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10 Cards in this Set
- Front
- Back
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Inotropic Agents
common |
CHF DRUG
most common: digoxin/lanoxin Antidote – digibind |
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Inotropic Agents
MOA |
increase cardiac contractility. Inhibits enzyme – sodium-potassium ATPase ->
promotes Ca accumulation, incr contractility -> incr CO. HR decrease, ventricular filling increases. Slows down heart, increases contractility--- greater CO |
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Inotropic Agents
IND |
CHF, dysrhythmias
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Inotropic Agents
AE |
predisposing factor for toxicity:
Hypokalemia; low K increases digoxin toxicity. Digoxin & K bind at same sites Signs of digoxin toxicity: early S&S: visual changes (changes in color, halos, yellowish or green caste), anorexia, n/v, fatigue. late S&S: ventr dysrhythmias b/c digoxin slows HR-can lead to AV block |
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Inotropic Agents
DI |
diuretics b/c of risk of low K—so must be on K supplement
Quinidine (antidysrhythmic drug) Verapamil- increases digoxin levels |
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Inotropic Agents
NC |
Monitor/assess apical HR for one minute before giving.
Hold if HR<60 monitor serum K level, digoxin level |
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Diuretics
common |
CHF DRUGS
Loop (lasix most common), thiazide diuretics Patients frequently on lasix, digoxin and K |
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Diuretics
MOA |
Increase renal excretion of water, Na, other e-lytes.
Increase urine output, decrease fluid overload – decr pulmonary & peripheral edema. |
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Ace Inhibitors
MOA |
block enzyme that converts angiotensin I to angiotensin II -reduces aldosterone
Causes: vasodilation – dec BP decrease blood volume since it reduces retention of Na and H2O reverses pathological changes in heart and blood vessels |
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Ace Inhibitors
AE |
some patients w/kidney issues cannot take
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