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117 Cards in this Set

  • Front
  • Back
ONE OF THE TOP THREE CAUSES OF LAWSUITS
Inadequate preop assessment is now one of the top three causes of lawsuits!
TYPES OF SURGERY THAT ARE LOW RISK < 1%
skin, breast, urologic, and minor ortho, cataract surgery
TYPES OF SURGERY THAT ARE INTERMEDIATE RISK <5%
abdominal (lap chole, intrathoracic, ortho, and carotid
High risk – usually >5%
emergent, aortic and other vascular surgery (AAA, CABG), anticipated prolonged surgical procedures associated with large fluid shifts and/or blood loss
Anesthesia History: any complications
MH, slow wakeup, nausea/vomiting, difficult airway
Family History
Inheritable diseases
NPO Guidelines
LONGER FOR...
Longer for pregnant, DM, renal pts, hx of GERD, hiatal hernia, esophageal narrowing
NPO GUIDELINES
Clear Liquids (water, pulp-free juice, clear tea, black coffee, carbonated beverages) 2 HOURS
Breast milk 4 HOURS
Infant formula/nonhuman milk 6 HOURS

Light meal (toast, crackers, clears) 6 HOURS
Fried/fatty foods, meat > 8 HOURS
TYPES OF ANAPHYLAXIS
Type I – immediate hypersensitivity reaction; IgE mediated release
Atopic – dermatitis, allergic rhinitis
Anaphylactoid – generalized hives, edema, erythema associated with antigen-antibody process
25% will get allergic rxn to cefazolin—give vanco or clindamycin(less SE than vanco) Do not push clinda will get IGG rxn
Shellfish/seafood: not linked to IV iodine contrast allergy
Dermatitis after topical iodine may alert to IV allergy
WHICH BLOOD PRESSURE MED WOULD YOU TELL A PATIENT TO HOLD DAY OF SURGERY?
ACE inhibitors, especially angiotensin II antagonists (AIIAs)
ACE inhibitors were associated with hypotension in 100% of pts during induction vs. only 20% in whom ACE inhibitors were withheld on morning of surgery (1994)
Bertrand and colleagues demonstrated severe hypotensive episodes requiring vasoconstrictive therapy after induction in pts chronically treated with an AIIA and receiving this drug on morning of surgery than in those whom AIIAs were withheld day before and morning of surgery
WHICH DRUGS SHOULD A PATIENT CONTINUE TO TAKE MORNING OF SURGERY?
Antihypertensive
Antianginals
Antiarrhythmics
Anticonvulsants (Alter the hepatic metabolism of many drugs and induce cytochrome P450 enzyme activity)
Hormone therapy
ORAL HYPOGLYCEMIC MEDS AND DAY OF SURGERY?
Oral hypoglycemic medications - may produce hypoglycemia for long as 50 hours after intake
TOLINASE, ORINASE, MICRONASE, GLUCOTROL, DIABINESE
Implanted insulin pump - ask pt how controlled is diabetes; ask rate; time of surgery; time usually between each meal
insulin pump just decrease the dose in half.
Discontinue drugs
NSAIDS – seven days
ASA – should be stopped 10-14 days before surgery
Coumadin/Plavix – up to surgeon; PT/INR; usually within 24 hours
Tricyclics/MAOI’s – if possible stop 2 weeks prior; or continue but no indirect acting sympathomimetics or demerol
Tricyclics/MAOI’s
if possible stop 2 weeks prior; or continue but no indirect acting sympathomimetics or demerol
Excitatory or depressive interaction
Excitatory – agitation , headache, hemodynamic instability, fever, rigidity, convulsions, and coma - Thought to be due to excessive central serotoninergic activity – meperidine blocks neuronal uptake of serotonin
Depressive – respiratory depression, hypotension, and coma as result of MAOI inhibition of hepatic microsomal enzymes and meperidine accumulation
LIFE SPAN OF A PLATELET
11 DAYS
Steroids
If on supplemental steroids >one month, give stress dose
100mg on induction, then 50 mg every 6 hours x , or
Stoelting, 25mg on induction then total of 100mg over next 24 hours
Minimum drug dose that would produce and appropriate effect is desirable
Avoid Etomidate!!
Steroids for at least a month long for less than 6 months ago.
