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38 Cards in this Set

  • Front
  • Back
how common is incontinence
-prevalence increases with age
-25-30% of community dewling older women and 10-15% of men
-Common cause of institutionalization, social isolation, and decline in function
-it is underdiagnosed and undertx
-80% of urinary incontinence can be cured or improved!
importance of incontinence
1. social stigma
2. medical complications: skin breakdown, inc UTIs, falls
3. institutionalization
4. economic
5. effects ALL aspects of QOL
urethrovesicle junction
-support by fascia
-normally an acute angle is formed between bladder and urethra, which helps maintain continence
neural control
-local stretch receptors (reflex arc)
-parietal recognition center
-frontal lobe inhibits
-micturition center in pons
-detrusor under cholinergic control
-urethra under alpha adrenergic control
-external sphincter under voluntary control
FILLING OF BLADDER -> SYMPATHETIC
EMPTYING --> PARASYMPATHETIC
Filling of the bladder
-Bladder neck, sphincters close, detrusor relaxes
-fill under low P
-first urge around 250cc
-capacity 400-600cc
emptying of bladder
-Bladder neck, sphincters relax, detrusor contracts
-When bladder pressure exceeds urethral pressure, voiding occurs
requirements for continence
1. intact GU anatomy
2. intact neural control
3. mobility/dexterity
4. reasonable environment
5. motivational
urologic changes with age
1. dec bladder compliance and capacity
2. inc residual vol
3. uninhibited bladder contractions
4. prostatic enlargement
5. dec urethral P
6. dec estrogen
7. pelvic laxity with vaginal childbirth
8. neurologic: dec nerve cell #, dec NT concentration, dec autonomic function
9. altered immune function
basic causes of incontinence
1. failure to store
2. failure to empty
3. failure of coordination
risk factors for incontinence
1. female
2. >60
3. parity
4. UTI/CU surgery
5. chronic illness/immobility
6. meds
types of incontinence
1. acute (reversible/ transient)
2. fixed
potentially reversible causes
D delerium
I infx/inflam
A atrophic vaginitis or urethritis
P phamaceuticals
P psyc disorders
E endocrine disorders
R restricted mobility
S stool impaction
meds that may cause incontinence
1. diuretics
2. anticholinergics
3. sedatives
4. CNS depressants
5. alcohol
6. narcotics
7. aadrenergic agonists
8. CCBs
categories of fixed incontinence
1. urge incontinence
2. stress
3. overflow
4. reflex
5. functional
6. mixed
urge incontinence
-most common cause if UI <75 yo
-sudden urge to void
-idiopathic (uninhibited bladder contractions)
-other causes: infection, tumor, stones, atrophic vaginitis or urethritis, stroke, Parkinson’s Disease, dementia
stress incontinence
-most common type in women >75
-occurs with increase in abdominal P, cough, sneeze
-hypermobility of bladder neck and urethra
-intrinsic sphincter problems
overflow incontinence
-over distention of bladder
-bladder outlet obstruction
-non-contractible bladder
reflex incontinence
-Involuntary loss of urine without urge or stress
-Patient is unaware that they need to void
-Usual cause: suprasacral spinal cord lesion
functional incontinence
-Does not involve pathology of lower urinary tract
-Result of psychological, cognitive or physical impairment
mixed incontinence
-combo
-esp see urge + stress in older women
eval of incontinence
1. hx
-Onset, duration, severity, previous treatment, medications, GU surgery
-assoc sx
-3 P's: position when leakage, protection, problem)
2. PE
3. labs/xrays
4. beside urologic disgnostic test
4. formal urodynamics
how to ask
Do you lose (leak) urine with very little warning?
Do you lose (leak) urine before you can reach the bathroom?
Do you lose (leak) urine when you cough, sneeze, …………………?
Do you constantly lose small amounts of urine?
After you go to the bathroom, do you dribble small amounts or feel you need to go again?
Are there times you feel you need to void, but are unable or unwilling to get to the bathroom ?
Location/accessibility of toilet?
Difficulty removing clothing?
PE
-mental status
-mobility
-fluid overload
-abd exam
-neuro exam
-genital/pelvic
-rectal
diagnostic tests
1. blood tests: lytes, Ca, glucose, BUN, Cr
2. UA and culture
3. ? radiologic testing
4. bedside cystometrics:
-flow rate (male)
-post-void residual
-filling under low P
intepretation of post-void residual
PVR < 50cc - Adequate bladder emptying
PVR > 150cc - Avoid bladder relaxing drugs
PVR > 200cc - Refer to Urology
PVR > 400cc - Overflow UI likely
stress test
-remove catheter, pad in front of urethra, cough
-look for leakage on pad
-if leakage, repeat after Marshall maneuver
-if not leakage, but suspicion of SI, standing
characteristics- Urge
1. sudden urge to void
2. leakage frequent, mod to large amts
3. nocturnal frequency
4. sacral sensation/ reflexes preserved
5. PVR low
6. evidence of uninhibited contractions
characteristics- reflex
No urge to void, no sensation of fullness, no stress
Leakage frequent, moderate amounts
Nocturnal as well as during the day
Perineal sensation may be decreased; reflexes are preserved
Detrussor hyperreflexia, sphincter dyssynergia
PVR increased
characteristics- stress
Daytime loss of small to moderate amounts with increased intra-abdominal pressure
PVR low
Positive stress test
Correction with Marshall maneuver is 90% predictive of correction with surgery
Look for stress-induced detrussor instability
characteristics- overflow
+/- hesitancy, decreased/interrupted flow
sensation of fullness, need to strain to void, incomplete emptying
Leakage frequent, small amounts
Nocturnal as well as during the day
If neurologically mediated, perineal sensation, sacral reflexes, and anal sphincter control may be decreased
Palpable bladder
PVR LARGE
referral for urodynamics
1. any pt with relfex incontinence
2. any pt with overflow incont
3. when initial dx is unclear
4. when risk from empiric therapy is high
5. when empiric therapy has failed
treatment goals for incontinence
1. maintain renal function
2. help pt become dry
3. establish nml voiding pattern
4. improvement of sx vs. cure
treatment options
1. eval amt and timing if fluid intake
2. avoid bladder stimulants
3. use diuretics judiciously
4. reduce physical barriers to toilet
5. resolve other functional prob
6. bladder training
7. pelvic floor (Kegel)
8. biofeedback
9. caregiver interventions
Meds for stress incontinence
Phenylpropanolamine (Ornade)
Pseudo-Ephedrine (Sudafed)
Estrogen (orally, transdermally or transvaginally
meds for urge incontinence
Propantheline (Pro-Banthine)
Imipramine (Tofranil)
Oxybutynin (Ditropan)
Tolterodine (Detrol)
Trospium (Sanctura)
Solifenacin (Vesicare) , darifenacin (Enablex)
Alpha-adrenergic antagonists (men with BPH)
Adverse effects of meds
1. dry mouth
2.HA
3. dizziness
4. GI complaints
5. somnolence
C/I: urinary retention, GI disturbances, glaucoma
surgical interventions
-Surgery is reported to “cure” 4 out of 5 cases, but success rate drops to 50% after 10 years.
1. Urethral hypermotility:
-Marshall-Marchetti-Kantz procedure
-Needle neck suspension
2. Intrinsic sphincter deficiency
-sling procedure
other interventions
1. Pessaries
2. Periurethral bulking agents
-injection of collagen, fat or silicone
3. Diapers or pads (can be temporary)
4. Chronic catheterization
-Last resort!
-Periurethral or suprapubic
-Indwelling or intermittent