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218 Cards in this Set
- Front
- Back
When does hyperemesis typically occur in early pregnancy?
|
6-12 weeks
|
|
What can be used to treat hyperemesis in pregnancy?
|
An anti-emetic, like maxalon which is safe in pregnancy
|
|
When does a mother generally start to feel foetal movements?
|
18-20 weeks, depending on placental location
|
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What is a pregnancy test measuring?
|
beta-HCG
|
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How early can home pregnancy tests detect a pregnancy?
|
Two weeks
|
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When taking an obstetrics history what does G3 P2 mean?
|
Gravidity= 3: number of pregnancies
Parity: 2: live births (at any stage of gestation) or still births after 24 weeks. |
|
What antenatal serology is taken?
|
Antentatal serology
- Rhesus - HIV - HepB, C - Syphillus - Variecella - FBC |
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How long does bHCG stay +ve after abortion or miscarriage?
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Remains positive for ≈5d. after miscarriage/abortion or foetal death.
|
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When is a dating US done?
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8-10 weeks
|
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In a nm pregnancy what happens to B-hCG, and for how long?
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B-hCG doubles every 48 hours for the first 8 weeks
|
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What key pre-existing medical conditions need to be addressed in pregnancy? (4)
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1. diabetes
2. hypertension 3. epilepsy 4. thyroid disease |
|
What day of the menstrual cycle does ovulation occur on?
|
Day 14
|
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How long does an egg live for in utero?
Semen? |
Egg: 12-24 hours
Semen: 3 days |
|
What days should a couple have sex on when trying to conceive?
|
10, 12, 14, 16
|
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How long does corpur luteum persist for in pregnancy?
|
10 weeks
|
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After conception, when does implanation occur?
|
Day 6
|
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How does the maternal system react to conception?
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1. progesterone to keep corpus luteum alive
2. modify trophoblast expression of HLA antigens to facilitate maternal tissue acceptance of the fetal graft. 3. occasional there is an implantation bleed |
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How long does the embryonic period last?
|
8 weeks post conception
10 weeks post last menstrual period |
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What does the primitive streak occur in embryonic development?
|
2 weeks
|
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When does the heart develop?
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3 weeks
|
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How long is the human gestational period? (days, weeks)
|
280 days
40 weeks |
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Are maternal and foetal circulation in continuation?
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Nope, they are completely separated.
|
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How does the placental develop?
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Trophoblasts invade the endometrium as fingerlike projections (vili)
|
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What are the functions of the placenta? (5)
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1. gas exchange
2. provision of nutrients 3. waste disposal 4. hormone synthesis: HCG, progesterone, oestrogen, etc. 5. drug transfer |
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What percentage of the worlds population are rhesus -ve?
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15%
|
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How do you treat rhesus -ve women, and when?
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Administer Anti-D at 28 weeks, and 34 weeks.
|
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When is the uterus palpable above the symphis? at the umbilicus?
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symphis: 12 weeks
umbilicus: 18-20 weeks |
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How long is the sac making amniotic fluid?
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until about week 16, after than the baby is making it.
|
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what changes occur in the cervix during pregnancy?
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- softening
- cyanosis - shortening - proliferation of glands - formation of mucus plug |
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What changes occur in the vagina during pregnancy?
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- increased vaginal discharge
- changes colour - becomes more vascular - thickening of vaginal mucosa - loosening of connective tissue - hypertrophy of smooth muscle |
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What skin changes occur during pregnancy?
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1. stria gravidarum
2. diastasis recti 3. pigmentaion |
|
Cervical changes in pregnancy
|
Reduction in cervical collagen towards term enables its dilatation.
Hypertrophy of cervical glands leads to the production of profuse cervical mucus and the formation of a thick mucus plug or operculum that acts as a barrier to infection. Vaginal discharge ↑ due to cervical ectopy and cell desquamation |
|
Breast changes in pregnancy?
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- tenderness
- antenatal secretion of clostrum - increase from week 8 - areolar becomes more pigmented |
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Metabolic changes in pregnancy?