Alzheimer’s therapy
prolongs sux
Lithium
potentiates NMB; check sodium level
Clonidine
decreases anesthetic requirements
Aminoglycosides
potentiates NMB
HIV meds (protease inhibitors end with “vir”):
potentiate versed
Recommendation is to D/C herbals
2 weeks before surgery
Echinacea (echinacea purpura
Increased toxicity of drugs dependent on hepatic metabolism (phenytoin, phenobarbital, rifampin)
Garlic (Allium sativum)
Uses: Lipid lowering, vasodilatory, antihypertensive, antiplatelet, antioxidant, antithrombotic/fibrinolytic qualities
Issues:
Inhibition of platelet aggregation
Increased fibrinolyismay increase risk of bleeding
Ginkgo (Gingko biloba)
Uses: circulatory stimulant, antioxidant, anti-inflammatory effects; used to treat claudication, tinnitis, vertigo, memory loss, dementia, sexual dysfunction
Issues:
Inhibition of platelet activating factor may increase risk of bleeding
May decrease effectiveness of anticonvulsants
Ginseng (Panax ginseng)
Uses: enhances energy level, anitoxidant, aphrodisiac; lowers blood glucose
Issues:
Ginseng abuse syndrome: sleepiness, hypertonia, edema; also tachycardia, hypertension with other stimulants
Intraop hypotension
Hypoglycemia in diabetics
Inhibition of platelet aggregation
May interfere with effect of warfarin makes effects more
Kava-kava (Piper methysticum)
Uses: sedation, anxiolysis; treatment for gonorrhea, skin diseases
Issues:
May inhibit norepinephrine
Potentiates sedating effects of barbiturates, benzos, alcohol
Saw Palmetto (Serenoa repens)
Uses: treatment for BPH
Issues:
Inhibition of 5-α reductase
Inhibition of cyclooxygenasemay increase risk of bleeding
Vitamin E
Uses: slows aging process, prevention of stroke and pulmonary emboli, prevention of atherosclerosis, promotion of wound healing
Issues:
May increase bleeding
TIAs
5% - 6% annual mortality rate after TIAs is due to mainly MI
Parkinson’s
Avoid drugs that inhibit release of dopamine or compete with dopamine at the receptor avoid reglan***
Reglan causes pyramidal side effects like shaking and drooling
Dementia/Alzheimer’s
Usually on cholinergic mediations
PORPHYRIA
– type 1,3, and 4, could cause life threatening neurologic abnormalities (THIOPENTAL & SULFA CAUSES IT)
Autosomally inherited lack of functional enzymes active in the synthesis of hemoglobin
Certain drugs can induce the enzyme ALA synthetase – exacerbating the disease
Colicky pain, n/v, severe constipation, psychiatric disorders, and lesions of the lower motor neuron that could progress into bulbar paralysis
Avoid barbiturates, diazepam, phenytoin, ergotamine prep, sulfanomides
Adm glucose suppresses ALA synthetase activity and prevents and ablates acute attacks
Myasthenia Syndrome
Eaton-Lambert syndrome
Associated with small cell carcinoma of lung
A prejunctional defect in the release of acetylcholine; body produces antibodies against ca channels of tumor; however these antibodies cross react to ca channels at the NMjunction.
Muscle weakness improves with repeated effort; unaffected by anticholinesterase drugs
Sensitive to depolarizing and nondepolarizing agents
Myasthenia Gravis
Destruction or inactivation of postsynaptic acetylcholine receptors leading to reduce number of NMJ sites
Unpredictable reaction to NMB
Resistant to succ, but lead to phase II
Sensitive to nondepolarizers
Good response to anticholinesterases
EX: pyridostigmine tx (NMB reversal)
If people use sux they are more likely to get a phase II block b/c of the larger dose. Require less of non depolarizing. They are treated with anticholinesterases, so when trying to reverse from nondepolarizers they don’t respond as well. May have an unpredictable response to the reversal.
MURMURS?
20-50% number of adults with undiagnosed PDA
Cardiovascular
Stress response of surgery
Physiological response of surgical assault
Blood is diverted from areas of body to head & heart, BP & HR increase – CV system has to be in optimal health
Determination of Ischemic heart disease/CAD
Recent MI
Perioperative infarction rate is higher in the first 6 months after a previous MI
American College of Cardiology and AHA indicate a liberalization of surgery to 6 weeks after infarction to ‘low-risk pts’
Due to changes in tx of MI – thrombolytics, PTCA
Six weeks will allow myocardium to heal - reduces risk of arrhythmias and rupture of ventricular aneurysms
Recent cardiac event
you are more at risk in surgery for perioperative cardiac re-event.
