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121 Cards in this Set

  • Front
  • Back
What is the most inaccurate way to take temp measurement?
Axillary
What temp measurement has a wide variability?
TM
What temp measurement is affected by ventilation?
Oral
What temp measurement needs a hypothermia thermometer?
Rectal
__ is the temp of deep structures of body that usually don't vary >1-2 degrees from normal temp and maintained by metabolism
Core temp
__ is generation of energy where heat is the byproduct
Metabolism
What regulates body temp?
Hypothalmus
__ is when heat production exceeds heat loss
hyperthermia (inc body temp; core temp>101 degrees F)
__ is when heat loss exceeds heat production
hypothermia (dec body temp; core temp<35 degrees C/ <95 degrees F)
What are the 5 mechanisms of heat loss/transfer?
convection, radiation, respiration, evaporation, conduction
__ is heat loss directly into the environment by electromagnetic waves; larger the temp gradiant, the larger the heat loss
Radiation (55-60% heat loss)
__ is heat transfer from object to another by direct physical contact (i.e. water chill and water immersion)
Conduction (3% heat loss)
__ is transfer of heat thru circulating air; effect dec as temp inc (i.e. wind chill)
Convection (10% heat loss)
__ conversion of liquid to gas by sweating and cooling effect occurs thru drying of sweat on surface of body; ambient temp inc, primary way to eliminate heat (i.e. humidity)
Evaporation (25% heat loss)
__ accounts for 2-9% heat loss
Respiration
What are the factors that affect body temp?
physical condition, age, nutrition/hydration, environmental condtions
What are the 4 types of local cold emergencies?
nonfreezing:
Chilblains
Trench foot

freezing:
Frostnip
Frostbite
__ is a localized condition in which specific body tissues freeze; water lies in and around the cells damaging the cells when the water freezes eventually causing gangrene
Frostnip
__ is caused by chronic exposure to damp nonfreezing ambient temp leading to painful, inflammatory lesions on skin, pruritus, burning, and paresthesias
Chilblains (Pernio)
tx of Chilblains
supportive: gentle rewarming, bandage, elevate
__ is caused by prolonged exposure of foot to cold water leading to tissue damage thru soft tissue cell, blood vessels, and nerves; skin becomes pale, mottled, and anesthetic (sloughing, gangrene possible)
Trench foot (immersion foot)
tx of Trench foot
local skin care, elevation, rest
__ is freezing of the skin but not the deeper surface; extremity appear pale, discomfort present, no extracellular ice crystal formation
Frostnip
tx of Frostnip
rewarming
__ is localized cold injury causing freezing of tissue (usually toes, feet, hands, fingers, nose, ears); permanent inflammation, tenderness, cold intolerance (gangrene possible)
Frostbite
tx of Frostbite
save as many cells as possible

pre-hospital tx: remove from cold/wet clothing, rapid transport, protect extremity/elevate, splint extremity, remove jewelry (DON"T rub/massage, allow refreezing, allow smoking, puncture blisters, apply heat, rewarm, use affected extremity)

ED tx: correct hypothermia, rapid rewarm (104-108 degree F bath), wound care, parenteral analgesics, tetanus prophylaxis, debridement, aloe vera cream, elevation, ibruprofen, hydrotherapy, penicillin G
What is the difference between superficial and deep Frostbite?
Superficial: affect dermis & shallow subcutaneous layer

Deep: affect dermal & subdermal layers
S/S of local cold injury
Early/Superficial: blanching, feeling/sensation loss, soft skin, tingling sensation on rewarming

Late/Deep: white/waxy skin, firm/frozen, swelling, blisters, flush/purple/blanche/mottled/cyanotic when thawed
What is Phase I of Frostbite?
exposure to cold, vasoconstriction, dec blood flow to periphery, ice crystal formation in extracellular space, ischemia, cellular dehydration, hyperosmolarity, edema, inc pressure, blood vessel damage, worsened ischemia, cellular component destruction
What is Phase II of Frostbite?
tissue rewarmed, blood flow return, damaged capillaries leak fluid, swelling, sludging blood, thrombus formation
Name Frostbite injury:
__ is partial skin freezing; redness, mild edema; firm white/yellow plaque in area of injury, lack of blisters
1st degree injury
Name Frostbite injury:
__ is superficial skin blisters with clear/milky fluid, surrounding erythema/edema
2nd degree injury
Name Frostbite injury:
__ is full-thickness skin/ subcutaneous freezing; hemorrhagic blisters, skin necrosis, bluish-gray discoloration; extend into deep dermis
3rd degree injury
Name Frostbite injury:
__ involves tissue below dermis, with muscle/bone involvement; digit/extremity eventually becomes dry, black, mummified
4th degree injury
What is the difference between primary hypothermia and secondary hypothermia?
Primary (accidental): spontaneous reduction in body temp from cold exposure without adequate protection