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1. weight changes: uterine hypertropy & contents, fet deposition, fluid retention
2. fluid retention: - decreased plasma osmolality - increased maternal blood volume - pitting oedema |
|
Haematological changes in pregnancy? (6)
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Prothrombotic state:
1. blood volume up by 40-45% 2. RBC increase by 33% 3. increased platalets 4. increased ESR 5. increased fibrinogen 6. decreased fibrinogen |
|
Cardiovascular changes in pregnancy?
|
- HR up 10-15bpm
- increased SV & CO - decreased peripheral resistence - decreased BP for first 20 weeks - exaggerated splitting of the first heart sound. - systolic ejection murmur |
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What percentage of pregnant woman have a systolic ejection murmur?
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90%
|
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Respiratory changes in pregnancy?
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There aren't actually any, but they may be perceived due to the baby pressing up against the diaphragm
|
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Renal changes in pregnancy?
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- increased renal size
- increased renal blood flow - increased GFR - decreased serum creatinine and urea |
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GI changes in pregnancy?
|
Displacement
Delayed gastric emtpying and intestine transit times Reflux & heart burn are common Constipation/ haemorrhoids |
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What are some minor disorders of pregnancy?
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1. nausea
2. vomiting 3. oesophageal reflux 4. constipation 5. epistaxis (nose bleed) |
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What is the standard antenatal visit schedule in a normal low risk pregnancy?
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First visit: 12-16 weeks
4 weekly till 28 weeks 2 weekly till 36 weeks weekly until term |
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When is nuchal translucency investigated?
|
11-14 weeks
|
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When is the foetal morphological scan undertaken?
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18-20 weeks
|
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When are routine FBC's done?
|
First visit
week 28 week 36 |
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When is Anti-D given
|
to rhesus -ve mothers
week 28 week 36 |
|
when is glucose testing done?
|
28 weeks
|
|
How do you test for Group B strep?
|
Low vaginal swab
|
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What are the phases of labour in the first stage?
How long does the first stage last on average? |
Latent phase & active phase (when cervix is >3cm dilated)
Nulliparis: 12 hours Multiparis: 6 hours |
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How long does the second stage last on average?
|
Nulliparis: 20-120 minutes
Multiparis: 10-60 minutes |
|
When is oxcytocin administered in the 2nd stage, and why?
|
Administered with the delivery of the anterior shoulder and helps with the delivery of the placenta.
|
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How long should the 3rd stage of labour last?
|
less than 30 minutes.
|
|
prevalence of ovarian cysts worldwide?
|
7%
|
|
What genetic mutations are associated with ovarian cancer?
|
BRCA-1
BRAC-2 Lynch II syndrome |
|
What are the two common functional ovarian cysts?
|
follicular cysts
corpus luteum cysts |
|
What is most common type of ovarian cyst?
|
Follicular
24% |
|
What makes a follicular cyst?
|
An unruptured Graddian follicle that continues to secrete fluid
|
|
Are follicular cysts generally bilateral?
|
Nope, usually unilateral.
|
|
What are the most common types of benign ovarian neoplasms?
|
serous cystadenoma & mucinous cytadenomas make up 40% and are the most common.
|
|
Follicular cysts, features:
|
unilateral
<5cm secrete oestrogen may result in longer menstrual cycle |
|
40 year old with bilateral moderately sized cyts, and no disruption of her cycle. What is it?
|
Serous cystadenoma
- 30% bilateral - moderate size - 35-55yrs - secrete a thin watery fluid |
|
35 yr old women with a large multilocular ovarian cyst. What is it most likely?
|
Mucinous cytadenoma
- big and multilocular - usually unilateral - secretes mucin |
|
What causes pseudomyxoma pertonei?
|
when a mucinous cytadenoma
ruptures and mucinous cells attach to the peritoneum and omentum. |
|
What are chocolate cysts?
|
endometriomata
|
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A 30yr old presents with bilateral ovarian cysts or moderate size, which contains atypical tissue. What is it likely?
|
Benign teratoma
|
|
What is a fibromata?
characteristics? |
a connective tissue neoplasm of mesenchymal origin.
