Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
111 Cards in this Set
- Front
- Back
U waves...
Upright -> Inverted-> |
U- hypokalemia, hypercalcemia, digitalis, phenothiazines, quinidine, epi, thyrotoxicosis
I- ACS, strain, hypertension, increased ICP |
|
Temperature at which hypothermia starts?
EKG findings? |
35 C (95 F); J wave, prolongation of PR, QRS and QT, sinus brady, slow a-fib
|
|
CPR in hypothermia?
|
CPR in all monitored with v-fib, pulseless v-tach, or asystole; if no monitor, check pulse x 45 seconds before begining CPR
|
|
V-fib in hypothermia...
cardiovert? |
yes, but may not work; rewarm and will prob convert on own; can use mag as antidysrhythmic
|
|
EKG in hypo-k
|
u wave, flat then inverted t, QT and PR prolongation, ectopy, st depression
|
|
EKG in hyper k
|
peaked T, prolongation of PR, loss of P, QRS widening, sine wave
|
|
EKG in hypocalcemia
|
prolonged QT, T-wave inversion, torsades; treat with mag
|
|
EKG in hypercalcemia
|
QT shortening, QRS widening
|
|
EKG in hypomag
|
QT and PR prolongation, T wave inversion, ectopy
|
|
Digitalis effect on EKG
|
sagging ST seg, QT shortening, PR prolongation
|
|
2 mechanisms of digitalis' toxicity
|
poisons Na/K/Mg ATPase -> K eggress, Ca builds up in cell -> irritability; Increases vagal tone -> brady and blocks
|
|
Factors that increase dig toxicity
|
hypo-K, hypo-Mag, hyper-Ca, hyper-K, hypoxia, alkalosis, age, sick heart; drugs- quinidine, CCB, e-mycin, captopril, amiodarone, ibuprofen
|
|
EKG in dig tox; most common; pathognomonic; other common
|
most common - PVCs, either multiform or bigeminy/trigeminy; pathognomonic- atrial tach with block; regular/slow a-fib, bidirectional v-tach
|
|
Clincial symptoms of dig toxicity
|
flu-like with N/V, diarrhea, malaise, yellow-green color abberrancies, halos, AMS
|
|
Prognosis of dig tox in acute ingestion is dependant on what?
|
degree of hyper-k, not dig level
|
|
Treat dysrhythmias in dig tox (other than fab frag); what to avoid
|
phenytoin or lidocaine #1; mag can work too
cardioversion, calcium, procainamide, beta-blockers |
|
Treatment of bradycardia in dig toxicity
|
#1- atropine (remembter increased vagal tone)
#2- trans-q pacing (not invasive pacing) |
|
EKG of WPW
|
short PR (< .12), delta wave, wideish QRS, kent bundle bypass
|
|
EKG of LGL
|
short PR, no delta wave, normal QRS, James bundle bypass
|
|
Treatment of bradycardia...
when not to use atropine |
atropine, trans-q pacing, epi gtt (2-10 mcg/min), dopamine 5-20 mcg/kg/min, transvenous pacing
NO- acute iscemia, mobitz II or third degree AVB |
|
Dobutaimine
action, dose |
B1 agonist -> inotrope with mild chronotropy
2.5-40 mcg/kg/min |
|
Dopamine
action, dose |
dopamine receptor, plus beta and/or alpha
0.5-2 mcg/kg/min - vasodilation; 2-10 mcg/kg/min- beta; 10-20 mcg/kg/min alpha |
|
Epi gtt
action, dose |
inotrope, chronotrope > vasoconstrictor
1-20 mcg/min |
|
Norepi gtt
action, dose |
inotrope, chronotrope < vasoconstrictor
8-30 mccg/min |
|
Phenylephrine
action, dose |
pure alpha-agonist
40-180 mcg/min; push dose 50 mcg q 2 min 10 mg into 100 ml bag of saline -> 100 mcg/ml, push 0.5 ml at a time |
|
vassopressin
dose |
40 units is code dose
0.01 - 0.04 units/min |
|
Tx to consider in PSVT
|
valsalva, adenosine, diltiazem, esmolol, amiodarone, procainamide
|
|
Drug that potentiates adenosine?
|
carbamazepine
|
|
Esmolol
bolus and gtt |
500 mcg/kg bolus/ 1 min
50-200 mcg/kg/min |
|
Propranolol
|
crosses brb; use in thyrotoxicosis
1 mg IV q min to 0.3 mg/kg |
|
Digoxin loading dose
|
0.5 mg IVP then 0.25 mg IVP q 30 min up to effect or 0.02 mg/kg
|
|
Diltiazem
bolus and gtt |
0.25 mg/kg IV over 2 min then 0.35mg/kg IV over 2 min 15 min later; gtt at
80-250 mcg min |
|
How much Ca to give before dilt?
