Reading...
Play button
Play button
Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
267 Cards in this Set
- Front
- Back
|
Clostridial Enteritis CD
Age |
Day 1 to 7
|
|
|
Clostridial Enteritis CD
Agent |
Clostridia difficile
|
|
|
Clostridial Enteritis CD
Clinical Signs |
Diarrhea Sporadic
Weight Loss Thin Appearance of Backbone Abdominal Distension Edema Dyspnea Edema Edema Subcutaneous Scrotal |
|
|
Clostridial Enteritis CD
Control |
Lactation Disruption Minimize
Fostering Reduce Antimicrobial Minimize Stop Routine Injections esp Broad Spectrum ie Tets Vaccination Injection Autogenous Pre Farrowing Normal Flora Re-establish Probiotics, Yogurt Antimicrobial Feed 2.5 Weeks Pre Farrow to 3 Weeks Post Farrowing Sow Bacitracin Methylene Disalicylate (BMD) |
|
|
Clostridial Enteritis CD
Diagnosis |
Diffuse Edema Suckling Pig
Mesocolonic Edema Suckling Pig Remarkable and Indicative Immuno Histo Chemistry ELISA (Feces, Gut Loops, Anaerobic) Toxin A, Toxin B PCR Feces, Gut Loops, Anaerobic Medium Toxin A, Toxin B Genes Culture Difficult ie Difficile =; ) |
|
|
Clostridial Enteritis CD
Epidemiology |
Colonizes Upon Disruption or Non Establishment Normal Flora
Gilt Start Up Herds Antimicrobial Use Chronic Gestation Pens Antimicrobial Use Excessive Piglets Fostering Excessive Disruption During Lactation |
|
|
Clostridial Enteritis CD
Etiology |
Emerging Disease Suckling Pig Neonatal
Zoonotic NIET |
|
|
Clostridial Enteritis CD
Pathogenesis |
Non Invasive
Enterotoxins Toxin A Fluid Filled Intestines Cytotoxins Toxin B Inhibition Protein Synthesis Cell Division Enterocyte Desquamation Exfoliation Degranulation Mucosal Mast Cells DIFFUSE EDEMA |
|
|
Clostridial Enteritis CD
Treatment |
Unrewarding Frustrating Kind of Like Studying Pigs
|
|
|
Clostridial Enteritis CPA
Age |
Day 1 to 2 Diarrhea within 48 Hours of Birth
|
|
|
Clostridial Enteritis CPA
Agent |
Clostridia perfringens Type A
|
|
|
Clostridial Enteritis CPA
Clinical Signs |
Diarrhea Creamy Pasty
|
|
|
Clostridial Enteritis CPA
Control |
Vaccination Injection Difficult May not Contain CPA Alpha or Beta 2 Toxoids
Vaccination Injection Conditionally Licensed Toxoid (CPA Alpha Toxoid) Novartis Antimicrobial Feed 2 to 3 Weeks Pre Farrow Sows Bacitracin Methylene Disalicylate (BMD) |
|
|
Clostridial Enteritis CPA
Diagnosis |
Histology Villous Atrophy Superficial Necrosis
Small Intestines NON Hemorrhagic Cytology Mucosal Smears Gram +ve Rods Culture Anaerobic PCR Toxin Genes Alpha and Beta2 Genes Immuno Histo Chemistry ELISA Intestinal Contents Alpha and Beta2 Toxins |
|
|
Clostridial Enteritis CPA
Differentials |
ETEC Indistiguishable
|
|
|
Clostridial Enteritis CPA
Epidemiology |
Normal Flora Traditional Belief
Ubiquitous Gut Contents Soil Mortality Rare |
|
|
Clostridial Enteritis CPA
Etiology |
Diarrhea Mild Suckling and Nursery NON Hemorrhagic
|
|
|
Clostridial Enteritis CPA
Pathogenesis |
Toxin Production Alpha Beta 2 Toxin (Mild)
Small Intestines Epithelium Necrosis Attachement Villous Atrophy Superficial Necrosis |
|
|
Clostridial Enteritis CPA
Stage |
Suckling ONLY FOR EXAM
Nursery Emerging Evidence |
|
|
Clostridial Enteritis CPA
Treatment |
Supportive Care Rewarding Usually
|
|
|
Clostridial Enteritis CPC
Age |
Day 1 to 2 Peracute
Day 3 Acute Day 4 Plus Chronic |
|
|
Clostridial Enteritis CPC
Agent |
Clostridia perfringens Type C
|
|
|
Clostridial Enteritis CPC
Clinical Signs |
Death Acute Survive for 2 to 3 Days Die at about 3 Days
Death Peracute < 36 Hours From Birth Severe Hemorrhagic Enteritis Diarrhea Reddish Brown, Necrotic Debris Acute Diarrhea Intermittent or Persistent Chronic Diarrhea Acute Necrotizing Hemorrhagic Enteritis Peracute |
|
|
Clostridial Enteritis CPC
Control |
Antimicrobial Feed 2.5 Weeks Pre Farrow to 3 Weeks Post Farrowing Sow Bacitracin Methylene Disalicylate (BMD)
Vaccination Injection Dams Commercial or Autogenous |
|
|
Clostridial Enteritis CPC
Diagnosis |
Abdominal Skin Black Discolouration Prior to Death
Culture Anaerobic Cytology Mucosal Smears Gram +ve Rods Immuno Histo Chemistry ELISA Intestinal Contents Beta Toxin PCR Toxin Genes Alpha and Beta Small Intestines Nectrotic Membrane Blood Tinged Intestinal Contents Small Intestines Intensely Hemorrhagic |
|
|
Clostridial Enteritis CPC Epidemiology
|
Acute Necrotizing Hemorrhagic Enteritis Suckling Pig ONLY CAUSE
Epizootic Outbreaks Historically USofA Persistent in Environment Year 1 Plus Western Canada Rare |
|
|
Clostridial Enteritis CPC
Etiology |
Encapsulated Gram +ve Spore Forming
|
|
|
Clostridial Enteritis CPC
Pathogenesis |
Attachement Villous Tip Jejunal Enterocytes
Enterocyte Desquamation Lamina Propria Necrosis Hemorrhage Proliferation Basement Membrane Toxin Production Alpha and Beta Necrotizing |
|
|
Clostridial Enteritis CPC
Stage |
Suckling
|
|
|
Clostridial Enteritis CPC
Treatment |
Clinical Cases Unrewarding
|
|
|
Coccidiosis
Age |
Day 5 Plus ONLY Suckling Pigs
Week 1 to 3 Typical Suckling Pigs Week 5 to 6 Possible Nursery Pigs |
|
|
Coccidiosis
Agent |
Isospora suis
|
|
|
Coccidiosis
Clinical Signs |
Dehydration
Diarrhea Day 5 Plus ONLY Diarrhea Loose Pasty Diarrhea Week 1 to 3 Typical Suckling Pigs Hair Coat Rough Wait Gain Impaired |
|
|
Coccidiosis
Control |
Sanitation Disinfection Vigourous
Seal Porous Surfaces Concrete Floors Wooden Partitions |
|
|
Coccidiosis
Diagnosis |
Cytology Mucosal Smears Merosoites
Cytology Later Stages of Disease Fecal Exam Oocysts Histology Mucosal Smears Merosoites |
|
|
Coccidiosis
Epidemiology |
Age Relationship Severity Decreases with Age
Morbidity High Within Litter Mortality Low Oral Transmission Patency 5 Days from Ingestion Sporulated Oocysts Poor Farrowing Room Hygiene Traditional Association Source Sows Environment Ubiquitous |