Conditions that could/should be corrected/stable before surgery – (ideally
Recent MI/severe ICD
Severe CHF (rales, an S3 gallop, or JVD)
Severe angina (unstable angina)
Cerebrovascular disease
Heart rhythm other than sinus
Chronic renal insufficiency
Serum creatinine > 2.0 mg/dL (at higher risk for further renal damage)
Does adequate control of HTN result in complications that could be prevented with some control?
Pt should be educated regarding importance of lifelong treatment of HTN to prevent arterial aging/damage
Perioperative hemodynamic fluctuations occur less frequently in treated than untreated hypertensive pts
Hemodynamic fluctuations have some relationship to morbidity
Pacemaker / AICD (settings, firing, manufacturer)
Demand pacemakers can sense electrocautery therefore will inhibit pacemaker firing
Convert to fixed rate or default program – know pt’s settings, manufacturer, and type
Magnet or programming device  contact EPS (turns device off)
Grounding pads should be as far from the generator and leads as possible; should use Bipolar cautery
Drug eluting stents (DES) vs bare metal stents
Challenges with bleeding vs perioperative stent thrombosis
Cardiology consult
Elective surgery should be delayed if less than 6 months since placement of DES
If emergent, ASA and Plavix should be continued if possible
Obstructive Sleep Apnea
Airway collapse d/t loss of muscle tone
During REM sleep – pronounced in obese pts
Snoring caused by airway vibration from turbulent flow
Repeated collapse (obstruction) of upper airway develops during sleep with cessation of airflow, however resp efforts continue.
Obstructions and apnea lead to SNS activation and arousal from sleep, restoring muscle tone.
OSA pt have 7 x increase in mortality
Obesity and Plastic bottle use increase risk
Review sleep study/Respiratory Distress Index (RDI)
RDI > 10 monitored bed overnight vs ICU
ANESTHESIA AND OSA
Anesthetic agents worsen OSA by decreasing pharyngeal tone and attenuating normal responses to hypoxia and obstruction
Supine position worsens OSA
OSA pt have redundant pharyngeal tissue: difficult airway mgt
Grey zone: mgt of OSA in ambulatory surgery setting
GA may increase number and duration of sleep apneas
Inhibits arousal which would occur during sleep
Judicious use of sedatives and narcotics; use short acting agents; non steroidal analgesics if not contraindicated
Difficult airway precautions
Regional or local anesthesia when appropriate
CPAP in PACU available
Smoking
SMOKING – risk factor for cardiac and pulmonary disease
Cigarettesnicotine + carbon monoxide
Nicotine – adrenergic effects
Carbon monoxide – occupies oxygen-binding sites of Hgb
Long Term:
Hypersecretion of gastric acid and hepatic enzyme stimulation
Persists for 6-8 weeks after cessation
Development of obstructive airway disease
Chronic bronchitis
Emphysema – over production of sputum & reduction of ciliary motility & distal airway function
Decreased CO only benefit of decreasing smoking hours before surgery
SMOKING ANESTHETIC CONSIDERATIONS
Anesthetic considerations
Airway is hyperactive
Excessive coughing & bucking
Bronchospasm and rapid desaturation
Cessation for 24hrsreduces carboxyhgb and may improve oxygenation
Cessation 24hrs – 6 weeks increases incidence of morbidity
Cessation >6weeks returns oxygenation and mucociliary clearance but not to normal.
Post-op complications: atelectasis, pleural effusions, and pneumonia
Post thoracic/abdominal cases
Need 8 week cessation before drastic reduction of post-op complications
Ulcerative colitis, Crohn’s Disease
Associated with electrolyte imbalances
Dehydrated, malabsorption & malnourished
If active, could have GI bleeding, GI obstruction, perforation of colon, toxic megacolon
Anorexia / Bulemia
Malnourished, dehydration, electrolyte imbalances
Hyperthyroidism – Thyroid Storm
Does your face flush or get red every now and then, even when you’re not exercising? Hyperthyroidism?
