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128 Cards in this Set

  • Front
  • Back
Primary Heart block
etiology: diseased infranodal conduction system
signs/symptoms
EKG Findings
treatment
etiology: occurs in normal individuals, increased vagal tone
sings/symptoms asymptomatic
treatment: not necessary
Secondary heart block (Mobitz type I)
etiology
signs/symptoms
EKG Findings
treatment
etiology: drug effect, increased vagal tone
sings/symptoms: usually asymptomatic
EKG findings: increasing PR interval until a dropped beat occurs (wenckebach); PR then resets
Treatment: stop offending drug
Secondary Heart block (Mobitz type II)
etiology
signs/symptoms
EKG Findings
treatment
Etiology: diseased infranodal conduction system
Signs/symptoms symptoms are rare
EKG findings: Unexpected dropped beat without change in PR interval
Treatment: ventricular Pacemaker
Tertiary Hear block
etiology
signs/symptoms
EKG Findings
treatment
etiology: no electrical communication between the atri and ventricles
signs/symptoms: syncope, dizziness, acute heart failure, hypotension, cannon A waves
EKG Findings: no relationship between P waves and QRS complexes
Treatment: ventricular pacemaker
Atrial fibrillation
Sings/Symptoms
EKG findngs
Treatment
Sings/ Symptoms: asymtomatic, shortness of breath, chest pain, palpitations, irregularly irregular pulse
EKG findings: wavy baseline without discernible p waves ; variable and irregular QRS respons
Treatment: anticoagulation, rate control drugs, cardioversion
Atrila flutter
EKg findings
regular rhythm "sawtooth appearnce"
ventricular fibrillation
etiology
signs/symptoms
ekg
treatment
etiology - associated with CAD/MI
signs/symptoms - syncompe, hypotension, pulselessnes
EKG findings: totally erratic tracing
treatmetn: immediate electricla cardioversion, cpr, lidocaine and/or epinephrine
premature ventricular contraction
EKG findings
early wid QRS compexes not preced by a P wave
What is a sign of perforated ulcer on a abdominal x-ray
free air under the diaphragm
What is the most likely cause fo acute lower GI bleed in pateints > 40 years old
diverticulosis
What are complications of diverticulitis
abscess, perforation, fistulas to bladder, skin or vagina, sepsis
Does diverticulits bleed?
no
What are risk factors for cholelithiasis
fat, female, fertile, forty, flatuelnt
What is a sign of perforated ulcer on a abdominal x-ray
free air under the diaphragm
What is the most likely cause fo acute lower GI bleed in pateints > 40 years old
diverticulosis
What are risk factors for cholelithiasis
fat, female, fertile, forty, flatuelnt
Does diverticulits bleed?
no
Does diverticulits bleed?
no
What is a sign of perforated ulcer on a abdominal x-ray
free air under the diaphragm
Does diverticulits bleed?
no
What is the most likely cause fo acute lower GI bleed in pateints > 40 years old
diverticulosis
Does diverticulits bleed?
no
What are risk factors for cholelithiasis
fat, female, fertile, forty, flatuelnt
What are risk factors for cholelithiasis
fat, female, fertile, forty, flatuelnt
What are complications of diverticulitis
abscess, perforation, fistulas to bladder, skin or vagina, sepsis
What are risk factors for cholelithiasis
fat, female, fertile, forty, flatuelnt
What are risk factors for cholelithiasis
fat, female, fertile, forty, flatuelnt
What are risk factors for cholelithiasis
fat, female, fertile, forty, flatuelnt
Does diverticulits bleed?
no
What are risk factors for cholelithiasis
fat, female, fertile, forty, flatuelnt
Intestinal inflammation presenting wth watery diarrhea and weight loss; fistulas between bowel and skin, urinary tract, or other parts o fbowel may ocur
crohn's disease
inflammatory disease of the colon with increased risk of clon cancer
ulcerative colits
immunosuppressive agent used for both ulcerative colits and crohn's
sulfasalzine
dull, waxing/waning perumbilical pain that then shifts to the RLQ and becomes sharp and continuous
appendicitis
Ulcerative Colitis/Crohn's Disease:
Sit of involvement
ulcerative colitis: the rectum, may extend in a continuous fashion to involves ome or all of he colon

