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37 Cards in this Set

  • Front
  • Back
What is an arrhythmia?
abnormal in rate, rhythm or flow of depolarization
Where does a sinus arrythmia originate?
SA node

variable RR interval see in on inspiration (vagal tone)
no treatment needed but can give atropine to stop vagal tone
Where does a wandering atrial pacemaker originate?
Atria due to decrease PR interval and variable P waves
Seen on respiration and there is not treatment
Where does sinus bradycardia originate?
Sinus node causing the heart rate to slow
causes heart problems, electrolyte abnormalities, hyperthyroidism, certain drugs

treat by stopping drugs, treating the problem and inserting a pacemaker
Where does a sinus arrest originate?
SA node

caused by a dropped heart beat and will cause an escape beat due to heart problems, drugs, electrolyte abnormalities or hyperthyroidism

treat with atropine, pacemaker it atropine doesn't work
Where does junctional or nodal escapes beat originate?
AV node or bundle of His

QRS is normal with no P wave

looks wide and bizarre
What is a differential diagnosis for sinus arrest?
respiratory sinus arrythmia
What would a P wave from the AV node look like?
From the SA node?
negative
positive
What are ECG qualities of hyperkalemia?
Spiked T wave (should be 1/4 of R)
Absent P wave
Prolonged QRS

differential diagnosis is hypoxia
What are the four differential diagnoses for a prolonged QRS?
right bundle branch block
left bundle branch block
sinus arrest
hyperkalemia
What are the characteristics of a 1st degree AV block?
Prolonged PR interval

*the problem is in the AV node*
What is a second degree AV block?
What is a mobitz type I?
intermittent failure of conduction
slowly progressing increase in PR interval until QRS is dropped
What is a second degree AV block mobits type II?
What is a high grade block?
no prolonged PR interval preceding a dropped QRS
ever two P waves get conducted to ventricles
What are the abnormality of a third degree AV block?
Treatment?
no communication between the atria and the ventricles
P wave is normal, QRS are escape beats from the AV node or from ventricle making them wide

-insert a pacemaker
How do you differentiate a 3rd degree block with AV junctional escapes (normal QRS) and 2nd degree block?
measure PR interval, if it is regular than its 2nd not 3rd
What are the clinical signs of sick sinus syndrome?
weakness and syncope cause by fibrosis of the SA node

see brady or tachycardia, arrest escapes beats

treat with a pacemaker
What are two physiological causes of tachycardia?
Pathological?
fear and pain
fever,anemia, thyroid
How is tachycardia defined by heart rate?
a heart rate 160-180
greater than 240 in cats
What are the abnormalities of sinus tachycardia?
increased heart rate

waves are normal
What is the main problem with supraventricular premature depolarization?
beats that occur earlier than expected from atria outside SA node

treatment by slowing the heart with diltiazem
What does the ECG look like with SPD?
abnormal p waves with normal QRS and premature PQRS

can sees superimposed P waves on T waves

see signs of syncope and weakness
What is a common arrhythmia in dogs with dilated cardiomypathy?
atrial fibrillation

hearts sounds are chaotic
What are the ECG abnormalities of atrial fibrilation?
no P waves, wavy baseline, random RR and normal QRS
How does tachycardia effect the O2 requirements of the heart?
How is the cardiac output affected?
increase
decrease
Can you have a normal HR with A-fib?
How do you tx animal with A-fib and >150 beats/min?
Do these drugs increase or decrease contractility?
Yes (monitor with Halter monitor)
Slow heart to allow to fill with diltiazem or beta blockers
Decrease
In a dog with Dilated cardiomyopathy what drugs do you give?
What drug is contraindicated in cats?
Digoxin (anti-arrythmic effects) and diltiazem

Digoxin
What is the pattern of atrial flutter?
saw-tooth

this is a precursor to A fib
What is the origin of ventricular premature depolarization?
Ectopic focus in ventricles

there is no P wave
What will the QRS look like with VPDs?
wide because it is not going through normal conducting pathways

right ventricle (travels in pos direction)
left ventricle (travels in neg direction)
What are the four clinical signs of VPDs?
dropped heart sounds
irregular pulses
dropped pulses
sudden death from transition to A fib
What drug do you want to use for VPDs in acute settings?
Stable?
lidocaine (slow bolus)

-Mexilitene
-Procainamide
-Beta blocker
What is the definition of ventricular tachycardia? What do they arise from?
Which side?
-Runs of > 3 VPDs
-Ectopic focus in ventricles
-Left vent b/c mainly in negative direction
What is a capture beat?
when the P wave activates the AV node and the ventricales are in a refactory period

normal QRS complex
How does an accelerated idioventricular rhythm/tachycardia occur?
Run of VDP (ectopic vent beats) too slow to be ventricular tachycardia

Normal to wide and bizarre QRS with capture beats
What is the treatment for accelerated idoventricular rhythm/tachycardia?
no treatment necessary

heart rate is normal and allows for diastolic filling so CO is maintained
How is ventricular fib different from ventricular tachycardia?
Ventricular fib is random electrical impulses, ventricular tachycardia has regular VPDs

Treat ventricular fib with External fibrilator, Epi, CRP
What is a fusion beat?
the combination of a normal beat and an ectopic beat

there are p waves present