|
|
Coccidiosis
Pathogenesis |
Diarrhea Malabsorptive
Enteritis Catarrhal Mild to Severe Enteritis Fibrinonecrotic Membrane Enteritis Necrotizing NON Hemorrhagic Invasive Sporozites Penetrate Enterocytes Villous Atrophy Villous Fusion |
|
|
Coccidiosis
Stage |
Nursery
Suckling |
|
|
Coccidiosis
Treatment |
Anticoccidial No Longer Available Bacycox Removed from Market
|
|
|
Colibacillosis ETEC Enterotoxic
Age |
Day 1 to 7
|
|
|
Colibacillosis ETEC Enterotoxic
Agent |
Escheria coli Enterotoxic
|
|
|
Colibacillosis ETEC Enterotoxic
Clinical Signs |
Death Acute
Death Peracute Before Onset Diarrhea Dehydration Diarrhea Emesis Curdled Milk Severe Cases Only Lethargy Lateral Recumbancy Severity Virulence, Vaccination Status, IgA Dam Environment, Piglet Age |
|
|
Colibacillosis ETEC Enterotoxic
Control |
Environment Chilling > 25 C
Fostering Stop Immediately Management Sanitation Hygiene Vaccination Injection Autogenous Bacterins (No Toxoid) Farm Specific (Gallant) Vaccination Injection KIlled Bacterins Adhesins (F4, F5, F6, F41) Toxoids (Sta, STb, LT) Vaccination Injection Sows 2 Weeks Pre Farrowing Gilts 5 and 2 Weeks Pre Farrowing |
|
|
Colibacillosis ETEC Enterotoxic
Diagnosis |
Age Day 1 to 7
Congestion Small Intestines Vasculature Culture and Sensitivity GUT LOOP Rectal Swabs Dehydration Dilated Fluid Filled Small Intestines Dilated Stomach +/- Undigested Curd Fecal pH Alkaline Gilt Litters Histology Fresh Gut Villus Atrophy Minimal Necropsy Piglets in Acute Stage 12hrs Not Tx 2 - 3 Min, Typical CS, History PCR Genes Fimbria Enterotoxin |
|
|
Colibacillosis ETEC Enterotoxic
Epidemiology |
Environment Farrowing Crate Skin of Dam
IgA Protective tf Pre Farrowing Vaccination More Common Parity 1 and 2 Sows Milk IgA Levels Lower |
|
|
Colibacillosis ETEC Enterotoxic
Pathogenesis |
Enterotoxins STa, STb (Heat Stable Toxins) LT (Heat Liable Toxin)
Fimbrial Adhesins F4 (K88), F5 (K99), F6 (987P), F41 Secretory Diarrhea tf Usually No vilus atrophy |
|
|
Colibacillosis ETEC Enterotoxic
Stage |
Suckling
|
|
|
Colibacillosis ETEC Enterotoxic
Treatment |
Antimicrobial Feed Second Choice Neomycin
Antimicrobial Parenteral Prefered Ceftiofur Antimicrobial Parenteral Prefered Gentamycin Antimicrobial Parenteral Prefered TMS Electrolyte Dextrose Syringe or Dish |
|
|
Edema Disease ED
Agent |
Escheria coli Shiga Toxin Producing
Escheria coli Verotoxigenic VTEC Exotoxins Stx1, Stx2, Stx2e |
|
|
Edema Disease ED
Clinical Signs |
Death Peracute < 24 Hours Otherwise Healthy Pigs
Edema Eyelids Face Edema Proximal Stomach Wall Characteristic but < 10% Neurologic Ataxia, Recumbancy, Paddling, Disorientation Cerebral Edema Self Limiting Spontaneous Recovery Possible |
|
|
Edema Disease ED
Control |
Antimicrobial Feed
Antimicrobial Water Feed Reduce Energy Density Short Term Restricted Feeding Feed Zinc Oxide May Help Vaccination Injection NIET |
|
|
Edema Disease ED
Differentials |
Clostridia difficile Differentiate by Age
Salt Poisoning Differentiate by Number Affected |
|
|
Edema Disease ED
Epidemiology |
Morbidity Low
Mortality Case Mortality High Stressors High Energy Diet Environment Ubiquitous Disease is Rare Weeks 4 to 6 (1 to 3 Weeks Post Wean) |
|
|
Edema Disease ED
Etiology |
Enterotoxemia Exotoxin producing Escheria coli
Fimbrial Adhesins F4, F18 Most Common Specific Serotypes O138, O139, O141 Traditionally Reported |
|
|
Edema Disease ED
Pathogenesis |
Adherence Epithelium F4, F18
Colonization Edema Various Tissues Exotoxins Stx1, Stx2, Stx2e Toxemia Cytotoxic Endothelial Cells |
|
|
Edema Disease ED
Treatment |
Unrewarding Peracute Death Healthy Pigs
|
|
|
Gastric Ulcers Acute Stage 3
Clinical Signs |
Death Acute Hemorrhage Intragastric
Pale Weak Melena Possible |
|
|
Gastric Ulcers All Forms
Agent |
Helicobacter LIKE Bacteria May Have Some Significance
|
|
|
Gastric Ulcers All Forms
Clinical Signs |
Pale Puking Pig
|
|
|
Gastric Ulcers All Forms
Control |
Concurrent Disease Heliobacter Like Bacteria ??? Reduced Feed Intake
Environment Hot Weather = Decreased Intake Feed - Avoid Feed Interuptions Empty Stomachs Partical Size Want > 400 microns |
|
|
Gastric Ulcers All Forms
Epidemiology |
Glandular Fundic and Pyloric Uncommon Associated with Systemic Dz (Salmonellosis, Erysipelas, Hog Cholera)
Non Glandula Pars Esophagus Most Common |
|
|
Gastric Ulcers All Forms
Stage |
Finisher
Grower |
|
|
Gastric Ulcers Chronic
Clinical Signs |
Death Chronic Emaciation Euthanasia
Weight Loss Esophageal Stricture |
|
|
Gastric Ulcers Subclinical Stage 0 to 2
Clinical Signs |
Constipation Fecal Balls Melena Possible
Emesis Inappetance Most Common Detected at Slaughter |
|
|
Gastrosplenic Torsion
Age |
Kg 150 Plus
|
|
|
Gastrosplenic Torsion
Clinical Signs |
Death Peracute
Splenic Torsion Subclinical Incidental Slaughter |
|
|
Gastrosplenic Torsion
Epidemiology |
Feed Speed and Frequency
Excessive Activity (Regrouping) |
|
|
Gastrosplenic Torsion
Etiology |
Torsion 180 to 360 Degree Stomach +/- Spleen
|
|
|
Gastrosplenic Torsion
Stage |
Adult
|
|
|
Mesenteric Torsion
Age |
Kg 50 to 120
|
|
|
Mesenteric Torsion
Clinical Signs |
Bloat Abdomen
Death Peracute Non Agonal Pale Blood Pools GIT Rectal Prolapse Common |
|
|
Mesenteric Torsion
Diagnosis |
Congestion ENTIRE GIT Pathognomic
Torsion Root of Mesentery |
|
|
Mesenteric Torsion
Epidemiology |
Feed Interuption
Feed Changes Sudden Excessive Activity |
|
|
Mesenteric Torsion
Etiology |
Torsion 189 to 360 Degree Root of Mesentery
|
|
|
Mesenteric Torsion
Stage |
Finisher
Grower |
|
|
Necroproliferative Enteritis All Forms
Control |
Antimicrobial Feed Pulsed or Continuous Tetracyclines
Antimicrobial Feed Pulsed or Continuous Tylosin Vaccination Injection Long Duration of Activity 22 Weeks Plus Vaccination Injection No Antimicrobials 3 Days Pre and Post Vaccination |
