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30 Cards in this Set

  • Front
  • Back
  • 3rd side (hint)
deoxygenated blood
right atrium through the tricuspid valve, through right ventricle to the pulmonary valve to the lungs
oxygenated blood
through the pulmonary veins to the left atrium,to the mitral valve to the to left ventricle to the aortic valve to the aorta
factors that can alter cardiac rate and rythm
drugs, acidosis, oxygen to the cells, change in electrolytes and accumulation of waste in the system
elements that determine BP
heart rate, stroke volume, total pherpial resistance
ACE inhibitors
keeps ACE from converting angiotension 1 to angiotension 2, leads to decreased bp and aldostrone production
ACE inhibitor
lisinopril- Zestril
catopril- Capoten
angiotension II receptor blocker
binds to the recptors in vascular muscle and adernal cortex to block vasoconstriction and aldosterone
ARBS
valsartan-Diovan
losartan-Cozaar
Calcium Channel Blockers
inhibits the movemnet of calcium iions across the of the cardiac and atrerial muscle cells, depressing the impulse and slowing the conduction, decreased cardiac contraction and dialation of arterioles, therefore decreasing BP
CCB's
amlodiopine-Norvasc
nefediopine-Procardia XL
'
thiziade like products
HCTZ
potassium sparing diruetic
spironolactone
loop diruetics
fursomide- Lasix
Beta-blockers
block the SNS, slower HR , decrease in arrythmias,decrease cardiac workload and oxygen consumption
Beta-blockera
propranolol-Inderal
Treatments for Heart Failure
Vasodialators (ACE and Nitrates)
diuretics
beta-andrenegic agonist
Cardiac Glycosides
increase force of heart contraction,increase cardiac output and renal perfusion,slows heart rate, decrease conduction of AV node
cardiac glycosides
increases intracellular calcium, allows more calcium to enter they myocardial cell during repolarization; postive inotropic effect, increases renal perfusion with a diuretic effect and decreases in renin release and slowedconduction through the AV node
can cause arrhythmia development, HA and vision change
cardiac glycosides
dig- lanoxin
stable angina
no damage to heart, stressed induced
unstable angina
pre-infarction or risk of blockage of o2 to the heart, unpredictable
prinzmetal angina
spasms of hte heart happens at rest
types of angina meds
nitrates
beta-adrenergic blockers
calcium channel blockers
nitrates
acts directly on smooth muscle to causes relaxation and depress muscle tone
used to prevent angina pectoris
nitrates
nitroglycerin
beta-blockers
used to treat stabe angina and hypertension
beta-blockers
metropolol- lopressor toprol XL
calcium channel blocker
used for prinzmetal angina
calcium channel blocker
diltiazem (Cardizem)
antihyerlipidemic actions
HMG-CoA- lipitor
bile- acid sequestrants- questran