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24 Cards in this Set
- Front
- Back
Valvular disease is when
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the heart valves (mitral and aortic) dont work right
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Stenosis in valvular disease results in
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narrowing of the orifice and failure of the valve leaflets to open normally
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Regurgitation in valvular disease results in
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backward flow of blood through the valve when the valve is suppose to be closed.
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Cause of valvular disease
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congenital defects, trauma, ischemic damage, degenerative change, inflammation or infection
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Mital stenosis mechanism
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stiffness and fusion of the valve leaflets resulting in the mitral cusps to fuse at the edges and involves the chorda tendinae to shorten and pull the valves deep into the ventricles
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What is the result of mitral stenosis mechanism
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left atrial dilation and elevated left atrial pressure causing pulmonary congestion
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Manifestation of mitral stenosis
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nocturnal paroxysmal dyspnea and orthopnea, palpitations and chest pain, weakness and fatigue, PAC's, PAT, A. Fib, and a diastolic murmur sounding like low pitched rumbling best heart at the apex.
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Must known mitral stenosis manifestations
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PAC's, PAT, A. Fib, and a diastolic murmur sounding like low pitched rumbling best heart at the apex.
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Mitral regurgitation mechanism
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blood is ejected back to the atria with each systole which causes a decrease in SV ejected from LV during systole
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Overtime you will see _____ with mitral regurgitation?
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Left atrial and left ventricular enlargement.
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Acute mitral regurgitation manifestation
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acute volume overload, increased LV preload, little increase in LV stroke volume and overall forward flow is diminshed. acute rish in LA pressure = pulmonary edema
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chronic mitral regurgitation manifestations
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more gadual change, LA dilates.... increase in LVEDV to maintain forward volume= increases workload = hypertrophy.
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As chronic mitral regurgitation progresses you will see
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dilation of the LV and SV decrease, LA pressure increase, and pulmonary congestion development
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You can tell is a mitral regurgitation by
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Predisposititon to a. fib and pansystolic murmur
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Mitral valve prolapse
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2.4-7% of population, more frequent in women and has a familiar base
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Mitral valve prolapse caused by
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unknown, with marfan syndrome, osteogenesis imperfecta, and other connective tissue disorders.
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mechanism of mitral valve prolapse
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valve leaflets degenerate and become larger and floppy. During systole the leaflets will prolapse/ balloon back into he atrium= leaking
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manifestation of mitral valve prolapse
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asymptomatic but midsystolic click on auscultation and some have chest pain, palpitations and light headedness.
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Aortic stenosis
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the end result: narrowing of aorta orifice causing flow obstruction from LV = decrease SV, systolic Bp and pulse pressure
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Manifestations of aortic stenosis
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aysmptomatic for years, HR is often slower, harsh systolic murmur heart at left sternal border, and eventually signs of angina, syncope and heart failure. Alll tissue hypoxic b.c peripheries are cut off so lower body will be white/blue while upper body is hot and warm
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Acute Aortic regurgitation mechanism
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large volume regurgitated back to LV, increased LVEDP severely causing increased LA pressure and pulmonary edema. Decrease in CO
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Decreased co with acute aortic regurgiation activates
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sympathetic system to increase HR and systemic vascular resistance = worsens aortic regurgitation
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Chronic aortic regurgitation mechanism
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regurgant flow back to the LV, diastole pressure decreases, and LV enlarges.
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Chronic Clinical manifestations of aortic regurgitation
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asymptomatic, soft systolic aortic murmur, will eventually see LV failure : exertional dyspnea, orthopnea, and paroxymal nocturnal dyspnea. drop in diastole pressure affecting the coronary arteries = angina. Also, uncomfortable heart beat awareness, elevated systolic pressure and falling diastolic pressure
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