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18 Cards in this Set

  • Front
  • Back
Peripheral vertigo
CN VIII or vestibular apparatus affected
Central vertigo
Brain stem [vestibular nuclei in medulla and lower pons] and cerebellum affected
Peripheral vertigo: differentiating features
(1) Sudden onset; (2) Severe intensity; (3) Paroxysmal duration; (4) Frequent nausea; (5) Absent CNS signs; (6) Tinnitus/hearing loss may be present; (7) Nystagmus: torsional/horizontal; (8) Nystagmus is fatigable
Central vertigo: differentiating features
(1) Slow, gradual onset; (2) Intensity is ill-defined; (3) Constant duration; (4) Infrequent nausea; (5) CNS signs usually present; (6) Tinnitus/hearing loss usually present; (7) Nystagmus may be verticular; (8) Nystagmus is non-fatigable
DDx of peripheral vertigo
(1) BPPV; (2) Vestibular neuronitis; (3) Labyrinthitis; (4) Meniere's disease; (5) Head trauma; (6) Drug-induced (aminoglycosides, phenytoin, phenobarb, carbamazepine, salicylates, quinine)
DDx of central vertigo
(1) Cerebellar infarction; (2) Cerebellar hemorrhage; (3) Lateral medullary infarction; (4) Other lower brainstem ischemia; (5) MS
Characteristic Story for BPPV
First, Pt turns their head - after a few seconds of delay, the vertigo occurs; it will resolve within 1 minute if Pt doesn't move, if you turn your head back, the vertigo occurs in the opposite direction
What causes BPPV?
Otoliths become detached from hair cells in utricle; otoliths inappropriately enter the posterior semicircular canal
Pathophysiology of BPPV
"(1) Stop turning head, but otoliths keep moving and drag endolymph. The receptors continue to fire inappropriately and say that the “head is still moving”! The eyes say “head is NOT moving”! The brainsays “room must be spinning in the opposite direction” --> VERTIGO
(Modified) Epley maneuver
Moves otoliths out of the posterior semicircular canal and back into utricle. Must Remain upright for 8-24 hours; complications - can convert to horizontal canal BPPV
Contraindications to Epley Maneuver
(1) Unstable heart disease; (2) High grade carotid stenosis; (3) Severe neck disease; (4) Ongoing CNS disease; (5) Pregnancy beyond 24th week gestation (relative)
Summary of BPPV
(1) Rotatory vertigo (<30 secs) precipitated by head movements; (2) Positive Dix-Hallpike; (3) One treatment is with Epley maneuver. IV phenergan is probably the best ED medication if one is needed
Differentiating BPPV from labyrinthitis and vestibular neuritis (VN)
(1) BPPV requites head movement, labyrinthitis/VN does not; (2) BPPV lasts seconds, labyrinthitis lasts hours/days; (3) BPPV usually in elderly, labyrinthitis can occur at any age; (4) BPPV has no relation to viral syndrome, in labyrinthitis, viral prodrome precedes; (5) BPPV responds to Epley, Labyrinthitis/VN does not
Lab Studies for Vertigo Workup
NONE NEEDED! May get Hgb, D-stick, Electrolytes if prolonged vomiting
Imaging in Posterior Circulatory Cerebrovascular Events
CT is good for cerebellar hemorrhage, but can miss brainstem acute cerebellar infarctions; MRI is much more sensitive than CT for acute infarction with DWI
Acute vertigo and dizziness are associated with what type(s) of strokes?
(1) Lateral medullary infarctions and (2) Cerebellar strokes
Nystagmus due to peripheral causes has all of the following features except: [answer: being horizontal, rotary, or vertical]
Peripheral nystagmus is typically horozonto-rotary, not pure horizontal or rotary...and not vertical.
Nystagmus due to central causes has all of the following features except: [answer: can be dramatically accentuated by head movement]
Vertigo and nystagmus produced by central causes do not significantly worsen with head movement.