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15 Cards in this Set
- Front
- Back
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most common glom nephrosis in adults
EM shows |
spike and dome
membranous (diffuse membranous) |
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signs of glom nephrosis
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>3.5g protein/d
hyperlipid fatty casts edema frothy urine recurrent inf think major loss of proteins (Ig, albumin, etc) |
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most common casue of G nephrosis in kids
tx: |
minimal change
csc |
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changes on light microscopy of DM
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nephropathy
gbm thick mesaggial expansn kimmelstiel wilson lesion (nodular sclerosis) |
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mcc death in SLE
what do you see |
diffuse proliferative glom nephritis
granular IF (immune complex deposition) |
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most common glomerular diseaes in HIV
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gocal segmental glomerulosclerosis (type of nephrotic syndrome)
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granular IF
tram track subendothelial immune complex deposition type of nephrotic syndrome disease associ? |
membranoproliferative glomerulonephritis (can present as nephrotic)
HBV>HV |
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male with hematuria, hemoptyisi and pneumonitis and linear IF
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goodpasture's (rapidly progressive or crescentic G nephritis)
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nephritis + p-ANCA
nephritis + c-ANCA nephritis post URI or GI bug with hematuria and low grade proteinuria |
microscopic polyarteritis
Wegeners bergers (IgA nephritis |
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sx of nephritis
most common in kids |
inflamm (htn and periorbital puffy, red cell casts, oliguria, hematuria) <3.5/d azotemia.
acute poststrep. |
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SE of statins
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increases LFTs, myositis, warfarin potentiation
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lipoprotein lipase inhibitors are called ____
function? SE |
fibrates
increase VLDL and TG breakdown GI upset, cholelithiasis, myositis, increased LFTs |
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cholesterol absorption inhibotr that lowers LDLs
SE |
ezetimibe
ab pain, D, angioedema |
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this drug decrease hepatic synthesis of LDL and decreases fatty acid release from adipose lower LDL and raise HDL
SE |
Niacin
flushing (prevent with admin of ASA prior to NIacin), paresthesia, purrutis, GI upset, elevated LFTs |
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bile acid resins decrease LDLs by?
se |
binding intestinal bile acids and increasing catabolism of LDLs from plasma
Gi, constipation, decrased absorption of other drugs in sm intesting |
