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24 Cards in this Set
- Front
- Back
Inguinal Hernia
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a. Labs
i. None; usually a clinical diagnosis b. Imaging i. Herniography, ultrasound, MRI c. Treatment i. Truss, reduction, or surgery |
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Epididymitis/orchitis
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a. Labs
i. CBC (leukocytosis) ii. Urinalysis, urine culture (pyuria in epididymitis) iii. Gram stain, culture, nucleic acid amplification for STDs b. Imaging i. Doppler ultrasonography, scintigraphy (for perfusion) c. Treatment i. Gonorrhea: ceftriaxone 125mg IM; azithromycin 2g PO ii. Chlamydia: azithromycin 1g IM; doxycycline 100mg PO bid |
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Hydrocele/varicocele
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a. Labs
i. None; usually a clinical diagnosis b. Imaging i. Doppler ultrasonography, scintigraphy (for perfusion) c. Treatment i. Varicocele: observation; ligation of gonadal vein ii. Hydrocele: excision of hydrocele sac |
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Meckel's diverticulum
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a. Labs
i. None; usually a clinical and radiographic diagnosis b. Imaging i. Small bowel (barium) follow-through ii. Enteroclysis (fluoroscopy of small bowel) iii. CT shows mass with air/fluid level c. Treatment i. Surgical excision |
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GI duplication
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a. Labs
i. None; usually a clinical and radiographic diagnosis b. Imaging i. CT scan, not barium studies as these usually do not communicate with bowel lumen c. Treatment i. Surgical excision |
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Intussusception
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a. Labs
i. None; usually a clinical and radiographic diagnosis b. Imaging i. Ultrasonography – shows “bull’s eye” or “coiled spring” lesion c. Treatment i. Fluoroscopic-guided hydrostatic/pneumatic enema |
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Juvenile polyps
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a. Labs
i. None; usually a clinical and radiographic diagnosis b. Imaging i. Colonoscopy; flexible sigmoidoscopy; hydrocolonic ultrasound (US after warm saline enema) c. Treatment i. Removal at colonoscopy; recurrence < 5% |
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Gastroenteritis
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a. Labs
i. Fever suggests infection with invasive bacteria or viruses; onset after food is clue ii. Stool cultures; fecal lactoferrin (for fecal leukocytes, controversial); occult blood b. Imaging i. Endoscopy can help distinguish IBD from infectious enteritis/colitis c. Treatment i. Oral rehydration solutions; abx; loperamide (in absence of fever, bloody stools); bismuth; probiotics |
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Irritable bowel disease
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a. Labs
i. Routine blood chemistries are normal ii. Stool cultures to rule out GI infection; lactose breath testing iii. C-reactive protein elevated in IBD, Crohn’s > UC b. Imaging i. Colonoscopy including terminal ileum; biopsy confirmatory rather than diagnostic c. Treatment Anti-inflammatory, steroids, MABs |
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Peptic ulcer disease
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a. Labs
i. Urea breath test for H. pylori infection b. Imaging i. Endoscopy; barium radiography if ineligible for endoscopy c. Treatment i. Thermal coagulation for bleeding ulcers; absolute alcohol or epinephrine injection ii. Fibrin sealant, endoclips introduced by endoscopy iii. Acid inhibition: PPIs >> H2 receptor antagonists iv. Somatostatin, octreotide |
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Celiac disease
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a. Labs
i. Second-generation anti-gliadin antibody test, Deamidated Gliadin Peptide (DGP) b. Imaging i. Upper endoscopy with 2nd/3rd portion duodenal biopsy c. Treatment i. Eliminate gluten from diet |
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Cholecystitis
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a. Labs
i. CBC (leukocytosis), fever; LFTs elevated; amylase and lipase b. Imaging i. Ultrasound: gallstones, thickened gallbladder wall (>3mm), pericholecystic fluid ii. Hepatobiliary iminodiacetic acid (HIDA) scan: acute if no uptake of radiolabeled bile c. Treatment i. Surgical excision, preferably laparoscopy |
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Cholangitis
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a. Labs
i. CBC (elevated WBCs), elevated alkaline phosphatase, GGT, bilirubin ii. Serum amylase elevation may suggest associated pancreatitis b. Imaging c. Treatment i. Charcot’s triad: RUQ pain, fever, jaundice ii. Reynold’s pentad: Charcot’s triad + hypotension and mental status change 1. Heralds significant morbidity and mortality ii. Carbapenem; 3rd gen cephalosporin and metronidazole |