- usually small - 10% are bilateral |
|
How many women presenting with polycystic ovaries have PCOS?
|
1/3
|
|
What are s/s of polcystic ovaries? (4)
|
Pelvic pain
Bloating Early saitey Palpable adnexal mass |
|
what are risk factors of polycystic ovaries? (7)
|
pre-menopausal
early menarche 1st trimester of pregnancy PCOS increased intrinsic/extrinsic gonadotropins tamoxifen therapy |
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What is the equation for the risk of malignancy index?
|
RMI= UxMxCA125
U: ultrasound score M: menopausal CA-125: serum CA-125 measurement as marker of epithelial ovarian ca. |
|
US signs that increase the risk of an ovarian cyst having malignant potential?
|
1. multi-locular cyst
2. evidence of solid areas 3. evidence of metastases 4. presence of ascites 5. bi-lateral lesions |
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How to treat polycystic ovaries?
|
Observe with repeat US
Suppress with OCP Laparoscopic ovarian cystectomy Laparoscopic oviectomy |
|
Fibroids: details?
|
Aka. leimyomata, fibromyomas
Most common pelvic tumour in women (30-70%) Malignant changes are rare Related to E and P Shrink after menopause |
|
Most common pelvic tumour in women (30-70%)
|
Fibroids
|
|
When do you treat fibroids?
|
When the symptoms are unpleasant (intramenstrual bleeding, miscarriage, compression of bladder, pain, constipation)
when it is affecting fertility |
|
What are some drugs therapies for fibroids?
|
NSAIDs, tranexamic acid
OCP, mirena IUD, progestins GnHR agonist/antagonists |
|
what is preterm?
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<37 weeks
|
|
what is postterm?
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>42 weeks
|
|
low birth weight=
very low birth weight= extreme low birth weight= |
LBW=<2500gm
VLBW=<1500gm ELBW= <1000gm |
|
Risk factors for small babies (both small for gestational age and premature)
|
Socioeconomic: poverty, teenage mums, single mothers, smoking, drug use
Medical: hypertension, renal, autoimmune Placental/uterus: infection, multiple gestation, placental abruptio, placenta praevia Foetal: chromosomal abnormalities, cardiac problems, etc. |
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What are the broad issues involved with preterm delivery for the feotus?
|
1. Asphyxia
2. Thermal instability 3. Pulmonary disease: respiratory distress syndrome/ hyaline membrane disease 4. Metabolic disturbances 5. GI: jandice, necrotising enerocolitis 6. Renal immaturity 7. Cardiac: patent ductus arterosus 8. Haematological 9. Infections 10. Neurological/ neurosensory 11. Retinopathy of prematurity 12. Sensori-neural hearing loss |
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How do manage potential metabolic disturbances in a premature baby?
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Monitor blood sugars and electrolytes
|
|
When do babies get their suck reflex?
|
36 weeks
|
|
What is a potential serious neurological result of prematurity?
|
Intraventricular haemorrhage: bleeding into the ventricles which results in hydrocephalus and potentially a stroke.
|
|
What causes retinopathy of prematurity?
|
vessels being grown too fast in response to oxygen, leads to retinal detachment resulting in blindness
|
|
What is the accepted cut off for potential survival of premmie?
|
23 weeks
|
|
When is a preterm baby considered viable? ie. parents can't decide to withdraw care
|
25 weeks
|
|
Preterm babies between 23-25 weeks suffer what complications?
|
1. chronic lung disease: 50-70%
2. infection: 30-50% 3. intraventricular haemorrhage:10-30% 4. retinopathy of prematurity: 10-50% 5. necrotising enterocolitis: 5-10% |
|
What are some long term respiratory problems associated with premature birth?
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Asthma
Chronic lung disease Home oxygen therapy Increased risk of infection |
|
Do respiratory problems associated with premature birth persist?
|
overt lung abnormalities resolve over 1-2 years
|
|
Do premature babies catch up in terms of growth?
