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35 Cards in this Set

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in conditions associated with a decrease in lung compliance (e.g., pulmonary edema, pneumonia), positive-pressure lung inflation tends to
compress the heart and intrathoracic blood vessels
decrease in lung compliance (e.g., pulmonary edema, pneumonia), positive-pressure lung inflation tends to
compress the heart and intrathoracic blood vessels
decrease in lung compliance (e.g., pulmonary edema, pneumonia), positive-pressure lung inflation tends to
compress the heart and intrathoracic blood vessels
Positive-pressure lung inflation can reduce ventricular filling in several ways, as indicated in Figure 24.3. First, positive intrathoracic pressure decreases the
ecreases the pressure gradient for venous inflow into the thorax (although positive-pressure lung inflations also increase intra-abdominal pressure, and this tends to maintain venous inflow into the thorax
Positive-pressure lung inflation can reduce ventricular filling in several ways, as indicated in Figure 24.3. First, positive intrathoracic pressure decreases the
ecreases the pressure gradient for venous inflow into the thorax (although positive-pressure lung inflations also increase intra-abdominal pressure, and this tends to maintain venous inflow into the thorax
Positive-pressure lung inflation can reduce ventricular filling in several ways Second,
positive pressure exerted on the outer surface of the heart reduces cardiac distensibility, and this can reduce ventricular filling during diastole
Positive-pressure lung inflation can reduce ventricular filling in several ways Second,
positive pressure exerted on the outer surface of the heart reduces cardiac distensibility, and this can reduce ventricular filling during diastole
Positive-pressure lung inflation can reduce ventricular filling in several ways Second,
positive pressure exerted on the outer surface of the heart reduces cardiac distensibility, and this can reduce ventricular filling during diastole
Positive-pressure lung inflation can reduce ventricular filling in several ways Second,
positive pressure exerted on the outer surface of the heart reduces cardiac distensibility, and this can reduce ventricular filling during diastole
Positive-pressure lung inflation can reduce ventricular filling in several ways lastly
compression of pulmonary blood vessels can raise pulmonary vascular resistance, and this can impede right ventricular stroke output. In this situation, the right ventricle dilates and pushes the interventricular septum toward the left ventricle, and this reduces left ventricular chamber size and left ventricular filling. This phenomenon, known as ventricular interdependence, is one of the mechanisms whereby right heart failure can impair the performance of the left side of the heart
Positive-pressure lung inflation can reduce ventricular filling in several ways lastly
compression of pulmonary blood vessels can raise pulmonary vascular resistance, and this can impede right ventricular stroke output. In this situation, the right ventricle dilates and pushes the interventricular septum toward the left ventricle, and this reduces left ventricular chamber size and left ventricular filling. This phenomenon, known as ventricular interdependence, is one of the mechanisms whereby right heart failure can impair the performance of the left side of the heart
Positive-pressure lung inflation can reduce ventricular filling in several ways lastly
compression of pulmonary blood vessels can raise pulmonary vascular resistance, and this can impede right ventricular stroke output. In this situation, the right ventricle dilates and pushes the interventricular septum toward the left ventricle, and this reduces left ventricular chamber size and left ventricular filling. This phenomenon, known as ventricular interdependence, is one of the mechanisms whereby right heart failure can impair the performance of the left side of the heart
Positive-pressure lung inflation can reduce ventricular filling in several ways lastly
compression of pulmonary blood vessels can raise pulmonary vascular resistance, and this can impede right ventricular stroke output. In this situation, the right ventricle dilates and pushes the interventricular septum toward the left ventricle, and this reduces left ventricular chamber size and left ventricular filling. This phenomenon, known as ventricular interdependence, is one of the mechanisms whereby right heart failure can impair the performance of the left side of the heart
Positive-pressure lung inflation can reduce ventricular filling in several ways lastly
compression of pulmonary blood vessels can raise pulmonary vascular resistance, and this can impede right ventricular stroke output. In this situation, the right ventricle dilates and pushes the interventricular septum toward the left ventricle, and this reduces left ventricular chamber size and left ventricular filling. This phenomenon, known as ventricular interdependence, is one of the mechanisms whereby right heart failure can impair the performance of the left side of the heart
Whereas compression of the heart from positive intrathoracic pressure impedes ventricular filling during diastole, this same compression facilitates
ventricular emptying during systole.
