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9 Cards in this Set

  • Front
  • Back
Endproduct of aerobic glycosis vs. anaerobic glycolysis
Pyruvate vs. Lactic acid
Mechanism by which glucose is transported into cells
GLUT-1: RBCs, brain
GLUT-2: Liver
GLUT-4: Muscle, adipose tissue (insulin dependent)
Differences in location, Km, Vmax, regulation of hexokinase vs. glucokinase
Location: Hexo in periphery; Gluco in liver
Km: Hexo low; Gluco high
Vmas: Hexo low Vmax; Gluco high
- Regulation: Hexo: G6P; Gluco: F6P
+ Regulation: Gluco: insulin & glucose
Function,+ and - regulation of PFK
Function: F6P -> F1,6BP
- Regulation: ATP, citrate
+ Regulation: AMP, F2,6BP
Function,+ and - regulation of pyruvate kinase
Function: PEP -> pyruvate
- Regulation: Glucagon (-)
+ Regulation: F1,6BP, insulin
Effects of pyruvate kinase deficiency
Varying degrees of hemolytic anemia
Function,coenzymes, + and - regulation of pyruvate dehydrogenase
Function: Form acetyl CoA from pyruvate
Coenzymes: TPP, lipoic acid, CoA, NAD+, FAD
+ Regulation: NAD+, CoA, pyruvate
- Regulation: ATP, Acetyl CoA, NADH
Most common cause of congenital lactic acidosis AND mechanism of arsenic poisoning
Pyruvate dehydrogenase deficiency --> Deprives brain of acetyl CoA
Total number of NADHs/FADH2s/ATPs from TCA and subsequent electron trans chain ATPs
3 NADHs (9 ATP), 1 FADH2 (2 ATP) + 1 ATP = 12 ATP / 1 acetyl CoA