• Shuffle
    Toggle On
    Toggle Off
  • Alphabetize
    Toggle On
    Toggle Off
  • Front First
    Toggle On
    Toggle Off
  • Both Sides
    Toggle On
    Toggle Off
  • Read
    Toggle On
    Toggle Off
Reading...
Front

Card Range To Study

through

image

Play button

image

Play button

image

Progress

1/150

Click to flip

Use LEFT and RIGHT arrow keys to navigate between flashcards;

Use UP and DOWN arrow keys to flip the card;

H to show hint;

A reads text to speech;

150 Cards in this Set

  • Front
  • Back
accomodation
when pupils/lens adjust to see objects at different depths (near/far)

ABILITY TO FOCUS AND REFOCUS
the leading causes of vision changes are associated with...
AGING

-glaucoma
-cataracts
-macular degeneration
upper eyelid innervated by which CN
CN III
what forms tears

what are the 3 parts of tears
lacrimal gland (outer/upper part)

1)lipiod
2)aqueous
3)mucoid
conjuctiva and it's 2 parts
mucus membrane whose goblet cells secrete mucus

1) bulbar-covers sclera
2) palpebral-lines both lids
sclera and color

what covers it
the white of the eye serves as protection

may be blue in children

may be a dull white or kinda yellow in old

covered by conjuctiva which contains the vessels
limbus
part around iris where conjuctiva meets the cornea
main refractory surface of eye

how many layers does it have
cornea

avascular clear lens that has 5 layers -the endothelium replaces itself every 7 days
anterior chamber what is in it and why
area behind cornea filled with aqueous humor that nourishes the cornea
aqueous humor is made by .....

produced by it's relationship to..
ciliary body

IOP
normal IOP (intra ocular pressure)
10-21 mm Hg
what controls pupil constriction/dilation

are they the same size?
sphinchter-parasympathetic

dilator pupillae-sympathetic

only in 80% of us but should respond the same if normal
OD
oculus dexter =RIGHT EYE
OS
oculus sinister =LEFT EYE
OU
oculus uterque= both eyes
age and the lens

when do you start to see these s/s
the lens retains ALL cells formed throughout life and GROWS throughout life forming rings (like a tree)
=loss of accomodation

5th decade
what fluid is anterior and posterior to lens
anterior-aqueous humor

posterior-vitreous humor
posterior chamber

what is made here

where does it go
between vitreous and iris

ciliary body makes aqueous humor

from here into anterior chamber then trabecular meshwork into the canal of Schlemm
ocular fundus
largest chamber of eye contains vitreous humor
vitreous humor
clear, gelatinous substance, encased by hyaloid membrane that helps maintain the shape of the eye

BEHIND THE LENS

it is attached to retina by scattered collagen filaments
what happens to vitreous humor with age
loses gel-like quality
may see "floaters" d/t cells/casts
loses shape and effects retina
retina
innermost surface of ocular fundus

10 microscopic layers that are like "wet tissue paper

NEURAL TISSUE EXTENSION OF THE OPTIC NERVE
optic disc

what does it look like
where optic nerve enters retina
AKA optic nerve, optic head

"PHYSIOLOGICAL BLIND SPOT"

looks like a oval/circular opening w/sharp edges
macula
part of retina used for central vision the rest of retina used for peripheral
retinal pigment epithelium (RPE)

sensory retina
many Fs including absorbing LIGHT

contain photoreceptor cells rods and cones
rods
retina photoreceptor cells =night vision and low light
cones
retina photoreceptor cells=bright light, color, fine detail

everywhere in retina but MOST in fovea (Macula)
*there are NO RODS in fovea
optic chiasm
if this does not function
point at which the nasal fibers from the nasal retina of each eye cross to the opposite side of the brain

pt cannot see out of left side or right side of eye BUT OPPOSITE in both eyes=bitemporal hemainopsia
if pt cannot see out of one eye
unilateral blindness d/t OPTIC NERVE on that side
optic tract does not function
homonymous hemianopsia =OPTIC TRACT

cannot see on on SAME SIDE of BOTH EYES -problem will be on opposite side of visual cortex
what if pt cannot read any letters on Shnellen chart at 20'
move chart closer and record point when pt can read largest letter on chart
what if pt can ONLY read 'E" on chart at 10'
record as 10'/200
what if pt cannot see "E" at any distance
-count fingers

record as CF/X X=number of feet
what if pt cannot see to count fingers
move arm up or down or side to side as pt which direction it is being moved

