Vet Gen Path Cell Injury

Front 1

1.     Which of the following statements about cellular response are true; (P6,7)
A. Cells capable of division can respond to stress by hyperplasia or hypertrophy.
B. The increase in tissue mass due to hyperplasia after cell loss is caused only by proliferation of the remaining cells, but not by the development of stem cells.
C. Physiologic hyperplasia which increases the functional capacity of a tissue when needed is mentioned to the compensatory hyperplasia.
D. Hypertrophy takes place if the cellular population is capable of synthesizing DNA.
E. The increased cell size in hypertrophy is due to cellular swelling.

Back 1

(1:A)

Front 2

2. All of the following statements about cardiac muscle hypertrophy are true EXCEPT: (P7,8)
A. Both mechanical triggers and trophic triggers induce hypertrophy.
B. Both cardiac and skeletal _-actin increase muscle activity.
C. The _-myosin heavy chain is replaced by the _-myosin heavy chain, which leads to decreased myosin ATPase activity.
D. Ventricular ANF gene, which is down-regulated after birth, is reinduced.
E. Reinduced ANF serves to increase hemodynamic load.

Back 2

(2:E)

Front 3

3. All of the following statements about metaplasia are true EXCEPT: (P10,11)
A. Retinoic acid deficiency induces squamous metaplasia in the respiratory epithelium.
B. Refluxed gastric acid cause replacement of intestinal-like columnar cells by squamous epithelial cells.
C. Metaplasia is the result of a reprogramming of stem cells present in normal tissue.
D. Metaplasia is the result of a reprogramming of undifferentiated mesenchymal cells present in connective tissue.
E. Bone morphogenetic proteins induce chondrogenic or osteogenic expression in stem cells.

Back 3

(3:B)

Front 4

4. All of the following statements about cellular injury are true EXCEPT: (P11~15)
A. Reduced oxidative phosphorylation, ATP depletion, and cellular swelling due to ion concentration changes are all hallmarks of reversible cell injury.
B. Ischemic tissues are injured more rapidly than are hypoxic tissues.
C. Liver is the most susceptible (easily injured) to the ATP decrease by hypoxia.
D. Decreased ATP causes cellular influx of Na+ and efflux K+.
E. Morphologic consequence due to decreased pH is a clumping of nuclear chromatin.

Back 4

(4:C)

Front 5

5. Which of the following statement about oxidative stress is true?
(P16,17)
A. Inflammation-induced xanthine oxidase breaks down hydrogen peroxide.
B. Cu catalyzes free radical formation.
C. Most of the intracellular free iron is in the ferric state, which can catalyze hydroxyl ion production.
D. NO which is generate in inflammation act as a free radical by itself, and can be converted to non-reactive peroxynitrite anion.
E. Free radicals promote reduction of the protein backbone, resulting in protein fragmentation.

Back 5

(5:B)

Front 6

6. Which of the followings work as free radical scavenger? (P17)
1. Vitamin A
2. Vitamin C
3. Vitamin E
4. Glutathione reductase
5. Xanthine oxidase

A. 1,3
B. 1,2,3
C. 1,3,4
D. 1,2,3,5
E. All of above

Back 6

(6:B)

Front 7

7. Which of the following antioxidant enzymes are located in mitochondria? (P17,18)
1. Catalase
2. Manganese-superoxide dismutase
3. Cu-Zn-superoxide dismutase
4. Glutathione peroxidase

A. 1,2
B. 1,3
C. 2,3
D. 2,4
E. 3,4

Back 7

(7:D)

Front 8

8. Which of the following type of necrosis is most frequently seen by the hypoxic cell death within central nervous system?
(P21,22)
A. Coagulative necrosis
B. Liquefactive necrosis
C. Gangrenous necrosis
D. Caseous necrosis
E. Fat necrosis

Back 8

(8:B)

Front 9

9. Which of the following statement about cellular injury is true?
(P24, 25)
A. Ischemia-reperfusion injury occurs after resume of blood supply only when cells are irreversibly injured.
B. Some IgM antibodies prone to deposit in ischemic tissue, resulting ischemia-reperfusion injury after resume of blood supply.
C. Carbon tetrachloride (CCl4) act directly on target cells.
D. CCl4-induced injury is severe, but extremely slow in onset because this reaction have to pass through lipid peroxidation by produced free radical.
E. Acetaminophen is detoxified by cytochrome P-450.