|
|
Necroproliferative Enteritis All Forms
Diagnosis |
Histology Intracellular Bacteria Special Stains (Silver)
Immuno Histo Chemistry PCR Feces Tissue Serology Timing of Exposure Vaccination Planning |
|
|
Necroproliferative Enteritis All Forms
Treatment |
Antimicrobial Feed Herd Treatment Lincomycin
Antimicrobial Feed Herd Treatment Tetracyclines Antimicrobial Feed Herd Treatment Tylosin Antimicrobial Parenteral Valuable Animals Animals Demonstrating Clinical Signs Antimicrobial Water Herd Treatment Tetracyclines |
|
|
Necroproliferative Enteritis NE Necrotic Enteritis
Age |
Week 6 to 20
|
|
|
Necroproliferative Enteritis NE Necrotic Enteritis
Agent |
Lawsonia intracellularis
|
|
|
Necroproliferative Enteritis NE Necrotic Enteritis
Clinical Signs |
Death Increasing Involvement of Mucosa and Muscle Layers in Severe Cases
Diarrhea Homogeneous Normal Colour 50% of Cases Growth Rate Impaired Hose Pipe Gut Increasing Involvement of Mucosa and Muscle Layers in Severe Cases Necrotic Enteritis Increasing Involvement of Mucosa and Muscle Layers in Severe Cases Pseudomembrane Increasing Involvement of Mucosa and Muscle Layers in Severe Cases Regional Ileitis Increasing Involvement of Mucosa and Muscle Layers in Severe Cases Sub Clinical Fecal Shedding Can Start at 3 Weeks Seroconversion ins Sows Increases Risk of PIA in Grow Finish Weight Loss |
|
|
Necroproliferative Enteritis NE Necrotic Enteritis
Epidemiology |
Infection Prevalent
|
|
|
Necroproliferative Enteritis NE Necrotic Enteritis
Onset |
Chronic
|
|
|
Necroproliferative Enteritis NE Necrotic Enteritis
Pathogenesis |
Adenoma Like Mucosa
Crypt Cell Invasion Exposure in Nursery Incubation 2 to 3 Weeks Intermittent Fecal Shedding Subclinical Animals Intestinal Mucosa Thickened Inflamed Necrotic Penetrates Dividing Crypt Cells Crypt Cells Fail to Mature > Hyperplastic > "Ademoma" Proliferation of immature epi cells in Crypt Proliferative and Necrotic Lesions Thickening of Mucosa Small Intestines and Colon |
|
|
Necroproliferative Enteritis NE Necrotic Enteritis
Stage |
Finisher
Grower |
|
|
Necroproliferative Enteritis PHE Proliferative Herorrhagic Enteropathy
Age |
Kg 80 Plus Older Finisher Hogs
Week 16 to 52 Breeding Herd |
|
|
Herorrhagic Enteropathy
Agent |
Necroproliferative Enteritis PHE Proliferative Lawsonia intracellularis
|
|
|
Necroproliferative Enteritis PHE Proliferative Herorrhagic Enteropathy
Clinical Signs |
Death Acute
Death Peracute Dysentery Possible Melena Pale Anemia Acute Blood Loss |
|
|
Necroproliferative Enteritis PHE Proliferative Herorrhagic Enteropathy
Control |
Vaccination Injection Breeding Herd Enterisol Ileitis BIVM
Vaccination Injection Gilt Replacements Enterisol Ileitis BIVM |
|
|
Necroproliferative Enteritis PHE Proliferative Herorrhagic Enteropathy
Diagnosis |
Necropsy Melana Blood
|
|
|
Necroproliferative Enteritis PHE Proliferative Herorrhagic Enteropathy
Epidemiology |
Mortality Case Fatality 50%
New Gilt Start Up Herds Season Autum |
|
|
Necroproliferative Enteritis PHE Proliferative Herorrhagic Enteropathy
Onset |
Acute
|
|
|
Necroproliferative Enteritis PHE Proliferative Herorrhagic Enteropathy
Pathogenesis |
Degeneration of Intestinal Epithelium
Hemorrhage Acute |
|
|
Necroproliferative Enteritis PHE Proliferative Herorrhagic Enteropathy
Stage |
Adult
Finisher |
|
|
Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis
Age |
Week 6 to 20
|
|
|
Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis
Agent |
Lawsonia intracellularis
|
|
|
Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis
Clinical Signs |
Death Increasing Involvement of Mucosa and Muscle Layers in Severe Cases
Diarrhea Homogeneous Normal Colour 50% of Cases Growth Rate Impaired Hose Pipe Gut Increasing Involvement of Mucosa and Muscle Layers in Severe Cases Necrotic Enteritis Increasing Involvement of Mucosa and Muscle Layers in Severe Cases Pseudomembrane Increasing Involvement of Mucosa and Muscle Layers in Severe Cases Regional Ileitis Increasing Involvement of Mucosa and Muscle Layers in Severe Cases Sub Clinical Fecal Shedding Can Start at 3 Weeks Seroconversion ins Sows Increases Risk of PIA in Grow Finish Weight Loss |
|
|
Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis
Diagnosis |
Necropsy Ileum Thickened Hose Pipe
|
|
|
Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis
Epidemiology |
Infection Prevalent
|
|
|
PIA Porcine Intestinal Ademonatosis Necroproliferative Enteritis
Onset |
Chronic
|
|
|
Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis
Pathogenesis |
Adenoma Like Mucosa
Crypt Cell Invasion Exposure in Nursery Incubation 2 to 3 Weeks Intermittent Fecal Shedding Subclinical Animals Intestinal Mucosa Thickened Inflamed Necrotic Penetrates Dividing Crypt Cells Crypt Cells Fail to Mature > Hyperplastic > "Ademoma" Proliferation of immature epi cells in Crypt Proliferative and Necrotic Lesions Thickening of Mucosa Small Intestines and Colon |
|
|
Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis
Stage |
Finisher
Grower |
|
|
Necroproliferative Enteritis RI Regional Ileitis
Age |
Week 6 to 20
|
|
|
RI Regional Ileitis
Agent |
Necroproliferative EnteritisLawsonia intracellularis
|
|
|
Necroproliferative Enteritis RI Regional Ileitis
Clinical Signs |
Death Increasing Involvement of Mucosa and Muscle Layers in Severe Cases
Diarrhea Homogeneous Normal Colour 50% of Cases Growth Rate Impaired Hose Pipe Gut Increasing Involvement of Mucosa and Muscle Layers in Severe Cases Necrotic Enteritis Increasing Involvement of Mucosa and Muscle Layers in Severe Cases Pseudomembrane Increasing Involvement of Mucosa and Muscle Layers in Severe Cases Regional Ileitis Increasing Involvement of Mucosa and Muscle Layers in Severe Cases Sub Clinical Fecal Shedding Can Start at 3 Weeks Seroconversion ins Sows Increases Risk of PIA in Grow Finish Weight Loss |
