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77 Cards in this Set

  • Front
  • Back

leukoplakia


-most common oral precancer

leukoplakia

leukoplakia


*lip vermilion, buccal mucosa, gingiva

early/thin leukoplakia

homogeneous/thick leukoplakia



granular leukoplakia


(invasive SCC)

verruciform leukoplakia


(well differentiated SCC)

benign alveolar ridge keratosis



smokeless tobacco keratosis

smokeless tobacco keratosis


*SCC is most common malignancy resulting from this


*also verrucous carcinoma (she says)

severe smokeless tobacco keratosis


-first you'll get perio disease


-then you'll get caries


-then tissue will dry out (tell them to change spot)

mild smokeless tobacco keratosis

nicotine stomatitis


-not premalignant


-develops in response to heat


-palatal mucosa


-men 45+



nicotine stomatitis


-reversible (these are salivary ducts)

leukoedema


-unknown cause


-darkly pigmented patients (AA)


-diffuse, gray-white, milky, opalescent appearance of the mucosa


-folded wrinkles

leukoedema

-bilateral buccal mucosa extending forward


-stretch cheek, lines go away


-epithelial cells have a lot of fluid in them



leukoedema


-very common/variation of normal

linea alba


-2nd most common oralmucosal pathosis


-may be scalloped


-usually bilateral

Morsicatio Buccarum


(chronic cheek chewing)


-Thickened,shredded, white areas combined with intervening zones of erythema, erosion orfocal traumatic ulceration

Morsicatio Linguarum


(tongue chewing)

aspirin burn


-very thin white sloughing

aspirin burn

hydrogen peroxide burn


-skin will peel off


-long-term use

silver nitrate burn


-usually you use to stop bleeding


-lightly tap where it's bleeding or else you will burn off normal tissue

silver nitrate burn

formocresol burn


-in peds they may use this to burn off pulp in kids to do partial pulpotomy

exfoliative cheilitis


-younger patients


-constantly licking lips


-scaling/flaking of lips

actinic cheilosis


-premalignant alteration of lower lip (SCC)


-UV exposure



lichen planus (wickham's striae)


-immunologically mediated


-Hep C associated


-associated skin lesions (purple, pruritic papules)

reticular LP


-no symptoms


-Oral mucosal surfaces –lateral and dorsal tongue, gingiva, palate and vermillion border


-White lesions appear aspapules



reticular LP


-ask if patient has any other itching lesions


-vulvar mucosa, glans penis, nails

lichenoid contact stomatitis


-amalgam hypersensitivity



lichenoid contact stomatitis

drug reaction to tetracycline


(lichenoid drug reaction)

drug reaction to tetracycline


-focal


-white in lips

erythema migrans


geographic tongue


benign migratory glossitis


-completely benign


-immune reaction


-looks like psoriasis


-may have burning tongue

erythema migrans


geographic tongue


-can come and go


-move from place to place

geographic tongue

geographic tongue


-anterior 2/3 dorsal tongue


-well demarcated zones



squamous papilloma


-benign proliferation of stratified squamous epithelium


-induced by hpv (6,11)





squamous papilloma


-soft palate, tongue, lips


-painless, pedunculated, cauliflower


-low risk hpv


-not an STD

squamous papilloma


-IT WILL RECUR



verruca vulgaris


-this is a skin wart; not common in mouth



verruca vulgaris


-Whenoral mucosa is involved sites include vermillion border,labial mucosa and anterior tongue


-contagious

condyloma acuminatum


(venereal wart)


-this is an STD


-high risk HPV


-Oral lesions occurmost frequently on labial mucosa, soft palate and lingual frenum

condyloma acuminatum


(venereal wart)


-monitor patient for cervical or oropharyngeal cancer

pseudomembranous (thrush) candidiasis


-buccal mucosa, palate, dorsum of tongue


-cottage cheese


-unpleasant taste


-painful


-as result of long-term antibiotics



hyperplastic candidiasis


-hyperkeratotic lesion


-anteriorbuccal mucosa

hyperplastic candidiasis

hairy leukoplakia


-due to EBV


-lateral tongue


-white streaks, corrugated surface


-immunosuppressed host


-swollen lymph nodes

hairy tongue


-Elongated,hyperkeratotic filiform papillae on tongue dorsum producing a “furred” to“hairy” texture

oral submucous fibrosis


-Chronic,scarring high-risk precancerous condition of oral mucosa


-Reduction of tobacco does not reduce likelihood ofdeveloping oral submucousfibrosis


-linked to betel nut


-mucosal rigidity!

oral submucous fibrosis

white sponge nevus


-genetic (defect in keratinization)


-Symmetrical, thickened,white, corrugated or velvety, diffuse plaques affect buccal mucosa bilaterally


-looks like cheek biting (but no habit)


-ask if family members have it

proliferative verrucous leukoplakia


-high risk form of leukoplakia


-rarely regress


-females


-leathery


-most of time just genetic



PVL

verrucous carcinoma


(snuff dippers cancer)


-low grade SCC


-doesn't mets


-better to get verrucous than scc

verrucous carcinoma


(snuff dippers cancer)


-may develop from PVL

fordyce's granules


(ectopic sebaceous glands in the oral mucosa and vermilion of lip)



fordyce's granules

lymphoid tissue


-yellow balloon-like


-lymphoid chains are around tonsils and base of tongue

ectopic lymphoid tissue

lymphoepithelial cyst


-FOM, ventral/lateral tongue


-normal/accessory sites


-small yellow/white swellings

parulis/gum boil

lipoma


-buccal mucosa and vestibule


-rare recurrence



(acute) pseudomembranous candidiasis


-when you scrape off, leave red bleeding mucosa

chronic candidiasis (erythematous)

chronic candidiasis (erythematous)

smuff dipper's pouch

idiopathic leukoplakia


(don't know what causes it-no habit)


-could be nutrition, smoking, alcohol, unknown

-lichen planus (ask about any other skin lesions)


-lichenoid drug reaction (investigate med history)

reticular LP



cutaneous LP



erosive LP



atrophic LP

frictional hyperkeratosis



-tongue chewing


-hairy leukoplakia