Back 9

(9:B)

Front 10

10. Which of the following is correct order of CCl4-induced fatty liver; (P25)
1. Free radical production
2. Decreased apoprotein synthesis
3. Polysome detachment
4. Membrane damage to RER
5. Lipid peroxidation

A. 1→2→4→5→3
B. 1→5→2→4→3
C. 1→5→4→3→2
D. 2→3→1→5→4
E. 2→4→1→5→3

Back 10

(10:C)

Front 11

11. In apoptotic cells, endonucleases cleave DNA depending on; (P27)
1. Copper
2. Zinc
3. Magnesium
4. Calcium

A. 1,2
B. 1,3
C. 2,3
D. 2,4
E. 3,4

Back 11

(11:E)

Front 12

12. Which of the following factor bridge between cell-surface death receptor and mitochondria (; bridge between extrinsic and intrinsic pathway); (J of Vet Path)
A. FADD
B. Bid
C. PTPC
D. Caspase 8
E. Ced

Back 12

(12:B)

Front 13

13. Apoptotic cells are identified by special dye, Annexin V, binding to which of the following in their outer layer of membrane? (P27)
1. Phosphatidylserine
2. Thrombospondin
3. CD31

A. 1
B. 2
C. 3
D. 1,2
E. 1,2,3

Back 13

(13:A)

Front 14

14. Which of the following statements about lysosomal catabolism are wrong? (P32)
1. Lysosomal enzymes are synthesized in the Golgi apparatus and transferred to ER.
2. Phagolysosomes are formed in heterophagy, but not in autophagy.
3. In most of acquired lysosomal diseases, decreased internal pH of the lysosome result in inactivation of enzymes.

A. 1
B. 2
C. 3
D. 1,2
E. All of above

Back 14

(14:E)

Front 15

15. Which of the following cytoskeletal constituent is essential for cell division; (P34)
A. Actin
B. Myosin
C. Microtubules
D. Clathrin
E. Keratin

Back 15

(15:C)

Front 16

16. Which of the following statement about subcellular response is true; (P37-39)
A. Xanthoma is the visceral or subcutaneous benign neoplasia of adipocytes.
B. Russell body is the eosinophilic large distended lysosome due to excessively produced immunoglobulins.
C. In protein loosing renal diseases, reabsorption droplets of protein are seen in proximal renal tubles.
D. Hyaline changes happens only intracellularlly, but not extracellularlly.
E. Lipofuscin is the sign of free radical injury or lipid peroxidation, and injurious to the cell.

Back 16

(16:C)

Front 17

17. Which of the following statement about chaperon or ubiquitin is true; (P37,38, NFJM 2005)
A. Ubiquitin aid both in proper folding and transporting of protein.
B. In cystic fibrosis, mutation delays dissociation of a sodium channel protein from its chaperon, resulting in the increase of partially folded intermediates.
C. Ubiquitin marks for degradation by proteasome complex in lysosome.
D. The activation of the unfolded protein response leads to cell death by activating death receptor.
E. Ubiquitin is covalently linked to the target protein by ubiquitin ligase.

Back 17

(17:E)

Front 18

1. All the following statements regarding lysosomes are correct EXCEPT:
A) Small membrane-bound vesicles
B) Hydrolytic enzymes
C) Enzymes essential for intracellular digestion
D) Similar to peroxisomes
E) Play a role in energy metabolism

Back 18

(1:E p.8, Peroxisomes also play a role in energy metabolism, but lysosomes do not.)

Front 19

2. Diameter of the intermediate filaments are about:
A) 0.01 nm
B) 0.1 nm
C) 1.0 nm
D) 10.0 nm
E) 100.0 nm

Back 19

(2:D p.8)

Front 20

3. Extracellular matrix includes:
a. Proteoglycans
b. Adhesion glycoproteins
c. Collagens
d. Basement membranes

A) None
B) a
C) a, b
D) a, b, c
E) a, b, c, d

Back 20

(3:E p.8)

Front 21

4. All the following are general mechanisms of cell injury EXCEPT:
A) ATP depletions
B) Membrane damage
C) Decreased intracellular Ca2+
D) Distribution of cellular metabolism
E) Genetic damage

Back 21

(4:C p.9, 10 Fig. 1-6, Increased intracellular Ca2+)

