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36 Cards in this Set

  • Front
  • Back

Panic Attack (PA)

- not a disorder


- intense fear/discomfort


- sudden onset and peak rapidly (<10 min)


- accompanied by 4+ physical symptoms: sweating, increased heart rates, gastrointestinal distress, muscle tension


cued v.s. uncued


- prevalence: female > male

Panic Disorder (PD)

- recurrent and unexpected PA


- followed by 1+ month of at least one from the following: persistence concern about having another PA, persistence concern about implications of PA, significant change in behaviors


- prevalence: female > male

Agoraphobia

- anxiety about places / situations where they can't escape and help may not be available


- may caused by PD


- situation avoided or endured with distress


- e.g. being in crowd, on a subway, being or leaving the house alone


- prevalence: female > male

Separation anxiety disorder

- excessive anxiety concerning the separation from primary caregiver or home


- 4+ weeks (6 months for adults)


- duration of the symptoms, abnormality in behaviours and physical impairment can serve as key indications of the disorder


- e.g. kids acting sick so they don't have to leave their parents for going to school


- prevalence: female > male

Selective Mutism

- consistent failure to speak in social situations


- interfere with achievement


- duration > or = 1 months


- other disorders need to be ruled out so such behaviour can be considered to be "selective mutism"


- prevalence: female = male; lowest



Specific phobia

- marked and persistent fear


- exposure triggers fear and anxiety


- situation avoided / endured with distress


- fear/anxious object is not an actual danger


- has subtypes e.g. animal, blood-injection-injury, situational, natural/environmental

Social Anxiety Disorder (social phobia in DSM-4)

- fear of being focus of the attention / scrutiny


- fear of being humiliated


- exposure triggers fear and anxiety


- fear is persistent (6+ months)


- prevalence: female> male in community setting, female=male in clinical setting (patients reaching help); highest



Generalized anxiety disorder (GAD)

- chronic / exaggerated worry / tension (6+ months)


- unable to control worry


- 3+ physical symptoms for adults and 1+ for children (restlessness, easily fatigued, difficulty concentration, irritability, muscle tension, sleep disturbance)

Order of Age of Onset (early to late)

Separation anxiety (~ preschool)


Selective Mutism (<5)


Specific Phobia (7-11)


Social Anxiety (8-15)


Agoraphobia (17)


Panic Disorder (20-24)


Generalized Anxiety (<30)

Internalizing disorder

- comorbidity of anxiety disorder and depression in kids


- the difference between anxiety and depression in children is quite ambiguous so it's usually address under the umbrella of internalizing disorder

Biological Approaches of Anxiety Etiology


Structural theory: Dual Pathway Model of Fear

stimulus --> thalamus


--> amygdala (reflexives / store emotional info) or cerebral cortex (slow though processing / cognition)




Biological Approaches of Anxiety Etiology


Psychophysiological Theory

- poorly regulated fight or flight


- overreactive autonomic nervous system (either the body respond too sensitively or paying to much attention to those normal body response)

Biological Approaches of Anxiety Etiology


Genetic Theory

- you are more likely to have anxiety disorder if your family has it


- gene candidate: serotonergic system (5-HT, SLC6A4), dopaminergic system (DRD4, DRD2), modulation of monoamine metabolism (MAOA, COMT)


- serotonergic for negative emotion, dopaminergic for positive emotion, monoamine for degradation of serotonin

Biological Approaches of Anxiety Etiology


Neurotransmitter Theory

Poor regulation of NT: high NE (stimulating), low 5-HT(mood regulating), GABA (inhibitory so calm body down / GAD deficient in GABA and GABA receptor)

Personality Trait Theory

- Neuroticism / negative affectivity


- Behavioral inhibition in early stages increase risk getting social phobia

Psychological Approaches of Anxiety Etiology


Cognitive Theory

- high anxiety sensitivity: catastrophic interpretation to body response or event -


- hyper-vigilant to potential threat


- cognitive self-evaluation model (social phobia) e.g. too sensitive in social rejection, have high standards of social values about themselves

Triple Vulnerability Model

- Biological vulnerability


- Specific Psychological vulnerability: maladaptive interpretation on events of response


- Generalized psychological vulnerability: external locus of control

Psychological Approaches of Anxiety Etiology


Behavioural Theory

- acquired through classical conditioning (can be via observation), maintained by operant conditioning


Biological Treatments to Anxiety

- Tricyclic antidepressant: increase level of NTs (NE, don't work with PD but work with GAD)


- SSRI: increase serotonin (less neg emotion)


