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99 Cards in this Set
- Front
- Back
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This inhibits the actions or formation of clotting factors and prevents clot formation.
a. anticoagulants b. antiplatelets c. hemorheologic drugs d. thrombolytic drugs e. hemostatic or antifibrinolytic drugs |
a. anticoagulants
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inhibits platelet aggregation
a. anticoagulants b. antiplatelets c. hemorheologic drugs d. thrombolytic drugs e. hemostatic or antifibrinolytic drugs |
b. antiplatelets
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alters platelet functions.
a. anticoagulants b. antiplatelets c. hemorheologic drugs d. thrombolytic drugs e. hemostatic or antifibrinolytic drugs |
c. hemorheologic drugs
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lyse bread down existing clots
a. anticoagulants b. antiplatelets c. hemorheologic drugs d. thrombolytic drugs e. hemostatic or antifibrinolytic drugs |
d. thrombolytic drugs
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Promote blood coagulation
a. anticoagulants b. antiplatelets c. hemorheologic drugs d. thrombolytic drugs e. hemostatic or antifibrinolytic drugs |
e. hemostatic or antifibrinolytic drugs
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This is used to prevent clot formation in certain settings where clot formation is likely. MI, unstable angina, atrial fibrillation, indwelling devices, such as mechanical heart valves, major orthobpedic surgery
a. anticoagulants b. antiplatelets c. hemorheologic drugs d. thrombolytic drugs e. hemostatic or antifibrinolytic drugs |
a. anticoagulants
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The adverse effects/interactions: bleeding, risk for increases with increased dosages, may be localized or systemic, Heparin induced thrombocytopenia, N/V, abdominal cramps,
a. anticoagulants b. antiplatelets c. hemorheologic drugs d. thrombolytic drugs e. hemostatic or antifibrinolytic drugs |
a. anticoagulants
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Thes prevents platelt adhesion, and has antithrombotic effects. It reduces risks of fatal and nonfatal strokes, actue unstable angina and MI.
a. anticoagulants b. antiplatelets c. hemorheologic drugs d. thrombolytic drugs e. hemostatic or antifibrinolytic drugs |
b. antiplatelets
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This drug prevents the lysis of fibrin with the result in promoting clot formation. Used for prevention and treatment of excessive bleeding, resulting from hyperfibrinolysis or surgical procedures.
a. anticoagulants b. antiplatelets c. hemorheologic drugs d. thrombolytic drugs e. hemostatic or antifibrinolytic drugs |
e.antifibrinolytic drugs
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This drug is used to treat excessive bleeding like: hyperfibrinolysis, surgical complications, excessive oozing from surgical sites, reducing total blood loss and duration of bleeding in the post-op periods.. treats, hemophilia or VWdisease.
a. anticoagulants b. antiplatelets c. hemorheologic drugs d. thrombolytic drugs e. hemostatic or antifibrinolytic drugs |
e. antifibrinolytic drugs
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this drug has uncommon and mild interactions and has rare thrombotic events. dysrhythmia, orthostatic hypotension, bradycardia, h/a, dizziness, fatigue, n/v, abdominal cramps, diarrhea
a. anticoagulants b. antiplatelets c. hemorheologic drugs d. thrombolytic drugs e. hemostatic or antifibrinolytic drugs |
e.
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These drugs break down or lyse performed clots. Older drugs are streptokinase and urokinase. New drugs are t-PA, anisoylated plasminogen-streptokinase activator complex
a. anticoagulants b. antiplatelets c. hemorheologic drugs d. thrombolytic drugs e. hemostatic or antifibrinolytic drugs |
d. thrombolytic drugs
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activate the fibrinolytic system to break down the clot in the blood vessel quickly. Activate plasminogen and convert it to plasmin wich can digest fibrin. Reestablish blood flow to the heart muscle via coronary arteries preventing tissue destruction.
a. anticoagulants b. antiplatelets c. hemorheologic drugs d. thrombolytic drugs e. hemostatic or antifibrinolytic drugs |
d. thrombolytic drugs
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Adverse effects/interactions are bleeding: internally, intracranially, and superficially. n/v, hypotension, anaphylactoid reactions, cardiac dysrhthmias
a. anticoagulants b. antiplatelets c. hemorheologic drugs d. thrombolytic drugs e. hemostatic or antifibrinolytic drugs |
d. thrombolytic drugs
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Iv doses may be given by bolus or IV infusions.