Secondary: occurs as complication with systemic dz that compromises body's thermoregulatory mechanisms
Risk factors for hypothermia
extreme ages
chronic illness
drug use/poisons (etoh, barbs, benzos, phenothiazines)
cold environment
dementia
insufficient clothes/shelter
outdoors sports
What are the 3 levels of hypothermia?
mild: core body temp 93.2-96.8 degree F
mod: core body temp 86-93.2 degree F
severe: core body temp <86 degree F
S/S of mild hypothermia
mild-strong shiver
fine motor/complex motor skill loss
lack of sound judgment
confusion, apathy
pale/cool/red/cyanotic skin
piloerection
rapid pulse/resp
nausea
visual disturbances
lethargy
warm sensation/paradoxical undressing
S/S of mod-severe hypothermia
fine motor coordination loss
slurred speech/violent shiver
irrational behavior
dazed consciousness
mood changes/lethargy/impaired judgment
stiff muscle/joints
shiver stops
muscular activity dec/stop
pale, cyanotic lips/ears/fingers/toes
S/S of severe hypothermia
dec vital signs
shallow/absent resp
cardiac arrhythmias
slow pupillaryrxn
cardioresp activity cessation
skin pale
pupils dilated
bradycardic
shiver stops
fetal position
muscle rigidity (dec peripheral blood flow/inc lactic acid & CO2)
What is seen on ECG with hypothermia?
bradycardia
small/absent P waves
abnormal ST seg/ T waves
J (Osborn) waves
What are the 3 methods of rewarming?
Passive external rewarming (PER)
Active external rewarming (AER)
Active core rewarming (ACR)
When and how is PER used?
mild hypothermia
includes: shelter, blankets, heated environment, removal of wet clothing
When and how is AER used?
mod-severe hypothermia
includes: warm bath, hot water bottles, heat lamp, heating blankets
When and how is ACR used?
mod-severe hypothermia
includes: heated humidified O2, warmed IVF, warm fluid lavage (NG, peritoneal, thoracic, colonic), extracorporeal rewarming
How is extracorporeal rewarming accomplished?
hemodialysis, arteriovenous extracorporeal rewarming, cardiopulm bypass
Causes of dec heat dispersal
dehydration, obesity, heavy/impermeable clothing, poor physical fitness, lack of acclimatization, CVD, skin changes, extreme ages, lack of mobility, febrile illnesses, hyperthyroidism, alcoholism, drug use (cocaine/amphetamines/opiates/LSD/PCP), poor socioeconomic, prolonged exertion, meds (antipsychotics/anticholinergics/CCB/BB/diurectics/alpha agonists/sympathomimetics)
What are 4 types of heat illnesses?
minor heat illness
heat exhaustion
heat stroke
unusual heat d/o
What is included in minor heat illness?
heat rash
heat edema
heat cramps
heat syncope
heat tetany
__ is a skin ailment marked by small/itchy rashes that is especially common in tropics/summer and children/infants
Miliaria (sweat rash/prickly heat)
tx of Heat rash
cool/less humid environment
cool showers/baths
loose clothes/light fabric
mild cortisone cream/calamine lotion/oral antihistamines/powder
__ is d/t cutaneous vasodilation/ orthostatic pooling of interstitial fluid in extremities most often seen in elderly/non-acclimated travelers; tx self limited/ leg elevation
Heat edema
__ is painful spasmodic contractions of skeletal muscle that usually occurs after exercise/latent period; tx with rest, oral salt(0.1%) rehydration, IVF(0.9% saline)
Heat cramps
__ occurs (esp elderly) to hot exposure resulting from cumulative effect of peripheral vasodilation, relative hypovolemia, dec cardiac output, and dec cerebral perfusion; tx with rehydration, removal from heat, rest, support hose, flex legs, and sitting
Heat syncope
__ is hyperventilation resulting in resp alkalosis, paresthesia, and carpopedal spasm; tx with removal from heat and dec resp
Heat tetany
__ is a clinical syndrome characterized by vol depletion by heat stress exposure that can occur at rest with young/old people;
Heat exhaustion
S/S of heat exhaustion
dizziness, weakness, fainting, dry tongue/mouth, inc thirst, cold/clammy skin, normal-inc vitals/body temp
dx and tx of heat exhaustion
dx: H+P, CBC, lytes, LFT, BUN/Cr, urine specific gravity