|
5-10 cc of CaGluconate
|
|
Phenytoin
dose |
20 mg/kg IVPB in NS and run at 50 mg/min or less
|
|
Rx to control rate in A-fib
|
diltiazem, esmolol, metoprolol, digoxin
*magnesium is an adjunt for all of the above |
|
Rx to control rate in A-fib with low EF
|
diltiazem, digoxin
|
|
Causes of MAT?
|
COPD, CHF, sepsis, theophyline toxicity
|
|
Tx of MAT? What is inneffective?
|
correct hypoxia, magnesium, CCB, BB;
don't - cardiovert, digoxin |
|
Causes of PVCs?
|
hypo-k,mg; hypoxia, MI, ETOH, cocaine, meethyxanthines, dig, tobacco, hyperthyroid, chf, cardiomyopathy, contusion
|
|
Procainamide
dose? |
17 mg/kg at 35 mg/min to qrs widening or hypotension
gtt of 1-4 mg/min |
|
Lidocaine
dose? |
1.5 mg/kg IVP; re[eat .5 mg/kg q 5 min to 4mg/kg
gtt 2-4 mg/min |
|
Algorhithm for stable v-tach
|
lidocaine followed by amio or procain followed by mag, followed by cardioversion
|
|
Magnesium dose?
precaution? |
2 g slow IVP followed by .5-2 g/hr
renal failure |
|
H's and T's?
|
hypovolemia, hypoxia, hydrogen ion, hyper/hypo-k, hypothermia; thrombosis (mi, pe), tension pneumo, tamponade, tablets (drugs)
|
|
Amiodarone
Dose? |
code - 300 mg IVP then repeat 150 mg IVP
non-code- 150 mg over 10 min then 1mg/min x 6 hr then .5 mg/min x 18 hrs |
|
First degree AVB
causes? Danger? |
normal or high vagal tone, dig, myocarditis; not dangerous
|
|
Mobitz I
cause? Dangerous? |
typically transient and intranodal; inferior MI, dig, vagal tone, myocarditis
No tx needed unless symptomatic -> atropine |
|
Mobitz II
Cause? Dangerous? |
often permanent and infranodal; anteroseptal MI -> complete block; pacer placement; no atropine
|
|
Complete block?
Cause? Dangerous? |
narrow- typically inferior MI or drugs and transient
wide- typically anterior MI and permanent -> pacer |
|
Tx of WPW with narrow or wide SVT?
|
narrow- orthodromic; tx same as regular SVT
wide- antidromic; tx with amio (EF <40%) or procaine |
|
Tx of WPW in A-fib or flutter?
|
unstable -> defib
stable -> amio (EF < 40%) or procaine |
|
First two letters of pacer code?
|
chamber paced, chamber sensed
V, A, D (dual) or O (none) |
|
Major risk factors for CAD/AMI?
|
cigarettes, HTN, DM, hypercholesterolemia, 1st degree relative < 55, other vascular occlusion
|
|
Order of EKG changes in AMI?
|
large T -> ST elevation +/- reciprocal, q waves, T-wave inversion
|
|
isolated t wave without ST changes or Q waves?
|
ischemia not infarction
|
|
Inferior MI association?
|
RCA lesion; bradydysrhythmias, vagal tone, AV blocks
|
|
Right ventricular MI association?
|
shock, hypotension
|
|
Definition of pathologic q?
|
greater than 0.04 sec long and > 25% of r wave
|
|
CK-MB peak and duration?
|
peak at 4 hours, lasts 24 hours
|
|
Troponin peak and duration? I v. T?
|
peak in 6 hours, lasts weeks; I is very specific, T is less so but more than CK-MB
|
|
Heparin dosing?
|
60 U/kg up to 4,000 units and 12U/Kg/hr (max 1000 U/hr) to PTT of 50-70 sec
|
|
contraindication for lovenox?
|
HIT, obesity, renal failure, planned CABG
|
|
Contraindication for NTG?
|
use of viagra etc in last 24 hours, SBP < 100 or inferior/RV infarct
|
|
Metoprolol dose?
|
5 mg IV q 5 min to 15 mg total
|
|
Reperfusion rhythms post lytic? Tx?
|
PVCs, accelerated idioventricular, V-tach, V-fib
tx v-vib always; others if unstable only |
|
nicardapine
action? dose? |
peripheral CCB
80 - 250 mcg/hr IV |
|
Slovis' PEA
|
1. oxygenate/ventilate
2. IV fluids 3. think hyper-k, hypothermia 4. epi q 3 min 5. h's and t's |
|
Slovis' v-fib
|
200J -> O2/tube -> epi -> CPR -> 200J -> amio 300 -> cpr -> 200 J -> ? mag -> cpr -> amio 150/lido
|
|
Slovis' V-tach
|
lido -> amio or procaine -> mag -> 200 J
if convert -> load with amio post conversion |
|
Contraindications to ACE-I?