Given subcutaneously in the abodomen a. LMWH b. Heparin |
b
a. |
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anticoagulant effects seen immediately
a. LMWH b. Heparin |
b
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you need to roate injections istes
a. LMWH b. Heparin |
a.
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Lab values done daily to monito coagulation effects aPTT
a. LMWH b. Heparin |
b.
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Protamine sulfate can be given ias in case of excessive anticoagulation.
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heparin
lmwh |
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Nursin implications for what type of drugs
dipyridamole should be taken on an empty stomach. monitor for abnormal bleeding, monitor IV sites for bleeding , redness and pain monito for bleeding around gums mucous membranes, nose and injection sites a. antiplatelet/anticoagulant b. antifibrinlytic c. thrombolytic |
a.
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This drug alleviates the symptoms of this type of anemia. does not correct it but helps it.
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Iron
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most common cause of pediatric poisoning deaths. causes nausea, vomiting, diarrhea, constipation, stomach cramps, and pain.
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iron
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causes black tarry stools due to a GI bleed. LIquid oral preparations may stain teeth. injectable forms may stain skin and cause pain upon injection.
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iron
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This is a water soluble, b-complex vitamin. Essential for erythropoiesis, primarily used for folic acid deficiency, during pregnancy to prevent nerual tub defects.
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folic acid
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Malabsorption syndromes are th most common cause of deficiency. Should not be used until actual cause of anemia is determined. May mask symptoms of pernicious anemia, which requires treatment other then this vitami. Untreated pernicous anemia progresses to neurologic damage
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folic acid
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Oral forms of this supplement should be taken between meals for maximum absorption but may be taken with meals if GI stress occurs. Oral forms should be given with juice, but not with milk or antacids.It blocks the absorption of this
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iron
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True or false. patietns taking a liquid iron prep should drink it through a starw to avoid staining tooth enamel
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true
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Keep away from chlidren because oral forms may look like candy.
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iron
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This drug acclerates the rate of urin formation
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diuretics
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This enzyme helps to make H+ ions available for exchange with sodium and water in the proximal tubules
a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
a.
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This diuretic creates H+ ions thus preventing exchange with sodium and water resulting in higher H+ ions and releasing more sodium, bicarbonate, water and potassium
a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
a.
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Adjunct drugs in the longterm managment of open angle glaucoma. Used with miotics to lower intraocular pressure before ocular surgery in certain cases. Used also in treatment of edema, epiliepsy, high altitude sickness.
a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
a.
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Adverse reactions of this diuretic is.
metabolic acidosis, anorexia, hematuria, phtosensitivity, melena, hypokalemia, drowsiness, parethesias, uticaria. a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
A.
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This diurectic acts directly on the ascending limb of the loop of hele to inhibit chloride and sodium resorption. Increase renal prostaglandins, resulting in the dilation of blood vessels and reduced peripheral vascular resistance.
a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
B.
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This is a potent diuresis and subsequent loss of fluid. decreased fluid volume causes a reduction in:
BP, Pulmonary vascular resistance, central venous pressure, left ventricular end diastolic pressure and potassium and sodium depletion a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
b.
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This is indicated in edema associated with HF or hepatic or renal disease. To control hypertension. to increase renal excretion of calcium in pts. with hypercalcemia, in cases of HF resulting from diastolic dysfunction.
a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
b.
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adverse effects:
CNS: dizzziness, h/a, tinnitus, blurred vision GI: Nausea, vomiting, diarrhea hematologic: agranulocytosis, neutropenia, thrombocytopenia metabolic: Hypokalemia ( see most often), hyperglycemia, hyperuricemia a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
B.
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Works mostly in the proximal tubule, prouces an osmotic efffect. Pulls water ino the renal tubules fromt eh surrounding tissues. Inhibits tubular resorption of water and solutes, thus producing rapid diuresis and is used to reduce excessive intraocular pressure.
a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
c.
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This diurectic increases glomerular filtration rate and renal plasma flow- helps to prevent kidney damage durig acute renal failure.
a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
c.
