Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
93 Cards in this Set
- Front
- Back
What is spina bifida?
|
Posterior end of neural tube does not close properly.
|
|
What is the function of the temporal lobe?
|
Auditory, speech, memory. Contains hippocampus.
|
|
Funcion of Occipital lobe?
|
Visual processing.
|
|
How many pairs of spinal nerves are there? and whats the breakdown?
|
31 pairs. C8, T12, L5, S5, C1
|
|
Where do retinal fibers go after the optic chiasm?
|
Synapse at LGN. Then to Visual Cortex.
|
|
What is Meyers loop?
|
After leaving LGN, inferior retinal fibers take this indirect path through the temporal lobe to the cortex.
|
|
2 other names for visual cortex:
|
Area 17, Striate Cortex
|
|
Where is lesion that causes a "pie in the sky" VF defect?
|
Temporal lobe. Affects inferior retinal fibers of Meyers loop.
|
|
Where is the lesion that causes a "pie on the floor" VF defect?
|
Parietal lobe.
|
|
Parietal lobe lesions are associated with what other ocular finding?
|
Asymmetric OKN responses.
|
|
Where does the macular region of the occipital lobe recieve blood supply?
|
2 sources: 1) Middle cerebral artery 2) Posterior cerebral artery.
Thus, VAs are only affected in a lesion if both sources are lost. (rare) |
|
What is the main cause of macular sparing homonymous hemianopia? Macular involving?
|
Sparing = Stroke. Involving = tumor...compress both blood supplies.
|
|
Where is the lesion that produces a junctional defect?
|
At junction of optic nerve and optic chiasm. (Anterior Knee of Wilbrand)
|
|
Where is lesion if VF defect respects horizontal midline?
|
Anterior to chiasm. (optic nerve)
|
|
Where is lesion if VF defect respects vertical midline?
|
Posterior to chiasm.
|
|
Most common cause of post chiasmal lesion?
|
Stroke
|
|
Which 2 sets of arteries supply the brain?
|
Internal carotids and vertebrals
|
|
Whats in the circle of Willis?
|
Basilar Artery, internal carotids, Ant. and Post. Communicating Arteries
|
|
What is the most common VF defect seen with early glaucoma?
|
Nasal steps
|
|
Port-Wine Stains are associated with what OCULAR disease?
|
Ipsilateral glaucoma (45%) (Only 5% of those have Sturge Weber Syndrome.)
|
|
Two types of secondary open angle glaucoma:
|
Psuedoexfoliation Syndrome, Pigmentary Dispersion Syndrome
|
|
2 causes of Angle Closure Glaucoma:
|
#1 = Pupillary block. #2 = Plauteau iris Syndrome
|
|
2 sources of secondary Acute Closure Glaucoma
|
Neovascular and Uveitic sources.
|
|
Most common causes of Neovascular Glaucoma:
|
#1 = CRVO, #2 = DM, others - CRAO, Carotid Dz. (anything w/ severe retinal ischemia)
|
|
How does uveitis cause glaucoma?
|
Iris gets inflamed and sticky....leads to Anterior and Posterior Synechia...leads to secondary angle closure.
|
|
2 most common inflammatory glaucomas:
|
Glaucomaticocyclitic Crisis (Posner-Schlossman Syndrome), Fuch's Heterochromic Iridocyclitis
|
|
What causes glaucomatocyclitic crisis? Uni or bilateral?
|
Acute trabeculitis, unilateral
|
|
How do pupil size and depth of focus relate?
|
Smaller pupils = increased depth of focus. (inversely)
|
|
How do pupil size and spherical aberrations relate?
|
Decreased pupil size = decreased spherical and chromatic aberrations. Spherical aberrations scale with the Square of diameter of entrance pupil.
|
|
What part of what bone houses the pituitary gland?
|
Sella turcica of sphenoid bone.
|
|
The optic chiams lies _______to the pituitary gland.
|
Superior
|
|
Where do 4 of the EOMs originate? Which ones don't?
|
Common Tendinous Ring. SO and IO.
|
|
Where does Inf. Oblique originate?
|
Maxillary bone.
|
|
Most common benign orbital tumor in adults? in children?
|
Adults - Cavernous hemangioma. Kids - Capillary hemangioma.
|
|
3 things that pass through Sup. Orbital fissure and Common Tend. Ring:
|
NOA
Nasociliary N. Oculomotor N. Abducens N. |
|
Which side of orbit is thinnest? Smallest? Weakest? Strongest?
|
Thinnest = medial
Smallest - medial Weakest - Floor Strongest - Lateral |
|
Why is there vision loss with temporal (Giant cell) Arteritis?
|
Short Post. Cil. Arteries that supply the Optic disc are damaged.
|
|
Which CN is most likely affected by an internal carotid artery aneurysm in the cav. sinus?
|
6 - Abducens
|
|
What provides blood to the optic disc?
|
Circle of Zinn. (Zinn Haller) Its a network of SPCAs.
|
|
What causes OIS?
|
Carotid or Ophthalmic Artery blockage. DM, HTN, Cardiac dz. (d/t atherosclerosis)
|
|
Vortex veins drain the...
|
choroid
|
|
CRV drains the
|
retina (inner 6 layers)
|
|
Flow of venous blood out pathway:
|
CRV + Anterior Ciliary V + Vortex V. => Sup. and Inf. Ophthalmic V. => Cav. Sinus =>Petrosal Sinus => Int. Jugular V.
|
|
2 main threats to vision with CRVOs and BRVOs:
|
Macular Edema, Neovasc. (CRVOs lead to Neo. Glaucoma)
|
|
What goes through Cav. Sinus?