tx: remove clothes/hot environment, cool mist/fan/packs to neck/axilla/groin, correct fluid/lytes, rest
__ is an acute dangerous rxn to heat exposure that results when body fails to cool sufficiently (105 degree F/ 40 degree C) leading to brain damage, hot/flush skin, behavior change, dec LOC, slowing of rapid pulse, drop BP, and death
Heat stroke
heat stroke classic triad
temp>40.5 degee C/ 104.9 degree F
CNS dysfunction
anhidrosis
What are the 2 types of heat stroke?
exertional
classic
What is exertional heat stroke?
d/t vigorous activity
heat builds up faster than removed
hypothalamus damage
heavy sweating
What is classic heat stroke?
slow evolution/onset insidious
inc exogenous heat gain with dec heat dispersal
risks: elderly, poor, infant, chronically ill, lack of AC, CV dz, CV/anticholinergic drugs
S/S of heat stroke
hot dry flush skin
rapid-slow pulse
HA/dizziness/irritability
weakness, n/v
confusion, disorientation
behavior change, unresponsiveness
coma, death
sweating, sz, hypoglycemia, resp alkalosis, lactic acidosis
DDx of heat stoke
meningitis, encephalitis, status epilepticus, falciparum malaria, meds, malignant hyperthermia, DTs, typhoid fever, hypothalamic hemorrhage, thyroid storm
dx and tx of heat stroke
dx: H+P, CBC, lytes, cx, clotting studies, LFT, lactate, CK, myoglobin, pancreatic enzymes, LP, ECG

tx: stabilization, monitor, correct lytes, cooling, supportive (no antipyretics)
benzos - shiver
fluid/mannitol - myoglobinuria
Insects, spiders, and scorpions are what type of species?
arthropods
Pit viper, coral snakes are what type of species?
reptiles
What are the MC bite/sting death?
insects (50%)
snakes (30%)
spiders (14%)
other (6%)
Hymenoptera includes what families?
honeybees, wasps, yellow jackets, hornets, fire ants, harvester ants
What is contained in Hymenoptera venom?
histamine, serotonin, amines, phospholipase, hyaluronidase
What are the 5 types of reactions from bite/sting?
local rxn
toxic rxn
anaphylactic rxn
delayed rxn
unusual rxn
__ is sudden sharp, burning pain that usually disappears over few hrs accompanied with edema, erythema, itching, wheal at site and airway obx (if around throat)
local rxn
__ is dramatic swelling, redness, and pain that persist for up to a wk and area adjacent to site may be involved including airway loss with tongue/throat sting
large local rxn
__ is when >10 stings induce systemic rxn secondary to large toxin load leading to vomiting, dizziness, syncope, edema, diarrhea, and possible convulsions/death
toxic rxn
__ is major cause of death assoc. with bee/wasp sting usually occurring within 1/2 hr of sting which induces antigen-antibody, IgE mediated systemic response (histamine/SRS-A released)
anaphylactic rxn
S/S of anaphylaxis
pruritis, generalized urticaria, dry cough, wheezing, angioedema, laryngeal stridor, anxiety, distant edema, dyspnea, bronchospasm, cyanosis, ab cramps, n/v, shock, LOC, hypotension, death
__ is serum sickness like presentation appearing ~2wk after sting with sx of HA, malaise, fever, polyarthritis, generalized pruritus
delayed rxn
__ may present similar to encephalopathy, vasculitis, neuritis, autonomic dysfunction
unusual rxn
tx of local rxn
scrape out stinger
wash wound
ice packs
oral antihistamines/analgesics
elevation/oral steroids (mod swelling)
tx of anaphylactic rxn
ABCs
endotracheal intubation/surgical airway (Severe)
local wound care
IVF
antihistamine
steroids
H2 blockers
epinephrine (life-threatening)
albuterol nebulizer (bronchospasm)
dopamine infusion (hypotensive)
T/F: All spiders are venomous
True
What are the 2 dangerous spiders in the US?
black widow (Latrodectus mactans)
brown recluse (Loxosceles reclusa)
__ are found in all states except Alaska; forms irregular webs in wood piles/trash dumps/outdoor structures/under rock; only females bite humans; glossy black with red hourglass on abdomen; neurotoxin venom causing painful muscle spasm
black widow
Sx of black widow venom
local: immediate sharp stinging pain at site followed by cramping and numbness

systemic: dizziness, HA, nausea, itching, inc salivation, weakness

mimic acute abdomen/MI
What is the timing of sx with black widow venom?
onset: immediate
peak: few hrs
duration: <24hrs - 4 days
*most recover completely
tx of black widow venom
Pre-hospital: ice/ ID arachnid
stabilization
monitor
nitroprusside (BP>130mmHg)
local cleansing/tetanus admin
muscle relaxants/analgesia (muscle spasm)
anti-venom (old/young/pregnant/pre-exist conditions)
__ is a fiddle-back spider; violin shape on thorax; mc in SE/south central US; shy/nocturnal found in woodpiles, dry bark, dark closets, basements, attics; causes local tissue damage
brown recluse
What is the timing of s/s in brown recluse?
onset: sharp little/no pain with mild stinging
within 2 hrs: local pain, blue-gray halo with central dark blister/bleb leading to rupture/ulcer with severe pain
12-18hrs: bleb formation with ischemic zone
5-7d: aseptic necrosis, necrotic ulcer
What is the difference between mild/ severe systemic effects of brown recluse?
mild: fever, chills, malaise, n/v, joint pain

