• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/24

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

24 Cards in this Set

  • Front
  • Back
  • 3rd side (hint)
MC sites of atherosclerosis
Aortic bifurcation (proximal common iliac arteries)
Adductor (Hunter's)
Carotid bifurcation

Where is the MC place for stenosis of these sites?
Superficial femoral artery (SFA): MC place of stenosis is adductor (HUNTER'S) canal
workup of peripheral arterial occlusive dz
Continuous - wave doppler (duplex U/S)
ABI
CT angiography (CTA - only for candidates of invasive intervention - stent placement

what are results indicative of claudication vs. rest pain/necrosis on ABI
normal: >1
claudication: < 0.8
rest pain/necrosis < 0.4
where would you expect stenosis in a smoker with HTN & hyperlipidemia have stenosis?
aortoiliac

what about a 70 yo pt with DM, ESRD
tibial - DISTAL Dz
intermittent claudication is most improved by?
smoking cessation & exercise

how is ischemic rest pain relieved during physical exam?
dependent positioning of leg
ulcer on toes & heels with pale necrotic base?
ischemic ulcer from arterial insufficiency

tx?
pt needs offloading with a boot protecting area & keeping the pressure off of it.
ulcer on medial/lateral malleolus

associated findings: brown discoloration, itching, scaly skin
venous ulcer.
with stasis dermatitis

what if the associated findings were edema and scarring of skin?
venous stasis ulcer w/ lipodermatosclerosis from longstanding edema & venous congestion.

venous ulcers result from venous insufficiency --> venous pooling --> increased capillary pressure --> and pressure necrosis of the skin
presentation of diabetic neuropathic ulcer
plantar or lateral foot
foot becomes atrophic & deformed (CHARCOT FOOT*), dry, insensate
deforminity results in new pressure points
Ulcer is PAINLESS & cannot detect further injury

what does a Mal perforans ulcer refer to?
result from pressure of the deformed, insensate portions of foot
difference btw wet and dry gangrene
dry: mummification

wet: infected, malodorous, etiology of SEPSIS

tx?
removal followed by revascularization
Treatment of PAD?
stop smoking, control HTN, exercise, statins, cilostazol, ASA, clopidogrel

when would PTA be used
best in high flow vessels (carotids, iliacs, renals)
indications for surgical treatment of PAD
lifestyle limiting claudication, ischemic rest pain, or tissue loss.

pre op?
imaging to plan procedure:
MRA or contrast angiography (angiogram)
bypass for Arteroiliac occlusive dz?
artobifemoral bypass

what are the extra - anatomic bypass options?
axillobifemoral bypass
femorofemoral bypass
femoropopliteal dz?
FEM POP bypass (use saphenous vein)

bypass for tibial dz?
femorotibial or popliteal-tibial bypass
foot only amputation?
TMA or BKA

knee?
AKA
toe only for amputation?
may be able to do toe only if necrosis is isolated.

amputation of bed - ridden pt?
AKA!
due to permanent knee contracture/ulceration
presentation of acute arterial occlusion?
pain
pallor
paresthesia
pulselessness
paralysis
poikilothermia (cold)

what is the immediate treatment?
heparinization with postop coags
AGGRESSIVE fluid resuscitation
tx if AAO is caused by arterial embolus?
embolectomy
use fogarty cath.

remove ALL of the clot then send for pathologic exam! (atrial myxoma)
treatment if AAO is caused by diffuse PAD and in - situ thrombosis?
BYPASS!
duration of limb ischemia over 4 - 6 hrs?
perform FASCIOTOMY of the 4 compartments of the lower leg

why?
prevents compartment syndrome
amaurosis fugax, TIA or CVA on presentation would make you suspect what?
carotid stenosis

when would you do a carotid duplex US?
symptoms (TIA)
carotid bruit
pt undergoing CABG or other major procedures
medical tx for carotid stenosis?
HTN, lipids, ASA/clopidogril

what about if pt is asymptomatic w/ >60% occlusion or symptomatic > 70%?
carotid endarterectomy (CEA) -
**CAN NOT be done 1/ complete occlusion
when would carotid angioplasty or carotid stenting be indicated?
High risk pt.

risk compared to a CEA?
lower risk of MI
AAA < 5cm tx?
follow up CT every 6 - 12 months

indications for AAA repair?
>5cm
sxs (pain)
growth rate >1cm per year
open vs. endovascular repair of AAA?
open is shorter, cheaper, and proven.
EVAR: shorter recovery, less morbidity, more expensive, extensive f/u care

complications of EVAR?
graft migration, ENDOLEAK, graft kink/thombosis, aneurysm rupture
risks of popliteal aneurysms?
50% contralateral popliteal & 50% AAA

SxS: thromboembolism, compression of vein/nerve - tx?
ligate aneurysm proximally and distally.
DO FEM POP or POP POP bypass.

high amputation rate if thromboembolism