|
CNs 3, 4, 5(1), 5(2), 6, Int. carotid, sympathetic plexus (postgang. fibers) CN 6 runs freely, the rest in the lateral wall.
|
|
Infections in the ______can invade the cav. sinus.
|
cheek
|
|
4 destination for fibers after chiasm crossing:
|
1) LGN => Primary Visual Cortex
2) Pretectal Nucleus for pupil innervation 3) Superior Colliculus for saccades 4) Hypothalamus for circadian rhythm |
|
Edinger Westphal nucleus?
|
Parasymp. innervation to ciliary and sphincter muscles. CN 3.
|
|
What is unique about levator muscle nuclei?
|
1 subnucleus controls both. (See bilateral ptosis, keep in mind possible nuclear lesion)
|
|
How do you examine for superior oblique palsy?
|
Have pt. adduct and look down. Pt. will try to tilt head away from affected side.
|
|
Which structure do sensory nerves from eye synapse in?
|
Trigeminal ganglia. (CN 5) (Final destination is thalamus)
|
|
Pathway of sensory nerves from cornea, iris, and CB
|
long ciliary nerves => Nasociliary N. => Ophthalmic N. => Trigeminal N.
|
|
What goes through Sup. Orbital fissure?
|
CN, 3, 4, 5(1), 6, Ophthalmic Veins
|
|
Which CN is most susceptible to increased intracranial pressure?
|
6 (lateral rectus palsy)
|
|
Which CN sends parasympathetic info to lacrimal gland?
|
7 (with help from 5(2)
|
|
Two main causes of pupil-involving CN 3 lesions?
|
1) Aneurysm (main artery is post. communicating) 2) tumor (anything that compresses nerve!)
|
|
2 main causes of pupil sparing CN 3 lesions?
|
DM and HTN. Affect small vessels that are nourishing inner nerve.
|
|
Which ganglion sends parasymp info to lacrimal gland?
|
Pterygopalatine
|
|
Sympathetic Pathway to eye
|
Hypothalamus => Ciliospinal Center of Budge => above lung => Superior Cervical Ganglion
|
|
Function of orbital septum?
|
Protect from infection.
|
|
Function of tarsal plate?
|
Provide rigidity (encloses Mei. glands too)
|
|
What type of glands are Meibomian glands?
|
Sebacious. (oil)
|
|
The lacrimal sac is __________ to the orbital septum.
|
Anterior
|
|
General symptoms seen in orbital cellulitis not seen in preseptal cellulitis pts.
|
Fever and malaise
|
|
What acts as the fulcrum of the levator?
|
Whitenalls Ligament
|
|
Which contributes more to holding the eye open? Levator or Muellers?
|
Levator
|
|
Postgang. sympathetic fibers enter the skull through the...
|
carotid canal. (Surround internal carotid)
|
|
Which structure causes the eyelid furrows?
|
Levator aponeurosis.
|
|
Where and what are glands of zeis?
|
Sebacious glands at eyelash follicles. Lube eyelid so it doesn't become brittle.
|
|
Where and what are glands of moll?
|
Modified sweat glands near lid margin. Apocrine.
|
|
What type of glands are Krause?
|
Accessory lacrimal. Merocrine
|
|
What is a chalazion?
|
Painless, sterile inflammation of Meibomian gland. Often caused by obstructed duct.
|
|
What is an external hordeolum?
|
Staph. infection of Zeis of Moll.
|
|
What is an internal hordeolum?
|
Staph. infection of of Meibomian gland.
|
|
What does frontalis muscle do?
|
Raises eyebrows - surprise expression.
|
|
What is function of corrugator muscle?
|
Moves eyebrows medially - concentration.
|
|
Function of procerus muscle?
|
Moves medial portion of eyebrown inferiorly for menacing look.
|
|
Pinguecula is from what layer? What can cause?
|
Submucosa. Wind, dust, UV.
|
|
What is dacroadenitis? Symptoms?
|
Inflammation of lacrimal gland. Pt. has discomfort in upper, lateral eyelid and reduced tearing on that side.
|
|
What layer of the tear film does ocular pemphigoid and stevens johnson syndrome disrupt?
|
Mucin
|
|
Low TBUT indicates deficiency of what layer of tears.
|
Lipid. (Oil, from Mei. gland) evaporation quickly
|
|
Low Schirmer strip time indicates deficiency of what layer?
|
Aqueous
|
|
What type of gland is a goblet cell?
|
Mucous.
|
|
What type of gland is meibomian gland?
|
Lipid
|
|
What straddles lacrimal sac?
|
Limbs of medial palpebral ligament.
|
|
What is dacrocystitis? Symptoms?
|
Inflammation of lacrimal sac. Pain and epiphora.
|
|
Where does nasolacrimal duct terminate?
|
Inferior meatus of nasal cavity.
|
|
3 components of acute inflammation:
|
1) Vascular size change - Quick VC, followed by VD.
2) Structural changes in microvascularization to increase arrival of leukocytes and plasma proteins 3) Immigration of leukocytes (PMNs) |
|
Possible outcomes of acute inflammation:
|
Scar, Chronic Infl. progression, Abscess formation (pus), Resolution ("SCAR")
|
|
3 Components of Chronic Inflammation:
|
1) Infiltration of mononuclear cells (Macrophages, lymphocytes, plasma cells)
2)Tissue destruction 3) New vessel proliferation and fibrosis. |
|
True/False: All Acute Inflammation reactions are identical.
|
True
|
|
True/False: All chronic inflammation reactions are identical.
|
False.
|
|
2 common causes of binocular, granulomatous uveitis:
|
TB and Sarcoidosis
|