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Hepatitis
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a. Labs
i. Chronic LFT elevation, think medications, ETOH, viral ii. Isolated elevation in bilirubin, get CBC to check for hemolysis iii. Elevated alkaline phosphatase may herald PSC or PBC, antimitochondrial antibody (AMA) suggests PBC b. Imaging (ultrasound) c. Treatment |
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Pancreatitis
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a. Labs
i. CBC: WBCs elevated, Hct depressed in setting of hemorrhage ii. Amylase elevated more than lipase b. Imaging iii. CT with oral, IV contrast (presence of pseudocysts, necrosis, masses) c. Treatment i. Aggressive fluid and electrolyte replacement; surgery only for infection or severe pancreatic necrosis |
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Appendicitis
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a. Labs
i. Mild fever, CBC shows mild leukocytosis b. Imaging i. CT with oral, IV contrast c. Treatment i. If presenting 24-72 hours since onset of symptoms, surgery ii. If greater than 5 days since onset of symptoms, wait 6 months then remove |
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Pyelonephritis
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a. Labs
i. Urinalysis for pyruia, white cell casts (if no pyuria, consider obstruction) ii. Urine culture, antimicrobial susceptibility; urine gram stain b. Imaging i. Only in severe or refractory cases: ultrasound, CT (obstruction, abscesses) c. Treatment i. Levofloxacin 500-750mg PO qd; ciprofloxacin 500mg PO bid |
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Pneumonia
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a. Labs
i. Outpatients: usually unnecessary due to effectiveness of empiric treatment ii. Inpatients: sputum for gram staining, blood and sputum cultures b. Imaging i. Chest radiograph – lobar consolidation, interstitial infiltrates, and/or cavitation c. Treatment i. Hospitalization if can’t maintain oral intake, substance abuse, cognitive impairment ii. Azithromycin 500mg qd x3 days or 2g IM |
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Pelvic inflammatory disease
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a. Labs
i. Urine beta-hCG; urinalysis; stool hemoccult ii. CBC not very useful, less than half of PID patients have elevated WBCs iii. Vaginal fluid: gram stain, WBC elevation b. Imaging i. For very ill patients; transvaginal ultrasound shows thickened, fluid-filled oviducts c. Treatment i. Abx as dictated by culture results |
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Ovarian torsion/ruptured ovarian cyst
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a. Labs
i. Urine beta-hCG ii. Hemorrhage = anemia; ovarian necrosis = leukocytosis; vomiting = electrolyte abnormalities iii. Ovarian torsion associated with elevated IL-6 b. Imaging i. Ultrasound (+/- Doppler) for lesions; CT or MRI to detect torsion c. Treatment i. Laparotomy and detorsion; removal if obvious necrosis |
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Nephrolithiasis/ureterolithiasis
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a. Labs
i. Urinalysis for hematuria in patients with unilateral flank pain b. Imaging i. Non-contrast helical CT scan; ultrasound for patients who must avoid radiation; abdominal plain film will miss small or radiolucent stones c. Treatment i. Many stones pass on their own ii. Shock wave lithotripsy (SWL), ureteroscopic obliteration, percutaneous nephrolithotomy, laparoscopy |
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Lung cancer (1˚ or 2˚)
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a. Labs
i. Sputum cytology (screening) ii. BMP, LFTs for metastatic workup (increased Ca2+ may indicate bone metastasis) b. Imaging i. Chest X-ray, chest CT ii. PET, radionuclide bone scintigraphy for metastasis c. Treatment i. Lobectomy or other resection when possible, concurrent chemotherapy for more advanced ii. Prophylactic radiation for SCLC may reduce brain metastasis iii. SCLC is usually disseminated at presentation; NSCLC may be local |
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Diverticulosis/-itis
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a. Labs
b. Imaging i. CT scanning with oral and IV contrast: bowel wall thickening, increased soft tissue density within pericolic fat; phlegmon, abscess ID c. Treatment i. Uncomplicated: bowel rest, abx (Cipro+metro PO; Zosyn IV); clear liquids, high fiber (1/3 recurrence) ii. Complicated (peritonitis, obstruction, perforation, abscess): amp/gent/metro iii. Percutaneous drainage then surgery for abscess |
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Acute myocardial infarction
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a. Labs
i. ECG: ST elevation, ST depression, T-wave inversion ii. Elevated troponin I/T (within 2-3 hours) more specific than creatine kinase-MB (CK-MB) b. Imaging i. None c. Treatment i. Medical treatment including aspirin |