Front 22

5. Ischemia causes in cells:
A) Increased both glycogen and pH
B) Increased glycogen and decreased pH
C) Decreased glycogen and increased pH
D) Decreased both glycogen and pH
E) No change either glycogen or pH

Back 22

(5:D p.11 Fig. 1-8; ATP depletions → increased glycolysis → decreased both glycogen and pH)

Front 23

6. All are characteristic cytomorphologic changes of irreversible cell injury EXCEPT:
A) Disruption of cell membranes
B) Nuclear condensation
C) Swelling of endoplasmic reticulum and detachment of ribosomes
D) Swollen mitochondria with amorphous densities
E) Lysosomal rupture

Back 23

(6:C p.9, p.16 Fig. 1-13; Detachment of ribosomes from ER and chromatin clumping are reversible; myelin figures first appear during the reversible stage)

Front 24

7. Cyanide toxicosis cause hypoxia due to:
A) Heart failure
B) Respiratory failure
C) Reduction of blood supply
D) Reduced transport of O2 in blood
E) Blockage of cell respiratory enzymes

Back 24

(7:A p.10; Reduced transport of O2 in blood are results of anemia / carbon monoxide toxicity)

Front 25

8. Na+-K+ ion pumps within cell membranes move:
A) 3Na+ into the cell and 2K+ out of the cell
B) 2Na+ into the cell and 3K+ out of the cell
C) 2Na+ into the cell and 2K+ out of the cell
D) 2Na+ out the cell and 3K+ into the cell
E) 3Na+ out the cell and 2K+ into the cell

Back 25

(8:E p.12)

Front 26

9. All the following cells highly vulnerable to hypoxia and cell swelling EXCEPT:
A) Oligodendrocytes
B) Endothelial cells
C) Cardiac myocytes
D) Distal renal tubular epithelial cells
E) Hepatocytes

Back 26

(9:D p.15; Proximal renal tubular epithelial cells; In CNS, also neurons and astrocytes)

Front 27

10. All the following are features of apoptosis EXCEPT:
A) Nuclear chromatin condensation
B) Nuclear chromatin fragmentation
C) Cytoplasmic budding
D) Cytoplasmic blebs
E) Phagocytosis

Back 27

(10:D p.17 Fig. 1-14; Necrosis/oncosis: cytoplasmic blebs & digestion and leakage of cellular components)

Front 28

11. Increased cytosolic Ca+ due to cell injury cause all of the following EXCEPT:
A) Activation of phospholipase A resulting in membrane damage
B) Activation of proteases resulting cytoskeleton and membrane damage
C) Activation of ADP phosphatase resulting in increased ATP
D) Activation of endonucleases resulting in chromatin degradation
E) Activation of phospholipase resulting in generation of arachidonic acid

Back 28

(11:C p.18; Activation of ATPase resulting in decreased ATP)

Front 29

12. All of the following are intracellular antioxidants that control free radical injury EXCEPT:
A) Superoxide dismutase (SOD)
B) Vitamin C
C) Ferritin
D) Ceruloplasmin
E) Cytochrome p450

Back 29

(12:E p.19, 20; Also glutathione peroxidase, vitamins E and A, β-carotene, and catalase)

Front 30

13. Fenton reaction is:
A) H2O2 + Fe3+ → OH + OH- + Fe2+
B) H2O2 + Fe2+ → OH + OH- + Fe3+
C) OH + OH- + Fe2+ → H2O2 + Fe3+
D) OH + OH- + Fe3+ → H2O2 + Fe2+
E) H2O2 + Fe2+ → H2O + O- + Fe3+

Back 30

(13:B p.19 Fig. 1-16)

Front 31

14. Match the following postmortem changes:
A) Rigor mortis
B) Algor mortis
C) Livor mortis
a. Gradual cooling of the cadaver
b. Contraction of muscles occurring after death
c. Gravitational pooling of blood to the down side of the animal

Back 31

(14: A-b; B-a; C-c = hypostatic congestion, p.26)

Front 32

15. Pseudomelanosis is the green discoloration by what?
A) Hemoglobin
B) Bile
C) Hydrogen sulfide (H2S)
D) Iron sulfide (FeS)
E) Melanin

Back 32

(15:D p.27)

Front 33

16. Which followings are associated with activation of the extrinsic pathway of apoptosis?
a. FasL
b. CD95
c. FLIP
d. TNFR1
e. FADD
f. Caspase-8
g. Caspase-9