-HM-hand motion
LP

NLP
light perception

no light perception

(hand motion)
retinitis pigmentosa
night blindness
cellophane reflex
apperance of macula in young person
crazy stuff seen in eye with direct opthalmoscopy
cup should be 1/3 size of disk
veins are bigger than arteries
silver/cooper disc=arteriosclerosis
red smuges/flames=HTN
lipids=lipidemia or diabetes "cotton wool"
red dots/nevi=microaneurysms
DRUSEN
Drusen
seen in retina, made of hyaline globular deposits may be yellowish seen in macular degeneration
slit lamp examination
thing that sits on a table where the magnification can be changes like they did at school
is color vision loss more a problem in central or peripheral conditions
CENTRAL- because central vision identifies color
most common test for color vision
Ishihara polychromatic plates

uses "hidden images" using primary colors on background of secondary colors
Amsler Grid
often used for those w/macular problems

grid of squares
stare at center
if changes are seen in peripheral
this is not normal
pt may be told to to this qd at home
color fundus photography

for what/ side effects
to detect retinal lesions
pupil is dilated
visual acuity diminished for about 30 minutes after d/t "retinal bleaching" from light
neovasculurization

test for it

how/side effects
growth of abnormal new blood vessels

(EX: macular degeneration )

Fluorescein Angiography

dye injected into eye
may turn skin yellow/urine yellow or orange
Indocyanine green angiography

for/ side effects
to ID abnormalities of choroidal vasculature

injected IV
may cause N/V
NOT given if IODINE ALLERGY
applanate
flatten
Tonometry
used to measure IOP
by amount needed to applanate globe
scotomas
blind areas of visual field
perimetry testing
tests visual field an tests for scotomas
average visual field when eye is in primary gaze
65 upward
75 downward
95 outward

(degrees)
refraction
determination of the refractive errors of the eye and correction by lenses
refractive errors
eye is too long or short and light is not focused right
-may result in blurred vision
-may be corrected by lenses/glasses
emmetropia
no refraction errors=normal eye/vision
myopia
-deeper eyeball
-light focused before retina
-NEARSIGHTED
-things at distance are blurred
hyperopia
-shorter eyeball
-light focused after retina
-FARSIGHTED
-things at up close are blurred
astigmatism
-irregularly curved cornea
-can cause refractive errors
-may have both problems
-contacts/sx to correct
-hard or conical lenses
BCVA
best corrected vision at
best seen with glasses/etc
low vision
needs more than just glasses/etc
BCVA 20/70 to 20/200
blindness
BCVA 20/400 or less
clinical absolute blindness
no light perception
legal blindness
BCVA 20/200 MAX in better eye
OR
visual field diameter 20 degrees or less
travel vision
person has some significant visual impairment but can manage without the use of aides
glaucoma

patho

AKA

common s/s
-condition causes optic nerve damage r/t increased IOP

1) direct mechanical=direct pressure to optic nerve
2) ishemic=pressure to vessels of nerve

"silent thief of sight"

blurred vision
"halos" around lights
difficulty focusing in low light
loss of peripheral vision "tunnel vision"
pain in eye and headache

cupping of optic nerve disc
pallor of optic nerve
how does aqueous humor drain

what does flow depend on
1) Trabecular Network
normally aqueous humor flows between iris and lens then flows out of anterior chamber through the spongy trabecular meshwork into the canal of Schlemm and episceral veins

2) Uveoscleral route
10% from ciliary body into venous system

open angle of 45 between iris and cornea
DX glaucoma
tonometrey
opthalmoscopy
gonioscopy=to view angle
perimetry
miotics
cause pupils to contstrict and trabecular network to open
mydraitics
medications that cause pupils to dilate
open angle