|
renal artery stenosis, creatinine > 2.5
|
|
Which side is CHF effusion most often on (if unilateral)?
|
right
|
|
Tx cardiogenic pulmonary edema?
|
ntg, O2, bipapa, lasix, morphine; if shock then dopamine or norepi (better)
|
|
Order of sx in HCM?
|
D.O.E. -> CP with exertion -> syncope -> cardiac death
|
|
Exam for HCM?
|
systolic murmur increases with decreased preload (valsalva) and decreases with knee to chest/squatting
|
|
Tx HCM? Avoid in HCM?
|
beta blockers, avoid sports
avoid- dig, inotropes, vasodilators |
|
Phlegmasia cerulea dolens?
|
massive dvt -> ischemia; tense blue leg with petechiae and bullae
|
|
Phlegmasia alba dolens
|
massive dvt with arterial spasm -> not tense, white, petechiae -> progresses to cerulea dolens
|
|
O2 sat/paO2 rule?
|
60 -> 90%
50 -> 80% 40 -> 70% |
|
A-a gradient calculation
|
150 - (pO2 + (pCO2/.8))
|
|
Normal A-a grad?
normal pOs? |
age/4 + 4
100 - age/4 |
|
P pulmonale?
|
sharply peaked p wave, seen best in V2 = COPD or chronic pulm dz; also PE
|
|
D-dimer in PE?
|
low pretest prob = dd (-) and done; mod/high = just get a doppler or ct
|
|
heparin or lovenox in pregnancy?
|
yep; doesn't cross the placenta
|
|
Work up for pt with (+) PE?
|
troponin, echo; if shock or (+) troponin/echo then lytic or thrombectomy
|
|
2 most common causes of pericarditis?
|
idiopathic and viral
|
|
History in pericarditis?
|
sharp CP, pleuritic, positional and worse with swallowing; dyspnea, fever
|
|
ECG in pericarditis?
|
diffuse ST seg elevation with PR depression except aVR, V1; diffuse T-wave inversion (late)
|
|
Steroids in pericarditis?
|
not first line since 25% will have rebound sx after course is finished
|
|
Out patient v. in for pericarditis?
|
Idiopathic/viral in the young can go home on NSAID
|
|
Beck's triad for tamponade?
|
distant sounds, JVD, hypotension
|
|
Risks for endocarditis?
|
valve dz(fake), IVD use, Ca degeneration, catheters, dialysis, card surg, HIV, burns
|
|
Most common bacteria in native-valve endocarditis?
|
non-viridans strep -> staph -> strep viridans -> enterococci
|
|
Most common bacteria in prosthetic-valve endocarditis?
|
coag neg staph -> staph -> strep viridans -> enterococci
|
|
Most common bacteria in IVD use endocarditis?
|
staph ->. strep -> gram (-)
|
|
Most common left-sided valve affected by endocarditis? Right-sided?
|
mitral; tricuspid
|
|
New guidlines for abx prophylaxis against IE?
|
prosthetic valve, hx of congenital heart disease, hx of IE or heart tx with valve dz
|
|
Risks for dissection?
|
HTN, connective tissue d/o, 3rd trimester, coarc, bicus AV, Turner's, trauma, cocaine/meth, syphilis, tobacco
|
|
Connective tissue d/o that increase risk for dissection?
|
Ehlers-Danlos, Marfan, Lupus, Ciant cell arteritis, Cystic medial necrosis
|
|
Classification of dissection?
|
DeBakey 1- ascending + distal, 2- a only 3- d only
Stanford A- includes a B- d only |
|
Tx of dissection?
|
esmolol to HR of 60 and nitroprusside to systolic of 100 or single agent labetalol
|
|
Dx of thrombosis of prosthetic valve?
|
acute CHF, muffling of prosthetic heart sound, hypotension, embolic phenomenon
|
|
Symptoms of mitral stenosis?
|
D.O.E., hemoptysis, orthopnea, PND, fatigue, emboli, arrhythmias
|
|
Most common cause of AS?
|
bicuspid (<65), calcium (>65), rheumatic dz
|
|
The most common reason for failure to pace?
|
oversensing
|
|
Findings of CHF in order of appearance?
|
cephalization -> hilar congestion, kerley-B, frank edema
|
|
Most common type of dissection?
|
proximal (Stanford A or DeBakey I or II)
|
|
Treatment of chest pain from mitral valve prolapse?
|
beta blocker
|
|
Adult blood volume?
|
70 ml/kg
|
|
Successful placement of transvenous pacer on EKG is?
|
ST elevation
|
|
Janeway? Oslers?
|
J- nontender macules on fingers, palms, soles
O- tender nodules on volar aspect of fingers |
|
Most common complication of mitral stenosis?
|
a-fib
|