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15 Cards in this Set
- Front
- Back
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Differentiate between Gross and Histopathology And Ultrastructural.
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Gross - visible to the naked eye
Histo - only seen under a slide Ultrastructural - under electron microscope |
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Define Pathognomonic lesions.
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those so characteristic as to identify a cause without a doubt.
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Name the dimensions of description
(8) |
1. Size
2. Color 3. Consistency 4. Shape 5. Surface 6. Margins 7. Distributions 8. Location |
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Name the 6 descriptors of morphological diagnosis
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1. Severity
2. Duration 3. Distribution 4. Modifier 5. Anatomic site 6. Lesion type |
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In pertaining to etiology, MINI VAN DITTY stands for?
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M - metabolic
I - inflammatory N - neoplastic I - infectious V - vascular A - anomaly of development N - nutritional D - degenerative I - idiopathic T - traumatic T - toxic I - iatrogenic |
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K9 Parvovirus pathogenesis
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1. Virus infects rapidly dividing cells
2. necrosis of intestinal mucosa 3. diarrhea and entrance of Gram negative bacteria into bloodstream 4. endotoxic shock and dehydration 5. Death |
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Free radicals interact with thymine to do what?
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Produce single strand breaks.
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Selenium is a component of which enzyme? What does the enzyme do?
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glutathione peroxidase. It reduces the hydroxyl radical to water.
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What does aflatoxin do to DNA?
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binds to it, forming adducts. This also prevents fatty acid oxidation.
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What does ricin do on a cellular level?
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Inhibits ribosomes.
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What is the meaning of life?
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42.
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What are Heat Shock Proteins?
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Molecular chaperones produced in response to almost any stress that act intracellularly to manipulate proteins. Often targeted by Autoimmune diseases.
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What types of cells have high susceptibility? Low?
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High - neurons.
Low - fibroblasts, epidermis, skeletal muscle |
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What is the pathogenesis for Feline Hepatic Lipidosis?
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Fat cat stops eating
Mobilization of fat from LARGE stores excessive fatty acids enter liver lipid processing is overwhelmed protein availability reduced by anorexia decreased lipid export fatty change Liver dysfunction icterus and anorexia fat yellow cat still not eating Repeat |
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What is the pathogenesis for Ruminant hepatic lipidosis?
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Over-conditioned animal (usually in late pregnancy or early lactation)
intake doesn't keep up with nutritional demand Large fat stores mobilized fatty liver |