Crohn's diease: may involve any portion of the GI tract, particularly the ieocecal region, in discontinuous patter.
ulcerative colitis/ crohn's disease
symptoms and signs
ulcerative colits: bloody diarrhea, lower abdomina cramps and urgensy, abdomina tenderness, frank blood on rectal exam, extraintestinal mafestations

Crohn's Disease: abdomina pain, abdomina mass, low grade fever, watery diarrhea, , abdominal tnederness or mass, preanal fissues, fistulas, extraintestinal manifestations
ulcerative colitis/ crohn's disease
extraintestinal manfestations
ulcerative colitis: apthtous stomatitis, episceritis/uveiits, arthritis, sclerosing cholangitis, erythema nodosum, and pyoderma gangrenosum

Chron's disease: nephrolithiasis and fistulas to the skin, biliary tract, urinary tract, or between bowell loops
Ulcerative colits/crohn's disease
what would you see on a colonoscopy
ulcerative coliits: diffuse and continuous colorectal involvement, friability, edema, pseudopolyps

crohn's disease: ulcers, strictures, "cobblestoning" and "skip lesions."
Crohn's disease/ ulcerative colitis
treatment
ulcerative colitis: sulasalzine, total colectomy is curative

Crohn's disease: sulfasalzien, resection may be necessary, but crohn's disease may recur lesewhere in the GI tract
Which is more likely to cause cancer ulcerative colitis or crohn's disease
ulcerative colitis has a higher risk for colon cancer, although they both have some risk
What is normal range of pH?
7.36 to 7.44
ph and pCO2 for metabolic acidosis
pH < 7.36
pCO2 < 40 mmHg
ph and pCO2 for respiratory acidosis
pH < 7.36
pCO2 > 40
pH and pCO2 for metabolic alkalosis?
pH > 7.44
pCO2 > 40
pH and pCO2 for metabolic acidosis
pH > 7.36
pCO2 < 40mmHg
Treatment for metabolic acidosis
IV fluids
possibley HCO3, for very low pH
trat underlying disease
treatemnt for rspiratory aciosis
Iv flids, O2
Peaked T waves on an EKG means
hyperkalemia
capral spasm upon inflation of lood pressure cufff
hypocalcemia
first line treatrment for moderat yhpercalcemia
IV hydration adn loop diuretics (furosemide)
49 year old male presents with acute onset flank pain and microscoic hematiure, what the most likely diagnosis
IV hydration and loop diureticsnephrolithiasis
the presence of red cell casts in uirn sediment indicates
glomerluonephirits
eosinophis isn urine sediment indicates
allergic interstiial nephirits
low uirine specific gravity in the presence of high serum osomolaitiy indicates
diaebetes insipidus
salicylate ingestion results in wht type of acid base diorder
anion gap acidosis
acid base distubance commonly seen in pregnant women
respiratory alkalosis
risk factors for DVT
stasis, endothelial injry and hypercoagulability
What acid base disorder do you get in pulmonary embolism
respiratory alkaosis
Acute onset tachypnea, pleuritic chest pian in tall, thin young men
spontaneous pneumothorax
what test do you use for cystic fibrosis
sweat chloride test
How is TLC affected in obstructive vs restrictive lung disease
TLC increased in obstructive, decreased in restrictive
what is the most common caue of headaches in chldren age 2-10
infection
What is the most common cause of headaches in children age 18-35
trauma
tension headache
chonic headaches, vise-like or tigh pain, genrally in occipitalnad neck region,
diagnosis of exclusion
migraine headache
throbbing headache that lasts > 24 hours. typically associated with nausea and vomiting, photophobia

