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108 Cards in this Set

  • Front
  • Back
Primary function of the eye
-encode light and carry information to brain, the brain interprets the information
Extraocular Structures
-protect from the foreign bodies
-regulate entry of light
-distribute tears by blinking
Extraocular Structures-Conjuctiva
-thin, transparent mucous membrane that lubricates the eye
-lines inner surfaces of eyelids and folds over anterior surface of eyeball
-palpebral conjunctiva lines upper and lower eyelids
-bulbar conjunctive-loosely covers anterior sclera
Extraocular structures-extrinsic eye muscles
-control eye movement
-help maintain shape of eyeball
-controlled by cranial nerves
Intraocular structures-sclera
-protects and gives shape to eyeball
-gives way to cornea over iris and pupil
Intraocular structures-cornea
-transparent, avascular, sensitive to touch
-allows light to enter eye and ispart of eye's light-bending apparatuus
-corneal reflex-eyelids blink when cornea is touched, tears are also secreted
Intraocular structures-Iris
-a disc of muscle surrounding pupil and lying between cornea and lens
-gives eye its color
-regulates light entry by contracting pupil size
Intraocular Structures-Pupil
-constricts when light enters and when used for near vision
-dilates in dim light and when used for far vision
Intraocular structures-anterior cavity
-consists of anterior chamber, posterior chamber, and canal of schlemm
-anterior chamber-space between cornea and iris
-posterior chamber -space bwtween iris and lens
-a network of channels that circle the eye in the angle at the juction of the sclera and cornea
-drainage system for fluid moving between anterior and posterior chambers
Intraocular structures-Aqueous Fluid/Humor
-in anterior cavity of eye
-clear fluid
-constantly formed and drained to maintain a relatively constant pressure of 15-20 mmHg in eye
-provides nutrients and oxygen to cornea and lens
Intraocular structs-internal chamber
-contains lens, posterior cavity, vitreous humor, ciliary body, uvea, and retina
Intraocular structures-Lens
-convex, avascular,transparent
-located directly behind pupil
-can change shape to focus and refract light onto the retina
Intraocular structures-posterior cavity
-lies behind lens
-filled with vitreous humor (clear, gelatinous)
Vitreous humor
-lies behind the lens in the posterior cavity
-supports posterior surface of lens
-maintains position of retina
-transmits light
Intraocular structures-Uvea (Vascular tunic)
-the middle layer of the eyeball
-three components-iris, ciliary body, choroid
Three components of uvea
-iris
-ciliary body
-choroid
Ciliary Body
-encircles lens
-along w/ iris, regulates amount of light reaching retina by controlling shape of lens
Choroid
-pigmented and vascular
-its blood vessels nourish layers of eyeball
-its pigmented layers absorb light, preventing it from scattering within eyeball
Rods and cones
Ishihar test-evaluates color vision
Rods-dim light and peripheral vision are more sensitive than cones, so we don't see colors as well in the dark
cones-bright light and perception of color
Optic disc (blind spot)
-cream-colored, round
-the point at which the optic nerve enters the eye
-physiologic cup-a slight depression in the center of the optic disc
-macula-lateral to optic dis-contains cones
-fovea centralis-a slight depression in center of macula-contains cones-a main receptor of detailed color vision
Physiologic cup
-part of optic disc
-a slight depression in the center of the optic disc
Macula
-part of the optic disc
-lateral to optic dis-contains cones
Fovea Centralis
-in optic disc
-slight depression in center of macula
-contains cones
-main receptor of detailed color vision
In your visual patway your optic nerves meet where? Where is the location of the tracts?
-meet at the optic chiasma (in intersection of 2 tracts)
-just anterior to the pituitary gland
-at the optic chiasma, axons from the medial half of each retina cross to opposite sides to form pairs with axans of the other
-these pairs continue as the right and left optic tracts
-these tracts carry information from both eyes
-visual impulse travels in optic tracts to thalamus and synapse with neurons, forming pathways called optic radiations
-optic radiations terminate in visual cortex of occipital lobe
-visual cortex fuses information from each eye into one image
Refraction
-bending of light rays as they pass from one medium to another medium of different optical density
-because the lens in convex, the image is projected onto the retina upside down and reversed from left to right
-the brain decodes the image so it is perceived as it actually occurs
Accommodation
-the eys are best adapted to see distant objects
-for persons w/ normal vision, the distance from the viewed object at which the eyes require no accommodation is 20 feet (perfect eyes)
-accommodation is accomplished by contraction of ciliary muscles
-near point of vision-closet point on which a person can focus, usually 8-10 " in young adults with normal vision
-pupillary constriction also eliminated most divergent light rays and sharpens focus.