|
|
Necroproliferative Enteritis RI Regional Ileitis
Epidemiology |
Infection Prevalent
|
|
|
Necroproliferative Enteritis RI Regional Ileitis
Onset |
Chronic
|
|
|
Necroproliferative Enteritis RI Regional Ileitis
Pathogenesis |
Adenoma Like Mucosa
Crypt Cell Invasion Exposure in Nursery Incubation 2 to 3 Weeks Intermittent Fecal Shedding Subclinical Animals Intestinal Mucosa Thickened Inflamed Necrotic Penetrates Dividing Crypt Cells Crypt Cells Fail to Mature > Hyperplastic > "Ademoma" Proliferation of immature epi cells in Crypt Proliferative and Necrotic Lesions Thickening of Mucosa Small Intestines and Colon |
|
|
Necroproliferative Enteritis RI Regional Ileitis
Stage |
Finisher
Gower |
|
|
Post Weaning Colibacillosis
Age |
Day 24 to 26 (3 to 5 Days Post Wean) Associated with Feed Change
Week 6 (21 Days Post Wean) Associated with Feed Change |
|
|
Post Weaning Colibacillosis
Agent |
Escheria coli
Enterotoxic Fimbria F4ac, F18ab, F18ac |
|
|
Post Weaning Colibacillosis
Clinical Signs |
Death Peracute No Previous Signs
Dehydration Eyes Sunken Skin Discoloured Diarrhea Pasty to Watery Endotoxemia |
|
|
Post Weaning Colibacillosis
Control |
Antimicrobial Feed per
Culture and Sensitivity Apramycin Antimicrobial Water per Culture and Sensitivity TMS or other Sulfonamide Environment Prevent Chilling Improve Sanitation Feed Adhesion Competitive Inhibition Mannoligosaccharides Feed Acidify Gastrointestinal pH Reduce Calcium Carbonate Week 3 to 5 (1st 2 Weeks Post Weaning Substrate Reduction Poorly Digested Absorbed CHO Excess or Poor Protein Vaccination Injection Live Avirulent Escheria coli (O149:K91:F4) 5 and 12 Days Post Weaning Prevtec Microbia can Leave AM in Feed Water Acidifcation |
|
|
Post Weaning Colibacillosis
Diagnosis |
PCR Feces, Gut Loops Enterotoxin STa, STb, LT Genes
PCR Feces, Gut Loops Fimbria F4, F18ab, F18ac Genes Small Intestines Contents Yellow Pasty to Watery Mucoid Small Intestines Dilated Small Intestines Edematous Small Intestines Hyperimic |
|
|
Post Weaning Colibacillosis
Epidemiology |
Enzootic
Feed Antigen Increase Elevated Soybean Meal Elevated Canola Meal Feed Antimicrobial Decrease Absence Zinc Oxide Antimicrobial Activity Feed pH Increase Elevated Calcium Elevated Magnesium Morbidity Multifactorial Effects Enviro, Concurrent Enteric Dz, Feed Mortality Multifactorial Effects Enviro, Concurrent Enteric Dz, Feed Peaks Day 24 to 26 (3 to 5 Days Post Wean) Associated with Feed Change Peaks Week 6 (21 Days Post Wean) Associated with Feed Change Sporadically Herd Outbreaks |
|
|
Post Weaning Colibacillosis
Pathogenesis |
Enterotoxins STa, STb (Heat Stable Toxins) LT (Heat Liable Toxin)
Receptors Genetic Resistance Many Pigs Do Not Express F4, F18 Receptors Week 3 Plus (>20 Days) F18 Receptor Expressed |
|
|
Post Weaning Colibacillosis
Stage |
Nursery
|
|
|
Post Weaning Colibacillosis
Treatment |
Antimicrobial Feed
per Culture and Sensitivity Neomycin |
|
|
Post Weaning Diarrhea
Agent |
Multifactorial ETEC, Rotavirus, TGE, Coccidiosis, Salmonellosis, Porcine Circovirus
Nutritional, Inappropriate Formulations, High Nutrient Density |
|
|
Post Weaning Diarrhea
Epidemiology |
Declining Immunity IgG IgA
Dietary Change Liquid to Solid Environment Change in Environment Change in Sanitation Normal Flora Change in GI Flora Social Stress of Weaning Decreased Villi Size |
|
|
PRCV Porcine Respiratory Corona Virus
Significance |
Partial Protection from TGE Cross Reacts with Serum Neutralization Reduces TGE Infection
|
|
|
Rectal Stricture
Clinical Signs |
Bloat
Emaciation Always Present Chronic Case |
|
|
Rectal Stricture
Pathogenesis |
Rectal Prolapse Secondary To
Salmonellosis Secondary To |
|
|
Rotavirus
Age |
Day 1 to 7 Recently
Day 7 Plus Classically |
|
|
Rotavirus
Agent |
Porcine Rotavirus
Group A Most Significant (A - G) Non Enveloped |
|
|
Rotavirus
Clinical Signs |
Death Day 14 Plus Uncommon
Diarrhea Day 1 to 7 Recently via Modern Management Diarrhea Day 7 Plus Classically Diarrhea Severity Decreases with Age Diarrhea Severity Increased with Concurrent Escheria coli |
|
|
Rotavirus
Control |
Sanitation Uber Important Environmental Resistance
Vaccination Injection Sows Pre Farrowing Combined with ETEC |
|
|
Rotavirus
Diagnosis |
Fecal pH Acidic via Villus Atrophy
Histology Villous Atrophy Less Severe than TGE Immuno Histo Chemistry Definitive PCR Antigen Identification, Nucleic Acid Definitive = Best |
|
|
Rotavirus
Differentials |
ETEC
TGE Virus |
|
|
Rotavirus
Epidemiology |
Endemic Most Herds
Fecal Oral Ubiquitous Stable Resistent |
|
|
Rotavirus
Etiology |
Non Enveloped = Tough Group
A Most Significant (A - G) Multi Serotypes Often on Farm |
|
|
Rotavirus
Pathogenesis |
Diarrhea Malabsorptive Osmotic
Enterocytes Lysis (Villus TIP) Desquamation Small Intestines Thinwalled Fluid Filled Villous Atrophy Moderate Varies with Severity Villous Atrophy SI Distal |
|
|
Rotavirus
Stage |
Suckling
|
|
|
Rotavirus
Treatment |
Antimicrobial
Non Responsive +/- Co-infections - ETEC |
|
|
Salmonellosis All Forms
Control |
Biosecurity Breeding Stock Incoming = Neg Status Vermin Control,
Fomites = People Feed Coarse Grind = Low pH = Better Meal Better than Pelleting Management Sanitation, Slats AIAO, Stocking Density Reduce Gut pH Fermented Wet Feed +/- Whey Water Acidification Nursery Best Vaccination Injection Killed Bacterins = NIET NOT = Cell Mediated Vaccination Injection Live Attenuated Bacterin BIVM Enterisol SC54 - X protects |
|
|
Salmonellosis All Forms
Diagnosis |
Culture Enriched 50 g Best
