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53 Cards in this Set
- Front
- Back
list the primary endocrine organs
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1. hypothalamus
2. pituitary gland 3. pineal gland 4. thyroid gland 5. parathyroid gland 6. thymus 7. adrenal gland |
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list the parts of the pituitary gland
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anterior pituitary and posterior pituitary
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list the parts of the adrenal gland
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adrenal cortex and adrenal medulla
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TSH, ACTH, FSH, LH, GH, prolactin
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anterior pituitary gland
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ADH, oxytocin
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posterior pituitary gland
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trophic hormones (releasing and inhibiting hormones)
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hypothalamus
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secretes melatonin
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pineal gland
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secretes thyroxine (T4), triiodothyronine (T3); calcitonin
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thyroid gland
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PTH
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parathyroid gland
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thymosin, thymopoietin
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thymus
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aldosterone, cortisol, androgens
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adrenal cortex
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epinephrine & NE
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adrenal medulla
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list the primary endocrine glands in other organs
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1. pancreas
2. ovaries/testes 3. placenta |
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produces insulin, glucagon
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pancreas
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estrogen & progesterone (females)/ androgens (testosterone) (males)
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ovaries/testes
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estrogen & progesterone, hCG
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placenta
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list the secondary endocrine organs
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GI organs, heart, kidneys, liver, skin
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GI hormones, ANP, erythropoietin, vitamin D3, etc.
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GI organs, heart, kidneys, liver, skin
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part of the brain (diencephalon), controls the pituitary gland
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hypothalamus
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major endocrine gland; infundibulum attaches to hypothalamus
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pituitary gland
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attaches to hypothalamus
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infundibulum
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-neurohypophysis
-direct NEURAL CONNECTION to hypothalamus -neurosecretory cells originate in hypothalamus, axons in infundibulum, axon terminals in posterior pituitary secrete neurohormones -ADH (vasopressin) and oxytocin |
posterior pituitary gland
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-CIRCULATORY CONNECTION to hypothalamus via HYPOTHALAMIC-HYPOPHYSEAL PORTAL SYSTEM
-neurosecretory cells in the hypothalamus secrete releasing hormones into portal system which control anterior pituitary secretion -secretes trophic hormones that control other endocrine glands and tissues |
anterior pituitary gland
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TRH + TSH = ?
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thyroid gland --> TH
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CRH + ACTH = ?
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adrenal cortex --> cortisol
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GnRH + FSH & LH = ?
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ovaries/testes
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GHRH + growth hormone = ?
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liver, bone, muscle, etc., protein synthesis
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PRH + prolactin = ?
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breasts
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inhibits hypo and anterior (hormone 3)
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long loop negative feedback
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anterior inhibits hypo (tropic hormone 2)
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short loop negative feedback
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tell me what you know about the thyroid gland
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1. production of thyroid hormones T3 and T4 (thyroxine)
2. effects of thyroid hormones 3. feedback control of thyroid hormone secretion |
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effects of thyroid hormones
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-increase basal metabolic rate
-stimulate protein synthesis -developmental effects: nervous and reproductive systems |
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what contains mineralcorticoids, glucocorticoids, and androgens?
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adrenal cortex
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-connection to sympathetic ANS
-chromaffin cells secrete epinephrine and norepinephrine -epi and NE act via adrenergic receptors on target cells |
adrenal medulla
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constriction of blood vessels
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alpha-adrenergic receptors
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increase in heart rate and contractility
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beta 1 adrenergic receptors
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bronchodilation
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beta 2 adrenergic receptors
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endocrine portion consists of pancreatic islets
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pancreas
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secrete insulin, most abundant
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beta cells
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secrete glucagon, somewhat less abundant
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alpha cells
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these are major actors in the maintenance of glucose homeostasis
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insulin and glucagon
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-stimulates glucose uptake (facilitated diffusion) into most body cells
-stimulates synthesis of glycogen, protein, and lipids (energy storage) -decreases plasma glucose concentration |
insulin
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-stimulates glycogenolysis and gluconeogenesis in liver (releases glucose into blood)
-stimulates lipolysis in adipose tissue (mobilizes energy stores, spares glucose) -increases plasma glucose concentration |
glucagon
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insulin deficiency disease
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diabetes mellitus
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insulin dependent (IDDM), "juvenile onset"
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type 1 diabetes
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non-insulin dependent (NIDDM), "adult onset"
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type 2 diabetes
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high blood glucose levels
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hyperglycemia
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excretion of glucose in the urine, osmotic effect results in excessive urinary water loss (diuresis)
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glycosuria
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effects and complications of diabetes mellitus
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-hyperglycemia
-glycosuria -elevate ketones and ketoacidosis --cells are "glucose starved" because of insufficient glucose uptake --->increased fat and protein catabolism --> excess production of ketone bodies -vascular degeneration |
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promotes Na+ retention and K+ excretion by kidneys, zona glomerulosa
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mineralcorticoids (aldosterone)
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"stress hormones", promote gluconeogenesis, anti-inflammatory, zona fasciculata
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glucocorticoids (cortisol)
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for example, testosterone
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androgens
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secrete epinephrine and norepinephrine
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chromaffin cells
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