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10 Cards in this Set
- Front
- Back
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What is the Presentation of Conns syndrome? BP?
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pt complained of severe headaches and muscle weakness
High blood pressure 180/100 |
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What was the blood chemistry like in Conn's syndrome?
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Low potassium,
High Bicarbonate, High PH, slightly high PaCO2 Eleveated K+ excretion Increased Aldosterone |
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What is the core cause of Conns Syndrome?
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this due to hyperaldosteronism.
too much aldosterone increased Na+ uptake Also increases K+ excretion. Aldosterone also stimulates H+ ATP ase in the distal tubule |
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What is the blood pressure and pulse like in DKA?
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low blood pressure, and high pulse rate
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What is the blood chemistry like in DKA?
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high potassium
high glucose LOW bicarb High osmolatirty Low pH High urine flow Anion Gap INCREASED (due to concompensated H+ production. normal Cl-) |
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Why is there hyperkalemia in DKA?
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this is due to low insulin, which promotes K+ efflux from cells
Also high ADH |
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What causes contraction alkalosis, what are the lab values like?
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this is caused by vomiting.
Low potassium low chloride HIGH bicarb High pH slightly high PaCO2 |
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How does volume contraction increase H+ loss?
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via angiotensin release, this stimulates Na+/H+ antiporter push H+ out.
Aldosterone- stimulates section of H+ from type A cells, and K+ from prinicple cells |
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How does hypokalemia affect contraction alkalosis?
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volume contraction stimulates aldosterone release
aldosterone stimulates Na uptake. and K+ secretion. this lowers blood K+ further. Also, hypokalemia stimualtes the secretion of H+ by intercalated type A cells (using H+ excretion to take up K+) |
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how do hypochloremia affect contraction alkalosis?
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this will exaccerbate it due to a lack of Cl- preventing the Na/K/Cl symporter in the collecting duct.
this facilitates excretion of K+ and H+ |
