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34 Cards in this Set
- Front
- Back
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What is the source of rotenone?
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roots of the Derris plants
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What is the use for rotenone?
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in flea control products
premise insecticides control of external parasites, intentional killing of fish in lakes |
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What is the absorption and metabolism of rotenone?
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absorption- good in oily food, metabolized in the liver to toxic(-OH) and nontoxic products
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What spp. is most sensitive to rotenone toxicity?
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cats and birds
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What are some clinical signs you can see with rotenone toxicity?
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vomiting, gi irritaion, respiratory depression, and dyspnea
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How is rotenone toxicity best diagnosed?
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blood analysis reveals incidence of hypoglycemia
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What is the treatment given for rotenone toxicity?
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diazepam-control seizures
dextrose- given IV |
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What is contraindicated with rotenone toxicity?
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phenothiazine tranquilizers
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What is the specific antidote given for rotenone toxicity?
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there is no specific antidote available
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What spp. is Amitraz used on?
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cattle, dogs, adn pigs
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What is the MOA of Amitraz
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its an alpha-adrenergic agonist and MAO inhibitor
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What organs are targeted with Amitraz?
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CNS and cardiovascular system
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What clinical signs can be seen with Amitraz intoxication?
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vomiting, diarrhea, ataxia, seizures
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What is Citrus Oil Extracts used for?
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control of lice, ticks, and fleas
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What system is most effected with Citrus Oil Extracts?
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Nervous system
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What clinical signs are seen with citrus oil extract toxicity?
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ataxia, vasodilation, CNS depression
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What is boric acid used for?
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used to control ants and roaches
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What spp. is most effected by toxicity of boric acid?
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cats, and to a lesser extent dogs
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Where is the site of greatest damage in boric acid toxicity?
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in the kidney due to accumulation
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What are some clinical signs that are observed with boric acid toxicity?
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GI tract disturbances
Muscle weakness CNS depression Comprimised renal function Metabolic acidosis Occassional tremors and seizures |
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What are the characteristic lesions associated with boric acid toxicity?
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gastroenteritis
nephrosis cerebral edema hepatic and renal fatty degeneration |
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What is the common name for N,N-diethyltoluamide?
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Deet
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What is the use of deet?
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used as an insect repellant
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What is the MOA of Deet?
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it is neurotoxic
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What are some clinical signs that are evident in people affected with deet toxicity?
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confusion, acute manic psychosis, headaches, ataxia, tremors, encephalopathy, convulsions
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What signs are seen with acute toxicity to deet?
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seizures, hypotension, bradycardia, coma
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What agent is present in mothproofing products?
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napthalene
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What are clincal signs seen with napthalene?
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vomiting, hemoglobinuria, Heinz bodies
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What is the source of molluscacides:metaldehyde?
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pellets and baits for slugs and snails
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What three things are decreased in the brain due to toxicity?
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GABA, Norepinephrine, and Serotonin
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What imbalances can be observed with metaldehyde toxicity?
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high fever, and electrolyte and acid-base imbalance
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What metabolic process is seen with metaldehyde toxicity?
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metabolic acidosis
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What is the most popular molluscacide which has no specific hazard?
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copper sulfate
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What molluscacide is receiving a growing amount of attention?
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pentachlorophenol
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