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113 Cards in this Set

  • Front
  • Back
spinal cord malformation; neural tube defect
spina bifida
non progressive CNS damage before age 3. lack of postural control. Brain damage decreases the brains ability to monitor and control nerve and voluntary muscle activity
cerebral palsy
Lower brachial plexus injury involving C-7, C8, and T-1. Results in weakeness of the intrinsic muscles of the hand and long flexors of the wrist and fingers. The biceps reflex is present but grasp is absent. (claw hand)
klumpke's paralysis
autosomal, chromosome 19. Affects face, neck, hand, and foot. characterized by myotonia, a delay in muscle relaxation time, and muscle weakness.
myotonic dystrophy
abnormal developement of trochanter
trochanteric dysplasia
non progressive neuromuscular disorder estimated to occur in first trimester in utero.
Charasteristics- cylinder like extremeties, significant and multiple contractures, dislocation of joints, muscle atrophy, muscle weakness, and imbalance.
arthrogryposis multiplex congenita
unilateral shortening of SCM muscle. limited cervical ROM. causes lateral flexion to the same side of the contracture and chin rotation to opposite side.
torticollis
results from abnormal development and growth of the hip joint. Testing for this condition includes Ortolani test, barlow maneuver
hip dysplasia
method of decreasing spascity in mucscle of cerebral palsy patients- phenol and botulinum toxin A
neuromuscular blocks
pump faclofen into abdomen and intrathecal space
intrathecal baclofen
trisomy 21 extra chromosome. Clinical manifestations: hypotonia, flattened nasal bridge, Simian line (palmar crease) epicanthal fold, enlargement of the tongue, and developmental delay
down syndrome
lag progrssion as compared to full term. takes 2 years to catch up. due to prematurity
developmental delay
a self limiting disease of the hip initiated by avascular necrosis of the femoral head- 4 stages
legg calve perthes disease
x gene linked recessive by boys. Dystrophin gene missing. destruction of muscle cells collage, adipose laid in muscle, pseudohypertrophic calf muscle
duchenne muscular dystrophy
dislocation at birth
congenital dislocation
results when chromosome 5 is missing. abnormal laryngeal; development of weak and high pitched cry in infants; causes hypotonia, scoliosis, delayed development, small head size, low birth weight.
chr-du-chat syndrome
congenital disorder of collagen synthesis that affects all connective tissue in the body. can cause osteoporosis, excess fractures, bowing legs, spinal deformity, and muscle weakness.
osteogensis imperfecta
congenital talipes equinovarus, ankle plantarflexion, hindfoot varus, and forefoot adduction
club foot
alteration developent of blood vessels in the eye. a vasoproliferative disease of the immature retina.
retinopathy of prematurity
surgery for cerebral palsy; a surgical approach that focuses on the spinal reflex arch and its modulation at the level of the anterior horn cell by supraspinal and segmental influences.
selective dorsal rhizotomy
injections into the dominant agonist muscle at the nerve terminals to causes temporary paralysis of the muscles lasting 3-6 months. improve function of children with cerebral palsy
botulinum A toxin
typical development that followa a predictable sequence. there are variations in the sequence and the rate at which each child moves through the sequence varies
motor development
underutilized areas of the brain take over functions of damaged tissue
vicariance
areas of brain reprogrammed, increased sensitivity of hands to blind
functional subsititution
reaccqusiton of movement skills lost through injury
recovery function
sympathetic nervous system
fight or flight
performance of task visualized
mental practice
study dealing with neural, physical, and behavioral aspects
motor control
ability of brain to change or repair itself
neuroplasticity
sending signals adcance of movement to ready system; allows anticipatory adjustments of postural activity
feed forward
performing two tasks at once (catching and throwing a ball)
dual task training
what is the spinal level for functional ambulation with spina bifida?
L3-L4
what is most important to remember with down syndrome patients?
no approximation through the head
how low to heal fractures in infants?

in children?
4-6 wks

6-8 wks
aversive response to tactile stimuli manifested by hyperactivity or distractibility. children who show this may display avoidance reactions around the hands, feet, and face. in the oral area may cause child to reject textured or flacored food in preference to smoother blander foods.
tacticle defensiveness
serial casting needs to be changed...
every 5-7 days
goals of positioning preterm infant (2)
1. enhance flexor tone with flexed trunk and protracted scapula

2. increase midline orientation
how to measure leg length difference
asis to medial malleolus

1-2 cm
when does mother first feel fetus move?

when do movements decrease?
20 weeks

32 weeks
why do we promote balanced strength in opposing muscle groups for children?
for postural control
primitive reflexes appear after..
1st month
is w sitting good for kids?
no
primitive reflex that affects feeding if it doesn't integrate?
Asymmetrical tonic neck reflex