and

angle-closure glaucoma
3 types-all have open anterior chamber

(Pupillary Block) 3 types-all obstruction of aqueous humor and increased IOP
risks for cataracts
-smoking
-long term corticosteriod use
-sunlight/ionizing radiation
-diabetes
-eye injury
-obesity
3 most common types of senile cataracts -major differences
-nuclear (center of lens/ myopia)
-cortical (little effect on vision/bright light)
-posterior subcapsular (from posterior capsule/hyperopia/bright light and glares)
brunescens
color value shifts to yellow-brown
diplopia
double vision
major s/s of cataracts
-clouding of lens (as it progresses)
-dimmer surroundings like "dirty lens"
-light scattering=reduced contrast
-sensitive to glare
myopic shift (return of near vision)
-astigmatism
-monocular diplopia
-color shift (more blue light absorbed)
-brunescens
-decreased visual acuity
aphakic
without lens
binocular vision
ability for both eyes to focus on an object and fuse the two images into one
keratoconus
conical growth that sticks out of cornea
more commonly seen in younger
lens used to flatten it out
strabismus
"lazy eye"
deviation in ocular alignment
retinal detachment
seperation of retinal pigment epithelium (RPE) from sensory layer (rods,cones,photoreceptors)
4 types of retinal detachment
rhegmatogenous
traction
COMBO: rhegmatogenous/traction
exudative
rhegmatogenous retinal detachment
-most common
-hole or tear allows fluid to move between layers
-risks include myopia or aphakia, also trauma and retinopathy associated w/diabetes
traction retinal detachment
is d/t a pulling force or tension
-often d/t scar tissue
exudative retinal detachment
production of serous fluid under retina from choroid from uveitis or macular degeneration, etc
s/s retinal detachment
-shade/curtain coming across vision
-cobwebs/floaters
-bright/flashing lights
-NO PAIN
scleral buckle
-type of sx
-band placed around eye
-brings layers of retina together
-used for retinal detachment
age related macular degeneration
-most common cause of vision loss >60
-drusen
-central vision loss
-2 types (dry and wet)
age related macular degeneration
DRY
(nonneovascular,nonexudative)
-outer layers of retina break down
-drusen occur outside of macular area
-drusen outside macular=NO vision LOSS
-drusen inside=vision loss

gradual blurring when pt tries to read
often a slow onset
age related macular degeneration
WET
(neovasular, exudative)
-may have abrupt onset
-blood vessels grow under retina
-choriodal neovascularization
-straight lines look crooked/distorted
-letter/words look brocken
orbital trauma

s/s

assessment
tenderness, bruising, proptosis

check for signs of HEAD injury first then get at least a basic vision assessment
proptosis
downward displacement of eye ball
enopthalmos
eye ball deeper is socket

opposite is exopthalmus when it is popping out
worst type of material to get stuck in eye d/t purulent infection
-iron
-copper
-vegetable matter
chemosis
edema of conjunctiva
hyphema
hemorrhage within eye chamber
enucleation
removal of eye
sympathetic opthalmia
inflammation in UNIJURED eye d/t injury of AFFECTED eye

may be cause for enucleation
irrigating an eye after chemical contact until pH is....
7.3-7.6
dry eye syndrome
AKA keratoconjunctivitis sicca
tears or part of tears not right

scratchy, burning, itching, redness, pain, corneal erosion, infection

TX: artificial tears/punctal plug
Schirmer's test
filter paper used to measure tear production
Hordeolum and tx
AKA stye
abscess caused by Staph aureus
warm compress 10-15 min x4/day
may need to drain if more than 48hrs
Blepharitis
inflammation of the eye lid(s)
Conjunctivitis

must evaluate the type of ....

type of ......

and type of.....
AKA pink eye

discharge:
1) watery
2) mucoid
3) purulent
4) mucopurulent

conjunctival reaction:
1) follicular-lesions with vessels (RICE)
2) papillary-fine mosiac pattern

Membranes:
1) psuedomembranes-exudate sticks
2) true membranes-removal causes bleeding
Evisceration
removal of contents within the eye
allows for movement
exenteration
removal of eyelids, eye, and others
-most often with cancers/etc
when pt loses an eye what else is lost
depth perception
papilledema
swelling of optic nerve d/t increased IOP
function of auricle
transmits sound waves to ear
external auditory canal ends at
tympanic membrane
eustachian tube fucntions
drains stuff from middle ear
equalizes pressure
tympanic membranes

layers and fucntion
AKA eardrum

has three layers
pars flaccida lacks middle layers
pars tensa has all the layers

amplifies sound x22
ossicles
3 smallest bones in body

malleus, incus, and stapes
organ of hearing and location
cochlea -inner ear
organ of balance and location
semicircular canals -inner ear
end organ for hearing
organ of Corti
snail shaped structure in cochlea
transforms mech energy to neural
air and bone conduction
air is BETTER -vibration of tympanic membrane and ossicles

bone-travel through bone bypasses middle and external ear
balance and equilibrium

what part of brain

what 3 systems are needed
cerebellar (cerebral cortex)