Convergence
-refers to the medial rotation of the eyeballs so that each is directed toward the viewed object
-allow the focusing of the image on the retinal fovea of each eye
Diagnostic tests for the eye
-retinoscopy and/or refractometry-test for refractive errors
-tonometry-tests for increased intraocular pressure
-CT scan
Genetic eye disorders
-Glaucoma- ~3% is genetic
-gyrate atropy of choroid and retina
-best disease
amsler grid
-physical assessment
-used to ID early changes in vision related to diabetes and mascular degeneration
-hold grid 12-14", cover one eye, stare at the center dot
-done when pupils are dilated and pt is wearing best visual corrections
Abnormal amsler grid findings
-any of grid looks crooked or bent
-any of the boxes are different in size or shape
-if any of the lines are wavy, missing, blurry or discolored
Age related changes
-decreased elasticity of lens, decreaseing focus and accommodation for near vision (presbyopia)
-yellowing of lens, changes in retina affect color vision
-pupil-decrease in size and responsiveness to loght
-narrowing of visual fields
-distorted depth perception
-loss/decreased function of photoreceptors cells
-adaptation to light and dark takes longer
-decreased reabsorption of intraocular fluid
-decreased tear production
-vitreous body may pull away from retina
Myopia
-nearsightedness-changes in distant vision
hyperopia
-farsightedness
presbyopia
-changes in near vision resulting in changes in lens elasticity due to aging (another name for farsightedness occurring in persons over age 45)
Nystagmus
-involuntary rhythmic movement of eyes, associated with neurologic disorders, somemeds, sometimes seen in persons wo were premies
Foster safety and independence for a blind patient
-orient to environment verbally and physically
-use verbal communication freely
-provide other sensory stimuli per patient's preferences
-orient to food trays (clock face)
-when assisting w/ ambulation, ask pt to hold your arm while you walk slightly ahead
-ask pt what assistance is disired
-refer to community services if new loss of sight
Conjunctivitis
-bacterial
-viral
-fungal
Conjuctivitis-bacterial
-staph, hemophilus
-pink eye-very contagious
adenovirus
leading cause of conjunctivitis in adults
Systemic/ other infections that may affect eye
-herpes simplex, herpes zoster, gonococcus
Conjuctivitis symptoms
-pharyngitis
-fever
-malaise
-swollen preauricular lymph nodes
trachoma
-bacterial
-chronic conjunctivitis caused by chlamydia trachomatis
-preventable cause of blindness
-common in Sahara, Africa, Middle east, and parts of asia
Conjuctivitis-Infection control
-very contagious
-close personal contact transmission
-also transmitted by fomites suchh as towels, handkerchiefs, flies
Conjunctivitis-treatment
-antibiotics-drops/systemic
-antivirals as appropriate
-anti-inflammatories as needed
-eye irrigations
-warm normal saline compresses
Corneal disorders-Astigmatism
astigmatism-irregular or abnormal curvature of cornea
-light rays focus on more than one area of retina, distorting near and distance vision
Corneal disorders-Keratitis
-Inflammation of cornea
-causes infection, hypersensitivity reactions, ischemia, tearing defects, impaired innervation of cornea
Nonulcerative keratitis
-all layers of corneal epithelium remain intact (viral, TB, SLE)
Ulcerative keratitis
-tissue destruction and ulceration of cornea (bacterial conjunctivitis)
-corneas blood flow is low so it's hard to heal
Corneal disorders-Ulcer
-local necrosis
-causes-infection, exposure trauma, misuse of contact lenses, herpes viruses
-complications-fibrous tissue can form during healing, resulting in scarring and opacity of cornea
Corneal disorder-dystophies
accumulation of cloudy material
-typically inherited
-keratoconus-progressive thinning of cornea
-typically affects teens and yound adults, usually stabilized
-about 20% may require corneal transplant
keratoconus
-progressive thinning of cornea
-typically affects teens and yound adults
-usually stabilizes
-about 20% may require corneal transplant
Eye surgery-Cornea
-Laser-changes shape of eyes (lasik, prk, lasek, ltk) for refractive errors
PTK
-alternative to corneal transplant-diseased corneal tissue is vaporized and surface irregularities are corrected
Corneal disorders-corneal surgery
-usually done as outpatient
-eye patch for 24 hours
-minimize exposure to sources of infection
Lamellar keratoplasy
-superficial layer of cornea is removed and replaced with a graft
Penetrating keratoplasy
-a button or full-tickness of cornea is removed and replaced by donor tissue
Nursing care of post-op eye surgery patient
-assess dressing for bleeding, drainage
-maintain patch or shild as ordered
-place in semi-fowler's on unaffected side as tolerated
-assist pt to avoid coughing, sneezing, or straining if possible
-assess and medicate prn for pain
-approach pt on unaffected side
-place personal articles and call light within reach
-admin eye drops as ordered
-admin anti-emetic medication as ordered
How do you assess for potential complications for a post-op eye surgery patient?