classic migrains ar commonly unilateral, associated withn aura and preced by visual symtoms. Migrains more commonly present without these associated symptoms and may be bilateral and periorbital
who gets migrains more men or women
women
cluster headache
a brief, sever, unilateral periorbital headache. attacks ten d to occur in clusters affecting the the same part of the head and taking palce the same time of day. may be precipitated by alocohol or vasodialting drugs
what is the most important diganostic ttest for sizures
EEG
rocky mountain spotted fever
tic born, presents with headache, fever, maise, and a rash on the palms and soles. treat with doxycycline
woman has puruelnt cervical discharge, what could it be? treatment?
chlamyidia - doxycycline or azithromycin
gonrorrhea - ceftriaxone
causes of microcyitc anemia
thalassemia, iron deficiency, anemia of chornic disease
dietary suppplement ot geive to paitents with tthalasseimia
folate, but not iron
How doyou distinguish polycthemia vera from secondary polycythemai
normal oxygen saturation, EPO leve, high RBC mass and heamtocrit
Patient presents with excessive bleeding after dental work. Labs show normal Pt/PTT with pronolged bleeding time. Most liekly diganois
von willebrand's disease
Trsymotms of sicle cell crisis
cute onset boen/chest pain, fever
monoclonal gammopathy, bence jones proeinureia, "punched out lesions in ckull and long bones.
multiple myeloma
most common cancer of childhool
acute lympocyitic leukemia
auer rods are pathogomic for..?
acute myelogenous leukemia
A patient presents with early satiety, LUQ fullnes/pain, splenomegally, bleeding, and diathesis. cytogenics sow t9,22
chronic myelogenous leukemia withthe philadelphia chromosome
reed sternber cells are a sign of ?
hodgkin's lymphoma
risk factors for non hodgkin's lymphoma
EBV, HIV infection
Which has regional adenopathy, hodgkins or non-hodgkin's lymphoma
non-hodgkins lymphoma
treatment of von willebrand's disease
desmopressin
Disseminated Intraascular Coagulation
systemic disease with pahtologic coagulation and lack of physiologic coagulatin,
may occur secondary to sepsis, transfusion, neoplasia, trauma, obstttric compications

diffuse bleeding, digital cyanosis, tachycardia, tacypnea, respiratory failure,

low fibrinogen, elvated pt/PTT, low hemocrit
classic EKG findings in pericarditis
low-voltage, diffuse ST segment elevation
symptoms of pallor, high pressure, palpitations, headache pain, and perspiration
pheochromocytoma
first line treatment of rgrowth hormone secreint pituitary adnemoa
teansphenoidal surgical resection
patient complains of headache, weakness, and polyuria; exam reaveals hypertension andtentany. labs include hypernatremia, hypokalemai, and metabolic alkalosis
primary hyperaldosteronism due ot Conn's ynrome or bilateral adrenal hyperplasia
morning hyperglycemia that is a reboud response to nightime hypoclycemia, not a need for more insulin
somoyogi affect
signs and symptoms of hyperparathyroidsim
"stones, bones, groans, psychic overtones"

stoens: nephrolitiasis or nephrocalcinosis
bones - bone pain, muslce aches, arthralgias, fractues
groans, PUD, pancreatitis
psychic overtones - fatigue depression, anxiety, irrativility, sleep disturbances
patient preseins with wekness, nausea and vomiting, weigh loss, and new skin pgmentation. labs how yponatremai and hyperkalemai
addison's disease
peptic ulcer disease
damage ot the gastic or duodienal mucosa,

dul/burning/aching epigasric paintha t improves ieth meals,
hematemesis ("coffe-ground" emsis) and blood inthe stool (melena or hemtochezia
epigastric tenderness
hepatitis A
causes acute hedpatis
fecal-oral transmission (contaminated food/water/shellfish)
does NOT cause chronic hepatitis
temporal arteritis
most feared complicatin is blindess seconcary to occlusion f othe central retinal artery

you get temporal tenderness, jaw claudicatioon, monocula rblindess, ESR > 50

trat with prendisone
positively bifringent rhonboid crystals
pseudogout
negatively bfvely bifringent needle like crystals
gout
gout treatment
acutely: colchicinene, NSAIDS, sterois

long term - allpurinol and prbencid

avoid thizide / loop diuretics
Labs for SLE
positive ANA - sensitive but not specific
Anti-NDA and anti-SM andtibodies - specific but not sensitive
who gets SLE themost
women, especally black women
treatment of SLE
steroids,
hydroxychorquinie
cyclophosphamide