(ATNR)
3 stages of motor learning
1. cognitive

2. associative

3. autonomous
is it harder for adults to relearn developmental sequence than infants?
yes
modalities prior to stretching
heat, ultrasound, and E-STIM
what is a neonate period
first 28 days after birth
early childhood
3-6 years
later childhood
after 10 years
developmental transistion that lasts from about 10-11 intil the late teens or rarly twentites. the onset of puberty -- 13 YEARS
adolescence
increased tone, results from a motor cortex lesion- mild
spastic cerebral palsy
results from a cerebellar lesion, a balance disorder- moderate
ataxic cerebral palsy
flucating tone, basal ganglia lesion

severe
athetoid/dystonic cerebral palsy
affects one limb
monoplegia (S CP)
affects four limbs
quadriplegia (At CP)
affects two lower limbs- severe
diplegia (Ath CP)
affects one side
Hemiplegia- Mixed CP
flexion contractures are present at hips, and external ROM is slightly greater than internal rotation. knees exhibit genu varum, or bowing
0-9 months normal gait progression
wide base of support, hips in abduction, flexion, and slight external rotation, genu varus continues
9-15 months normal gait progression
genu varus straightens, may be limitation in hip extension ROM, base of support is decreasing, heel strike begins to develop and is normally consistent by 24 months
18-24 months normal gait progression
genu valgus, child normally has consistent heel strike and knee flexion in swing phase
3-3.5 years gait progression
the gait patterns should vbe fully mature, genu valgus returns to neutral
6-7 gait progression
the motor task is to develop an overall understanding of the skill.
1st stage of motor learning-

cognitive stage
motor strageties are refined through continued practice. "how to do"
associative stage

2nd stage of motor learning
motor performance that after practive is largely automatic. -runs themselves
3rd stage of motor learning

Autonomous stage
facilitation techniques to increase tone (7)
1. quick stretch
2. resistance
3. joint approximation
4. joint traction
5. light touch
6. manual contacts
7. fast vestibular stimulation
inhibition techniques to decrease muscle tone (7)
1. prolonged stretch
2. inhibitory pressure
3. maintained touch
4. slow stroking
5. prolonged icing
6. neural watmth
7. slow maintained vestibular stimulation
compares children to other "normal" children

has 4 test sections- assisted in referral to OT and SLP
Denver II
provides a comprehensive sequence of gross and fine motor skills from which the therapist can determine the relative developmental skill level of a child and plan an instructional program to develop those skills; for children from birth to 83 months
peabody developmental motor scale (PDMS)
evaluates muscle tone, primitive reflexes, automatic reactions, and volitional movement in the first year of life; provides a assesment of risk for motor dysfunction
movement assessment of infants (MAI)
assesment of gross motor performance designmed for the identification and evaluation of motor development in infants from term through the age of independet walking; focus is on the development of postural control relative to four postural positions; supine, prone, sitting and standing. from children from birth to 18 months
alberta infant motor scale (aims)
means of evaluating a child's current developmental status at a particular age; consists of scales composed of three parts; a mental scale, a motor scale, an infant behavior record, for children 1-30 months
bayley scales of infant development (BSID)
a motor assessment; used to evaluate child's motor repertoire and capacity for movement; examines quality of movement as opposed to attainment of motor milestones; for children 0-42 months
toddler and infant motor evaluation (TIME)
measures change over time, or after therapy, in gross motor function in children with CP; designed to assess motor function, or how much of an activity a child can accomplish; all item usually can be accomplished by a 5 year old with normal motor abilities
Gross Motor Function Measure (GMFM)
an assessment for detecting functional limitations and disability in age appropriate independence; measures both the capability and performance of functional activities in three content domains: self-care, mobility, and social function; for children 6 months to 7.5 years
Pediatric Evaluation of Disability Inventory (PEDI)
test of disability for assessing functions in self-cae, sphincter control, mobility, locomotion, and communication and social cognition; for children between the ages of 6 months and 7 years
functional independence measure for children (weeFIM)
used to determine developmental level of infants and young children, plan intervention strategies, and assess affects of instruction; birth to 8 years; tests personal-social, adaptive, motor, communication, and cognition
Battelle Developmental Inventory
(BDI)
used to assess function abd guide progran planning for students with disabilities within the educational environment; children with disabilities attebdibg grades K-6; areas testedL participation, task supports, activity performance, physical tasks and cognitive/behavioral tasks
School Function Assessment (SFA)
development of object manipulation (9)
1. rotation of held objects (2)
2. shaking of held objects (4)
3. bilateral hold of two objects (4.5)
4. two handed hold of a single object (4.5)
5. hand to hand transfer (4.5-6)
6. coordinated action with a single object in which one hand holds the object while other bangs it (5-6.5)
7. coordinated action with two objects such as striking two together (6-8.5)
8. deformation of objects (bending, squeezing) (7-8.5)
9. instrumental sequential actions(open a box with one hand take contents out with other) 7.5-9.5
developmental prone progression
(6)
1. prone lying
2. prone on elblows
3. prone on extended arms
4. pivot prone
5. quadruped
6. locomotion in prone
active four point that requires stability around the hip joints caused by cocontraction of the hip musculature. hands are aligned under flexed shoulders and his knees are aligned under flexed hips.
quadruped
infant must develop: stabilzation of the pelvis, head lifting with cephalocaudally progressing antigravity extensore control, and movement of the upper extremeties out of the neonatal position.
prone on elbows
equipment used for positioning (3)
1. sidelyer-(to encourage hands to midline)
2. corner chair- for back stability for sitting
3. bolster chair-(to keeps legs apart in case of tight hip adductors- scissors gait)
equipment used for standing(4)
1. vertical stander
2. a-frame
3. supine stander
4. prone stander (to improve head control)
all used for stability, if child has decreased LE strength
equipment used for LE orthotics (6)
1. heel cup
2. SMO
3. AFO
4. KAFO
5. HKAFO
6. THKAFO
walker equipment (2)
1. anterior walker
2. posterior walker- used more in pediatrics, improve upright posture
crutch equipment(2)
1. axillary
2. forearm
ambulation assistance equipment (3)
1. RGO --
2. Parapodium
3. Gait trainer
how to use standers
1. 20-30 mins 4-5 times per day initially
how to use standers for control of LE contractures
45 mins 2-3 times per day
how to use standers to facilitae bone development
60 mins 4-5 times per week
how to use LE orthotics
use as needed for standing or while placed in standers

