Random Cardo Questions Pt3

Front 1

Shorten phase 3 re-polarization, Fast disassociation from Na Channels, Used in arrhythmia s/p ischemic cardiac event. Class and examples

Back 1

Class IB (Lidocane, Mexilitne, and Tocainide)

Front 2

Increase AP duration, Increase relative refractory period, Good for both atrial and ventricular arrhythmia, (reentrant and ectopic). Class and examples

Back 2

IA (Quinidine, Procainide[SLE like symptoms], Disopyramide)

Front 3

No effect on AP duration, useful in V tach, good for patients with structural abnormalities. Class and examples and Contra?

Back 3

IC (Flecainide, Encainide, Propafenone) Contra s/p MI

Front 4

Hyperkalemia causes an increase in toxicity of which antiarrhithmics?

Back 4

All Class I

Front 5

Decrease cAMP, Decrease Ca2+ currents, Decrease slope of phase 4.

Class and examples

Back 5

Class II (Beta blockers), Propranolol, esmolol, metoprolol, atenolol, and timolol.

Front 6

Increase AP duration, Increase Effective Refractory period, Increase QT. Cause Torsade de points.
What to check? Class and Examples

Back 6

Class III K+ blockers (Sotalol, ibutilide, bretylium, dofetilide, amiodarone) Check, PFT, LFT, and TFT

Front 7

Pt. con cardiac hx on "some med" has pulmonary fibrosis, hepatotoxicity, and thyroid problems. Durg?

Back 7

amiodarone (doesn't cases torsade de points)

Front 8

Primalry affect AV nodal cells, Decrease conduction velocity, Increase Effective Refractory period, Used for nodal arrhithmias. Class and examples

Back 8

Class IV (Ca2+ blockers: Verapamil and deltiazam).

Front 9

Toxicity of a cardiac drug that causes flushing, hypotension, and chest pain. Short acting. Used to diagnose/abolish Supravent. Tach.

Back 9

Adenosine

Front 10

Ca2+ channel blockers that are vasodialators that don't affect conduction?

Back 10

Nifedipine, and Amylodipine.