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This diurectic is indicated of patients in early, oliguric phase of ARF, promotes excretion of toxic substances.
a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
c.
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Used to reduce intracranial pressure, cereblral edema but not indicated in peripheral edema. a. carbonic anhydrase inhibitors
b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
c.
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Adverse effects:
convulsions, thrombophlebitis, pulmonary congestion, h/a, chest pains, tachycardia, blurred vision, chills and fever a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
c.
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Work in collecting ducts and distal convoluted tubules, interferes with sodium-potassium exchange, competitively bind to aldosterone receptors, blocks resorption of sodium and water usually induced by aldosterone.
a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
d.
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Prevent potassium from being pumped into the tubule, thus preventing its secretion. Competitively block aldosterone receptors and inhibit their action. Promote the excretion of sodium and water.
a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
d.
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what drugs are these?
Spironolactone and triamterene amiloride a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
d.
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These diurectics cause gynecomastia, amenorrhea, irregular menses, postmenopausal bleeding.
a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
d.
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Ihibit tubular resorption of sodium, chloride and potassium ions. Act on distal convoluted tubule, potassium is also excreted to a lesser extent. Dilate the arterioles by direct relaxation
a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
e
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Lowered peripheral vascular resistance. Should not be used if creatinine clearance is less than 30 to 50 mL. Metolazone remians effective toa creatinine clearance.
a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
e
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INdicated in hypertension (one of the most prescribed group) of drugs for this. Idiopathic hypercalciuria, diabetes insipidus, HF due to diastolic dysfunction
a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
e.
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This diuretic cuases impotence and decreased libido
a. carbonic anhydrase inhibitors b. loop diuretics c. osmotic diuretics d. potassium sparing diuretics e. thiazide and thiazide like |
e.
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True of false. pt taking diuretics along with digitalis preps should be taught to monitor for digitalis toxicity
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true
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Thiazide and loop diretics increase blood glucose?
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true
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excessive consumption of licorice can lead to additive hypokalemia in pts taking thiazides?
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true
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this nervous system stimulates smooth muscle, cardiac muscle, glands.
a. PNS b. SNS |
a.
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this nervous system stiulates the heart, blood vessels, skelteal muscle.
a. PNS b. SNS |
b.
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Centrally acting alpha 2 receptor agonists in the brain. Decreases sympathetic outflow from the CNS, Decreases norepinephrine production. Stimulates aplha 2 receptors reducing renin activity in the kidney.. resulting in decreased BP.
a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators |
a.
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Clonidine, catapres
methyldopa (aldomet) can be used for hyperension in pregancy a. alpha 2 receptor agonists b. alpha 1 blockers/ antagonists |
a.
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doxazosin, cardura
terazosin, hytrin peipheral alpha1 blockers, antagonists results in decreased blood pressure thus working in the blood vessels. a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators |
a
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Thiazide and loop diretics increase blood glucose?
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true
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Reduces BP by reducing heart rate through beta 1 blockade. causes rduced secretion of renin. LOLs
a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators g. beta blockers. |
g.
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excessive consumption of licorice can lead to additive hypokalemai in pts taking thiazides?
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true
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This reduces the heart
this causes vasodilation a. alpha-2 b. alpha-1 c. beta 1 |
c.
b. |
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this nervous system stimulates smooth muscle, cardiac muscle, glands.
a. PNS b. SNS |
a.
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clonodine is useful in the managment of withdrawal symptoms in opiod or nicotine dependent persons
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true
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this nervous system stiulates the heart, blood vessels, skelteal muscle.
a. PNS b. SNS |
b.
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Thiazide and loop diretics increase blood glucose?
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true
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excessive consumption of licorice can lead to additive hypokalemai in pts taking thiazides?
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true
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Centrally acting alpha 2 receptor agonists in the brain. Decreases sympathetic outflow from the CNS, Decreases norepinephrine production. Stimulates aplha 2 receptors reducing renin activity in the kidney.. resulting in decreased BP.
a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators |
a.
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this nervous system stimulates smooth muscle, cardiac muscle, glands.
a. PNS b. SNS |
a.
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Clonidine, catapres
methyldopa (aldomet) can be used for hyperension in pregancy a. alpha 2 receptor agonists b. alpha 1 blockers/ antagonists |
a.