Tachycardia
Hyperthermia
Labile blood pressure –could be dehydrated and vasodilate during induction
Hyperthyroidism – Thyroid Storm
Treatment
Avoid surgery until euthyroid
Hydration and cooling
Beta blocker
Correction of precipitating cause
Avoid anticholinergics – could interfere with normal heat regulating mechanisms
Avoid ketamine, pancuronium, indirect-acting adrenergic agonist and other drugs that stimulate the SNS
Increase requirements of sedatives
anything that stimulates the sympathetic nervous system avoid.
DIABETIC ISSUES
Increases morbidity and mortality by 5-10 times
Accelerates atherosclerosis and end organ damage
Causes neuropathy, retinopathy
Often has silent ischemia – painless MI
50% likelihood of autonomic nervous system dysfunction with HTN
Reflex dysfunction: increase risk for CV instability
Delayed gastric emptying
Poor wound healing
Renal insufficiency
Joint collagen tissue abnormalities
DIABETES TREATMENT IN OR
Schedule early surgery
Check BS in pre-op holding
if brittle = every one hour in OR
Type II = every 3-4 hours in OR
Start IV NS (or LR is BG lower)
If taken insulin or oral agent
D51/2NS
Check blood sugar every 1-2 hours in OR
if you have peripheral neuropathy you also have
autonomic neuropathy. Sometimes people with parkinsons have this and also older people. Remember this. You would know that it was autonomic neuropathy b/c their heart rate didn’t increase which is a normal response when blood pressure drops. Tachycardia is the first sign of low blood sugar under anesthesia is tachycardia. If they are on beta blockers you won’t see this.
DMs who use NPH
(neutral protamine hagedorn) or protamine zinc insulin are at greater risk of allergic reaction to protamine sulfate
Massively hypotensive from protamine reaction
If they have had protamine before or they are taking NPH should check a test dose to make sure that they are not going to have a protamine reaction.
Each cc of D50, will rise BS of a 70kg person
2mg/dl approx.
If fragile diabetic
Start IV D5W at 1cc/kg/hr mix 50u Reg Insulin in a 250ccNS = 1unit/5cc; start at 5cc/hr
At one hour: check BS then divide by 150 to set insulin drip rate
If BS = 300; divide by 150 = 2; therefore insulin drip rate is 2u/hr or 10cc/hr
Target BS = 120-180
Watch K+, as insulin shifts potassium into the cell
Avoid LR, lactate converts to glucose; will see increase glucose levels 24-48hrs. after surgery
Need at least two functioning IV access
Adrenalcortical dysfunction
Cushing’s
glucocorticoid excess – either from endogenous oversecretion or chronic treatment of glucocorticoids (steroids) (DIFFICULT AIRWAYS)
Truncal obesity, thin skin, easy bruising
Adrenalcortical dysfunction
Addison’s Disease
– Adrenalcortical insufficiency or withdrawal of steroids or suppression of synthesis
Hyperaldosteronism – excess of mineralocorticoid hormones
Sometimes seen in excess of glucocorticoids
Adrenalcortical dysfunction
Cause fluid and electrolyte disturbances
Fluid retention and hypertension
Blood sugar elevation
Continue glucocorticoid or mineralocorticoid replacement therapy
Liver dysfunction
Coagulation dysfunction
Decrease albumin
Cardiomyopathy
Encephalopathy
Varicies
Decrease glucose, sodium, potassium
Renal dysfunction
Giving glucose to a malnourished alcoholic without thiamine
will cause irreversible brain damage!!!