1)body (proprioceptive system)
2) eyes (visual system)
3) labyrinth (vestibular system)
Weber test
-tests bone conduction
-placed on head or forehead
-normal=hears sound in head
-conductive loss=better in affected
-sensorineural=better in good ear
Rinne Test (rin-ay)
-mastoid until nothing then air

normal=air is louder
conductive loss=bone is longer
sensorineural=air is longer
best test for hearing loss
audiometry

2 types- tone and voice
3 parts to hearing evaluation
-frequency
-pitch
-intensity
frequency

how is it measured

what is normal range
-number of sound waves from source per second
-measured in Hertz (Hz)

normal 20-20,000
speech range 500-2000
pitch
used to describe frequency

100 Hz=low pitch
10000 Hz=high pitch
intensity

how is it measured

what is normal range

what is the corrected goal
AKA loudness or pressure exerted from sound

-measured in decibal (dB)

low conversation 40 dB
harsh and damaging 80 dB

goal is 30 db for tx/sx of loss
measuring hearing loss by dB
0-15 normal
15-25 slight
25-40 mild
40-55 moderate
55-70 moderate to severe
70-90 severe
>90 profound
presbycusis
progressive hearing loss associated with aging

loss ability to hear high pitched
"ch", "f", "p", "t", "k", "st"
otalgia
sense of fullness or pain in the hear with or without loss of hearing
external otitis (otitis externa)

s/s

teaching
AKA swimmers ear

-pain /aural tenderness
-discharge
(yellow/green bacterial)(black fungal)
-swelling redness

wick may be inserted to keep dry
dry ears
protect from wetness/cotton/jelly
malignant external otitis
AKA temporal bone osteomyelitis

infection gets into the bone
exostoses
-small,hard,bony growths in external ear canal
-may be caused by cold water
-must be sx removed
clear watery drainage from nose or ear after head trauma may be...
cerebrospinal fluid
tympanic membrane perforation
-often will heal on own
-must protect it until then
-tmpanoplasty=sx to fix
acute otitis media
-middle ear infection lasting less than 6wks
-most common in children

-PAIN-relieved if rupture of eardrum
-fever, runny nose
-bulging red eardrum
-may need to drain it
myringotomy
AKA tympanotomy

sx incision to tympanic membrane
serous otitis media
AKA middle ear effusion

-no infection (often)
-fluid stuck d/t blocked tubes
-fullness/air bubbles
cholesteatoma
skin from eardrum grows into middle ear
chronic otitis media
reccurent AOM
may cause damage to eardrum
and cholesteatoma
may also lead to other more serious infections

sx may be needed to correct it
ossiculoplasty
sx reconstruction of middle ear bones to restore hearing for conduction loss
pt teaching after middle ear or mastoid sx
-don't blow nose for 2-3 weeks
-sneeze/cough with mouth open
-avoid lifting/straining
-may hear popping/cracking 3-5wks
-temporary hearing loss is normal
-protect ear from water
otosclerosis

s/s
-involves the stapes (fixation)

-bone conduction better than air
-may have tinnitus
-low frequency loss
-tx with sx
dizziness
altered sensation of orientation
vertigo
illusion of motion of person/surroundings
Meniere's disease

s/s

dietary changes

sx
-inner ear fluid imbalance
-too much endolymph
-endolymphatic hydrops=space gets big

-sensorineural loss (fluctuation)
-tinnitus or roaring sound
-VERTIGO
-loss of low frequencies

low Na/diuretics/ increased K

-sac decompression
-vesicular nerve cutting
Labyrinthitis
-inflammation of inner ear

-ACUTE SEVERE VERTIGO
-N/V
-tinnitus
-hearing loss
ototoxic medications=most common
aspirin, quinine
aminoglycosides (-MICIN or -MYCIN)
acoustic neuroma
slow growing benign tumor of CN VIII
furuncle
bacterial infection of hair follicle
(often seen in ear)
Romberg's test
-tests vestibular function
-close eyes while standing
-some sway is normal
random stuff about hearing
-brain processes sound 1000x faster than images even during sleep
-B/P wil increase 5-8% in loud places
-loud noise=pupil dilation

SURVIVAL
microbial vs allergic conjunctivitis
microbial may start in one eye then spread and drainage is often cloudy/purulent

allergic often both eyes with clear drainage
otorrhea
drainage from ear