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17 Cards in this Set

  • Front
  • Back
Complex cardiovascular changes in OSA
1)increased venous return,increased afterload
2)decreased Spo2
Risks of Untreated OSA
1)Hypertension
2)Cardivascular dx.
3)stroke
4)daytime sleepiness
5)MVA
6)diminished quality of life
Treatments for OSA
1)Lifestyle modifications
--weight loss
--sleep position training
--alcohol, drugs, tobacco
2)CPAP
3)Oral appliances
4)Surgery
--airway
--cranio-facial
What CPAP reverses
1)Sleepiness
2)HTN
3)Cor pulmonale
4)respiratory failure
5)intellectual impairment
6)depression
7)increased mortality
CSA
1)Decreased or no respiratory effort
2)affects CNS
3)Increases incidence of
--degenerative muscle disorders
--strokes
--decompensated CHF
Risks of untreated CSA
1)Pulmonary htn
2)cardiac arrythmias
3)systemic htn
4)cardiac failure
Treatment for CSA
1) CPAP or Bilevel with timed mode
2)Adapto-servo
3)supplemental O2 for decompensated CHF
PLMS
1)Abnormal limb movements at night
2)EDS
3)complaints about sleep onset and maintenance
4)restlessness
PLMS signs and symptoms
1)Twitching
2)night-to-night variability
3)sleep fragmentation
4)bed partners complain
who is affected by6 PLMS
1)5-15 percent of pop
2)more frequent in elderly
3)prevalent in pregnant women
4)neuropathy
--renal failure
--liver disease
5)iron deficiency
causes of PLMS
1)abnormal iron metabolism
2)medications
--sedatives
--withdrawal from ssri's
3)90 percent of people with Restless Leg syndrome have PLMS
Diagnosis of PLMS
1)Sleep study
2)Leg EMG
3)May or may not be associated with arousal
4)Predominately first 1/3 to 1/2 of night
Narcolepsy
1)250,000 Americans
2)Familial cases
3)Genetic testing
4)80 percent have onset before 25 years
5)noncturnal sleep disruption
6)May have other sleep disorders
Narcolepsy atttributes
1)Marked EDS
2)Cataplexy (loss of skeletal muscle control, precipitated by emotion)
3)Hypnogogic/hyponopompic hallucinations
4)sleep paralysis
Pathology of Narcolepsy
1)blending of wake, REM, and NREM
2)cataplexy,hallucinations, and sleep paralysis are involuntary entry into REM
3)Sleep attacks are entry of NREM into wakefulness
chemistry of narcolepsy
deficit of orexin, a polypeptide in the brain, may be responsible for narcolepsy
Central Idiopathic Hypersomnia
1)Severe daytime sleepiness
2)need to nap multiple times a day
3)despite naps, continue to suffer from extreme sleepiness
4)Different from narcolepsy
--no sleep attacks
--unrefreshing naps or sleep times
--without cataplexy, hallucinations, or sleep paralysis