A) a, b, d, e, f
B) a, b, d, e, g
C) a, c, d, e, g
D) b, c, d, e, f
E) All

Back 33

(16:A p.30; CD95 = Fas; FLIP inhibitor of the extrinsic pathway by binding caspase-8)

Front 34

17. Which followings are proapoptotic?
a. Bim
b. Bcl-2
c. Bax
d. Bcl-x
e. Bak

A) a, b, c
B) a, c, d
C) a, c. e
D) c, d, e
E) a, c, d, e

Back 34

(17:C p.30)

Front 35

18. In diabetes, glycogen is found which following cells?
a. Hepatocytes
b. Epithelial cells of renal proximal tubules
c. B cells of the renal Islets of Langerhans
d. Myocytes
e. Neurons

A) a
B) a, b
C) b, c
D) a, b, c
E) All

Back 35

(18:D p.42)

Front 36

19. Amyloid is:
A) 0.075- to 0.100- nm filaments
B) 0.75- to 1.00-nm filaments
C) 7.5- to 10.0-nm filaments
D) 75- to 100-nm filaments
E) 750- to 1000-nm filaments

Back 36

(19:C p.45)

Front 37

20. Match regarding amyloid:
A) AL amyloid
B) AA amyloid
C) Hereditary amyloidosis
D) β-amyloid
a. In the brains of aged dog
b. In Shar-Pei dogs and in Abyssinian

c. SAA from liver
d. Response to IL-1 and IL-6
e. Immunoglobulin light chains (λ- and κ-light chains)

Back 37

(20: A-e; B-c,d; C-b; D-a, p.45-46)

Front 38

21. All followings are correct regarding to gout in birds EXCEPT:
A) Uric acid and urates are the end products of purine metabolism
B) Uric acid and urates are eliminated as semisolid urates
C) Hypervitaminosis A can be a cause
D) High-protein diets can be a cause
E) Renal injury can be a cause

Back 38

(21:C p.47, vitamin A deficiency)

Front 39

22. All followings are correct regarding pseudogout EXCEPT:
A) Deposits of sodium urate crystals
B) Recognized in humans and in dogs
C) In humans, it is inherited as an autosomal dominant trait
D) Grossly, chalky white deposits in joints
E) Histologically, chronic reaction

Back 39

(22:A p.47, Deposits of calcium pyrophosphate crystals)

Front 40

23. All followings are correct regarding calcification in or under the skin EXCEPT:
A) Calcinosis cutis occur in dogs with hyperadrenocorticism
B) Calcinosis cutis has a preference for German shepherds and Great Danes
C) In calcinosis cutis, there is mineralization of dermal collagen, and epidermal and follicular basement membranes
D) Calcinosis circumscripta is considered to be dystrophic
E) Calcinosis circumscripta can be associated with repetitive trauma and at the site of buried sutures of polydioxanone

Back 40

(23:B p.48, Calcinosis circumscripta is familial in German shepherds and Great Danes)

Front 41

24. All of followings are causes of metastatic calcification EXCEPT:
A) Renal failure
B) Vitamin D toxicosis by ingestion of Cestrum diurnum
C) Vitamin E/selenium deficiency
D) Parathormone (PTH) and PTH-related protein associated with canine malignant lymphomas and canine adenocarcinoma of the apocrine glands of the anal sac
E) Destruction of bone from primary or metastatic neoplasms

Back 41

(24:C, dystrophic calcification)

Front 42

25. All of followings are correct regarding exogenous pigments EXCEPT:
A) Anthracosis is a subtype of pneumoconiosis
B) Carotenoid pigments are also called lipochrome pigments
C) Carotenoid pigments are fat-soluble pigments of plant origin and include the precursors of vitamin A, namely β-carotene
D) Yellow to orange-yellow fat discoloration by carotenoid pigments are commonly seen in Holstein cattle, sheep, goats, and cats
E) Tetracycline-based antibiotics administered during development of teeth and bone may cause yellow or brown discoloration

Back 42

(25:D p.50, Fat discoloration with carotenoid pigments is common in horses, and Jersey and Guernsey cattle, and sometimes dogs; Holstein cattle, sheep, goats, and cats store little or no carotenoids and have white fat and clear serum)