avoid sun exposure
symtoms of SLE
MALAR RASH, joint nenderns, joint pian, photosensitvty, oral ulcer, pericaditis,
fibromyalgia
diagnosis of exclusion
multiple diffuse tender areas and standard "trigger areas" that produce pain on palpation and sleep disorders
what are the primary and dompensatory responses for metaboloic acidosis?
cause?
primary: decreased HCO3-

secondary: decreased pH, decresed PCO2
hyperventilation

cause: diabetic ketoacidosis; diarrhealactic acidosis; salicylate OD, acetazolamid OD
what are the primary and dompensatory responses forrespiratory acidosis?
cause?
Primary: Increased pCO2

secondary
decreased pH, increased HCO3-
Renal [HCO3-] reabsorption

cause: COPD; airway obstruction
what are the primary and dompensatory responses for respiratory alkalosis?
cause?
primary: decreased pCO2

secondary: increased pH, decreased HCO3-
Renal [HCO3-] secretion

cause: high altitude; hyperventilation
what are the primary and dompensatory responses for metaboloic alkalosis?
cause?
primary: increased [HCO3-]

secondary: increased ph; increased [HCO3-]
hypoventilation

causes: vomiting
chronic bronchitis
a productive cough lasting at least three months per year for two consecutive yeras
emphysema
pahtolgoic dignosis of termina airway destruction to smoking (cintrilobular) inherited alpha 1-antitrypsin deficiency (panlobular)
symptoms of emphysema
"pink puffer"
decreaed breath souns, minimal cough, dypnea, pursed lips, hypercarbia/hpoxia late, barrel chest
symptoms of chronic bronchitis
"blue bloater"
rhonic, productive cough, cyanotic but with mild dyspnea, hypercarbia/hypoxia early, frequently overweight with peripheral edema.
Sings and symptoms of ASthma
History: Cough, episodic wheezing , nad o chest tighntes, dyspnea, worse at night or early in the morning

PE: prolonged expriatory duration, accessory muxle use tachypnea, tachycardia, decrased O2 struation, hyper resonance
noncaseating granulomas indicate
sarcoidosis
treatment ofr tension pneumothorax
immediate decompression using a needle in the second intercostal space; chet tube placement
lung disease of coal miners
pneumoconiosis
increased risk of what infection with silicosis
M. tuberculosis
hepatitis A
causes acute hedpatis
fecal-oral transmission (contaminated food/water/shellfish)
does NOT cause chronic hepatitis
Hepatitis B
causes acute hepatitis or chronic hepatitis
major cuase of hepatocellular carnioma
spred by lood and other body fluids including saliva
there is a vacccine
Heaptitis C
causes acute or chronic hepatitis, major cause of cirrhosis and psot transfusion hepatitis
associated with Iv dug abuse, spread by blood
Hepatitis D
Only causes disease in asscoiatin wiht hepatitis B, acute or chronic, blood borne
Hepatitis E
normaly self - limted, dangerous in pregnant women, spread by fecal-oral tranmission
treatments for Tuberculosis
multiagnet therapy (INH, rifampin, pyrazinamide, ethambutol, streptomycin)
treatment for toxoplasmosis
pyremethamine, sulfadizine, clindamycin
What is somoygi effect
nocturnal hypoglycemia casuing elevated morning glucose due to release of coutnerregulatory hormones (reduce insulin to treat)
What is Dawn phenonomen
in diabetis, early moning hyperglycemia cased by reduced effectiveness of insulin at this time
Derm related difffering symptom for addison's disease and secondary adrenal insfficiency
addison's disease: increased skin pigmentation
secondary adrenal insfficency: no hyperpgmentation
presentign symptoms of adrenal inufficiency
inlude weakness, weight loss, nausea, and vomiting
Lab results ofr addison's disease and secondary adrenal insuffiency
addison's disease: ELEVATED plasmaACTH and low cortisol levels in respons to ACTH challenge

secondary adrenal insuffiency: DECREASED palsma ACTH and increased cortisol levels in response to ACTH

both can also get hypnatremia, hyperkalemia, and eosinophilia
common cause of secondary adrenal insufficiency
abrupt cesssation of chronic glucocorticoid treatment.