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Yes, over 1-3 years with additional nutrition
|
|
When are vaccinations given to a preterm baby?
|
According to chronological age, not gestational.
|
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What % of extreme low birth weight babies require hearing aids?
|
2-4%
|
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what is the relationship between birth weight and cerebral palsy?
|
ELBW: 50/1000
LBW: 8/1000 NmBW: 1.5/1000 |
|
Is cerebral palsy detected at birth?
|
Nope. May not be picked up on neurological exam until 8-12 months.
|
|
What % of miscarriages have a chromosomal abnormality?
|
50%
|
|
What % of still births have a chromosomal abnormality?
|
5%
|
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What % of live births have a chromosomal abnormality?
|
0.5%
|
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What is the best early screening test for chromosomal abnormalities?
|
NT+biochemistry
Detect 90-94% 2.5-5% false positives 7 unnecessary tests |
|
What is NT? When is it done?
|
nuchal translucency testing
11-13 weeks |
|
Can you use maternal age as a screening tool for T21?
|
No, 45% of DS babies are born to mothers <35yrs.
Only detect 55%, get 18-22% false positives, do 79 unnecessary tests. |
|
Why do you do first trimester tests?
|
Do look for foetal death, multiple pregnancies, or morphological abnormalities.
Early detection give parents privacy to make difficult decisions. |
|
What percentage of NT tests come back low risk?
|
95%
|
|
Where is it harder to get NT?
|
The country, as you require a very skilled sonographer
|
|
NT 3.9 at 12 weeks: high or low risk T21?
|
High, if maternal age is 31:
went from 1/528 to 1/24 |
|
First trimester screening: what should to look for chromosomal abnormalities?
|
Combined test:
11-13+6 weeks: Pregnancy-associated plasma protein A (PaPP-A) + free B-hCG- risk calculation Nuchal thickness (increase=+ve) Performance ˜90% detection for 5% FPR . May detect other abnormalities such as anencephaly. Acceptable detection of all trisomies. Disadvantage: expensive and difficult to perform nuchal scan |
|
Should you do T1 testing or T2 testing?
|
both is best, increases detection rate from 90 to 96.3%
|
|
Beyond NT, what else can be picked up on US to suggest T21?
(3) |
1. Nasal bone (5-6weeks) will be shorter and less mineralised in T21
2. Abnormal ductus venosus 3. Tricuspid regurg |
|
What is CVS and when is it performed?
|
It is sampling of chorionic vili, and chromosomes are analysed from cultured cells.
11-14 weeks EARLIER than amniocentesis |
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What is amniocentisis, and when is it performed?
|
Sampling of amniotic fluid (20mls). Chromosomes analysed from cultured cells.
15+ weeks |
|
Why is fetal blood sampling done? When is it done? Is it common?
|
Uncommon now
Done from 18 weeks Is done when there is risk of foetal transfusion (like in rhesus mothers). |
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What is the gold standard for analysis of chromosomes in samples (CVS, amnio)?
|
Classic cytogenics- but is expensive, and slow, and highly operator dependent.
|
|
What is the miscarriage risk with CVS?
|
<1%
|
|
What is the miscarriage risk with amnio?
|
<0.5%
|
|
What is the miscarriage risk with feotal blood sampling
|
2% is foetus is well
20% if hydrophobic foetus |
|
What normally happens to BP in pregnancy?
|
Drop in both SBP and DBP in first trimester
Normalises in second trimester Approaches pre pregnancy BP in third trimester |
|
Prevalence of gestational HTN?
|
9.8%
|
|
Severe HTN?
|
SBP > 170 mmHg
DBP>110 mmHg |
|
In pregnancy a change of how much is defined as HTN? And from when?
|
30/15mmHg
from BOOKING BP, not pre-pregnancy BP |
|
Define gestational HTN?
|
HTN after 20 weeks without associated systemic changes
|
|
If gestational HTN is still present 15 weeks after the birth, is this normal?