- Benzodiazepine: suppress CNS and influence NT (NE, 5-HT, GABA / addictive)



Behavioural treatments to Anxiety

- Modelling: therapist model appropriate behaviour


- Implosive therapy (flooding/ intense exposures)


- systematic desensitization (gradual exposure)

Systematic Desensitization

- Fear hierarchy development --> teach relaxation --> practice with gradual exposure --> practice relaxation while experiencing panic symptoms (behavioural)


- identify maladaptive cognition --> challenge catastrophic thoughts (cognitive)

Treatment effectiveness for Anxiety

GAD: short term, CBT > pharmacotherapy


Panic disorder: remain improved at follow up, CBT = pharmacotherapy (but pharm. has higher rate of relapse)

Obsessive-Compulsive Disorder (OCD)

- either obsession or compulsion


- time consuming symptoms ( > 1 hour/day)


- cause distress and impairment


- recurrent cycle of OCD:


stimulus (internal/external)


--> obsession <--> distress/anxiety


--> ritual behaviours <--> temporary relief (reinforcement of behaviour)




Obsession

- unwanted, anxiety-causing, recurrent and persistent urge, thoughts or images e.g germ contamination


--> thoughts are not worries to real life problems (GAD)


--> not delusional thoughts (use reality-testing)


--> may show attempt to suppress or to neutralize with actions/thoughts





Compulsions

- repeated behaviours or mental acts


- aim to reduce distress (e.g. neutralizing obsessive thoughts or reduce anxiety)



Biological Theory of OCD

- brain dysfunction = lack of control in primitive behaviours


- linked to 5-HT


- genetic predisposition

Cognitive-Behavioural Theory of OCD

- can not turn off the obsessive thought as their brain wired differently

Psychodynamic Theory of OCD

- Obsessions and compulsions represent symbolic conflicts


- formulation example: feel guilty of the past mistake leads her attempt to undo feelings of shame through cleansing rituals

Biological Treatments for OCD

- 5-HT enhancing / antidepressant drugs (e.g. prozac, zoloft, paxil)


- psychosurgery used for severe cases (anterior cingulotomy/capsulotomy)


- Treat the pathway from stimuli to obsession/distress



Behavioural Treatments for OCD

- Exposure and response prevention --> preventing from engaging rituals hen exposed with obsessive thoughts stimuli


- Treat the pathway from obsession<--> distress & ritual behaviours <--> temporary relief

Cognitive therapy for OCD

- Encourage change in thinking--> accepting the brain wire differently


- Treat the pathway from obsession <--> distress

Posttraumatic Stress Disorder (PTSD)

- experienced traumatic events with actual/threatened death or injury


-intrusive symptoms (nightmares/flashbacks)


- avoidance of stimuli


- negative alteration in mood/cognition (sudden change e.g. dropout of uni)


- marked alteration in arousal/activity (panic/difficulty in sleeping)


- fear/anxiety persisting after trauma (> 4 weeks)


- if <4 weeks = Acute Stress Disorder


- Cortisol level peak slow in stress --> not responsive to stress management

Other Stressor related disorder

Adjustment disorder - distress in major transition in life but not necessarily traumatic / within 3 months of stressor / youth diagnosis




Acute stress disorder - same symptoms as PTSD but <4 weeks

Treatments of PTSD

CBT: systematic desensitization (extinguish fear) / cognitive strategy (challenge irrational thoughts)




Stress management: problem solving to reduce stress / use of "thought stopping" to treat intrusive thoughts




Biological therapies: antidepressant/antianxiety drugs




Eye movement desensitization: side to side eye movements while the patient attends and react to traumatic stimuli (belief: once traumatize, you are not processing the thoughts in the right way and RAM sleep recovery can help that)


--> not proven more effective compare to exposure tech.

PTSD Vulnerability

determined by


- sociocultural factors (proximity, duration, severity, social support)


- psychological factor (personal assumption, distress,coping styles)


-biological factors (physiological hyperactivity, genetics, epigenetics affect DNA methylation to decrease capability in regulating stress hormone, HPA axis hypoactivity, developmental stages that are more susceptible or not)





Summary of Major Anxiety Disorders

Phobia: fear avoidance of harmless objects or situations




Panic Disorder: sudden physiological symptoms + recurrent panic attacks




Generalized Anxiety Disorder: persistent, uncontrollable worry




Separation Anxiety: worry about losing contact/proximity, esp. significant others (common in youth)




Agoraphobia: fear of being in public places (comorbid with PD)




Substance induced anxiety e.g. caffeine overdose