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60 Cards in this Set

  • Front
  • Back

A pt with spinal shock will manifest which of the following symptoms?


A.hypertension, headache, flushing


B.Hypotension bradycardia, and lack of tendon reflexes


C.Hypertension, bradycardia


D. A lack of deep tendon reflexes


E.Hypertension, flushing bradycardia

D. A, lack of deep tendon reflexes (rational; lack of reflexes, lack of sensation, flaccid paralysis)

Automomic dysreflexia will result in

a. Hypertensionb.Hypotension

c.loss of sensation

a. Hypertension

A complete spinal cord injury that involves C3 will result in


a.Diaphragmatic breathing


b.Ormal respiratory function


c. Ventilator Dependent


d.Decreased tidal volumes

c. Ventilator dependent

Early treatment of a spinal cord injury may include


a.Nsaids


b.Anticoagulants


c. Steroids


d.D. morphine

c. Steroids

An injury to the spinal cord that results in loss of pain and temperature on the contralateral side and loss of motor function on the isisilateral side. This is likely what type of cord injury?

A.Brown SequardB.Anterior cordC.Central cord

D.Lateral cord

a. Brown Sequard

What are the three meninges around the spinal column?

Pia - innermost layer


Arachnoid - delicate middle layer


Dura - toughest

There is usually a primary and secondary injury to the spinal cord, how does the primary commonly occur?

Mechanical disruption, stretching or lacerating axons


Torn by direct trauma like by stabbing or gun shots

There is usually a secondary injury following the primary, how does this occur?

Ongoing progressive damage caused by ischemia, hypoxia, microhemorrhaging, and edema



These further progress the primary injury

What results from the secondary injury?

Apoptosis that may go on for weeks and months



Complete cord damage usually due to autodestruction



Hemorrhaging that infarcts the grey matter within 4 hours



Edema and compression of the spinal column due to lack of space

At what time is the extent of injury and prognosis most accurately determined?

72 hours

Spinal shock is characterized by

Decreases reflexes below injury, including bowel and bladder function



Loss of sensation



Flaccid paralysis below level of injury

Neurogenic shock is characterized by

Damage at or above T5



Loss of ANS function below injury leading to


Hypotension


Bradycardia


Warm, dry extremities


Peripheral vasodilation


Venous pooling


Decreased CO

The goal for neurogenic shock management are

Adequate perfusion with the following parameters



BP systolic should be 90-100


HR should be 60-100 sinus rhythm


Bradycardia be treated with atropine if applicable


Urine output > 30 ml/hr


Prevent hypothermia

Classification is based on

Mechanism of injury



Level of injury both skeletally (vertebral with the most damage)


Neurologically (lowest segment with function on both sides of the body)



Completeness or degree of injury

What scale combines both motor and sensory function?

American Spinal Injury Association Impairment Scale

What level of injury is most common?



a. Cervical


b. Thoracic


c. Lumbar?

Lumbar

Complete cord lesion results in...

total loss of sensory and motor function below the the level of the lesion

Incomplete cord lesion results in...

mixed loss of sensory and motor function


Brown sequard syndrome is...

Any presentation of spinal injury that is an incomplete lesion (hemisection) can be called a partial Brown-Séquard or incomplete Brown-Séquard syndrome.

Anterior cord syndrome and brown sequard syndrome both involve half damage to the spinal cord, but what's the difference?

Anterior cord syndrome involves variable loss of motor function, pain, and temperature. Proprioception remains intact (sense of how body is positioned)



Brown sequard syndrome an ipsilateral loss of (same side) of proprioception and motor function, however contralaterally loses pain and temperature.

What does central cord syndrome involve?

Motor function weakness in the upper extremities more than the lower



And distal extremities more than proximal

What is Conus medullaris syndrome?

a sacral cord injury that may involve lumbar nerves. Areflexia (absence of reflexes) in the bladder, bowels, and to a lesser degree the lower limbs. Motor and sensory loss to the lower limbs is variable.

What is Cauda equina syndrome?

An injury to the lumbosacral nerve roots caused by a central lumbar disk herniation. Areflexia of bladder, bowel, and the lower limbs. Motor and sensory loss to the lower limbs is variable. This is a nerve root injury rather than a spinal cord injury

Quadra/tetraplegia is...

paralysis in all extremities

Paraplegia is...

paralysis of the lower half of body

Quadraparesis

Weakness in all extremities

Paraperesis

Weakness in legs

What are the biggest concerns immediately after an injury?