PCR Most Sensitive Confirm Species Serology Herd Testing Only Poor Sensitivity Individual |
|
|
Salmonellosis All Forms
Epidemiology |
Enzootic Reservoir Intestinal Tract of Warm and Cold Blooded Animals
Fecal Oral Fomites Persists Years 6 Plus |
|
|
Salmonellosis All Forms
Etiology |
2400 Serotypes
Clinical Salmoellosis in Pigs Salmonella cholerasuis, Salmonella typhimurium |
|
|
Salmonellosis All Forms
Significance |
Food Safety Number One Issue for Pork
Human Salmonellosis from Pork Low Multi Drug Resistance ACSSuT and AKSSuT |
|
|
Salmonellosis Enteric
Agent |
Salmonella california Possible
Salmonella many other species Possible Salmonella typhimurium (Group B) Non Host Adapted Salmonella worthington Possible |
|
|
Salmonellosis Enteric
Clinical Signs |
Diarrhea Initially Yellow Watery Becomes Bloody
Dysentery Enterocolitis Necrotizing Ulcerative Often Associated with Other Predisposing Factors Rectal Stricture Sequella Also Prolapse, Tenesmus, Tail Bite, Chronic Cough |
|
|
Salmonellosis Enteric
Differentials |
Spirochetal Diarrhea Colitis Only
Swine Dysentery Colitis Only |
|
|
Salmonellosis Enteric
Epidemiology |
Salmonella typhimurium
Pigs, Rodents, Birds Water, Feed, Bedding |
|
|
Salmonellosis Enteric
Pathogenesis |
Initial Fluid Secretion Independent of
Intestinal Mucosal Inflammation Microvascular Thrombosis Mucosal Ischemia Necrosis Mucosal Inflammation Neutrophil Recruitment Often Associated with Other Predisposing Factors |
|
|
Salmonellosis Enteric
Significance |
Antimicrobial Resistance
Salmonella typhimurium DT 104, DT 106, DT 193 |
|
|
Salmonellosis Enteric
Stage |
Suckling
Nursery Grower Finisher Adult |
|
|
Salmonellosis Enteric
Treatment |
Antimicrobial
Sick Animals Herd Outbreak Severe |
|
|
Salmonellosis Septic
Agent |
Salmonella cholerasuis (Group C)
Host Adapted Sepsis via Invasiveness |
|
|
Salmonellosis Septic
Clinical Signs |
Death Peracute
Enterocolitis Small AND Large Intestine Pneumonia Septicemia |
|
|
Salmonellosis Septic
Differentials |
Actinobacillus suis
Erysipelas Streptococcus suis |
|
|
Salmonellosis Septic
Epidemiology |
Salmonella cholerasuis
Intestinal Tracts of other Pigs |
|
|
Salmonellosis Septic
Pathogenesis |
Invasive Much More than other Serotypes
Pharyngeal Tonsil Invasion Septicemia 12 to 72 Hours Prior to Onset of Diarrhea |
|
|
Salmonellosis Septic
Stage |
Finisher
Grower Nursery |
|
|
Salmonellosis Septic
Treatment |
Antimicrobial Feed Aggressive Therapy Individuals Ceftiofur
Antimicrobial Feed Aggressive Therapy Individuals TMS Antimicrobial Parenteral Aggressive Therapy Individuals Ceftiofur Antimicrobial Parenteral Aggressive Therapy Individuals TMS Antimicrobial Water Aggressive Therapy Individuals Ceftiofur Antimicrobial Water Aggressive Therapy Individuals TMS |
|
|
Spirochetal Diarrhea
Agent |
Brachyspira pilosicoli
aka Porcine Intestinal Spirochetosis PIS |
|
|
Spirochetal Diarrhea
Clinical Signs |
Diarrhea Profuse, Watery, Bloody, Mucoid Severe Cases Only
Diarrhea Wet Cement Growth Rate Impaired Self Limiting 2 - 14 Days Possible |
|
|
Spirochetal Diarrhea
Control |
Erradication Depopulation
NOT Required |
|
|
Spirochetal Diarrhea
Diagnosis |
Colitis Hemorrhagic Severe Cases Only
Colitis Necrotizing Superficial Necrosis Colon ONLY Colon Epithelium Catarrhal Exudate Epithelial Surface Culture Hemolysis Weak Beta Blood Agar PCR Feces, Tissue Definitive and Specific |
|
|
Spirochetal Diarrhea
Differentials |
PPE (Lawsonia) Differentiate Ileal Lesion
Salmonella Differentiate Ileal Lesion Swine Dysentery Hemolysis Strong Trichuriasis (Whipworms) |
|
|
Spirochetal Diarrhea
Epidemiology |
Extremely Contagious Less Virulent than B. hyodysentariae Rapid Spread
Fecal Oral Transmission Fomites between Farms Morbidity Low Moderate Mortality Low |
|
|
Spirochetal Diarrhea
Etiology |
Beta-Hemolytic Spirochete Weakly Beta-Hemolytic
Diarrhea Mild Moderate |
|
|
Spirochetal Diarrhea
Pathogenesis |
Epithelium
Degeneration Necrosis |
|
|
Spirochetal Diarrhea
Significance |
Weight Gain Impaired
|
|
|
Spirochetal Diarrhea
Stage |
Finisher
Grower |
|
|
Spirochetal Diarrhea
Treatment |
Antimicrobial Parenteral Individual Animals Lincomycin
Antimicrobial Parenteral Individual Animals Tiamulin Antimicrobial Parenteral Individual Animals Tylosine |
|
|
Swine Dysentery SD
Agent |
Brachyspira hyodysenteriae
fna Vibrionic Dysentery |
|
|
Swine Dysentery SD
Clinical Signs |
Abdominal Pain
Death Acute Rare without Diarrhea Death Peracute Rare without Diarrhea Diarrhea Frank Blood Possible Diarrhea Mucofibrinous Exudate Diarrhea Mucohemorrhagic Diarrhea Acute Onset Mucus Copious Blood Flecks within 2 to 3 Days |
|
|
Swine Dysentery SD
Control |
Antimicrobial Feed Herd Prophylaxis Lincomycin
Antimicrobial Feed Herd Prophylaxis Salinomycin Antimicrobial Feed Herd Prophylaxis Tiamulin Antimicrobial Feed Herd Prophylaxis Tylosine Antimicrobial Feed Herd Prophylaxis Virginiamycin Depopulate Erradication Medicate Sanitate Weaning Off Site Transmission Prior to Weaning NIET |
|
|
Swine Dysentery SD
Diagnosis |
Colitis and Typhlitis ONLY Fibrinous Necrotizing Hemorrhagic
Culture Hemolysis Blood Agar Diarrhea Mucofibrinohemorrhagic Explosive Outbreak Fibrinonecrotic Membrane Cecum and Colin Chronic Gross Lesions Cecum and Colin ONLY Mesentery and Large Intestine Edematous Hyperemic Mucosa Mucus, Fibrin Blood Flecks within 2 to 3 Days PCR Feces, Tissue Definitive and Specific |
|
|
Swine Dysentery SD
Differentials |
PPE Differentiate Ileal Lesion
Salmonella Differentiate Ileal Lesion |
|
|
Swine Dysentery SD
Epidemiology |
Carriers Recovered Pigs Shed 70 Days
Extremely Contagious Infective Dose Low Rapid Spread Fecal Oral Transmission Fomites between Farms Persistent in Environment Many Reservoirs Rodents, Dogs, Flies, Birds, Pigs Severe Disease Traditionally Mild Variants Emerging |