|
Not for gestation HTN
Not it is chronic HTN This cut off is at 12 weeks post partum |
|
When is gestational HTN at risk of becoming pre-eclampsia?
|
If it is present early in pregnancy, or is severe.
|
|
What % of severe gestational HTN diagnosed <30weeks go onto PET?
|
40%
|
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If gestational HTN is dx >37 weeks what % will go onto PET?
|
10%
|
|
HTN <20 weeks, with no known cause is?
|
Essential HTN
|
|
secondary HTN <20weeks may be caused by what?
|
1. chronic kidney disease
2. renal artery stenosis 3. SLE 4. DM 5. Cushings syndrome 6. Primary hyperaldosteronism 7. coarctation of the aorta |
|
What variables should be controlled when measuring BP in pregnancy?
|
Right arm always
Patient sitting, relaxed |
|
When should you measure BP on both arms in pregnancy? Why?
|
On the first visit, to exclude coarcation of the aorta
|
|
If you have a woman with pre-exisiting HTN, and proteinuria, what do you do?
|
She is at higher risk of PET
Diagnose on other systemic/foetal features: such as IUGR |
|
Define pre-ecplamsia
|
HTN that occurs >20 weeks gestation with systemic signs, such as
- proteinuria - haematological involvement - liver signs: LFTs or pain - neurological involvement |
|
Is a raised Alk phos of concern in pregnancy?
|
Nope, it is normally raised in pregnancy
|
|
What is the prevalence of pre-eclampsia in aus?
|
4.2%
|
|
What % of maternal deaths in low and middle income countries is due to PET?
|
99%
|
|
Risk factors for PET
|
Previous PET
>10yrs since last baby Age: >40 BMI>35 Family history Underlying medical disorder |
|
What can be used as prophylaxis for PET?
|
Arpirin has a use
Calcium, Vit C and Vit E have been shown to NOT be useful |
|
What is involved in surveillance of a woman with PET?
|
1. BP measured 2-3x week in day assessment unit
2. Education about signs and symptoms 3. Blood investigations: FBC, UEC, LEFTs, Urate 4. Urinalysis: spot protein, creatinine 5. CTG and or US to monitor foetal growth |
|
Education about signs and symptoms of PET for mother should include what?
|
- headaches
- visual spotting - visual blurring - epigastric pain - dizziness |
|
Pathology of PET
|
1. immunological: foreign body reaction
2. Placental: poor implantation, poor invasion by throphoblastis |
|
Is oedema a key component of PET?
|
It occurs in PET, BUT is not part of the diagnosis because it is so common in pregnancy and is probably of little clinical importance.
|
|
How do you diagnose PET>
|
diagnosis of exclusion
|
|
What are the patho-physiological changes that distinguish PET from normal pregnancy?
|
1. intense vasopasm
2. intravascular coagulation |
|
Why is there increased intravascular coagulation in PET?
|
platelets are on hand to repair the vasculature in the placenta
|
|
what happens to the plasma volume in women with PET?
|
In normal pregnancy plasma volume increases (40-45%)
In women with PET there is slight or unchanged plasma volume Thus haemoconcentration!! |
|
How does the change in blood in PET affect the kidneys?
|
Kidneys are super sensitive little fellas
- protein > +1 - spot protein/creatinine ration >30mg - creatinine >90 |
|
What is HELLP syndrome?
|
HELLP syndrome is a group of symptoms that occur in pregnant women who have:
H -- hemolysis (the breakdown of red blood cells) EL -- elevated liver enzymes LP -- low platelet count |
|
What liver enzymes are we looking at in PET?
|
AST>40 U/L
ALT >40 U/L ALP will probably be up anyway as a result of pregnancy |
|
What is the end-point of PET?
|
ECLAMPSIA- Cerebral changes!
|
|
What occurs with eclampsia?
|
Seizures
|
|
What are the cerebral changes associated with PET?
|
hyperreflexia WITH clonus
persisting headache visual disturbances diminished level of conscoiusness |
|
What haematological problems are associated with PET?
