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36 Cards in this Set

  • Front
  • Back
Tracheal (Bronchial)
Heard at the intrethoracic trachea and major bronchi

loud, hard and tubular

High pitch

Exp. may be longer than insp. with a slight pause between
Bronchovesicular Sounds
Upper half of sternum anteriorly
-between scapula posteriorly

More muffled then tracheal sounds

Insp. and Exp are equal in length with no pause

Moderate pitch
Vesicular Sounds
Over lung parenchyma

soft muffled sounds

Low pitch

Insp. louder than exp

Insp lasts 3x longer than exp

Exp begins immediately after insp and rapidly fades out
Types of abnormal breath sounds
Absence of a normal breath sound

Normal BS heard in an area of the chest where it isnt normal

Adventitious BS
Causes of absence of normal breath sounds
Patient not breathing

Improper use/function of stethoscope

Lung Pathophysiology
-Atelectasis
-Pneumothorax
-Pleural effusion
Clinical Implications of hearing bronchial bs where you should hear vesicular bs
Bronchovesicular - Partial Consolidation

Bronchial/Tracheal - Complete Consolidation
Continuous Adventitious BS
(lasts longer than 25 msecs)

Rhonchi

Wheezes

Stridor
Discontinuous Adventitious BS
(Short duration)

Crackles (Rales)

Pleural Friction Rub
Rhonchi
Snoring sound

Associated with sputum producing diseases
-Resolving pneumonia
-Bronchitis

Can effect insp and exp phase

Cleared or lessened with
-coughing
-suctioning
Wheezes
Sounds generated by the vibration of the wall of a narrowed or compressed airway as air passes thru a high velocity

Airway can be narrowed by
-B. spasms
-secretions
-Foreign bodies
-Airway wall edema
Relationship between pitch and airway diameter
Inverse Relationship
Polyphonic Wheezes
Multi Airways

Begin and end simultaneously
Monophonic Wheezes
Indicates a partial obstruction of a single bronchus

ex. Endobronchial tumor
Common Diseases that produce wheezes
Asthma

Emphysema

CHF

Pneumonia

Partial airway obstruction with a foreign body
Stridor
high pitched cont. sound similar to a wheeze

produced by partial obstruction of the extrathoracic airway

Primarily an Insp. sound

Can often be heard without a stethescope

indicates vent. might soon be compromised

Emergency intubation equipment should be kept close
Common diseases producing stridor
Post ext. airway edema

Partial airway obstruction with a foreign body

Pediactric diseases
-croup (in kids)
-epiglottitis (infection of the glottis) (lethal)
Crackles (Rales)
Discont. sound produced in intrathoracic airways

produced by sudden explosive opening of airways that contain fluid or are collapsed
Early Insp. Crackles
Sudden opening of larger airways as seen in bronchitis
Late Insp. Crackles
Sudden opening of peripheral airways
Diseases that cause crackles
Atelectasis

Pulm. edema

CHF

Pulm. Fibrosis

Pneumonia
Pleural Friction Rub
Creaky Sound

Produced by visceral and parietal becoming inflamed and rub together during breathing

Mostly during insp.

Usually associated with sites of pt pleuritic pain

Can help confirm diagnosis of pleurisy
E to A Egophony
Abnormal sound transmissions in the presence of pleural effusion over an area of consolidation

pt is instructed to repeat the letter E

Affected areas will make it sound like the pt is saying A
Whispering Pectoriloquy
Abnormal BS in the presence of an area of patchy consolidation

pt is instructed to whisper 1,2,3

Over normal lung areas, no words will be heard

Over affected areas listener will here 1,2,3
Procedure for Chest Auscultation
1. Position pt in upright sitting position

2. Inspect areas areas of the chest

3. Position diaphragm of stethescope directly onto skin of the pt

4. Instruct pt to breath
-open mouth
-Insp. deeply
-Exhale passively

5. Listen for full resp. cycle per position

6. Move stethoscope to opposite symmetrical side of chest

7. Localize any adventitious breath sounds
-Type
-Area of chest
-Occurance during resp cycle
Inspection of the Abdomen
Check for scars:

How old and where is the scar

and abdominal distension:

May impair diaphragmatic function

May impair ability to breath deeply and cough

Causes: Hepatomegaly and Ascites
How to check for ab use during exhalation
Place hand on abdomen, and watch during exp phase

*a sign of increase WOB and forced exhalation
Signs of Resp disease that can be identified in the extremities
Clubbing

Cyanosis

Pedal Edema

Capillary Refill

Peripheral Skin Temp
Clubbing
Painless enlargement of the termianl phalanges of the fingers and toes

angle advances past 180 degrees

Mechanism is unknown

requires years to develop

best visualized by profile
Chronic Cardiopulmonary Diseases associated with clubbing
Cyctic Fibrosis

Bronchiectasis

Bronchogenic CA

Cyanotic Congenital Heart Disease
Acrocyanosis
cyanosis of the hands, feets, fingers, toes, and nail beds

Associated with:
-hypoxia
-poor peripheral perfusion
Things that cause poor peripheral perfusion
Low CO

Peripheral Vasoconstriction
Pedal edema
Accumulation of fluid in the subcutaneous tissues around the ankles

ankles and feet because they are gravity dependent

Edematous area will swell and leave a pit when a finger indentation is made

Pitting edema helps to decide how much swelling there is (1-4)
Pedal Edema indacates...
RSHF

Corpulmonale
Cor Pulmonale
RSHF caused by increased BP in pulm arteries
Capillary Refill
Assess CO thru perfusion to the digits

Less then 3 sec refill is normal

More than 3 sec refill is abnormal
Cool peripheral skin to touch means
Decreased peripheral blood perfusion