at night
PNF pattern D1 Flexion
-lower extremity
Flexion-adduction-external rotation

foot dorsiflexs and inverts; leg externally rotates and pulls up and across body moving into hip adduction and flexion
PNF pattern D1 Extension
lower extremity
extension-abduction-internal rotation

foot plantar flexes and everts; the leg internally rotates and pushes down and out, moving into hip abduction and extension. the knee remains straight.
PNF pattern D2 flexion
lower extremity
flexion-abduction-internal rotation

foot dorsiflexes and inverts; leg internally rotates and lifts up and out, moving into hip abduction and flexion. knee remains straight. (kick out)
PNF pattern D2 extension
LE
extension-adduction-external rotation

foot plantarflexes and inverts; leg externally rotates and pushes down and in moving into hip adduction and extension.
PNF D1 Flexion UE
flexion-adduction-external rotation

hand closes with wrist flexion; arm ext. rotates and pulls up and across the face, moving into shoulder adduction and flexion. elbow is straight
PNF D1 Extension UE
extension-abduction-internal rotation

hand opens with extension; arm internally rotates and pushes down and out, moving into shoulder abduction and extension. elbow is straight
PNF D2 Flexion UE
flexion-abduction-external rotation

hand opens with extension; arm externally rotates and lifts up and out moving into shoulder abductionand flexion. elbow is straight
PNF D2 extension UE
extension-adduction-internal rotation

hand closes with flexion; arm internally rotates and pulls down and across the body moving into shoulder adduction and extension. elbow is straight.
PNF Chop pattern
upper trunk flexion with rotation to right or left- chop is D1 extension
PNF lift pattern
lead arm is D2 flexion; trunk extend and rotate to right or left. both elbows are straight.
the use of active inhibition in conjunction with passive stretching
facilited stretching
deep tendon pressure with passive stretching
inhibitory pressure
an expansion of active assistive movement, manual contancts to provide support and stability
therapeutic guiding
pt moves limb to the end of pain free ROM. a strong isometic contraction of restricting muscles is resisted followed by voluntary relaxation and passive movement into the newly gained range of the agonist pattern.
hold-relax
used to improve mobility; pt is instructed to relax as therapist moves the pt passively thru the range establishing appropriate speed and rhythmn using verbal cues. finally movements are lightly resisted.
rhythmic initation
used to improve mobility; repeated isotonic contractions are performed directed to agonist muscles, initiated by a quick stretch and enhanced by resistance. (PNF Patterns)
repeated stretch
used to improve mobility; pt is positioned in the end position(shortened range) of a movement and is told to Hold don't let me move you. the isometric contraction is resisted followed by voluntary relaxtion and passive movement into the lengthened range. then instructed to push back into end position again.
replication
(hold-relax-active)
used to improve stability; alternating isotonic contractions of first agonists and then antagonists (don't let me push u backwars, then forward) against resistance allowing only very limited movement.
stabilizing reversals
(alternating isometrics)
used to improve stability; isometric contractions of anatgonist patterns, focusing on co-contraction of muscles. resistance applied to one segment while applying resistance to the other segment. a twisting motion
rhythmic stabilization
used to improve stability; isotonic contractions of first agonists then antagonists perfomed against resistiance. therapist resists contraction of one pattern, at the end of the desired range a command is given to guve reverse direction
dynamic reversals (slow reversals)
used to improve controlled mobility; resisted concentric contraction of agonist muscles moving thru the range is followed by a stabilizing contraction and then eccentric, moving slowly back to start position. (push up. now hold. now go down slowly.)
combination of isotonics (agonist reversals)