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8 Cards in this Set

  • Front
  • Back
treatment of torsades de pointes. what can predispose you to developing this/
mag sulfate

familial long QT syndrome
malnourished pt predisposed to hypomagnesemia (alcoholics,)
pts taking certain drugs (TCAs)
certain antiarrhythmics (amiodarone, sotalol)
anti-infective agents (moxifloxacin, fluconazole)
caustic ingestion: next step in mgmt
in absence of perforation or severe respiratory distress, early endoscopic eval within the first 12-24 hours is recommended in hemodynamically stable pts to assess the severity of esophageal damage.

mild or no esophageal injury: simple supportive measures

more severe injury: tube feedings and possible surgery (esophagectomy)

NO CHARCOAL
beta blocker overdose
causes AV block, bradycardia, hypotension, wheezing, and potential cardiogenic shock.

atropine and IV fluids are first line therapy, and if they do not completely reverse the cardiac sx, glucagon should be administered
overdose on fluphenazine
high potency typical antipsychotic that occasionally causes hypothermia by disrupting thermoregulation and the body's shivering mechanism

pts taking antipsychotics should be advised to avoid prolonged exposure to extreme temperatures
distinguishing bzd overdose from alcohol or phenytoin intoxication
alcohol and phenytoin -> nystagmus, whereas bzd does not share this
woman ingests 14 usual strength acetaminophen tablets two hours prior. next step in mgmt?
wait for two hours before obtaining serum acetaminophen -> decide treatment accordingly

Rumack-Matthew nomogram provides likeihood of hepatotoxic effects from acetaminophen overdose and the need for NAc. First data point on curve is at 4hrs. decision of whether or not to administer antidote can be made after 4 hrs, based on acetaminophen level.

as long as administered within 8hrs of ingestion, outcome will not be adversely affected.
PCP overdose
vertical nystagmus, can also cause dissociative feelings, psychotic and violent behavior, severe hypertension, and hyperthermia
mechanism of sodium bicarb in acute TCA poisoning
narrows the QRS complex by INCREASING THE EXTRACELLULAR SODIUM CONCENTRATION, preventing the development of arrhythmia in pts with antidepressant toxicity

alkalinization of the serum also DECREASES DRUG AVIDITY FOR THE SODIUM CHANNELS