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46 Cards in this Set

  • Front
  • Back

AV valves

tricuspid- Right


mitral - Left

Semilunar valves

Pulmonic - right


Aortic - left



What happens in S1(systole)



Semilunar valves ope


aortic


pulmonic




AV valves close


tricuspid (right)


mitral (left)

What happens in S2

Diastole




AV valves close


mitral (left)


tricuspid (right)




semilunar valves open


aortic (left)


pulmonic (right)

Systole

between S1 and S2



Diastole

between S2 and S1

S3

fluid overload


pregnancy


chf




Kentucky (S3=Y)



S4

S4




Stiff ventricular wall


Mi


LV hypertrophy


chronic HTN




Tennessee (S4=ne)

IV/VI murmur

thrill




I/VI - very soft


VI/VI - very loud, across the room

Murmur location: Left 5th ICS, MCL

Mitral




Middle of the chest




also known as the apex or base

2nd Right ICS

AORTIC

2nd Left ICS

Pulmonic

Systolic murmurs

Mitral


Regurg


Aortic
Stenosis
Systolic




+MVP (mitral valve prolapse)

Diastolic murmurs

Mitral
Stenoisi
Aortic
Regurg
Diastolic

Systolic heart failure

inability of the heart to contract




decreased cardiac output



diastolic heart failure



inability of the heart to relax




decreased cardiac output

Acute HF

Left heart


lungs


S3 (fluid overload




Abrupt onset


often after acute MI or valve rupture

Chronic HF

Right failure


most common cause of R failure = Left failure




a result of inadequate compensatory mechanisms over time

NYHA class I

no limitations, no ss

NYHA class II

symptomatic, but only slight




with physical activity

NYHA Class III

symptomatic - Marked




with physical activity




comfortable at rest

NYHA Class IV

Symptomatic at rest




severe symptoms

SS acute failure (left)

lungs




dyspnea at rest


rales


wheezing


S3 gallop (fluid overload)


murmur: mitral regurg (lt 5th ICS, MCL - systolic)

SS chronic failure (right)

swollen feet




JVD


Hepatomegaly


dependent edema (increased cap hydrostatic pressure)


PND


displaced PMI


fatigue


S3 +/- S4

CXR findings CHF

pulm edema


kerley B lines


effusions

pharm management of CHF

ACE


Diuretics (thiazide, loop)


Anticoag for A fib

non pharm mgmt CHF

Na+ restriction


rest/activity balance


wt reduction

HTN secondary

5% all cases




Causes:


estrogen use


renal disease


pregnancy


endocrine


Renal artery stenosis

HTN exacerbating factors

smoking


obesity


ETOH


NSAIDs

Severe HTN headache

suboccipital


pulsating


morning




resolves throughout the day

Heart sound with HTN

S4 (stiff heart)

threshold for => 60

150/90

Threshold for <60

140/90



Threshold for all adults (regardless of age) with:


CKD, DM

140/90

Non-AA HTN treatment

Thiazide


CCB


ACE/ARB

AA HTN treatment

Thiazide


CCB

CKD HTN treatment

ACE/ARB

reassess HTN Tx after how long

1 month




increase dose or add med


reassess monthly until at goal



When do you refer to HTN specialist?

if 3 or more drugs are needed

HTN thraputic lifestyle changes(TLCs)

restrict dietary sodium


wt loss


DASH diet


Exercise: 40 min/day most days

DASH diet

rich in:




fruits


veg


low fat dairy




reduced


saturated fat


total fat

Hypertensive urgency

180/110




without progressive target organ dysfunction



HTN urgency SS

May or may not be associated with:




severe HA


SOB


epistaxis


severe anxiety

HTN urgency mgmt

oral therapies:




lonidine


captopril


nifedipine



HTN emergency

180/130




or lowerwith evidence of impending or progressive target organ dysfunction

progressive target organ dysfunction

fudoscopic changes:


flame-shaped retinal hemorrhages


soft exudates


papilledema (swelling of disc)


hypertensive encephalopathy


intracranial hemorrhage


unstable angina


acute MI


acute LV failure with pulm edema


disecting AAA


eclampsia