|
DIC!
|
|
What effect can PET have on the foetus?
|
IUGR
Foetal hypoxia Prematurity Placental abruption Intrauterine death |
|
Is an epidural recommend or illadvised in PET patients?
|
Recommended, you dont want a pain related increase in BP
|
|
What antihypertensives are first line in PET?
|
Methyldopa- oldest
Oxyprenolol Labetolol |
|
Second line PET meds
|
Hydralazine
Nifedipine Prazosin |
|
Drug therapy for severe PET?
|
Jump straight to second line:
Hydralazine Nifedipine Prazosin |
|
What BP meds are contraindicated in pregnancy?
|
ARBs
ACEi |
|
Acute Rx for SEVERE HTN in pregnancy?
|
IV labetolol (50mg, repeat after 15-30mins)
IV Hydrazaline (5-10mg, repeat after 30 mins) IV Diazoxide (15-45mg, repeat after 5 mins, max 300mg) Tab Nifedipine (10-20mg, repeat after 45 mins) |
|
PET indications for delivery
|
GA>37weeks
Cant control the HTN Decrease in platelets- worry about DIC Decrease in LFTs, renal funct. Placental abruption Persistent neurological symptoms Pulmonary oedema Eclampsia IUGR |
|
Rx for eclampsia
|
Usually self limitiing seizures
If long: IV diazapan or clonazapem Magnesium sulphate- prevents further seizures! |
|
MgSO4
|
CNS depressant
used: - following a seizure, to prevent the next one - maintenance - in anticipation of delivery |
|
MgSO4 toxicity
|
hypotension
flushing slurred speech absent reflexes |
|
If a baby grows normally in initially, but is growth restricted in the last trimester, what has caused this?
|
placental dysfunction/ insufficiency
|
|
Fetal growth depends on?
|
1. genetics
2. nutrients (O2 and glucose esp) 3. A good foetal circulation 4. Functioning fetal pancreatic B-cells 5. a good blood supple to placental and transfer accross it |
|
What is the relationship between insulin and interutero growth
|
Insulin is one of the main regulators of feotal growth
|
|
What is the most common cause of IUGR?
|
maternal hypertensive disorders
|
|
Will all hypertensive women have small babies?
|
No, the degree to which this affects foetal growth depends on the amount of placental functioning reserve.
|
|
What makes a IUGR foetus likely to die in utero?
|
They are more likely to develop metabolic disorders:
- acidosis - hypoglycaemia - erythroblastosis |
|
macrosomic
|
large baby
DMII |
|
Is being a normal for size baby in a high risk pregnancy better or the same risk wise, than being IUGR?
|
Better
|
|
What additional tests should be done to access foetal well being in a high risk pregnancy?
|
>30 weeks
- foetal movement counts - cardiotocography - serial ultrasound examinations - doppler flow velocity wave forms |
|
How many times should a baby kick in a day, past 30 weeks?
|
10-130
<10, needs to have a cardiotographic assessment |
|
Naegele's rule?
|
(LMP + 1yr 7days) – 3 months
|
|
Gravidity
|
Number of pregnancies, including the current one
|
|
Parity
|
Number of births beyond 24 weeks gestation (including still births)
|
|
Linea nigra
|
dark pigmented line stretching from the xiphi sternum through the umbilicus to the suprapubic area
|
|
striae gravidarum
|
recent stretch marks are purplish in colour
|
|
striae albicans
|
Old stretch marks are silvery, white
|
|
What is the difference between foetal lie and presentation?
|
Lie: what direction the foetal pole is in
Presentation: part of foetus overlying the pelvic brim |
|
polyhdramnios
|
excess of amniotic fluid in the sac
|
|
Pawlick's manoeuvre
|
The presention foetal partis moved between the fingers and thumb of the examiners hand, to determine whether it is the foetal head or breech
|
|
Naegele's rule?