|
|
Swine Dysentery SD
Etiology |
Brachyspira hyodysenteriae Strongly Hemolytic
Flagella Involved in Virulence Hemolysins Involved in Virulence Motility Involved in Virulence Spirochete Gram -ve Anaerobic Aerotolerant |
|
|
Swine Dysentery SD
Pathogenesis |
Penetrates Mucous to Epithelial Cells Motility Required
Toxin Production = Malabsorption Hemolysin Lipooligosaccharide (LOS) |
|
|
Swine Dysentery SD
Stage |
Adult
Finisher Grower Nursery Week 8 Plus |
|
|
Swine Dysentery SD
Treatment |
Antimicrobial Parenteral Individual Animals Lincomycin
Antimicrobial Parenteral Individual Animals Tiamulin Antimicrobial Parenteral Individual Animals Tylosine Antimicrobial Water Herd Outbreaks Lincomycin Antimicrobial Water Herd Outbreaks Salinomycin Antimicrobial Water Herd Outbreaks Tiamulin Antimicrobial Water Herd Outbreaks Tylosine Antimicrobial Water Herd Outbreaks Virginiamycin |
|
|
TGE Transmissible Gastroenteritis All Forms
Agent |
TGE Virus
|
|
|
TGE Transmissible Gastroenteritis All Forms
Diagnosis |
Diarrhea Malabsorptive Osmotic
Enterocytes Lysis Desquamation Fecal pH Acidic Fluorescent Antibody Test Minimal Epi Desquamation ie Acute Cases Definitive Histology Villous Atrophy Severe SI Distal Immuno Histo Chemistry Minimal Epi Desquamation ie Acute Cases Definitive PCR Minimal Epi Desquamation ie Acute Cases Definitive Serology ELISA Competitive Does NOT Cross React with PRCV Serology Serum Neutralization Cross Reacts with PRCV Small Intestines Thinwalled Transparent Villous Atrophy Severe Blunting |
|
|
TGE Transmissible Gastroenteritis Enzootic
Clinical Signs |
Diarrhea Osmotic Malabsorptive
Diarrhea Transient Days a Few |
|
|
TGE Transmissible Gastroenteritis Enzootic
Control |
Eradication Clinical Observation Diarrhea
Eradication Feedback Intestines From Clinical Pigs Eradication Measure TGEV Serologic Response in Sentinals @ 30 Days Absence = Success Eradication Purchase ALL Breeding Replacements Close Herd Eradication Sentinals (TGE Naïve) @ 4 to 5 Months Expose to Feces from All Ages Eradication Wash and Disinfect Entire Barn Vaccination Injection NO Effective Vaccine Available |
|
|
TGE Transmissible Gastroenteritis Enzootic
Diagnosis |
Diarrhea
Persistent Non Responsive Post Weaning Scours |
|
|
TGE Transmissible Gastroenteritis Enzootic
Differentials |
Coccidiosis
Escheria coli Enterotoxic Rotavirus |
|
|
TGE Transmissible Gastroenteritis Enzootic
Epidemiology |
Mortality Low
Positive Farms Failure to Erradicate, Eliminate Transmission Oldest to Youngest in Nursery Loss of Access to Maternal IgA Viral Persistence Ongoing Infection Piglets Weaned Each Week |
|
|
TGE Transmissible Gastroenteritis Enzootic
Stage |
Nursery Generally
|
|
|
TGE Transmissible Gastroenteritis Enzootic
Treatment |
Supportive Care
Control Secondary Infections |
|
|
TGE Transmissible Gastroenteritis Epizootic
Clinical Signs |
Agalactia Sows 3 Weeks Onwards
Death Rare 3 Weeks Onwards Death Week 0 to 2 Near 100% Diarrhea Mild 3 Weeks Onwards Diarrhea Profuse, Watery Odoriferous +/- Curdled Milk Farrowing Emesis Transient Gastritis Farrowing Inappetance 3 Weeks Onwards Self Limiting 1 to 2 Days 3 Weeks Onwards |
|
|
TGE Transmissible Gastroenteritis Epizootic
Control |
Feedback
Feed Insetines of Scouring Piglets to ALL Pigs on Farm |
|
|
TGE Transmissible Gastroenteritis Epizootic
Diagnosis |
Mortality Near 100% < 2 to 3 Weeks
|
|
|
TGE Transmissible Gastroenteritis Epizootic
Epidemiology |
All Ages Clinically Affected
Mortality Near 100% < 2 to 3 Weeks Maternal IgA Response Lags Naïve Farm Susceptability 100% Season Spring Season Winter Short Incubation Transmission Through Farm Rapid 2 to 3 Days TGE Virus Preserved by Freezing |
|
|
TGE Transmissible Gastroenteritis Epizootic
Stage |
Adult Mild Disease
Finisher Mild Disease Grower Mild Disease Nursery Mild Disease Suckling Severe Disease High Mortality |
|
|
Age Day 1 to 2
|
Clostridial Enteritis CPA Diarrhea with 48 Hours of Birth
Clostridial Enteritis CPC Peracute |
|
|
Age Day 1 to 7
|
Clostridial Enteritis CD
Colibacillosis ETEC Enterotoxic Rotavirus Recently |
|
|
Age Day 24 to 26 (3 to 5 Days Post Wean)
|
Post Weaning Colibacillosis
Associated with Feed Change |
|
|
Age Day 3
|
Clostridial Enteritis CPC Acute
|
|
|
Age Day 4 Plus
|
Clostridial Enteritis CPC Chronic
|
|
|
Age Day 5 Plus
|
Coccidiosis ONLY Suckling Pigs
|
|
|
Age Day 7 Plus
|
Rotavirus Classically
|
|
|
Age Kg 150 Plus
|
Gastrosplenic Torsion
|
|
|
Age Kg 50 to 120
|
Mesenteric Torsion
|
|
|
Age Kg 80 Plus
|
Necroproliferative Enteritis
PHE Proliferative Herorrhagic Enteropathy Older Finisher Hogs |
|
|
Age Week 1 to 3
|
Coccidiosis
|
|
|
Age Week 16 to 52
|
Necroproliferative Enteritis
PHE Proliferative Herorrhagic Enteropathy Breeding Herd |
|
|
Age Week 5 to 6
|
Coccidiosis Possible Nursery Pigs
|
|
|
Age Week 6 (21 Days Post Wean)
|
Post Weaning Colibacillosis
|
|
|
Age Week 6 to 20
|
Necroproliferative Enteritis
NE Necrotic Enteritis |
|
|
Age Week 6 to 20
|
Necroproliferative Enteritis NE Necrotic Enteritis
Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis Necroproliferative Enteritis RI Regional Ileitis |
|
|
Stage Adult
|
Gastrosplenic Torsion
Necroproliferative Enteritis PHE Proliferative Herorrhagic Enteropathy Swine Dysentery SD TGE Transmissible Gastroenteritis Epizootic Mild Disease |
|
|
Stage Finisher
|
Gastric Ulcers All Forms
Mesenteric Torsion Necroproliferative Enteritis NE Necrotic Enteritis Necroproliferative Enteritis PHE Proliferative Herorrhagic Enteropathy Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis Necroproliferative Enteritis RI Regional Ileitis Salmonellosis Septic Spirochetal Diarrhea Swine Dysentery SD TGE Transmissible Gastroenteritis Epizootic Mild Disease |