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doxazosin, cardura
terazosin, hytrin peipheral alpha1 blockers, antagonists results in decreased blood pressure thus working in the blood vessels. a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators |
a
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this nervous system stiulates the heart, blood vessels, skelteal muscle.
a. PNS b. SNS |
b.
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Reduces BP by reducing heart rate through beta 1 blockade. causes rduced secretion of renin. LOLs
a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators g. beta blockers. |
g.
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Centrally acting alpha 2 receptor agonists in the brain. Decreases sympathetic outflow from the CNS, Decreases norepinephrine production. Stimulates aplha 2 receptors reducing renin activity in the kidney.. resulting in decreased BP.
a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators |
a.
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Clonidine, catapres
methyldopa (aldomet) can be used for hyperension in pregancy a. alpha 2 receptor agonists b. alpha 1 blockers/ antagonists |
a.
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This reduces the heart
this causes vasodilation a. alpha-2 b. alpha-1 c. beta 1 |
c.
b. |
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doxazosin, cardura
terazosin, hytrin peipheral alpha1 blockers, antagonists results in decreased blood pressure thus working in the blood vessels. a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators |
a
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clonodine is useful in the managment of withdrawal symptoms in opiod or nicotine dependent persons
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true
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Reduces BP by reducing heart rate through beta 1 blockade. causes rduced secretion of renin. LOLs
a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators g. beta blockers. |
g.
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This reduces the heart
this causes vasodilation a. alpha-2 b. alpha-1 c. beta 1 |
c.
b. |
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clonodine is useful in the managment of withdrawal symptoms in opiod or nicotine dependent persons
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true
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Carvedilol (coreg) is an dual action alpha1 and beta receptor blocker.
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true
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this adrenergic alpha 1 receptor agonist used to relieve symptoms of BPH. FLOMAX
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true
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This has a highh incidences in orthostatic hypotension. Dry mouth, drowsiness, sedation, constipation.
a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators g. beta blockers |
a.
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this is a large group of safe and effective drugs, often used as first-line drugs for HF and hypertension, maybe combined with thiazide diuretic or CCB
a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators |
b
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This inhibits angiotensin 1 to 2
a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators |
b.
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This is indicated in hypertension, HF( either alone or in combination with diuretics or other drugs, renal protective effects in patients with diabetes. Drugs of choice in hypertensive patients with HF and diabetic patients.
a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators |
b.
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catopril- very short half life
enalapril- lisnipril quinapril a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators |
b.
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catopril and lisniopril are not _____. Used in patietns with liver dysfunction, unlike other ace inhibitors that are these.
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prodrugs.
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These drugs are used after ace drugs are contraindicated.. because of a dry cough
a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators g. beta blockers |
c.
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allows angiotensin 1 to be turned in to 2 but blocks the receptor sites that receive a II.
a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators g. beta blockers |
c.
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This causes smooth muscle relaxation by blocking he binding of calcium to its receptors, preventing muscle contraction
a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators g. beta blockers |
d.
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this results in peripheral smooth muscle tone, decreased systemic vascular resistance, decreased bp
a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators g. beta blockers |
d.
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ainthing with PINES OR INES
a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators g. beta blockers |
D.
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Directly relax arteriole and or venous smooth muscle. results in decreased systemic vascular response, decreased afterload, peripheral vasodilation
a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators g. beta blockers |
f.
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Hyperstat, apresoline, loniten, nipride, nitropres
a. adrenergic drugs b. ACE c. ARBS d. CCBs e. diuretics f. vasodilators g. beta blockers |
f.
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Dizziness, h/a, anxiety, trachcycardia, n/v, diarrhea, anemia, dyspnea, edema, nasal congestion. Vasodilator
a. hydralazine b. sodium nitroprusside c. diazoxide. |
a.
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vasodilator
bradycardia, hypotension, possible cynaide toxicity a. hydralazine b. sodium nitroprusside c. diazoxide. |
b.
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dizziness, h/a anxiety, orthostatic hypotension, dysrhythmias, sodium and water retnetion, n/v, hyperglycemia, in diabetic pts.
a. hydralazine b. sodium nitroprusside c. diazoxide. |
c.
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