ABCs



Airway


Ventilation


Circulation

Cervical injury at or above C3 can result in ___________ and the pt would then need to be ____________

Respiratory arrest; intubated, mechanical vent, tracheostomy

Injury at C4 or below would result in what kind of breathing?

Diaphragmatic breathing ==> hypoventilation

Because of reduced muscle control, how do you prevent atelactasis and pneumonia in these patients?

Chest physiotherapy


Assisted coughing


Tracheal suctioning prn


Incentive spirometry

At what level of injury would you be concerned about cardiac functioning?

T6

What type of drug would you use to treat bradycardia? What would the physician apply to the pt heart?

Anticholinergic drugs to increase HR


Pacemaker to regulate HR

What would you do to treat peripheral vasodilation; what type of drug? What kind of therapy?

Fluids and vasopressors

Why would you use intermittent catheterization rather than just leaving one in the bladder?

To prevent urosepsis, intermittent catheters are done q3-4h

What is a neurogenic bladder and what can be done for it

A bladder that is hyperreflexic or areflexic



Besides catheterization, a hyperreflexic bladder can benefit from anticholinergics, kegal exercises, bladder reflex training, and even surgery

Above the level of T5 we are concerned with a pt GI and may have a paralytic ileus, how is this managed?

NGT


Monitor electrolytes


Gradual return of fluids and food


High calorie and high protein


Stress ulcers occur within 6-14 days, so how should we test pt for them? What are the prophylatic drugs we give patients?

Stool occult blood



PPI


H2 receptor blockers

How to manage a neurogenic bowel

use stool softeners


get them up for physical activity


get them on bowel routine


Valsalva maneuver


Suppository or mini enema at end of day

Monitor albumin levels in pt for



a. skin breakdown


b. liver function

Skin Breakdown!

Pt commonly can't control their body temperature t/f?

True

Pulmonary embolisms and DVT are common in pt? t/f

True

How are spinal cord injuries diagnosed?

x-ray


Spinal films


ct scan


MRI


comprehensive neurological examination

How do you move a patient with a spinal cord injury? It's ________ rolling

Log rolling

What device is screwed to the head like a halo?

Cervical traction/ Halo Brace

What is the goal of surgical therapy?

To stabilize the spinal column

Why is methyprednisone (MP) - Solu Medrol given

Improve recovery of neurological function


within 8 hours of injury


improves blood flow


reduce edema


What complications are associated with MP - Solu Medrol

Immunosuppression


GI Bleeding


Infection risk

Why are vasopressors used?

Maintain MAP for spinal cord perfusion

There are a **** ton of nursing assessments needed to be done, but name some key ones

Bradycardia


Hypotension


Ability to cough


hypoventilation


bowel sounds


urinary incontinence

A family sees a patient respond to reflexes you've tested. The family becomes excited and say that the patient is getting better, what must you do?

Tell the family that the return of reflexes is not indicative for the return of function

Autonomic dysreflexia occurs at what level?

It occurs at T6 or above. It is life threatening and is a response to noxious stimuli shutting down both the PNS and SNS

What are some symptoms of autonomic dysreflexia?

Severe HTN (up to 300 mmHg systolic)


Throbbing headache


Bradycardia


Flushing above level of lesion


Diaphoresis


Blurred Vision

How do you manage autonomic dysreflexia?

Notify physician


Elevate hob 45 degrees


assess to determine cause


Remove the stimulus - remove all stimuli

Respiratory rehabilitation for SCI pt includes

Ventilator care


Assisted Coughing


Incentive Spirometry


Deep breathing

Male sexuality complications may include

Absent


Psychogenic only


Reflexogenic (short lived)


Only 10% of men are fertile


Erectile Dysfunction


Poor sperm quality

Female sexuality complications may include

Usually remain fertile


50% have orgasms


Menses may cease upwards of 6 months


Uterine contractions not felt


Health promotion involves supporting legislation on seat belts? T/F

True

Acute nursing interventions for patients with SPC include

Immobilization


Respiratory


Cardiac


Fluid and nutritional maintenance


Bladder/Bowel management


Temp control


Stress ulcers


Reflexes assessment


Autonomic dysreflexia

Help patients through the grieving process by promoting

Independence


Assist in anger control


Allow mourning


Expect wide fluctuations of emotions


Regression at different stages