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33 Cards in this Set

  • Front
  • Back
what is syncope cause by?
global hypoperfusion and focal hypoperfusion of the reticular activating system
Things to differentiate syncope from?
siezures, vertigo, coma, drop attacks
Hemodynamics during syncope?
CO drops, SVR drops, MAP drops
4 causal agents of syncope?
reflex mediated, cardiac, orthostatic, cerebrovascular
Types of reflex mediated syncope?
vasovagal syncopy (faint), carotid sinus (hypersensitivity), situational, glossopharyngeal/trigeminal neuralgia
Primary vs secondary orthostatic syncope?
Prim: multi system atrophy, autonomic failure
Secondary: Parkinsons, vol depletion, meds, DM/neuropathy
cardiac syncope?
mechanical/structural, arrythmia, sinus node dysfunction
Vascular steal syndrome?
retrograde blood flow in the vertebral artery associated with subclavian occlusion/stenosis
causes of cerebrovascular syncope?
vertebrobasilar (TIA), vascular steal, seizure, migraine
High risk cardiac findings in syncope?
valve hrt dz, prior mi, prior cabg, heart failure, artificial pacemaker
High risk brain findings in syncope?
seizure, stroke, brain tumor, meningitis, brain injury
most specific and sensitive ways to eval syncope?
History and phys
H/P of syncope the acount must include?
circumstances surrounding the episode, precipitating factors, activity the pt was involved in prior to event, patients position when it occured, prior fainting, dizziness, light-headed, vertigo, weakness, diaphorisis
duration of symptoms preceeding a syncopal episode?
vasovagal: 2.5 min
arrythmia: 3 sec
Exstensive review of systmes should be conducted on ___ during a syncope exam?
cardiac, neurological, metabolic, medication, psychiatric
ROS of cardiac should include?
murmurs, rule out cardiovascular causes, difference in pulse pressures of each arm, subclavian steal, aortic dissection
ROS of Neuro should include?
parkinsons, orthostatic hypotension, epilepsy, narcolepsy
ROS of metabolic disorders should include?
DM, hypoglcemia
ROS of medictions should include?
antihypertensives, antidepressants, antiarrythmias, QT prolonging agents
Tachycardia may indicate?
pulmonary embolism, hypovolemia, tachyarrythmia, ACS
Bradycardia may point to?
vasodepressor cause of syncope, cardiac conduction defect, ACS
occurs in 70% of patients experiencing true syncope?
dizziness, light-headed, faintness
changes in BP and heart rate may indicate?
orthostatic cause of syncope
ECG abnormalities suggesting syncope?
bifasicular block, QRS duration >0.12 sec, mobitz type 1, pre-excited QRS complex, prolonged QT interval, RBBB, neg t waves (epsilon waves)
additional testing in syncope?
CT, MRI, EEG, carotid ultrasonagraphy (these should be ordered when nuero exam suggests it) Holter monitor, ECG, exercise test, EP study, HUTT
look at life threatening causes, non as well
??
MCC of non-life threatening syncope?
vasovagal
Probable seizure cause?
CT scan, EEG if event within pas 24hrs, neuro consult
probable cardiac syncope?
admit to telemetry, obtain ECG, cardio consult, EP consult if: previous MI, LVEF <40%, LBBB/IVCD on ECG, Hx of recurrent VT
Probable orthostatis syncope?
<40: eval blood loss, eval relative hypovolemia, discharge
>40: blood loss, polypharm likely, eval for hypvol, discharge
Other syncopal causes above 40 below 40?
below dischage home is H/P and ECG are unremarkable.
above consider 24 hr monitoring, if no dysrhythmias and normal ECG discharge home
How is global cerebral perfusion maintained?
feedback system involving CO, SVR, arterial pressure, volume status, cerebrovascular resistance.
Life threatening causes of syncope
Cardiac dysrhythmias: Long QT, brady/tachy, WPW
Cardiac outflow:
Cardiac ischemia
PE, dissection, hemmorhage