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79 Cards in this Set

  • Front
  • Back
what are the advantages of laproscopic sx
*mimimizes surgical trauma
*less post-op pain
*improved post-op pulmonary fxn
*shorter hospital stays
*earlier return to normal activities
with a laproscopy there is decreased tissue damage that is evidenced by what
*decreases in C-reactive protein

*decreaeses in interleukin-6
what occurs with the hyperglycemic response with laproscopy compared to laparotomy
it is decreased
is there a benefit in reduction of adrenocortical response with laproscopy
NO
what occurs with excretion of cortisol and catecholamines with a laproscopy compared to a laparotomy
they are UNCHANGED
a laparotomy has what type of pain
incisional pain
a laproscopy has what type of pain
*more visceral (spasm) pain

*shoulder pain from diaphragmatic irritation
what are some tech to minimize pain following a laproscopy
*pre-op NSAIDs

*intraperiotoneal local anesthetic

*more complete evacuation of pneumopertineum
pre-op NSAIDs for minimizing pain after a laproscopy are def effective in what procedures
*lap chole

*GYN laproscopy except BTL
intraperitoneal LA for minimizing pain after a laproscopy is particularly benefical in what procedures
GYN
a mesosapinx block is valuable for minimizing pain after a laproscopy in what procedure
Lap BTL
when is a mesosapinx block more effective
when perfomed at the beginning of the procedure
with a laproscopy greater severity of pulm dysfunction and slower recovery is reported in what pts
*elderly
*obesity
*COPD
*smokers
what are the disadvantages of a laproscopy
*PONV

*2 dimensional image

*diff in maneuvering instruments
fredelig
peaceful
what are methods to reduce PONV with a laproscopy
*propofol anesthetic
*intra-op antiemetics
*supplemental analgesia to reduce opioid consumption
*empty stomach intra-op
what positions are common with pelvic and lower GI sx with a laproscopy
trendelburg often accompanied by lithomy
what positions are common with upper GI sx with a laproscopy
reverse trendlenburg
with a laproscopy and positioning CV and resp changes are directly related to what
steepness of tilt
what resp changes are produced with tredelenburg
*decreased FRC
*decreased lung volume
*decreased compliance
resp effects from trendelenburg are exacerbated when
*elderly

*obese

*pre-existing pulm compromise
what are the CV changes related to trendelenburg in a normal heart
no sig compromise
what are the CV changes related to trendelenburg with poor ventricular fxn/CAD
increased myocardial o2 demand
what are the CV changes related to reverse trendelenbug
decreased CO
what may further decrease CO in reverse trendelenburg
increased SVR seen with pneumoperitoneum
venous stasis is exacerbated by what in regards to positioning
*reverse trendelenburg

*pneumoperitoneum

*lithtomy
what nerve injury is the most concern with trendelenburg
overextension of the shoulder
what nerves can be damaged in lithotomy position
*COMMON PERONEAL

*lateral femoral cutaneous

*femoral
what ventilatory changes occur with a pneumoperitoneum
*decreased compliance

*decreased FRC

*potential for VQ mismatch
what amt of change occurs with ventilatory compliance as a result of a pneumoperitoneum
30-50% reduction
to reduce the potential for a VQ mismatch with a pneumoperitoneum what should be done esp with CV compromise
avoid pressure greater than 14 mmHg
what occurs with PaCo2 with a pneumoperitoneum
it is INCREASED
what occurs with pH with a pneumoperitoneum
it is DECREASED
ETCO2 and PaCo2 plateau when with a pneumoperitoneum
about 20 min in
what is PaCo2 like with a local and a pneumoperitoneum and why
UNCHANGED d/t compensatory increase in MV
what is PaCo2 like with general anesthesia and spontaneous ventilation with a pneumoperitoneum
INCREASED despite increase MV d/t decreased compliance & vent depression from anesthetics
a greater change in PaCo2 with a pneumoperitoneum is seen with what pts
sicker one
what are the causes of increased PaCo2 with a pneumoperitoneum
*ABSORPTION OF CO2 (#1)

*impaired ventilation and perfusion
with a pneumoperitoneum what do you like to keep the IAP at
</= 14
what are the resp complications that can occur from a pneumoperitoneum
*sub-q emphysema

*pneumothorax

*endobronchial intubation

*gas embolism
sub-q emphysema with a pneumoperitoneum is suggested by what
increase in ETCO2 follwing the intial plateau
what are the causes of sub-q emphysema with a pneumoperitoneum
*accidental extraperitoneal insufflation

*necessary extraperiotoneal insufflation
what is the management of sub-q emphysema with a pneumoperitoneum
*hyperventilation

