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33 Cards in this Set
- Front
- Back
Cell death caused by a blockage of the oxygen supply to the injured area or caused by enzymatic damage and mitochondrial failure
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Secondary damage
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natural response to an injury through which dead or lost tissue is replaced by living tissue.
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Tissue Healing
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Most abundant type of tissue in the body.
Produced by fibroblasts |
Connective tissue
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The predominant type of connective tissue
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Collagen
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Secretes extracellular matrix components
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Fibroblasts
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Produces extracellular matrix components in cartilage
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Chondrocytes
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Produces extracellular matrix components with contractile properties
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Myofibroblasts
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Stores lipids
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Adipocytes
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Responsible for transferring physical loads of force
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Deep fascia
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An excessive accumulation of serous fluids.
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Edema
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Delicate tissue composed of fibroblasts, collagen, and capillaries formed during the revascularization phase of wound healing
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Granulation tissue
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Bleeding, from veins, arteries, or capillaries
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Hemorrhage
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A blood-thinning chemical released from damaged tissue during the inflammatory process. Its primary function is vasodilation of arterioles and increased vascular permeability in venules
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Histamine
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Local and temporary deficiency of blood supply caused by obstruction of circulation to a part.
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Ischemia
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Chemicals that act through indirect means
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Mediators
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A classification of scavenger cells that ingest and destroy unwanted substances in the body.
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Phagocyte
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A protein based connective tissue
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Collagen
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A cell concerned with the formation of new bone
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Osteoblast
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A cell that absorbs and removes unwanted bone
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Osteoclast
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A nerve that provides impulses to muscles
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Motor nerve
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A group of skeletal muscle fibers that are innervated by a single motor nerve
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Motor Unit
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Carrying impulses toward a central structure,the brain.
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Afferent
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Carrying impulses away from a central stucture. Nerves leaving the CNS.
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Efferent
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Dominate the first 6 to 24 hours of phagocytosis, releasing chemical "cluster bombs" that are intended to destroy bacteria but end up traumatizing all tissues in the area
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Neutrophils
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-Mechanism designed to rid the body of damaged tissue
-It is an immediate response to acute injuries -It is non-specific to site or stimulus -Essential for tissue repair |
What is the inflammatory response?
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fibroblastic-repair, fibroplasia (scar formation))
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Proliferation
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(maturation-repair, remodeling (scar maturation))
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Maturation
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-Short onset and duration
-Hemodynamic changes -Production of exudate -Presence of granular leukocytes -More extensive formation of scar tissue |
Acute inflammation
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-Occurs when the acute inflammatory response does not eliminate the injurious agent
-Longer onset & duration -Displays the replacement of leukocytes with macrophages, lymphocytes, & plasma cells |
Chronic Inflammation
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-Trauma followed by transient vasoconstriction to control hemorrhaging
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First process of acute inflammation
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-Vasodilatation and increased blood flow
-Coinciding with these vascular changes in an increase in vascular permeability |
Second stage of acute inflammation
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- Gaps then occur in the cell walls increase the intravascular hydrostatic pressure (IHP) which causes fluid to filtrate out into the interstitial space
-Fluid accumulates, causing edema (clinical manifestation of increase in vascular permeability) |
Third stage of acute inflammation
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-Trauma followed by vasoconstriction of capillaries, venules, and arterioles and = decrease in blood flow (transient)
-Occurs as norepinephrine is released into the tissue to prevent blood loss to the affected area |
Stage 1 Inflammation
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