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19 Cards in this Set

  • Front
  • Back
microbes which can infect healthy valves (4)
S. aureus
beta-hemolytic strep
N. meningitidis
H. influenzae
microbes which can only infect already damaged valves (2)
alpha-hemolytic strep
S. epidermidis
in contrast to RF, in infective endocarditis primary infection ___
can be anywhere in body
predisposing factors for IE (3)
pathological valve conditions
factors relating to general condition of host
immune compromise
pathologic valve conditions include (2)
previously damaged valves
pathologic jet streams
causes of pathologic jet streams (6)
mitral valve prolapse with regurg
hypertrophic cardiomyopathy with subaortic stenosis
coarctation of aorta
VSD
PDA
TOF
2 forms of IE
acute IE
subacute IE
___% of patients with AIE die
___% of patients with SIE live 5 years
50
90
differences of IE from RF (4)
vegetations are bulky
vegetations can be anywhere on valve
vegetations can be on any valve
inflammation is purulent
___ is a possible consequence of IE. that in turn can cause
autoamputation of valve
severe embolism
systemic complications of IE (4)
septic shock
septic emboli
nonseptic emboli
immune complications
immune complications from IE include (2)
small vessel vasculitis
glomerulonephritis
emboli from IE can cause ___
multiple infarcts
___ are an ocular sign of IE. they are ___
roth spots
retinal microemboli
___ (2) are signs of IE on the skin.
petechiae
subungual hemorrhages
nonbacterial thrombotic endocarditis is caused by ___, such as ___ (3). it is a misnomer because
hypercoagulable states
trousseau syndrome
DIC
extensive burns
it's not an inflammatory state
NBTE vegetations are __ and are located ___. like IE they ___. unlike IE, they ___ (2)
sterile
on free edge
can be on any valve
are small
do not cause global thickening of leaflet
Libman-Sacks endocarditis is associated with ___ (2). Both are ___ hypersensitivity reactions
SLE
antiphospholipid syndrome
type 3
LSE vegetations may be found (2)
on both sides of valve
on any part of valve