*suspend insufflation/sx briefly until CO2 blows off

*continue mech vent at the end of case until ETCO2 returns to normal
what causes a capothorax/pneumothorax with a pneumoperitoneum
*passage of gas through weak points in diaphragm or at aortic/esophageal hiatus secondary to increased IAP
*rupture of pre-existing bullae secondary to increased MV
what is the dx for a capnothorax/pneumothorax secondary to passage of gas through weak points in diaphragm or at aortic/esophageal hiatus with a pneumoperitoneum
*increased airway pressure

*increased ETCO2

*auscultation/CXR
what is the dx for capnothrorax/pneumothorax secondary to rupture of pre-existing bullae 2 to increased MV with a pnemoperitoneum
*increased airway pressure

*DECREASED etco2

*ausculation/CXR
what is the management of a CO2 pneumothorax (capnothorax) with a pneumoperitoneum
*stop N2O if using
*adjust vent to correct hypoxemia
*PEEP
*reduce IAP
*resolves spontaneously secondary to CO2 very diffusable
what is the management of ruptured bullae (pneumothorax) with a pneumoperitoneum
*thoracentesis

*NO peep
endobronchial intubation with a pneumoperitoneum caused by
cephalad displacement of diaphragm following insufflation
what is the dx for endobronchial intubation with a pneumoperitoneum
*decreased o2 sat

*INCREASED airway pressure

*auscultation
what is the management for a endobronchial intubation with a pneumoperitoneum
pull the tube back
when does a gas emobolism with a pneumoperitoneum usually present
at the start of insufflation
why does a gas emobolism occur with a pneumoperitoneum
d/t placement of insufflating needle into a blood vessel or organ
what is the lethal dose of Co2
~5x that of air
a large volume of Co2 in RA or vena cava does what
obstructs venous return resulting in decreased CO or circulatory collapse
what is the dx of a gas embolism during laproscopy
*decreased ETCO2

*tachycardia

*hypotension

*millwheel murmur
what is the DEFINITIVE dx for a gas embolism during laproscopy
aspiration of gas or foamy blood from a central line
what is the management of a gas embolism with a pneumoperitoneum
*stop insufflation
*relase pneumopertineum
*steep trendelenburg
*left lateral decubitis position
*stop N2O
*100% o2
*central venous line to aspirate
*consider hyperbaric o2 tx if gas in cerebral circulation
what are the hemodynamic effects of a pneumoperitoneum in a NORMAL pt
*decreased CO
*increased BP
*min to no change in HR
*increased SVR
what is the change initially in CO with a normal pt and a pneumoperitoneum
10-30% decrease with insufflation
the change in CO initially with insufflation of the abd in a pneumoperitoneum with a NORMAL pt is proportional to what
increase in intra-abd pressure
the change in CO initially with insufflation of the abd in a pneumoperitoneum with a NORMAL pt is INDEPENDENT of what
pt positioning
the change in CO with insufflation of the abd in a NORMAL pt is related to what
*increased SVR

*decreased venous return
decreased venous return with a pneumoperitoneum occurs with what IAP
>10 mmHg
decreased venous return with a pneumoperitoneum is attenuated by what
*fluid preload

*wrapping legs or SCDs
increased SVR with a pneumoperitoneum is NOT a result of what
decreased CO
increased SVR with a pneumoperitoneum is affected by what factors
*pt postion

*volume status

*mechanically and neurohumorally mediated
what occurs with the renal system with a pneumoperitoneum
~50% reduction in UO, GFR and renal blood flow
what occurs with cerebral blood flow with a pneumoperitoneum
increased d/t elevated PaCo2
what should be done with pts at risk for changes in cerebral blood flow with a pneumoperitoneum
maintain normocarbia
what is the most common cardiac arrythymia in sx with a pneumoperitoneum
bradycardia
what is the primary cause of bradycardia in laproscopy with pneumoperitoneum
increase of vagal tone from stretch of the peritoneum
what is the tx for bradycardia with a pneumoperitoneum
*stop insufflation
*release pneumoperitoneum if needed
*atropine
when is bradycardia with a pneumoperitoneum less likely to occur
with a deeper plane of anesthesia
what are the concerns of sx during pregnancy
*premature labor

*damage to uterus

*teratogenicity of anesthetic agents
what things should be done with sx in the pregnant pt
*involve the OB
*minilap for trocar access
*fluid load
*maintain normal maternal ETCO2
*left uterine displacement
what is the safest approach for anesthesia with a laproscopic sx
general with ETT
what are the advantages of using inert gases for laproscopy
*PaCo2 not elevated

*smaller increase in BP
what are the disadvantages of using inert gases for laproscopy
*greater decrease in CO

*low blood solubility