-increased pain/drainage from affected eye
-hemorrhage w/ blood in anterior eye chamber
-flashes of light, floaters, sensation of curtain being drawn over eye (retinal detachment)
-cloudy appearance (corneal edema)
Marginal blepharitis
-disorder of the eyelid
-inflammation of glands and lash follicles on eyelid margins
-caused by staph or seborrhea (dandruff)
Hordeoleum
-stye
-usually a staph abscess
-internal or external lid margin
Chalazion
-a granulomatous cyst or nodule of the lid
-temp/perm right on eyelid
Entropion
-inversion of the lid margin
-related to aging process, infection
-can lead to corneal irritation and scarring
-eyelashes invert and scrape cornea
Extropion
-eversion of lid margin
-related to aging
-can be caused by Bell's Palsy-facial nerve paralysis
-can cause infection/scarring of cornea
-eyelids flip out
Causes of eye burns
-chemicals
-explosions
-flash burns
-UV ray
Symptoms of eye burns
-pain
-partial/complete loss of vision
-possible bleeding
-possible extrusion of eye contents
Hyphema; and examples
-caused by trauma
-bleeding into the anterior chamber of the eye
-if the highly vascular uvea is disrupted by blunt force, hemorrhage mayoccur and fill anterior chamber
-eye pain, decreased visual acuity, seeing a red tint
orbital blowout fracture
-orbital contents can herniate into underlying maxillary sinus
nursing care of the eye trauma patient
-chemical burns
-irrigation
-chemicals-immediate priority flushing w/ NS
-water may be used if NS is not available
-flush from inner to outer canthus
-morgan lens

Irrigation; eyelid everted to remove material from conjunctival sac
-a topical anesthetic may be used
Penetrating Wounds, nursing priorities
-protect eye from further injury
-decrease pain
-no pressure an eye
-do not attempt to remove obj
-paper cup/shield over affected eye
-patch unaffected eye
-may be ordered Diamox to decrease ICP (osmotic diuretic)
Uveitis causes and symptoms
-causes; idiopathic, autoimmune, infections, parasites, trauma
-symptoms; pupillary constriction, erythema around limbus (edge), eye pain, photophobia
Cataracts Pathophysiology
-can be congenital or acquired (trauma), 2ndary to inhaled or systemic corticosteroids
-lens fibers and prteins change and degenerate
-lens may also yellow, affecting color discrimination
Immature cataract
-when only a portion of the lens is affected (periphery)
Mature cataract
-opacification (clouding) of entire lens
Cataract manifestations
-tend to occur bilaterally unless related to trauma
-one generally matures more rapidly than the other
-light rays are scattered as they pass through clouded lens
-if mature, pupil may appear cloudy gray or white
Intraocular lens (IOL) implant
-for cataracts
-an artificial lens to replace the diseased lens of the eye is placed in the posterior capsule behind the iris
Intracapsular cataract extraction (ICCE)
entire lens and its surrounding capsule removed-rarely done today
Extracapsular cataract extraction (ECCE)
-the anterior capsule, nucleus, and cortex of lens removed, and posterior capsule is left intact
Glaucoma symptoms, risk factors,
-symptoms are narrowing of visual field
-age is primary risk factor
-increased in hispanic and african american persons
Primary glaucoma
-most common in adults over age 60
-congenital
secondary glaucoma
-infection/inflammation of eye, cataract, tumor, hemorrhage, trauma
Glaucoma pathophysiology
-aqueous humor produced in ciliary body posterior capsule pupil anterior chamber outflow or aborption through trabecular meshwork and Canal of Schlemm
-when absorption or outflow are disturbed, IOP increases injury to optic nerve
Open Angle Glaucoma
-angle refers to the angle formed at the point where iris meets cornea in eye's anterior chamber
-also called chronic simple, most common in adults, cause is unknow
-flow of aqueous humor through trabecular meshwork and Canal of Schlemm is relatively obstructed
-generally affects both eyes, through pressures may be different in each eye
Anterior Angle
between iris and cornea is normal (open angle)
-however, flow of aqueous humor through
Angle Closure Glaucoma
-also called narrow angle or closed angle
-less common 5-10%
-becuase of corneal flattening orbuldging of iris into anterior chamber
-when the lens thickens during accommodation or iris thickens during pupillary dilation, the angle can close completely
What does Angle closure bock?