Front 43

26. All followings are correct regarding melanin EXCEPT:
A) Melanin is formed by the oxidation of tyrosine
B) Tyrosinase is a zinc-containing enzyme
C) Histologically the melanocytes appear normal in albino animals
D) Hyperpigmentation of skin is associated with chronic injury, endocrinopathies (hyperadrenalism), and in melanocyte neoplasms
E) Congenital melanosis occurs in the lungs and aorta (intima) of cattle, sheep, and pigs

Back 43

(26:B p.51, Tyrosinase is a copper-containing enzyme)

Front 44

27. All followings are correct regarding lipofuscin and ceroid EXCEPT:
A) Lipofuscin is a clear indicator of age of the cell, and ceroid is a pathological pigment, often associated with vitamin E deficiency
B) Lectin-binding histochemistry has shown differences between lipofuscin and ceroid from rats and humans
C) Intestinal lipofuscinosis is ceroid deposition in small intestine of dogs
D) Both lipofuscin and ceroid deposit intracellularly in lysosomes of postmitotic and stable cells
E) Lipofuscin deposition initially occurs infancy, but ceroid deposition occur anytime

Back 44

(27:D p.53, Only lipofuscin; Lipofuscin deposits only intracellular, but ceroid deposits both intracelluolar and extracellular)

Front 45

28. All followings are correct regarding hematogenous pigments EXCEPT:
A) Unoxygenated hemoglobin, hemosiderin, and bilirubin are produced normally but can accumulate excessively
B) Methemoglobin, carboxyhemoglobin, and hematin are pathological
C) In acute cyanide poisoning, cyanide binds to cytochrome oxidase, the enzyme in cell responsible for oxidative phosphorylation, and this results in paralysis of cellular respiration, and color of venous blood will be bright red
D) In carbon monoxide (CO) poisoning, the blood is a bright cherry red from the formation of carboxyhemoglobin. In nitrate poisoning, the blood and tissues are reddish-brown (chocolate brown) from the formation of methemoglobin, an oxide of hemoglobin.
E) Hematins, including formalin pigments and the excreta of parasites, such as Fascioloides magna (liver fluke) and Pneumonyssus simicola (lung mite), are positive for iron.

Back 45

(28:E p.55, Formalin pigments are negative for iron when stained by Prussian blue reaction)

Front 46

29. All followings are correct regarding Prussian blue reaction EXCEPT:
A) It is used to confirm iron
B) It is a chemical reaction
C) The end product is Prussian blue
D) Acid solution liberates ferric iron from hemosiderin
E) Ferric iron is reacted with oxygenerated ferrocyanide (colorless) to form ferric ferrocyanide

Back 46

(29:E p.56, Potassium ferrocyanide)

Front 47

1) All of the following are true about fibronectin, EXCEPT
A) Attachment cells to the connective tissue
B) Wound healing
C) Opsonized material for phagocytosis
D) Related to neoplastic formation
E) One of the structural fibrous protein

Back 47

(1:E p.21; Robbins p.51 Fibronectin is adhesive glycoprotein. Structure fiber proteins are collagen and elastin.)

Front 48

2) Which caspase is activated by granzyme B?
A) Caspase 2
B) Caspase 3
C) Caspase 8
D) Caspase 9
E) Caspase 10

Back 48

(2:E p.42)

Front 49

3) Which one is most important of the executioner caspase?
A) Caspase 2
B) Caspase 3
C) Caspase 8
D) Caspase 9
E) Caspase 10

Back 49

(3:B p.42-43)

Front 50

4) Which one is activated by caspase 3?
A) Caspase 8
B) c-myc
C) CAD
D) BAX
E) Apaf-1

Back 50

(4: p.42, CAD: caspase-activated deoxyribonuclease)

Front 51

5) Cytochrome P-450 located in
A) Cytoplasmic constituents
B) Peroxisomes
C) Plasma membrane
D) Endoplasmic reticulum
E) Mitochondria

Back 51

(5:D p.57)

Front 52

6) In Wilson’s disease in human and in a similar disease in some dog breeds, where is abnormal copper stored in the hepatic cells?
A) Lysosome
B) Peroxisomes
C) Plasma membrane
D) Endoplasmic reticulum
E) Mitochondria

Back 52

(6:A p.58)