THEY cannot metabolize the glucose and will cause irreversible brain damage
Liver dysfunction
goals
Goal is to avoid making hepatic disease worse and increasing the chance of renal failure, coma, and death
Studies of portal hypertension have shown that mortality can be 50% when preop serum albumin = <3g/dL, serum bilirubin >3mg/dL, and ascites and encephalopathy are present
NS can cause hyperchloremic acidosis!! If lots given… plasmalyte is the best (esp for liver tx)
Liver dysfunction
goals cont
Maintain fluid balance/colloids
Monitor electrolytes; arterial pH should be maintained to avoid systemic metabolic alkalemia – noted to increase diffusion of ammonia through BBB worsening degree of encephalopathy
RSI – especially if ascites present
Preoxygenate
Poor respiratory function; atelectasis
Careful with sedatives, narcotics, and drugs metabolized by liver
Low circulating protein/volume of distribution increased
Evaluation of coagulation disorder
Affects decision for regional vs GA; utilization of blood products intraop – FFP
Avoid use of meds than may affect platelet function
ASA / NSAIDs
Pheochromocytoma
catecholamine secreting tumor of chromaffin tissue
Usually benign and localized in adrenal gland; 20 – 30% are malignant and are extra-adrenal
Kidney removal (huge BP SPIKE-undx pheo)
Pheochromocytoma
Cardinal signs:
Paroxysmal headache
HTN – orthostatic hypotension
Sweating
Palpitations/tachycardia
Pheochromocytoma
Anesthetic Considerations
Adequate adrenergic blockade; then beta
Monitor volume status; cvp or swan; usually dehydrated
Avoid drugs that stimulate SNS, inhibit PNS (pancuronium), or release antihistamine
Carcinoid Syndrome (releases kinin cascade)
Tumors - 75% of the time originate in the GI tract – secrete serotonin (5-HT, 5-hydoxytryptamine) , histamine release, elevation of plasma kinins
7% of the pt with carcinoid tumors have carcinoid syndrome:
Flushing
Diarrhea
Valvular heart disease
Wheezing
Vasodilation / vasoconstriction – (sometimes effects of 5-HT on heart - chronotropic, inotropic effect
CARCINOID SYNDROME TREATMENT
Therapy of choice: somatostatin analog – powerful inhibitor of the release of peptides from carcinoid tumors and an inhibitor of the peptic effects on receptor cells
H2 blockers – combination tx H1/H2 (not H1 alone)
Steroids
Alpha adrenergic blockers
Beta adrenergic blocker
Vasopressin – for severe hypotension not responsive to somatostatin
(will increase pulmonary vascular resistance)
Sickle cell
– avoid hypoxemia, avoid vascular stasis, remain normothermic
Sickling causes patients to infarct body parts, they need to be warm and hydrated and stress of the body needs to be kept down. Even people with sickle cell trait they need to be taken care of lightly.
Usually dehydrated after dialysis
watch with induction!
Prone to CHF, increase K+ levels, platelet dysfx, low HCT
Preserve renal function in pts with renal insufficiency
Avoid frequent hemodynamic alterations – low CO, low renal blood flow
Acute renal failure is most likely to occur in pts who have renal insufficiency before surgery – greater if pt is >60years of age, and has left ventricular dysfunction.
Uremia
– the end result of renal tubular failure
CV - cardiac failure
Neuromuscular – neuropathy
Metabolic / endocrinologic – electrolyte changes, acidosis
Hematologic – anemia, coagulation and platelet dysfx
Lowered immune system
Birth control pills and smoking
have increased risk for blood clots
Most teratogenic period
3rd- 8th week during organogenesis
BZD’s are linked to
congenital anomalies
Cleft palate
All elective surgeries should be held until after delivery
6 weeks later
Cannot be avoided, regional when possible and/or after first trimester
Muscular Dystrophy
– unexpected effects to anesthetic drugs on myocardial function and skeletal muscle
Cardiac arrest & MH
Sux avoided – hyperkalemia, rhabdomyolysis, cardiac arrest
Increased risk of aspiration – degeneration of gastrointestinal smooth muscle
Osteoporosis
Limited range of motion
Increased risk of fracture during positioning or movement to and from operating room table
. Loud murmur that radiates out of their neck
they have aortic stenosis. (contra indication for a spinal)
Size of tongue
Large Tongue
Associated with acromegaly, cretinism, mongolism
Hard to perform laryngoscopy, or mask ventilate
Mandibular hypoplasia (micrognathia)
Associated with Pierre Robin
Temporomandibular joint (TMJ) dysfunctio
Degree of mouth opening
At least 4.0cm
Rheumatoid arthritis/Ankylosing spondylitis
Restricts ROM of neck
Restricts vocal cord movement
Tracheal stenosis
Thyromental Distance
The distance, with the neck fully extended, between the thyroid notch and the lower Mandibular border
Coma
Uremia, hypoglycemia, hyperosmolar nonketotic, metabolic encephalopathy,
Lethargic – avoid drugs that might worsen
Confusion / forgetful
Listen to heart sounds
S3 – left ventricular failure
Jugular Vein Distention
Carotid Bruits
Peripheral Edema
Exercise tolerance
BARREL CHEST
late manifestation of obstructive lung disease
GI/HEPATIC/RENAL/SKIN
INSPECT?
