All Lesions To Visual Pathways Ssom

Front 1

Left Optic Nerve

Back 1

Left Eye Monocular Anopia or
Left Monocular Vision Loss

Front 2

Optic Chiasm

Back 2

Heteronymous bitemporal hemianopia

Front 3

Right Optic Tract

Back 3

Left homonymous hemianopia

Front 4

Left Optic Tract

Back 4

Right homonymous hemianopia

Front 5

Right Meyer's Loop

Back 5

Left superior homonymous quadrantanopia
(pie in the sky disorder)

Front 6

Left Meyer's Loop

Back 6

Right superior homonymous quadrantanopia
(pie in the sky disorder)

Front 7

Right Parietal Loop

Back 7

Left inferior homonymous quadrantanopia
(pie in the floor disorder)

Front 8

Left Parietal Loop

Back 8

Right inferior homonymous quadrantanopia
(pie in the floor disorder)

Front 9

Right Optic Radiation

Back 9

Left homonymous hemianopia

Front 10

Left Optic radiation

Back 10

Right homonymous hemianopia

Front 11

Right lingual gyrus
(inferior banks of calcarine fissure of V1)
sparing macula region

Back 11

Left superior homonymous quadrantanopia with macular sparing

Front 12

Left lingual gyrus
(inferior banks of calcarine fissure of V1)
sparing macula region

Back 12

Right superior homonymous quadrantanopia with macular sparing

Front 13

Right cuneus gyrus
(superior banks of calcarine fissure)
sparing the macula region

Back 13

Left inferior homonymous quadrantanopia with macular sparing

Front 14

Left cuneus gyrus
(superior banks of calcarine fissure)
sparing the macula region

Back 14

Right inferior homonymous quadrantanopia with macular sparing

Front 15

Right V1
sparing macula region

Back 15

Left homonymous hemianopia with macular sparing

Front 16

Left V1
sparing macula region

Back 16

Right homonymous hemianopia with macular sparing

Front 17

Right Optic Nerve

Back 17

Right eye monocular anopia or
Right monocular visual loss

Front 18

pituitary adenomas

Back 18

lesions often compress the optic chiasm disrupting inferior nasal crossing fibers. Damage equals bitemporal superior quadrantanopsia

Front 19

injuries to the optic chiasm, lateral geniculate bodies, or occipital cortex cause what type of visual field loss

Back 19

contralateral quadrantanopsia or hemianopsia

Front 20

what is rare but might be the only physical manifestation of a contralateral temporal lobe lesion (i.e., residual temporal lobectomy)

Back 20

homonymous superior quadrantanopia (i.e., pie in the sky"

Front 21

when does macular sparing happen?

Back 21

in the occipital lobe (i.e., back part of optic radiations)

Front 22

eye deviation from cerebral lesions

Back 22

partial seizures push the eyes contralaterally; stroke pushes the eyes to look toward the lesion.

Front 23

conjugate eye movement

Back 23

supranuclear gaze centersinnervate pontine (nuclear) gaze centers, which innervated nearby oculomotor (CN3), trochlear (CN4), and abducens (CN6)

Front 24

progressive supra-nuclear palsy

Back 24

damage of the cortical bulbar tracks abolishing voluntary conjugate gaze. PSP patient's cannot voluntarily look upward or downward, and then later to either side.

seen in Parkinsonian and Dementia,

Front 25

what does the pontine gaze center due to?

Back 25

a pontine center pulls the eyes towards its own side. So a stroke on one side of the pons allows the eyes to be pulled to the opposite side. Ex: Damage to right pontine gaze Center the eyes would deviate to the left. Left-sided hemiparesis would also be evident. With brainstem lesions the eyes look towards the paralysis.

Front 26

what is medial longitudinal fasciculus syndrome (AKA intranuclear opthalmoplegia).

Back 26

spares the cranial nuclear eye and nerves and causes a classic pattern of ocular movement impairment identified by inability of the eye ipsilateral to the lesion to adduct past midline.

often seen in MS and brainstem strokes

Front 27

nystagmus

Back 27

rhythmic horizontal, vertical, or rotary oscillations of both eyes. While often seen in CNS injuries it may be the most prominent physical finding from alcohol or drug intoxication including Wernicke's-Korsakoff syndrome.

Front 28

oculomotor (CN3) nerve palsy

Back 28

Causes diplopia.

often comes with ptosis, lateral deviation of the eye, and dilated/blown pupil. Often most pronounced when the patient attempts to abduct the eye

Front 29

abducens (CN6) Nerve injury

Back 29

causes diplopia and inability of abduction

Front 30

what medications can cause transient visual difficulties

Back 30

tricyclic antidepressants due to anticholinergic side effects (i.e., amitriptyline)

Front 31

Optic Neuritius

Back 31

The loss of vision, accompanied by an impaired direct light reflex, but preserved consensual response, indicates that the problem is in the optic nerve. This cranial nerve forms the afferent limb of the light reflex. The pain indicates nerve inflammation, i.e., neuritis. In optic neuritis, the “patient sees nothing and the ophthalmologist sees nothing.” Optic neuritis as an isolated condition or a manifestation of multiple sclerosis (MS) occurs relatively commonly among young adults.