Does it effect one or two eyes?
when do episodes usually occur?
-abrupt blockage of trabecular meshowork and Canal of Schlemm, abrupt in IOP
-typically unilateral but incidence in one eye increases risk of occurrence inother eye
-episodes may occur in situations that cause pupil to dilate (darkness, emotional stress)
What meds are are used for the treatment of Angle Closure glaucoma?

Symptoms?
-no eye drops or OTC medications unless prescribed
-avoid meds that have mydriatic (pupil dilating) effect-atropine or other anticholinergics

-severe eye and face pain, malaise, aN/V, seeing colored halos around lights, abrupt decrease in visual acuity, pupil may be fixed
Tonometry
diagnosic tool for glaucoma
-contact or noncontact
Fundoscopy
-diagnosic tool for glaucoma
-visual inspection w/ an ophthalmoscope
-shows pallor and increase in size and epth of optic cup on optic disc
Gonioscopy
-diagnosic tool for glaucoma
-measures depth of anterior chamber
-differentiates open from closed -angle
Medications for glaucoma
-adreneric agonist-reduce production of aqueous humor
-beta blockers-reduce production of aqueous humor
-carbonic anhydrase inhibitiors-lower IOP
-prostaglandin analogs-relax ciliary muscle and increase outflow of aqueous humor
What is age-related macular degeneration (ARMD)
-macula area of retina that provide sharp central vision, receives light from center of visual field
-gradual degeneration of macula
Age related macular degeneration risk factors
-age
-smoking
-race
-possible genetics (caucasians)
Age related macular degeneration-non-exudative (dry)
-more common
-gradual, begins w/ accumulation of drusen deposits that gradually enlarge and become more numerous
-pigment epithelium detaches in small areas and becomes atrophic, interfering with sensory function of macula
-may progress to exudative
Age related macular degeneration-Exudative (wet)
-characterized by formation of new, weak blood vessels in the space between the choroid (vascular layer of the ye) and the retinal (neurosensory layer)
-these new vessels leak and elevate the retina from the choroid, causing vision distortion
-with significant or repeated bleeding episodes, scar tissue forms, and central vision is permanently lost
Diagnosis of age related macular degeneration
-Amsler grid
-Fluorescein angiogram-pictures taken as dye passes through blood vessels of retina; detects leaks
Treatment of ARMD
-dry
-wet
dry-in early or intermediate stages slowed by high dose antioxidants and zinc (vit C,E, A, Zn, Copper)
wet-laser or photodynamic therapy,
nonproliferative diabetic retinopathy
-venous capillaries of eye dilate and develop microaneurysms which leak and cause retinal edema or small ruptures within retina
Proliferative diabetic retinopathy
large areas of retinal ischemia occur and neovascularization occurs, spreading rapidly over the inner surface of the retina and into the vitreous body
-new vessels leak fluid and protein retinal edema and hemorrhage into vitreous humor
-vessels may become fibrous and cause retinal detachment
Symptoms of separation of retina from choroid
-floaters
-spots
-flashes of light
-blurred vision
-progressive decrease in vision
-sensation of curtain or veil across field of vision
Cryotherapy
treatment for separation of retina from choroid
-super cooled probe or laser photocoagulation
-these create an area of inflammation and adhesion to "weld" retina to choroid
scleral buckling
-treatment of retinal detachment
-an indentation or fold created in sclera, bringing choroid into contact w/ retina
-contact maintained w/ a local implant on sclera or an encircling strap or buckle
pneumatic retinopexy
-air injected into vitreous cavity. Pt positioned so that the air bubble pushes the detached part of the retina into the choroid
Laser for retinal detachment
-creates a bond between retinal and choroid
Retinitis Pigmentosa
-hereditary degenerative disease
-retinal atrophy and loss of retinal function
-rod cells degenerate at periphery of retina
-areas of degeneration and cell death expand, causing vision to narrow and central vision to be lost
Retinitis pigmentosa symptoms
-difficulty w/ night vision (even in childhood)
-progress to slow loss of visual fields, photophobia and disrupted color vision
-may be blind by age 40
-currently no effective tx
-low vision aids, couseling may be helpful
How does HIV effect the eyes?
-retinopathy
-kaposi's sarcoma
-cytomegalovirus (CMV) retinitis
-corneal ulcers
Enucleation
-caused by traum, infection, glaucoma, intractable pain, malignancy
-conjunctiva and eye muscles sutured onto an implant at time of surgery
-pressure dressing for 24-48 hrs
-most common complications are hemorrhage and infection