Assess abdominal girth/ascites
Jaundice
Bruising
Tatoos
Discoloration of skin – esp lower extremities
Rheumatoid Arthritis
Atlantoaxial subluxation and odontoid fracture – causes spinal cord impingement – usually degree if impingement does not correlate with pts symptoms or lack of symptoms
Affects joints of larynx – limits vc movement and high incidence of erythema and edema associated with intubation
Affects temporomandibular joint
Positioning issues with these patients
The best way to screen for disease
quality H & P
CBC/Platelet function
If expected to have large blood loss
Hx of renal disease, anemia, GI bleed, malignancies, bruising
Chemistry
Diuretic use
Renal disease
LFT’s for hepatic disease
Diabetic
Coags
Liver disease, blood thinners, malignancies
EKG
Goal is to establish baseline, pick up arrhythmias, blocks, ST changes, MI old or new; working pacemaker
Estimated ~30% of infarctions are silent with highest incidence in pts with diabetes and HTN
The presence of q waves in a high risk pt, has increased perioperative risk and the possibility of active ischemia
Influence anesthetic management and intraop monitoring
Proceed to exercise or pharmacologic stress test
Protocol of group
Men >45years, female >55years
Hx of CAD, MI, pulm disease, smoker, DM
If big case
Intrathoracic, major abdominal, big vascular case, lot of fluid shifting
CXR
Detects tracheal stenosis, mediastinal or lung masses, edema, atelectasis, cardiomegaly, severe disease, i.e. hyperinflation
>60 or > 50 yrs if smoker
Hx of pulm disease, malignancy, radiation therapy
PFT’s (for thoracic or sleep apnea pts)
Purpose is to determine degree of pulmonary disease; determine response to bronchodilators & help plan lung resection
To decide whether the removal of lung tissue can be tolerated without compromising pulmonary function
Simplest & most informative
PFT’s
FEV1 & VC
Flow volume loops
Increased risk: FEV1 < 2L; FEV1:FVC < 15cc/kg or max breathing capacity is <50% of predicted value
Coronary Angiography – anatomic info
Always for CABG/valve replacements
For noncardiac surgery – usually b/c +positive stress test
TEE/Echo – functional info (specific but not sensitive
Determine ventricular dysfunction, regional wall motion abn, ventricular wall thickness, and valvular function
EF
PS-1
Healthy patient
PS-2
Patient with mild systemic disease that results in no functional limitations
PS-3
Patient with severe systemic disease that results in functional limitations
PS-4
Patient with severe systemic disease that is a constant threat to life
PS-5
Moribund patient not expected to survive without operation
PS-6
Declared brain-dead patient for organ harvest
Emergency (E
Any patient for an emergency operation
ASA 4E in most places
likely a 5.
Delaying/cancelling case
Active URI
Newly “discovered” MI within last 6 months
New unstable cardiac rhythm
Coagulopathy
Hypoxia
Administrative issues
Jehovah’s Witness patient
Ethical procedure coverage
Anesthesia provider coverage
Premedications
Sedation/anxiety
Versed 1-2.5 mg IV
Ativan 0.5-2 mg IV/po
Valium 2-10 mg IV/po
Clonidine 0.1-0.3 mg p
Premedications
Antiemetics
Ondansetron 4 mg IV
Dolasetron 12.5 mg IV
Dexamethasone 4 mg IV (massive burning in the crotch)
Premedications
Anticholinergics
Glycopyrrolate 0.1-0.2 mg IV
Don’t’ give glycopyrrolate in patients with asthma.
Pulmonary aspiration prophylax
Proton pump inhibitors (particulates)
Omeprazole (Prilosec) 20 mg po night before surgery or > 2hours before
Pantoprazole (Protonix) 40 mg IV 20 min preop
Pulmonary aspiration prophylax
HISTAMINE (H2) ANTAGONISTS
< 1 hour pre-op
Cimetidine (Tagamet) 200-400 mg po, IM, IV
Ranitidine (Zantac)150-300 mg po/50-100 mg IV, IM
Pulmonary aspiration prophylax
Nonparticulate antac
Bicitra 30-60 ml 30min preop
Pulmonary aspiration prophylax
Metoclopramide (Reglan) (dopamine receptor antagonist)
10 mg po/IV 1-2 hours preop
Can not give particulates right before surgery
Reglan also increases esophageal sphincter pressure makes it harder to reflux.