|
|
Stage Gower
|
Necroproliferative Enteritis RI Regional Ileitis
Gastric Ulcers All Forms Mesenteric Torsion Necroproliferative Enteritis NE Necrotic Enteritis Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis Salmonellosis Septic Spirochetal Diarrhea Swine Dysentery SD TGE Transmissible Gastroenteritis Epizootic Mild Disease |
|
|
Stage Nursery
|
Clostridial Enteritis CPA Emerging Evidence
Coccidiosis Post Weaning Colibacillosis Salmonellosis Septic Swine Dysentery SD Week 8 Plus TGE Transmissible Gastroenteritis Enzootic Generally TGE Transmissible Gastroenteritis Epizootic Mild Disease |
|
|
Stage Suckling
|
Clostridial Enteritis CPA ONLY FOR EXAM
Clostridial Enteritis CPC Coccidiosis Colibacillosis ETEC Enterotoxic Rotavirus TGE Transmissible Gastroenteritis Epizootic Severe Disease High Mortality |
|
|
Abdominal Distension
|
Clostridial Enteritis CD Edema
|
|
|
Abdominal Pain
|
Swine Dysentery SD
|
|
|
Abortion Storm
|
Leptospirosis Pamona Maintenance
Still Born, Weak MUMMIES NIET |
|
|
Agalactia
|
TGE Transmissible Gastroenteritis Epizootic Sows 3 Weeks Onwards
|
|
|
Bloat
|
Mesenteric Torsion Abdomen
Rectal Stricture |
|
|
Constipation
|
Gastric Ulcers Subclinical Stage 0 to 2 Fecal Balls Melena Possible
|
|
|
Death Acute
|
Gastric Ulcers Acute Stage 3 Hemorrhage Intragastric
Swine Dysentery SD Rare without Diarrhea Colibacillosis ETEC Enterotoxic Necroproliferative Enteritis PHE Proliferative Herorrhagic Enteropathy |
|
|
Death Acute Survive for 2 to 3 Days
|
Clostridial Enteritis CPC Die at about 3 Days
|
|
|
Death Chronic Emaciation
|
Gastric Ulcers Chronic Euthanasia
|
|
|
Death Day 14 Plus
|
Rotavirus Uncommon
|
|
|
Death Peracute
|
Colibacillosis ETEC Enterotoxic Before Onset Diarrhea
Post Weaning Colibacillosis No Previous Signs Mesenteric Torsion Non Agonal Swine Dysentery SD Rare without Diarrhea Gastrosplenic Torsion Necroproliferative Enteritis PHE Proliferative Herorrhagic Enteropathy Salmonellosis Septic |
|
|
Death Peracute < 24 Hours
|
Edema Disease ED Otherwise Healthy Pigs
|
|
|
Death Peracute < 36 Hours From Birth
|
Clostridial Enteritis CPC Severe Hemorrhagic Enteritis
|
|
|
Death Rare
|
TGE Transmissible Gastroenteritis Epizootic 3 Weeks Onwards
|
|
|
Dehydration
|
Coccidiosis
Colibacillosis ETEC Enterotoxic Post Weaning Colibacillosis Eyes Sunken Skin Discoloured |
|
|
Diarrhea
|
Clostridial Enteritis CD Sporadic
Clostridial Enteritis CPA Creamy Pasty Clostridial Enteritis CPC Acute Necrotizing Hemorrhagic Enteritis Peracute Clostridial Enteritis CPC Intermittent or Persistent Chronic Clostridial Enteritis CPC Reddish Brown, Necrotic Debris Acute Coccidiosis Day 5 Plus ONLY Coccidiosis Loose Pasty Coccidiosis Week 1 to 3 Typical Suckling Pigs Colibacillosis ETEC Enterotoxic Necroproliferative Enteritis NE Necrotic Enteritis Homogeneous Normal Colour 50% of Cases Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis Homogeneous Normal Colour 50% of Cases Necroproliferative Enteritis RI Regional Ileitis Homogeneous Normal Colour 50% of Cases Post Weaning Colibacillosis Pasty to Watery Rotavirus Day 1 to 7 Recently via Modern Management Rotavirus Day 7 Plus Classically Rotavirus Severity Decreases with Age Rotavirus Severity Increased with Concurrent Escheria coli Salmonellosis Enteric Initially Yellow Watery Becomes Bloody Spirochetal Diarrhea Profuse, Watery, Bloody, Mucoid Severe Cases Only Spirochetal Diarrhea Wet Cement Swine Dysentery SD Frank Blood Possible Swine Dysentery SD Mucofibrinous Exudate Swine Dysentery SD Mucohemorrhagic TGE Transmissible Gastroenteritis Enzootic Osmotic Malabsorptive TGE Transmissible Gastroenteritis Enzootic Transient Days a Few TGE Transmissible Gastroenteritis Epizootic Mild 3 Weeks Onwards TGE Transmissible Gastroenteritis Epizootic Profuse, Watery Odoriferous +/- Curdled Milk Farrowing Swine Dysentery SD Mucus Copious Blood Flecks within 2 to 3 Days |
|
|
Dysentery
|
Necroproliferative Enteritis PHE Proliferative Herorrhagic Enteropathy Possible
Salmonellosis Enteric |
|
|
Dyspnea
|
Clostridial Enteritis CD Edema
|
|
|
Edema
|
Clostridial Enteritis CD Subcutaneous Scrotal
Edema Disease ED Eyelids Face Edema Disease ED Proximal Stomach Wall Characteristic but < 10% |
|
|
Emaciation
|
Rectal Stricture Always Present
|
|
|
Emesis
|
Colibacillosis ETEC Enterotoxic Curdled Milk Severe Cases Only
Gastric Ulcers Subclinical Stage 0 to 2 TGE Transmissible Gastroenteritis Epizootic Transient Gastritis Farrowing |
|
|
Endotoxemia
|
Post Weaning Colibacillosis
|
|
|
Enterocolitis
|
Salmonellosis Enteric Necrotizing Ulcerative
Salmonellosis Septic Small AND Large Intestine |
|
|
Growth Rate Impaired
|
Coccidiosis
Necroproliferative Enteritis NE Necrotic Enteritis Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis Necroproliferative Enteritis RI Regional Ileitis Spirochetal Diarrhea |
|
|
Hair Coat Rough
|
Coccidiosis
|
|
|
Hose Pipe Gut
|
Necroproliferative Enteritis NE Necrotic Enteritis Increasing Involvement of Mucosa and Muscle Layers in Severe Cases
Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis Increasing Involvement of Mucosa and Muscle Layers in Severe Cases Necroproliferative Enteritis RI Regional Ileitis Increasing Involvement of Mucosa and Muscle Layers in Severe Cases |
|
|
Inappetance
|
Gastric Ulcers Subclinical Stage 0 to 2
TGE Transmissible Gastroenteritis Epizootic 3 Weeks Onwards |
|
|
Lethargy
|
Colibacillosis ETEC Enterotoxic Lateral Recumbancy
|
|
|
Melena
|
Necroproliferative Enteritis PHE Proliferative Herorrhagic Enteropathy
|
|
|
Most Common
|
Gastric Ulcers Subclinical Stage 0 to 2 Detected at Slaughter
|
|
|
Necrotic Enteritis
|
