericardial effusion result in tamponade
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when the pressure in the pericardial space interferes with cardiac filling
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normally the pericardial space holds how much fluid
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15-50 ml
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symptoms of pericardial effusion and tamponade are related to what
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compression of adjacent structures
OR related to pericardial restraint |
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what are the symptoms of pericardial effusion and tamponade r/t compression of adjacent structures
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*dyspnea
*cough-hoarsness *anorexia-dysphagia *chest pain |
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what are the symptoms of pericardial effusion and tamponade r/t pericardial restraint
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*tachycardia
*JVD *hepatmegaly *peripheral edema |
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what is the mechanism for a cardiac tamponade
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diastolic collapse d/t pressure imposed by the pericardium impairs diastolic filling of the chambers thereby reducing SV and CO
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what are the compensatory mech with cardiac tamponade
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*tachycardia
*peripheral vasoconstriction |
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what are the physical findings with cardiac tamponade
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*kussmauls sign
*pulsus paradoxus *becks triad |
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what is becks triad
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1-quiet heart sounds
2-increased JVD 3-hypotension |
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how is cardiac tamponade dx
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*echo**
*CT/MRI *EKG (not a really good diagnostic tool) |
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what is the tx for cardiac tamponade
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*removal of fluid
*temporizing measures |
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how is fluid removed in cardiac tamponade
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*pericardiocentesis
*pericardiostomy (pericardial window) |
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what are temporizing measures for cardiac tamponade
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*intravascular volume expansion
*increase contractility *correction of any metabolic acidosis |
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what regarding anesthetic management in the presence of hemodynamically significant cardiac tamponade can result in life-threatening hypotension
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general anesthesia and positive-pressure ventilation
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in the anesthetic management of pts with cardiac tamponade what things do you want to maintain
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*CO
*contractility *adequate preload *SR |
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in the anesthetic management of pts with cardiac tamponade what things do you want to AVOID
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*decreases in systemic vascular resistance
*decreases in venous return *bradycardia |
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what types of monitors should be used in the anesthetic management of pts with cardiac tamponade
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*arterial line
*central venous pressure |
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what type of induction and maintenance drugs should be used in pts with cardiac tamponade
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*ketamine (good choice)
*bzd? *MR? |
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what is constrictive pericarditis
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pericardium becomes a rigid shell d/t adhesions &/or scarring
(over time may become calcified) |
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what is diastolic filling in constricitive pericarditis
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IMPAIRED like with cardiac tamponade
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what is the treatment for constrictive pericarditis
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stripping of the pericardium
(which may be closely adherent to the myocardium) |
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what may occur during the tx for constrictive pericarditis
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*may involve sig blood loss
*may require cardiopulmonary bypass |
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what are the goals of anesthetic management of constrictive pericarditis
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maintenance of:
*HR *contractility *SVR *venous return |
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anesthetic drug selection with constrictive pericarditis will depend on what
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severity of hemodynamic compromise
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with severe hemodynamic constrictive pericarditis anesthetic management should be treated similar to what
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cardiac tamponade
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when is pericardial laceration common
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in rapid deceleration injuries to the chest wall
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if a pericardial laceration is associated with laceration of adjacent structures it may result in what
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cardiac herniation
|
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cardiac herniation would be managed similar to what condition
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cardiac tamponade
|
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when should you have a high suspicion for cardiac contusion
|
with rapid deceleration injuries
|
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what should you be alert for in cardiac contusion
|
*ST-T abnormalities
*dysrhythmias |