severe: DIC, hemolysis, renal failure, convulsion, HF, death
tx of brown recluse
wound cleanse with soap and H2O
tetanus prophylaxis
monitor
steroids (within 24hrs) - no Dapsone
abx/analgesics
wound management/ surg consult
__ are nocturnal arachnids found mostly in SW US with only 1 potentially lethal type
scorpions
Presentation of Centuroides sculpturatus
neurotoxin venom
only lethal scorpion in US
Arizona/New Mexico mc
sx: extreme restlessness, agitation, roving eye movements, poor coordination, slurred speech, difficulty swallowing, salivation, wheezing, stridor, tachycardia, tachypnea, htn, n/v
tx of Centuroides sculpturatus
antivenin
ventilator support
atropine
diazepam
opiates (contraindicated)
When are snake bites mc?
apr-oct (jun/aug peak)
2pm-9pm
18-28 y/o
men
fingers/hands
ethanol intoxication (risk factor)
What are the 4 types of poisonous snakes in the US?
rattlesnake
cooperhead
cottonmouth
coral snake
Where are venomous snakes NOT found?
Maine, Alaska, Hawaii
Rattlesnakes, Copperheads, and Water moccasins (cotton mouth) are what type of venomous snakes?
pit vipers (crotalidae)
T/F: Colubrids are non-native to the US
True
Where do pit vipers store their poison and how do they inject it?
stored: pits behind nostrils
inject poison to victum thru movable front fangs
Characteristics of pit vipers
heavy bodies, diamond-shaped heads, vertical/elliptical pupil, heat sensing pit, erectile fangs, rattle tails, hemotoxic/neurotoxic venom
What are the 2 common rattlesnakes of US?
western diamondback and eastern diamondback
What is the difference between eastern diamondback and western diamond back?
eastern diamondback: largest, sullen disposition, 1in fangs, toxic venom, 5-7ft length

western diamondback: large amt venom in 1 bite, 4 1/2-6 1/2ft length
__ is CT species of mild temper that rattles with positive fatalities averaging 3 1/2-6 1/2ft length
timber rattlesnake
__ has very rare deaths, minimal edema and pain
Copperhead
__ is related to copperhead; >danger; highly toxic venom; South US; dark/black; 3 1/2-6 1/2ft length
water moccasin
__ are small thin bodies snakes with red/yellow/black bands, small fixed front fangs, small rounded head, injects venom with teeth, neurotoxic venom
coral snake (elapids)
S/S of coral snake bite
little/no pain/swelling
tingling
muscular incoordination
weakness
inc salivation
difficulty swallowing/talking
visual disturbances
resp distress/failure
shock
What is composed of crotalid venom?
mix of enzymes, polypeptides, glycoproteins (tissue/nerve/capillary damage, hemolysis, host cell/coagulation factors breakdown)
What is composed of coral snake venom?
neurotoxin and enzymes (systemic neuro sx)
__ is local swelling and other local changes; no systemic manifestations; normal lab findings
minimal envenomation
__ is swelling progressing beyond site of bite and >1 systemic manifestations (e.g. abnormal lab findings)
moderate envenomation
__ is marked local response, severe systemic manifestations, and significant alteration in lab findings
severe envenomation
tx of snake bite
pre-hospital: transport
ed management: stabilize, exam, prophylaxis, debridement, antivenin (within 4hrs-questionable after 12hrs)
Adverse rxn of snake bite antivenin
serum sickness
anaphylaxis
contraindications (low severity/known sensitivity to horse/horse serum)
What should not be utilized in snakebite management?
ice, arterial tourniquets, cut/suck, electrical shock, locating snake, bringing live snake to hospital
What are the 3 classes of marine envenomation?
envenomation by bite (sea snakes, octopus)
envenomation by nematocyst (coral, anemone, jellyfish)
envenomation by stinger (sea urchinsm, stingray, lionfish)
__ are jellyfish/Portuguese man-of-war; responsible for more envenomation than any other marine life; nematocyte stinging cells
coelenterates
S/S of nematocytes
intense burning pain
red hemorrhagic lesions
n/v
fever chills
angioedema
dyspnea, wheez, stridor
hypotension, shock
resp arrest
CV collapse
tx of nematocytes
Rinse affected area with seawater, inactivate nematocysts with 5% acetic acid (vinegar), and remove any tentacles to prevent further injury.
Avoid irrigating wound with fresh water b/c can stimulate release of toxin.
T/F: For Portuguese man-of-war stings, saltwater rinse can be used. Vinegar should not be used because it can activate undischarged nematocysts.
True