Necroproliferative Enteritis NE Necrotic Enteritis Increasing Involvement of Mucosa and Muscle Layers in Severe Cases
Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis Increasing Involvement of Mucosa and Muscle Layers in Severe Cases Necroproliferative Enteritis RI Regional Ileitis Increasing Involvement of Mucosa and Muscle Layers in Severe Cases |
|
|
Neurologic
|
Edema Disease ED Ataxia, Recumbancy, Paddling, Disorientation Cerebral Edema
|
|
|
Often Associated with Other Predisposing Factors
|
Salmonellosis Enteric
|
|
|
Pale
|
Mesenteric Torsion Blood Pools GIT
Necroproliferative Enteritis PHE Proliferative Herorrhagic Enteropathy Anemia Acute Blood Loss Gastric Ulcers Acute Stage 3 Weak Melena Possible |
|
|
Pale Puking Pig
|
Gastric Ulcers All Forms
|
|
|
Pneumonia
|
Salmonellosis Septic
|
|
|
Pseudomembrane
|
Necroproliferative Enteritis NE Necrotic Enteritis Increasing Involvement of Mucosa and Muscle Layers in Severe Cases
Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis Increasing Involvement of Mucosa and Muscle Layers in Severe Cases Necroproliferative Enteritis RI Regional Ileitis Increasing Involvement of Mucosa and Muscle Layers in Severe Cases |
|
|
Rectal Prolapse
|
Mesenteric Torsion Common
|
|
|
Rectal Stricture
|
Salmonellosis Enteric Sequella
Also Prolapse, Tenesmus, Tail Bite, Chronic Cough |
|
|
Regional Ileitis
|
Necroproliferative Enteritis NE Necrotic Enteritis
Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis Necroproliferative Enteritis RI Regional Ileitis |
|
|
Self Limiting
|
Edema Disease ED Spontaneous Recovery Possible
Spirochetal Diarrhea 2 - 14 Days Possible TGE Transmissible Gastroenteritis Epizootic 1 to 2 Days 3 Weeks Onwards |
|
|
Septicemia
|
Salmonellosis Septic
|
|
|
Splenic Torsion Subclinical
|
Gastrosplenic Torsion Incidental Slaughter
|
|
|
Sub Clinical
|
Necroproliferative Enteritis NE Necrotic Enteritis Fecal Shedding Can Start at 3 Weeks Seroconversion ins Sows Increases Risk of PIA in Grow Finish
Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis Fecal Shedding Can Start at 3 Weeks Seroconversion ins Sows Increases Risk of PIA in Grow Finish Necroproliferative Enteritis RI Regional Ileitis Fecal Shedding Can Start at 3 Weeks Seroconversion ins Sows Increases Risk of PIA in Grow Finish |
|
|
Weight Loss
|
Clostridial Enteritis CD Thin Appearance of Backbone
Gastric Ulcers Chronic Esophageal Stricture Necroproliferative Enteritis NE Necrotic Enteritis Necroproliferative Enteritis PIA Porcine Intestinal Ademonatosis Necroproliferative Enteritis RI Regional Ileitis |
|
|
Apramycin
|
Post Weaning Colibacillosis Control Antimicrobial Feed per Culture and Sensitivity
|
|
|
Bacitracin Methylene Disalicylate (BMD)
|
Clostridial Enteritis CPA Control Antimicrobial Feed 2 to 3 Weeks Pre Farrow Sows
Clostridial Enteritis CD Control Antimicrobial Feed 2.5 Weeks Pre Farrow to 3 Weeks Post Farrowing Sow Clostridial Enteritis CPC Control Antimicrobial Feed 2.5 Weeks Pre Farrow to 3 Weeks Post Farrowing Sow |
|
|
Ceftiofur
|
Salmonellosis Septic Treatment Antimicrobial Feed Aggressive Therapy Individuals
Salmonellosis Septic Treatment Antimicrobial Parenteral Aggressive Therapy Individuals Colibacillosis ETEC Enterotoxic Treatment Antimicrobial Parenteral Prefered Salmonellosis Septic Treatment Antimicrobial Water Aggressive Therapy Individuals |
|
|
Gentamycin
|
Colibacillosis ETEC Enterotoxic Treatment Antimicrobial Parenteral Prefered
|
|
|
Lincomycin
|
Swine Dysentery SD Control Antimicrobial Feed Herd Prophylaxis
Necroproliferative Enteritis All Forms Treatment Antimicrobial Feed Herd Treatment Spirochetal Diarrhea Treatment Antimicrobial Parenteral Individual Animals Swine Dysentery SD Treatment Antimicrobial Parenteral Individual Animals Swine Dysentery SD Treatment Antimicrobial Water Herd Outbreaks |
|
|
Neomycin
|
Post Weaning Colibacillosis Treatment Antimicrobial Feed per Culture and Sensitivity
Colibacillosis ETEC Enterotoxic Treatment Antimicrobial Feed Second Choice |
|
|
Salinomycin
|
Swine Dysentery SD Control Antimicrobial Feed Herd Prophylaxis
Swine Dysentery SD Treatment Antimicrobial Water Herd Outbreaks |
|
|
Tetracyclines
|
Necroproliferative Enteritis All Forms Treatment Antimicrobial Feed Herd Treatment
Necroproliferative Enteritis All Forms Control Antimicrobial Feed Pulsed or Continuous Necroproliferative Enteritis All Forms Treatment Antimicrobial Water Herd Treatment |
|
|
Tiamulin
|
Swine Dysentery SD Control Antimicrobial Feed Herd Prophylaxis
Spirochetal Diarrhea Treatment Antimicrobial Parenteral Individual Animals Swine Dysentery SD Treatment Antimicrobial Parenteral Individual Animals Swine Dysentery SD Treatment Antimicrobial Water Herd Outbreaks |
|
|
TMS
|
Salmonellosis Septic Treatment Antimicrobial Feed Aggressive Therapy Individuals
Salmonellosis Septic Treatment Antimicrobial Parenteral Aggressive Therapy Individuals Colibacillosis ETEC Enterotoxic Treatment Antimicrobial Parenteral Prefered Salmonellosis Septic Treatment Antimicrobial Water Aggressive Therapy Individuals Post Weaning Colibacillosis Control Antimicrobial Water per Culture and Sensitivity |
|
|
Tylosin
|
Necroproliferative Enteritis All Forms Treatment Antimicrobial Feed Herd Treatment
Necroproliferative Enteritis All Forms Control Antimicrobial Feed Pulsed or Continuous Swine Dysentery SD Control Antimicrobial Feed Herd Prophylaxis Spirochetal Diarrhea Treatment Antimicrobial Parenteral Individual Animals Swine Dysentery SD Treatment Antimicrobial Parenteral Individual Animals Swine Dysentery SD Treatment Antimicrobial Water Herd Outbreaks |
|
|
Virginiamycin
|
Swine Dysentery SD Control Antimicrobial Feed Herd Prophylaxis
Swine Dysentery SD Treatment Antimicrobial Water Herd Outbreaks |