|
(LMP + 1yr 7days) – 3 months
|
|
Gravidity
|
Number of pregnancies, including the current one
|
|
Parity
|
Number of births beyond 24 weeks gestation (including still births)
|
|
Linea nigra
|
dark pigmented line stretching from the xiphi sternum through the umbilicus to the suprapubic area
|
|
striae gravidarum
|
recent stretch marks are purplish in colour
|
|
striae albicans
|
Old stretch marks are silvery, white
|
|
What is the difference between foetal lie and presentation?
|
Lie: what direction the foetal pole is in
Presentation: part of foetus overlying the pelvic brim |
|
polyhdramnios
|
excess of amniotic fluid in the sac
|
|
Pawlick's manoeuvre
|
The presention foetal partis moved between the fingers and thumb of the examiners hand, to determine whether it is the foetal head or breech
|
|
What are the leading causes of death in indigenous women? in order
|
1. cardiovascular disease
2. malignant neoplasm 3. endocrine, nutritional, metabolic 4. smoking |
|
how do cervical cancer rates in indigenous women compare to their non indigenous counterparts?
|
5x
|
|
what is the prevalence of cervical cancer in indigenous women?
|
10.2%
|
|
Indigenous mothers:
Age? Delivery method? |
Younger. 24.8yrs is average
Less likely to be induces, less likely to have CS |
|
What are the socioeconomic complications of teen mothers?
|
<15 biologically immature
>15: poor preconception health care Poor education= financial difficulties socially vulnerable and stigmatised |
|
what % of indigenous mothers smoke?
|
57%
|
|
maternal mortality, indigenous vs non-indigenous?
|
2x as many indigenous
|
|
what is the average distance an indigenous mother travels to deliver?
|
4 hours
|
|
What % of indigenous mothers travelling to give birth, birth en route?
|
12%
|
|
Low birth rate risk factors
|
socioeconomic disadvantage
size & age of mother mothers nutrition illness during pregnancy duration of pregnancy drugs: EtOH, tobacco, others |
|
What is unique about STI's in indegnous women?
|
1/4 of women in remote communities have an STI
They present acutely: - disseminated gonococcal infection - PID - ICU admission for the complication of sepsis |
|
How much of a problem is domestic violence in indigenous populations?
|
24% of ASTI>15yrs have been the victim of threatened or physical violence
|
|
Risk factors for domestic violence?
|
being young
being removed from your family unemployment poverty |
|
What kind of incontinence is treated with retraining?
|
urge incontinence
|
|
How do you treat stress incontinence?
|
pelvic floor exercises
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If pelvic floor muscles fail to manage incontinence, what is your next step?
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Stress incontinence:
- Uridynamics - Surgical sling |
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If you have mixed type urinary incontinence, which do you treat first?
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Treat urge first (retraining, anticholinergics) then stress
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What happened in an overactive bladder?
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detruser over activity: urge incontenence
involuntary leakage of urine |
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What are pregnancy risk factors for incontinence?
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Large baby: >4000g
3rd degree tear forcepts long second stage |
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Which routine test gives you a clinical suspicion of urge incontinence due to overactive bladder?
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stress provocation test
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What is urodynamics?
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Pressure flow study to work out detrusor pressure
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How do you work out detrusor activity in urodynamics?**
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pressure in bladder= abdominal pressure+detrusor pressure
use a abdominal line (vaginal/rectal) and a bladder line and then subtract to get the detrusor pressure |
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when is improvement seen after commencing pelvic floor muscle exercises in stress incontinence?
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1 week
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what are the drugs used to treat OAB?
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antimuscarincs/ anticholinergics:
propanthene, oxybutin, solifenacin tricyclic antidepressants |
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define premature
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born before 37 weeks
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define preterm
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not in labour
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prevalence or premature labour?
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7.5%
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risk factors for prematurity?
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Multiple births is the big one! IVF
PROM spontaneous preterm labour cervical incompetency IUGR Pre-eclampsia Antepartum haemorrhage |
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Long term effects of prematurity
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Chronic lung disease
Neurological deficit: hearing, vision, epilepsy, cerebral palsy |