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101 Cards in this Set

  • Front
  • Back

Sensory receptors

specialized neuron that detects a particular category of physical events

*Sensory transduction

process by which sensory stimuli are transduced into slow, graded receptor potentials

Receptor potential

slow, graded electrical potential produced by a receptor cell in response to a physical stimulus

Vision stimulus

eyes detect the presence of light; electromagnetic radiation wavelength 380-760 nm is visible to humans

Wavelength

distance between waves; perception of color

Intensity

how strong is the wave; perception of brightness

*Hue

dominant wavelength, longer = more red, shorter= more blue

*Brightness

intensity; more intense= brighter color

*Saturation

purity; how many wavelengths are mixed together (e.g. all wavelengths= no hue sensation, appears white)

3 types of eye movements

vergence movement, saccadic movement, pursuit movement

Vergence movement

cooperative movement of the eyes; ensures that image of an object falls on identical portions of both retinas

Saccadic movements

rapid, jerky movements of the eyes used in scanning a visual scene

Pursuit movement

the movement the eyes make to maintain an image of a moving object on the fovea

Sclera

white outer coat of the eye; does not permit in light

Orbits

bony pockets in front of skull

Conjunctiva

mucous membranes that line the eye and fold back to attach the eye

Extraocular muscles

movement of the eyes

Iris

regulates amount of light that reaches the retina; contractile tissue, donut shaped, give eyes color

*Pupil

how light enters the eye; hole in iris, size responds to changes in illumination, compromise between sensitivity (detect presence of dimly lit objects) and acuity (ability to see details of objects)

*High illumination

sensitivity is not important, pupils constrict, sharper image, greater depth of focus

*Low illumination

receptors are activated successfully, pupils dilate to let more light in, worse acuity and depth of focus

Lens

behind the pupil, focuses incoming light on the retina, direct attention to something near

Ciliary muscles

contract- reduce tension on ligaments holding lens, helps the lens refract (bend) light, brings close objects into focus

Accommodation

process of bringing images into focus on the retina

Convergence

eyes move so that a point is projected to corresponding points on both retinas

Binocular (retinal) disparity

the difference in position of the same image on the 2 retinas, cocktail sausage illusion

Vision process

light passes through lens, goes through vitreous humor (clear glassy liquid), falls on retina

*Retina

interior lining of back of the eye, location of photoreceptors (rods and cones)

*Macula

center of retina

*Fovea

central portion of macula; region for acute vision, only contains cones (equal number of ganglion cells and cones); central vision

*Cones

6 million in retina, day time vision, small features, high acuity, color vision

Visual distortion

see wavy words, letters are distorted as in a funhouse mirror= damage to retina

*Come and go vision

small areas of vision loss that come and go= macular degeneration

Better night vision

rod cells are unaffected by macular degeneration, located in the peripheral retina and used in lower levels of light

Peripheral vision sensitivity

can see a speck of dirt on the floor, cannot recognize faces, damage to central retina impairs vision straight ahead but can use peripheral vision

*Rods

120 million in retina, more sensitive to light, used in dim light, color blind and lack foveal vision in dim light

Optic disc

location of exit point from retina of fibers of ganglion cells that form the optic nerve; responsible for the blind spot, no receptors there

*Blindspot

gap in the receptor layer where axons leave the eye

Completion (vision)

visual system uses info from receptors to fill in gaps in retina

Layers of retina

photoreceptors form synapses with bipolar cells; bipolar cells form synapses with ganglion cells, ganglion cells axons go through optic nerve to brain

Horizontal and Amacrine cells

combine messages from photoreceptors, responsible for lateral communication

Transduction

photopigments responsible for transduction, special molecules in photoreceptor lamellae (membrane)- Opsin (protein), retinal (lipid derived from vitamin A)

Rhodopsin

found in rods, pinkish hue, exposure to light breaks it into two component parts: rod (red) opsin and retinal, splitting causes change in membrane potential of photoreceptor, changes release rate of glutamate

Hyperpolarization of photoreceptor

-> depolarization of bipolar cells -> excitation of ganglion cells

Dorsal lateral geniculate nucleus (LGN)

thalamus; contains 6 layers of neurons, receives input from only one eye, cell bodies of inner 2 layers are larger than outer 4 layers

*Magnocellular layers

inner two layers of LGN (rods)

*Parvocellular layers

outer 4 layers in LGN (cones)

Koniocellular sublayers

ventral to each of the magno and parvo layers

Optic nerve

nerves join at base of brain to form optic chiasm

*Optic chiasm

x-shaped, joining of optic nerves

*Vision through optic chiasm

ganglion cell axons from inner half of retina (nasal), cross through the chiasm, ascend to contralateral LGN, ganglion cell axons from outer half of reina (temporal) remain on ipsilateral side= info from both eyes goes to both sides of the brain; image of the world on the retina is inverted

Meyers loop

axons from LGN go to primary visual cortex via optic radiations

*Primary visual cortex

V1; region surrounding the calcarine (spur shaped) fissue; also called striate cortex bc of dark striations (layers of cells); nerve fibers carry info from V1 to extrastriate areas (V2, V3, V4, and V5, MT (middle temporal))

*Blindsight

after damage to V1, some patients have the ability to respond to visual stimuli while reporting not seeing (e.g. person who is blind can walk through a room and avoid objects in the way, but can't report seeing them)

*Receptive field

portion of the visual field in which the presentation of visual stimuli will not produce an alteration in the firing rate of a particular neuron; part of visual field a neuron 'sees'; use microelectrodes to investigage individual neurons in receptive field; if neuron receives info in fovea= fixation point, periphery of retina= off to one side

Periphery of retina

many receptors converge on a simple ganglion cell, brings info from large area of retina and large part of visual field; peripheral vision not very precise