Whereas compression of the heart from positive intrathoracic pressure impedes ventricular filling during diastole, this same compression facilitates
ventricular emptying during systole.
Whereas compression of the heart from positive intrathoracic pressure impedes ventricular filling during diastole, this same compression facilitates
ventricular emptying during systole.
Positive-pressure lung inflation tends to reduce ventricular filling during diastole but enhances ventricular emptying during systole.
Positive-pressure lung inflation tends to reduce ventricular filling during diastole but enhances ventricular emptying during systole.
Positive-pressure lung inflation tends to reduce ventricular filling during diastole but enhances ventricular emptying during systole.
Positive-pressure lung inflation tends to reduce ventricular filling during diastole but enhances ventricular emptying during systole.
Positive-pressure lung inflation tends to reduce ventricular filling during diastole but enhances ventricular emptying during systole.
Positive-pressure lung inflation tends to reduce ventricular filling during diastole but enhances ventricular emptying during systole.
Positive-pressure lung inflation tends to reduce ventricular filling during diastole but enhances ventricular emptying during systole.
Positive-pressure lung inflation tends to reduce ventricular filling during diastole but enhances ventricular emptying during systole.
positive-pressure ventilation increases cardiac stroke output when
When intravascular volume is normal and intrathoracic pressures are not excessive, the effect on afterload reduction predominates, and positive-pressure ventilation increases cardiac stroke output
positive-pressure ventilation increases cardiac stroke output when
When intravascular volume is normal and intrathoracic pressures are not excessive, the effect on afterload reduction predominates, and positive-pressure ventilation increases cardiac stroke output
positive-pressure ventilation increases cardiac stroke output when
When intravascular volume is normal and intrathoracic pressures are not excessive, the effect on afterload reduction predominates, and positive-pressure ventilation increases cardiac stroke output
positive-pressure ventilation increases cardiac stroke output when
When intravascular volume is normal and intrathoracic pressures are not excessive, the effect on afterload reduction predominates, and positive-pressure ventilation increases cardiac stroke output
The increase in stroke volume causes an increase in systolic blood pressure during lung inflation; a phenomenon known as
reverse pulsus paradoxus
The increase in stroke volume causes an increase in systolic blood pressure during lung inflation; a phenomenon known as
reverse pulsus paradoxus
The increase in stroke volume causes an increase in systolic blood pressure during lung inflation; a phenomenon known as
reverse pulsus paradoxus
The increase in stroke volume causes an increase in systolic blood pressure during lung inflation; a phenomenon known as
reverse pulsus paradoxus
The increase in stroke volume causes an increase in systolic blood pressure during lung inflation; a phenomenon known as
reverse pulsus paradoxus
When intravascular volume is reduced, the predominant effect of positive intrathoracic pressure is to
reduce ventricular preload and, in this setting, positive-pressure ventilation decreases cardiac stroke output. This emphasizes the importance of avoiding hypovolemia in the management of ventilator-dependent patients.
When intravascular volume is reduced, the predominant effect of positive intrathoracic pressure is to
reduce ventricular preload and, in this setting, positive-pressure ventilation decreases cardiac stroke output. This emphasizes the importance of avoiding hypovolemia in the management of ventilator-dependent patients.
When intravascular volume is reduced, the predominant effect of positive intrathoracic pressure is to
reduce ventricular preload and, in this setting, positive-pressure ventilation decreases cardiac stroke output. This emphasizes the importance of avoiding hypovolemia in the management of ventilator-dependent patients.
When intravascular volume is reduced, the predominant effect of positive intrathoracic pressure is to
reduce ventricular preload and, in this setting, positive-pressure ventilation decreases cardiac stroke output. This emphasizes the importance of avoiding hypovolemia in the management of ventilator-dependent patients.
When intravascular volume is reduced, the predominant effect of positive intrathoracic pressure is to
reduce ventricular preload and, in this setting, positive-pressure ventilation decreases cardiac stroke output. This emphasizes the importance of avoiding hypovolemia in the management of ventilator-dependent patients.