Front 53

7) Selenium deficiency of cattle can cause muscle and myocardial necrosis, whereas Vit E deficiency in horse?
A) Muscle necrosis
B) Myocardial necrosis
C) Mortor neuron disease
D) Glycogen storage disease
E) Hepatic necrosis

Back 53

(7:C p.58)

Front 54

8) Xanthin dehydrogenase acts on hypoxanthine to generate oxygen-derived free radicals, where this enzyme locates?
A) Capillary endothelium
B) Myocytes
C) Hepatocyte
D) Platelets
E) Macrophages

Back 54

(8:A p.60)

Front 55

9) Which breed of dogs occur familial form of amyloidosis?
A) Shar-pei
B) German shepherd
C) Basenji
D) Great Danes
E) Siberian husky

Back 55

(9:A p.73, Familial forms of amyloidosis occur in the shar-pei dog and the Abyssinian cat.)

Front 56

10) Amyloid contains component other than the fibril protein, CHOOSE THREE
A) glycosaminoglycan
B) amyloid P component
C) apolipoprotein E
D) apolipoprotein B
E) fibronectine

Back 56

(10:A,B,C p.73)

Front 57

11) Which of the following are found in the inner membrane of the mitochondria?
1. Monoamine oxidase
2. Nucleoside diphosphokinase
3. ATP synthetase
4. Succinate dehydrogenase
5. Citrate synthetase

A) 1, 2
B) 2, 3
C) 3, 4
D) 4, 5
E) 3, 5

Back 57

(11:C p.22 Table 2-1, 1:outer membrane; 2:intermembranou compartment; 5:matrix)

Front 58

12) Lipofuscin represents accumulation in:
A) Primary lysosomes
B) Secondary lysosomes
C) Residual bodies
D) Mitochondria
E) Endoplasmic reticulum

Back 58

(12:C p.25)

Front 59

13) The protein found in coated pits is:
A) Actin
B) Tubulin
C) Desmin
D) Laminin
E) Clathrin

Back 59

(13:E p26)

Front 60

14) Directional movement of a cell primarily involves:
A) Actin
B) Tubulin
C) Desmin
D) Laminin
E) Clathrin

Back 60

(14:B p.27)

Front 61

15) Peroxisomes contain all except:
A) Nucleoid
B) Catalase
C) Superoxide dismutase
D) Urate oxidase
E) D-amino acid oxidase

Back 61

(15:C p.28, Nucleoid - a structure often contained in peroxisomes but not in lysosomes)

Front 62

16. Bcl-2 is the mammalian equivalent of which C. elegans apoptosis inhibitor:
A) CED-3
B) CED-4
C) CED-8
D) CED-9
E) CED-10

Back 62

(16:D p.41, Bcl-2 = CED-9; Apaf-1 = CED-4)

Front 63

17) Which of the following associates with cytochrome c and Apaf-1 to form the “apoptosome”:
A) Caspase-3
B) Caspase-4
C) Caspase-8
D) Caspase-9
E) Caspase-10

Back 63

(17:D p.43)

Front 64

18) Which of the following favor apoptosis:
1. Caspase-9
2. AIF
3. Puma
4. Bax
5. Cyclosporins

A) 1, 2, 3
B) 2, 3, 4
C) 1, 2, 3, 4
D) 2, 3, 4, 5
E) 1, 2, 3, 4, 5

Back 64

(18:C, p.44, AIF: apoptosis-inducing factor; p. 250, Cyclosporin: drug which blocks production of IL-2)

Front 65

19) Lipid peroxidation reactions are catalyzed by (2 answers):
A) Calcium
B) Copper
C) Iron
D) Magnesium

Back 65

(19:B,C p.58)

Front 66

20) Briefly, what is the pathogenesis of lipid accumulation in CCl4 liver toxicity?

Back 66

(p.59, Lipid peroxidation → RER membrane damage → decreased protein synthesis → decreased apolipoprotein necessary to transport triglyceride from the hepatocyte → intracellular lipid accumulation)

Front 67

21) Xanthine oxidase, a potentially important source of oxygen-derived free radicals, is not present in which species:
A) Cat
B) Cattle
C) Dog
D) Rabbit
E) Rat

Back 67

(21:D p.59)

Front 68

22) What is the name of an acidic membrane phospholipid that serves as a potential Ca-binding site and is also a receptor aiding in phagocytosis of apoptotic cells?

Back 68

(p.69, Phosphatidylserine)