*3 types of ganglion cells

(Hartline, 1938, frog retina)


On cells- excitatory, burst when retina illuminated


Off cells- respond when light is off


On/Off cells- respond when light goes on and again when light goes off

*Form of ganglion cells

(Kuffler, 1952-3, cat retina)


receptive field is a circle surrounded by a ring


On cells- excited by light in center, inhibited by light in surround


Off cells- opposite


On/Off cells- briefly excited when light turned on or off

Rebound effect

neurons that are inhibited when light is on, have burst of excitement when light goes off and vice versa

Center surround

good for seeing edges (contrasts), detect outlines even when contrast between items is low; visual system adds borders

Additive effects of color mixing

mixing colors of light= white; mixing colors of paint= brown/gray

Photopigment

special molecule embedded in lamallae, 2 parts: opsin and retinal; different opsins absorb different wavelengths

*Photoreceptors for cones

3 types: blue, green, red; based on amount of light absorbed by photopigment

Genetic defects in color vision

result from problems in one or more of the 3 types of cones; 2 involve genes on the x chromosome= more common in men, females have a normal gene on the other x chromosome to compensate

Protanopia

red and green hues confused; see world in shades of blue and yellow; red and green appear yellowish; visual acuity is normal- correct number of cones, red cones are filled with green cone opsin

Deuteranopia

red and green confused; visual acuity is normal, correct number of cones; green cones filled with red opsin

Tritanopia

not on x chromosome ( = likely for male and female)


rate, hues with short wavelengths are confused, see world in reds and greens; clear blue sky is bright green, yellow looks pink; blue cones lacking or faulty

Negative afterimage

image seen after a portion of retina is exposed to an intense visual stimulus, consists of colors complementary to those of the physical stimulus

Complimentary colors

colors that make white or grey when mixed together (e.g. red/green, yellow/violet, blue/orange)

Striate cortex

V1, contains dark staining layer, 6 layers, contain nuclei of cell bodies and dendritic trees; 25% analyzes foveal info, neurons in visual cortex respond selectively to specific features

Hubel & Weisel

Nobel prize winners; described neurons in V1, studied in cats

*Simple cell

receptive field is organized in an opponent fashion; respond best to bars of light of a particular orientation

*Complex cell

responds to the presence of a line segment with a particular orientation located within its receptive field, especially when line moves perpendicular to its orientation; detects movement

*Hypercomplex cell

responds to the presence of a line segment with a particular orientation that ends at a particular point within the cells receptive field

Sine wave grating

a series of straight parallel bands varying continuously in brightness according to a sine wave function, along a line perpendicular to their lengths; fuzzy, unfocused parallel bars

Spatial frequency

the relative width of the bands in a sine wave grating

*Neurons in V1

respond to orientation, movement, spatial frequency, retinal disparity, color

Visual association cortex

where info is received, combined, and analyzed; receives fibers from striate cortex and the superior colliculi-> projects to the inferior temporal cortex

*Dorsal stream (vision)

system of interconnected regions of visual cortex involved in perception of spatial location; begins in striate cortex, ends in posterior parietal cortex; "where" pathway, guides action

*Ventral stream (vision)

System of interconnected regions of visual cortex involved in perception of form, begins in striate cortex, ends in inferior temporal cortex; "what" pathway

Parvo and Konio cellular system

only found in primates

*Parvocellular system

receives info about wavelength from cones- analyze color; high spatial resolution, low temporal resolution; detect fine details but response is slow and prolonged

*Magnocellular system

found in all mammals; color-blind, does not detect fine detail, can detect small differences between light and dark, sensitive to movement

*V4

Zeki (1980); responds to color; regulates color constancy: relatively constant appearance of colors of objects viewed under varying light conditions, visual system compensates for source of light, not strictly a response based on the wavelength of the light reflected

Cerebral achromatopsia

inability to discriminate among different hues; caused by damage to area V8

Visual agnosia

deficits in visual perception in the absence of blindness; caused by brain damage

Apperceptive visual agnosia

failure to perceive objects, failure in high level perception, visual acuity is relatively normal (e.g. can't identify object without touching it); cannot copy

Associative visual agnosia

inability to identify objects that are perceived visually, disconnect between the perceptions and visual systems, form of perceived object can be drawn or matched with similar objects (can be copied)

Prosopagnosia

failure to recognize particular people by the site of their faces

Object agnosia

failure to recognize objects

*Fusiform face area

FFA; region of visual association cortex located in inferior temporal cortex (ventral stream); involved in perception of faces and other complex objects that require expertise to recognize

Extrastriate body area

EBA; region of visual association cortex located in lateral occipitotemporal cortex; involved in perception of the human body and body parts other than faces

Parahippocampal place area

PPA; region of medial temporal cortex; involved in perception of particular places (scenes)

*Experience dependent plasticity

integration of environmental stimuli into the normal patterns of development- more activity to stimuli have more experience with

V5 (MT)

responds to movement; receives input directly from striate cortex and several regions of extrastriate cortex; neurons in V5 respond much faster than those in V4

Akinetopsia

inability to perceive movement, caused by damage to area V5 (also called MST) of visual association cortex

Optic ataxia

difficulty in reaching for objects under visual guidance; can see it and recognize it but can't reach it

Ocular apraxia

inability to fixate on certain points in the peripheral visual field despite intact eye movements

Simultanagnosia

difficulty in perceiving more than one object at a time; even if you cross two items, subject only sees one

Balint's syndrome

syndrome caused by damage to parieto-occipital region (i.e. dorsal stream), includes optic ataxia, ocular apraxia, and simultanagnosia

Intraparietal sulcus

IPS; end of the dorsal stream of the visual association cortex, involved in perception of location, visual attention, control of eye and hand movements