Use LEFT and RIGHT arrow keys to navigate between flashcards;
Use UP and DOWN arrow keys to flip the card;
H to show hint;
A reads text to speech;
26 Cards in this Set
- Front
- Back
folliculitis
|
minor infection of the hair follicle
usually caused by staph aureus and some times pseudomonas aeruginosa from hot tubs |
|
Acne vulgaris
|
involves inflammation of hair follicles and associated sebaceous gland
*caused by propionibacterium acnes, the predominant anaerobe of the skin |
|
Furuncles
|
small staph abscess that develops in region of hair follicle. maybe solitary, multiple, and may be recurrent
|
|
carbuncle
|
furuncle spreads to dermis and subcutaneous tissue --> extensive multiloculated abscess.
genearally caused by dermatopphytes and candida |
|
impetigo or pyoderma
|
starts as small vesicle that spreads and dries to a honey colored crust
generally caused by S pyogenes but sometimes due to S aureus |
|
Bullous impetigo
|
large serum filled bullae (blisters) with skin layers at site of infection
common in children usually by S aureus |
|
staphylococcal scalded skin syndrome
|
due to toxins, exfoliation causes splitting of epidermis between stratum spinosum and stratum granulosum
obviously caused by staph |
|
erysipelas
|
rapidly spreading infection of deep layers of dermis. caused by strep pyogenes
|
|
Cellulitis
|
will be warm
accute inflamation of subcutaneous CT w/o predisposing trauma usually caused by S. areus and S. pyogenes with trauma usually caused by enterobacteriaceae and obligate anaerobes can be involved |
|
eikenella corrodens
|
usually found in human bites
|
|
pasteurella multocida
|
usually found in animal bites, especially cat bites
|
|
aeromonas hydrophila
|
usually found in fresh water injuries
|
|
vibrio vulnificus
|
usually found in saltwater injuries
|
|
necrotizing fasciitis
|
deep infection of subcutaneous tissue results in destruction of fascia and fat. may spare the skin AKA flesh eating bacteia of Group A strep. and sometimes by clostridium prefringes
|
|
granulomatous skin lesions
|
caused by either mycobacterium ulcerans, tuberculosis, marinum, disseminated fungal infections, sporothrix
|
|
how do you treat toxic shock syndrome
|
generally caused by staph aureus
saline, cefazolin and gentamicin after id in hospital was staph continue with cefazolin. after discharge continued with dicloxacillin |
|
ergosterols
|
make up the cell membrane of fungi, so it is the target of many antifungals.
|
|
how do azoles work
|
inhibit ergosterol synthesis in fungi
|
|
how do polyenes work
|
bind to ergosterol in fungi to increase permiability
|
|
amphotericin B
|
main anti-fungal therapy but is very toxic so fluconazole or itraconazole used
|
|
what makes up cell wall of fungi
|
mannan, glucan, and chitin
mannan- is major Ag of fungi glucans is a polymer chitin is long unbranched chains of glcNac |
|
caspofugin
|
antifungal that blocks glucan synthesis
|
|
conidia
|
asexual reproductive elements of fungi
|
|
blastoconidia
|
buds of yeast
|
|
mycelium
|
intertwined mass of hyphae
|
|
dermatophytes
|
mostfungi infections are non-communicable between humans except for dermatophytes.
Ab's may develope, but serve no protective role, cell mediated immunity important |